Search results for " Transgenic"

showing 10 items of 522 documents

Exercise and probiotics attenuate the development of Alzheimer's disease in transgenic mice: Role of microbiome

2018

Abstract It has been suggested that exercise training and probiotic supplementation could decelerate the progress of functional and biochemical deterioration in APP/PS1 transgenic mice (APP/PS1TG). APP/PS1TG mice were subjected to exercise training and probiotic treatments and functional, biochemical and microbiome markers were analyzed. Under these conditions the mice significantly outperformed controls on The Morris Maze Test, and the number of beta-amyloid plaques decreased in the hippocampus. B. thetaiotaomicron levels correlated highly with the results of the Morris Maze Test (p

Male0301 basic medicineGenetically modified mouseAgingalzheimer; exercise; microbiome; probioticsalzheimermicrobiomeMice TransgenicPlaque AmyloidDiseaseHippocampusBiochemistrylaw.inventionAmyloid beta-Protein PrecursorMice03 medical and health sciencesProbiotic0302 clinical medicineEndocrinologyAlzheimer DiseaselawPhysical Conditioning Animalmental disordersPresenilin-1GeneticsAnimalsHippocampus (mythology)MedicineMicrobiomeMolecular BiologyAmyloid beta-Peptidesexercisebusiness.industryMicrobiotaCell BiologyDisease Models Animal030104 developmental biologyprobioticsImmunologybusiness030217 neurology & neurosurgeryExperimental Gerontology
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Sexually Dimorphic Behavioral Profile in a Transgenic Model Enabling Targeted Recombination in Active Neurons in Response to Ketamine and (2R,6R)-Hyd…

2020

Background: Rapid-acting antidepressants ketamine and (2R,6R)-hydroxynorketamine ((2R,6R)-HNK) have overcome some of the major limitations of classical antidepressants. However, little is known about sex-specific differences in the behavioral and molecular effects of ketamine and (2R,6R)-HNK in rodents. Methods: We treated mice with an intraperitoneal injection of either saline, ketamine (30 mg kg&minus

Male0301 basic medicineHydroxynorketaminemedicine.medical_treatmentAntidepressantAnxietyHippocampuslcsh:Chemistry0302 clinical medicinelcsh:QH301-705.5Salineactivated neuronsSpectroscopyNeuronsRecombination GeneticSex CharacteristicsBehavior AnimalhydroxynorketamineGeneral MedicineComputer Science ApplicationsActivated neuronsAntidepressantFemaleKetaminemedicine.drugmedicine.medical_specialtyketamineMemory Episodicsex differenceGreen Fluorescent ProteinsIntraperitoneal injectionMice TransgenicIn situ hybridizationBiologyHydroxynorketamineArticleCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineketamine ; sex difference ; activated neurons ; antidepressant ; behavior ; BDNF ; rapid-acting ; hydroxynorketaminemedicineAnimalsKetamineRapid-actingPhysical and Theoretical ChemistrySocial BehaviorMolecular BiologyCell NucleusBehaviorantidepressantbehaviorBrain-Derived Neurotrophic FactorOrganic ChemistrySex differencerapid-actingSexual dimorphismDisease Models AnimalBDNF030104 developmental biologyEndocrinologylcsh:Biology (General)lcsh:QD1-999030217 neurology & neurosurgeryBehavioural despair test
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Protective effects of mirtazapine in mice lacking the Mbnl2 gene in forebrain glutamatergic neurons: Relevance for myotonic dystrophy 1

2019

Myotonic dystrophy type 1 (DM1) is a multisystemic disorder characterized by muscle weakness and wasting and by important central nervous system-related symptoms including impairments in executive functions, spatial abilities and increased anxiety and depression. The Mbnl2 gene has been implicated in several phenotypes consistent with DM1 neuropathology. In this study, we developed a tissue-specific knockout mouse model lacking the Mbnl2 gene in forebrain glutamatergic neurons to examine its specific contribution to the neurobiological perturbations related to DM1. We found that these mice exhibit long-term cognitive deficits and a depressive-like state associated with neuronal loss, increa…

