Search results for " T-Lymphocytes"

showing 10 items of 495 documents

Deficiency in the Transcription Factor Interferon Regulatory Factor (Irf)-2 Leads to Severely Compromised Development of Natural Killer and T Helper …

2000

Interferon (IFN) regulatory factor (IRF)-2 was originally described as an antagonist of IRF-1–mediated transcriptional regulation of IFN-inducible genes. IRF-1−/− mice exhibit defective T helper type 1 (Th1) cell differentiation. We have used experimental leishmaniasis to show that, like IRF-1−/− mice, IRF-2−/− mice are susceptible to Leishmania major infection due to a defect in Th1 differentiation. Natural killer (NK) cell development is compromised in both IRF-1−/− and IRF-2−/− mice, but the underlying mechanism differs. NK (but not NK+ T) cell numbers are decreased in IRF-2−/− mice, and the NK cells that are present are immature in phenotype. Therefore, like IRF-1, IRF-2 is required for…

CD4-Positive T-LymphocytesMaleInterferon Regulatory Factor 2Cellular differentiationImmunologyLeishmaniasis CutaneousBiologyNitric OxideTh1MiceInterleukin 21Immune systemBone MarrowInterferonmedicineAnimalsImmunology and AllergyLymphocyte CountLeishmania majorInterleukin-15Mice KnockoutLeishmaniaMice Inbred BALB Cnatural killer cellsCell DifferentiationTh1 CellsInterleukin-12Cell biologyDNA-Binding ProteinsKiller Cells NaturalMice Inbred C57BLRepressor ProteinsDisease Models AnimalInterleukin 15interferon regulatory factorImmunologyInterleukin 12FemaleOriginal ArticleDisease SusceptibilityInterferon Regulatory Factor-2interleukin 15Transcription FactorsInterferon regulatory factorsmedicine.drugJournal of Experimental Medicine
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Antibodies against tumor necrosis factor (TNF) induce T-cell apoptosis in patients with inflammatory bowel diseases via TNF receptor 2 and intestinal…

2011

Background & Aims The anti–tumor necrosis factor (TNF) antibodies infliximab, adalimumab, and certolizumab pegol have proven clinical efficacy in Crohn's disease. Here, we assessed the effects of anti-TNF antibodies on apoptosis in inflammatory bowel disease (IBD). Methods CD14 + macrophages and CD4 + T cells were isolated from peripheral blood and lamina propria mononuclear cells from patients with IBD and control patients. Cell surface markers and apoptosis were assessed by immunohistology and fluorescence-activated cell sorting techniques. Results Lamina propria CD14 + macrophages showed significantly more frequent and higher membrane-bound TNF (mTNF) expression than CD4 + T cells in IBD…

CD4-Positive T-LymphocytesMaleNecrosisCD14Anti-Inflammatory AgentsLipopolysaccharide ReceptorsApoptosisEnzyme-Linked Immunosorbent AssayBiologyAntibodies Monoclonal HumanizedReal-Time Polymerase Chain ReactionPeripheral blood mononuclear cellPolyethylene GlycolsImmunoglobulin Fab FragmentsYoung AdultmedicineHumansReceptors Tumor Necrosis Factor Type IIAntigen-presenting cellAgedLamina propriaHepatologyCluster of differentiationTumor Necrosis Factor-alphaMacrophagesGastroenterologyAdalimumabAntibodies MonoclonalMiddle AgedFlow CytometryInflammatory Bowel DiseasesInfliximabmedicine.anatomical_structureApoptosisCase-Control StudiesImmunologyCertolizumab PegolTumor necrosis factor alphaFemalemedicine.symptomGastroenterology
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Development of hapten-induced IL-4-producing CD4+ T lymphocytes requires early IL-4 production by alphabeta T lymphocytes carrying invariant V(alpha)…