Male0301 basic medicineMirtazapineGlutamic AcidHippocampusMice TransgenicMirtazapineMyotonic dystrophyAnimals Genetically ModifiedMice03 medical and health sciencesCellular and Molecular NeuroscienceGlutamatergicProsencephalon0302 clinical medicinemedicineAnimalsMyotonic DystrophyDentate gyrusInflammationMice KnockoutNeuronsPharmacologyDepressionbusiness.industryCognitive deficitsDentate gyrusNeurogenesisRNA-Binding Proteinsmedicine.disease3. Good healthMice Inbred C57BLNeuroprotective Agents030104 developmental biologynervous systemKnockout mouseForebrainNeuronal lossDrosophilaFemaleDM1businessNeuroscience030217 neurology & neurosurgerymedicine.drugNeuropharmacology
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Causal Link between n-3 Polyunsaturated Fatty Acid Deficiency and Motivation Deficits

2020

International audience; Reward-processing impairment is a common symptomatic dimension of several psychiatric disorders. However, whether the underlying pathological mechanisms are common is unknown. Herein, we asked if the decrease in the n-3 polyunsaturated fatty acid (PUFA) lipid species, consistently described in these pathologies, could underlie reward-processing deficits. We show that reduced n-3 PUFA biostatus in mice leads to selective motivational impairments. Electrophysiological recordings revealed increased collateral inhibition of dopamine D2 receptor-expressing medium spiny neurons (D2-MSNs) onto dopamine D1 receptor-expressing MSNs in the nucleus accumbens, a main brain regio…

Male0301 basic medicineN-3 PUFAPhysiology[SDV]Life Sciences [q-bio]DopamineMice TransgenicNucleus accumbensMedium spiny neuronMice03 medical and health sciences0302 clinical medicineDopamineDopamine receptor D2Fatty Acids Omega-3medicineAnimalsMolecular BiologyPathological030304 developmental biologyNeuronsMedium spiny neuronschemistry.chemical_classification[SDV.GEN]Life Sciences [q-bio]/Genetics0303 health sciencesMotivationReceptors Dopamine D2business.industry[SCCO.NEUR]Cognitive science/NeuroscienceCell BiologyMice Inbred C57BLElectrophysiology[SDV.GEN.GA]Life Sciences [q-bio]/Genetics/Animal geneticsElectrophysiology030104 developmental biologychemistryNucleus accumbensFemalePolyunsaturated fatty acids[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Causal linkbusinessNeuroscience030217 neurology & neurosurgerymedicine.drugPolyunsaturated fatty acidCell Metabolism
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Optogenetic Modulation of a Minor Fraction of Parvalbumin-Positive Interneurons Specifically Affects Spatiotemporal Dynamics of Spontaneous and Senso…

2017

Abstract Parvalbumin (PV) positive interneurons exert strong effects on the neocortical excitatory network, but it remains unclear how they impact the spatiotemporal dynamics of sensory processing in the somatosensory cortex. Here, we characterized the effects of optogenetic inhibition and activation of PV interneurons on spontaneous and sensory-evoked activity in mouse barrel cortex in vivo. Inhibiting PV interneurons led to a broad-spectrum power increase both in spontaneous and sensory-evoked activity. Whisker-evoked responses were significantly increased within 20 ms after stimulus onset during inhibition of PV interneurons, demonstrating high temporal precision of PV-shaped inhibition.…