1998

This paper investigates the mechanisms responsible for the generation of IL-4-producing CD4+ T cells during contact sensitization with the hapten trinitrochlorobenzene (TNCB). Lymph node cells taken 1 day after immunization spontaneously released IL-4 while lymph node cells taken 2 and 3 days after immunization did not produce IL-4. A second wave of IL-4 production that was both antigen-specific and MHC class II (I-A)-restricted was observed 4 days after immunization. The spontaneous release of IL-4 at day 1 was due to the alphabeta+ double-negative (CD4- CD8-) T lymphocytes that also expressed NK1.1 and showed V(alpha)14 rearrangement, while alphabeta+ CD4+ T lymphocytes were the source of…

CD4-Positive T-LymphocytesMaleReceptors Antigen T-Cell alpha-betaT cellImmunologyPicryl ChlorideCD8-Positive T-LymphocytesBiologyMiceInterleukin 21AntigenmedicineAnimalsImmunology and AllergyCytotoxic T cellMice Inbred BALB CT-cell receptorAntibodies MonoclonalGeneral MedicineT lymphocyteMolecular biologyInterleukin-10Mice Inbred C57BLmedicine.anatomical_structureImmunizationInterleukin-4Lymph NodesHaptensCD8Alpha chainInternational Immunology
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IFN-γ–Producing CD4+ T Cells Promote Generation of Protective Germinal Center–Derived IgM+ B Cell Memory against Salmonella Typhi

2014

Abstract Abs play a significant role in protection against the intracellular bacterium Salmonella Typhi. In this article, we investigated how long-term protective IgM responses can be elicited by a S. Typhi outer-membrane protein C– and F–based subunit vaccine (porins). We found that repeated Ag exposure promoted a CD4+ T cell–dependent germinal center reaction that generated mutated IgM-producing B cells and was accompanied by a strong expansion of IFN-γ–secreting T follicular helper cells. Genetic ablation of individual cytokine receptors revealed that both IFN-γ and IL-17 are required for optimal germinal center reactions and production of porin-specific memory IgM+ B cells. However, mor…

CD4-Positive T-LymphocytesMaleSalmonella VaccinesProtein subunitmedicine.medical_treatmentImmunologyCellBiologySalmonella typhiMicrobiologyInterferon-gammaMice03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansImmunology and AllergyTyphoid FeverReceptorB cell030304 developmental biologyMice KnockoutB-Lymphocytes0303 health sciencesGerminal centerSalmonella typhiGerminal Center3. Good healthVaccinationmedicine.anatomical_structureCytokineImmunoglobulin MbacteriaFemaleImmunologic Memory030215 immunologyThe Journal of Immunology
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Prospective Study of the Evolution of Blood Lymphoid Immune Parameters during Dacarbazine Chemotherapy in Metastatic and Locally Advanced Melanoma Pa…

2014

BackgroundThe importance of immune responses in the control of melanoma growth is well known. However, the implication of these antitumor immune responses in the efficacy of dacarbazine, a cytotoxic drug classically used in the treatment of melanoma, remains poorly understood in humans.MethodsIn this prospective observational study, we performed an immunomonitoring of eleven metastatic or locally advanced patients treated with dacarbazine as a first line of treatment. We assessed by flow cytometry lymphoid populations and their activation state; we also isolated NK cells to perform in vitro cytotoxicity tests.ResultsWe found that chemotherapy induces lymphopenia and that a significantly hig…

CD4-Positive T-LymphocytesMaleSkin Neoplasmsmedicine.medical_treatmentCancer TreatmentGene ExpressionNK cellsLymphocyte ActivationT-Lymphocytes RegulatoryLeukocyte CountCellular typesMedicine and Health SciencesCytotoxic T cellProspective StudiesNeoplasm MetastasisProspective cohort studyImmune ResponseMelanomaAged 80 and overMultidisciplinarymedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionMelanomaQRMiddle AgedFlow CytometryPrognosis3. Good healthDacarbazineKiller Cells NaturalTreatment OutcomeOncologyCutaneous MelanomaMedicineWhite blood cellsFemaleImmunotherapymedicine.drugResearch ArticleTumor ImmunologyAdultCell biologyBlood cellsCell SurvivalDacarbazineScienceImmune CellsImmunologyLocally advancedT cellsMalignant Skin NeoplasmsDermatologyCancer ImmunotherapyFlow cytometryImmune systemCell Line TumormedicineHumansAntineoplastic Agents AlkylatingAgedChemotherapyBiology and life sciencesbusiness.industrymedicine.diseaseAnimal cellsImmunologyCancer researchClinical ImmunologybusinessTranscription FactorsPLoS ONE
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Protein kinase CK2 enables regulatory T cells to suppress excessive TH2 responses in vivo

2014

The quality of the adaptive immune response depends on the differentiation of distinct CD4(+) helper T cell subsets, and the magnitude of an immune response is controlled by CD4(+)Foxp3(+) regulatory T cells (Treg cells). However, how a tissue- and cell type-specific suppressor program of Treg cells is mechanistically orchestrated has remained largely unexplored. Through the use of Treg cell-specific gene targeting, we found that the suppression of allergic immune responses in the lungs mediated by T helper type 2 (TH2) cells was dependent on the activity of the protein kinase CK2. Genetic ablation of the β-subunit of CK2 specifically in Treg cells resulted in the proliferation of a hithert…

CD4-Positive T-LymphocytesMaleT cellImmunologyMice TransgenicReceptors Cell Surfacechemical and pharmacologic phenomenaCell Growth ProcessesT-Lymphocytes RegulatoryCell LineMiceTh2 CellsImmune systemHypersensitivitymedicineAnimalsHumansImmunology and AllergyIL-2 receptorCasein Kinase IIMice Inbred BALB CChemistryPeripheral toleranceFOXP3Cell DifferentiationForkhead Transcription FactorsDendritic CellsAcquired immune systemCell biologyMice Inbred C57BLmedicine.anatomical_structureCell cultureInterferon Regulatory FactorsImmunologyLeukocytes MononuclearIRF4Nature Immunology
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The Transcription Factor T-bet Regulates Mucosal T Cell Activation in Experimental Colitis and Crohn's Disease

2002

The balance between pro and antiinflammatory cytokines secreted by T cells regulates both the initiation and perpetuation of inflammatory bowel diseases (IBD). In particular, the balance between interferon (IFN)-gamma/interleukin (IL)-4 and transforming growth factor (TGF)-beta activity controls chronic intestinal inflammation. However, the molecular pathways that evoke these responses are not well understood. Here, we describe a critical role for the transcription factor T-bet in controlling the mucosal cytokine balance and clinical disease. We studied the expression and function of T-bet in patients with IBD and in mucosal T cells in various T helper (Th)1- and Th2-mediated animal models …

CD4-Positive T-LymphocytesMalecolitisGenes RAG-1T-Lymphocytesmedicine.medical_treatmentMice SCIDGATA-3Polymerase Chain ReactionMiceInterleukin 210302 clinical medicineCrohn DiseaseT-Lymphocyte SubsetsImmunology and AllergyCytotoxic T cellIL-2 receptorIFN-γMice Inbred BALB C0303 health sciencesGene Transfer Techniqueshemic and immune systemsT-Lymphocytes Helper-InducerMiddle Aged3. Good healthCytokinemedicine.anatomical_structureFemaleAdultT cellImmunologychemical and pharmacologic phenomenaBiologyT-betArticleTCIRG103 medical and health sciencesmedicineAnimalsHumansColitisImmunity MucosalInterleukin 4DNA Primers030304 developmental biologyHomeodomain ProteinsBase Sequencemedicine.diseasecytokinesDisease Models AnimalGene Expression RegulationImmunologyT-Box Domain ProteinsSpleenTranscription Factors030215 immunologyJournal of Experimental Medicine
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WHOLE BODY IRRADIATION INDUCES IFN-γ PRODUCTION IN BALB/c MICE BY PREVENTING THE APPEARANCE OF A Vα14+NK T DOWNREGULATORY POPULATION

2000

Lymph node cells from TNCB-immune BALB/c mice fail to produce IFN-gamma when exposed to antigen in vitro. Conversely, lymph node cells of irradiated (550 rads) BALB/c mice produce IFN-gamma. Transfer experiments show that normal BALB/c mice contain cells which suppress IFN-gamma production. These downregulatory cells are CD4(+)alpha beta(+)and rearrange the invariant V alpha 14-J alpha 281 T cell receptor alpha chain, thus belonging to the NK T cell subset. Downregulatory cells probably act by producing IL-4 as their effect is blocked by mAb to IL-4.

CD4-Positive T-LymphocytesMalemedicine.drug_classCD8 AntigensReceptors Antigen T-Cell alpha-betaImmunologyPopulationWhole body irradiationAntigen-Presenting CellsDown-RegulationAntigens ProtozoanEnzyme-Linked Immunosorbent AssayCell SeparationMonoclonal antibodyBiochemistryBALB/cInterferon-gammaMiceAntigenmedicineAnimalsImmunology and AllergyeducationMolecular BiologyLymph nodeLeishmania majorMice Inbred BALB Ceducation.field_of_studybiologyChemistryT-cell receptorAntibodies MonoclonalHematologyFlow Cytometrybiology.organism_classificationMolecular biologyIn vitroKiller Cells NaturalPhenotypemedicine.anatomical_structureCD4 AntigensImmunologyInterleukin-4Lymph NodesSpleenCytokine
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Immunological pattern in patients with 21-hydroxylase deficiency.

1994

The aim of this work was to perform an immunological study in six patients with 21 hydroxylase deficiency in mild form (M210HD) and in 2 patients with 21-hydroxylase deficiency in classical form (C210HD) and in their parents, in whom a previous HLA,C4,Bf typing demonstrated high prevalence of DR5 and phenotypic absence of fraction C4B of complement (C4BQO). This study contains the evaluation of C3, IgA, IgG, IgM levels, anticardiolipin antibodies (IgG and IgM) and circulating immunocomplexes. A study of lymphocyte subsets was also performed. Among M210HD 1 patient showed presence of anticardiolipin antibodies both IgM and IgG; this patient had shown antinuclear antibodies in a previous stud…

CD4-Positive T-LymphocytesMalemedicine.medical_specialtyAnti-nuclear antibodyAdolescentEndocrinology Diabetes and MetabolismHuman leukocyte antigenImmunogeneticsBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunityPathogenesisEndocrinologyImmune systemImmunityInternal medicinemedicineHumansFamily HealthAdrenal Hyperplasia CongenitalImmunityT-Lymphocytes Helper-InducerEndocrinologyImmunoglobulin MAntibodies AnticardiolipinImmunoglobulin GImmunologybiology.proteinFemaleAntibodyJournal of endocrinological investigation
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Serum pancreatic enzymes in human immunodeficiency virus-infected children - A collaborative study of the Italian Society of Pediatric Gastroenterolo…

1998

Numerous studies have shown pancreatic disease in adult human immunodeficiency virus (HIV)-infected patients, but there are very few reports on pediatric patients. Our aim was to determine the prevalence of increased serum pancreatic enzyme levels and their relationship to clinical manifestations of acute pancreatitis in HIV-infected children.Forty-seven consecutive, symptomatic HIV-infected children (24 male; median age, 7.3 years; range, 1-17 years) and 45 sex- and age-matched controls without gastroenterologic disease were enrolled. In all subjects serum total amylase, pancreatic amylase, and lipase were assayed with commercial kits. The following were recorded: disease progression (CDC …

CD4-Positive T-LymphocytesMalemedicine.medical_specialtyPancreatic diseaseAdolescentNutritional StatusHIV InfectionsGastroenterologyImmunopathologyInternal medicineMedicineHumansChildPancreasPediatric gastroenterologyAIDS-Related Opportunistic Infectionsbusiness.industryGastroenterologyInfantLipaseHepatologymedicine.diseasemedicine.anatomical_structurePancreatitisChild PreschoolImmunologyAmylasesVomitingDisease ProgressionAcute pancreatitisFemaleViral diseasemedicine.symptombusinessPancreas
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