Male0301 basic medicineTime FactorsCognitive NeurosciencePopulationAction PotentialsMice TransgenicSensory systemOptogeneticsSomatosensory system03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineLateral inhibitionEvoked Potentials SomatosensoryPhysical StimulationparvalbuminmedicineAnimalseducationmouseeducation.field_of_studyinterneuronsbiologyChemistrymusculoskeletal neural and ocular physiologyOriginal ArticlesSomatosensory CortexBarrel cortexMice Inbred C57BLOptogeneticsParvalbumins030104 developmental biologymedicine.anatomical_structureTouch Perceptionnervous systemCerebral cortexconnectivityVibrissaebiology.proteincerebral cortexFemaleMicroelectrodesNeuroscience030217 neurology & neurosurgeryParvalbuminCerebral Cortex
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Dark exposure affects plasticity-related molecules and interneurons throughout the visual system during adulthood

2020

Several experimental manipulations, including visual deprivation, are able to induce critical period-like plasticity in the visual cortex of adult animals. In this regard, many studies have analyzed the effects of dark exposure in adult animals, but still little is known about the role of interneurons and plasticity-related molecules on such mechanisms. In this study, we analyzed the effects of 10 days of dark exposure on the connectivity and structure of interneurons, both in the primary visual cortex and in the rest of cerebral regions implicated in the transmission of visual stimulus. We found that this environmental manipulation induces changes in the expression of synaptic molecules th…

Male0301 basic medicinegenetic structuresinterneurons ()Mice TransgenicNeural Cell Adhesion Molecule L1Stimulus (physiology)PlasticityInhibitory postsynaptic potentialsensory deprivation ()Mice03 medical and health sciences0302 clinical medicineInterneuronsextracellular matrix ()medicineAnimalsVisual Cortexvisual pathways ()Neuronal PlasticitybiologyGeneral NeurosciencePerineuronal netAge FactorsDarknessPSA-NCAM ()030104 developmental biologyVisual cortexmedicine.anatomical_structureSialic Acidsbiology.proteinNeural cell adhesion moleculeneuronal plasticity ()Nerve NetSensory DeprivationNeuroscience030217 neurology & neurosurgeryParvalbumin
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A human relevance investigation of PPARα-mediated key events in the hepatocarcinogenic mode of action of propaquizafop in rats

2018

Propaquizafop is an herbicide with demonstrated hepatocarcinogenic activity in rodents. A rodent-specific mode of action (MOA) in the liver via activation of peroxisome proliferator-activated receptor α (PPARα) has been postulated based on existing data. Experience with PPARα-inducing pharmaceuticals indicates a lack of human relevance of this MOA. The objective of the present investigation was to evaluate the dependency of early key events leading to liver tumors on PPARα activation in wildtype (WT) compared to PPARα-knockout (KO) rats following 2 weeks exposure to 75, 500 and 1000 ppm propaquizafop in the diet. In WT rats, both WY-14643 (50 mg/kg bw/day) and propaquizafop (dose-dependentl…

Male0301 basic medicinemedicine.medical_specialtyPeroxisome proliferator-activated receptor010501 environmental sciencesBiologyToxicologyRisk Assessment01 natural sciencesMuscle hypertrophyRats Sprague-Dawley03 medical and health sciencesCytochrome P-450 Enzyme SystemInternal medicinemedicineAnimalsHumansAcyl-CoA oxidasePPAR alphaRelevance (information retrieval)Enzyme inducerReceptorMode of actionCarcinogen0105 earth and related environmental scienceschemistry.chemical_classificationGlutathione PeroxidaseHerbicidesGlutathione peroxidaseLiver NeoplasmsOrgan SizeGeneral MedicineGlutathioneDiet030104 developmental biologyEndocrinologyLiverchemistrybiology.proteinKey (cryptography)Acyl-CoA OxidasePropionatesRats TransgenicNeuroscienceToxicology Letters
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Neuropeptide FF increases M2 activation and self-renewal of adipose tissue macrophages

2017

The quantity and activation state of adipose tissue macrophages (ATMs) impact the development of obesity-induced metabolic diseases. Appetite-controlling hormones play key roles in obesity; however, our understanding of their effects on ATMs is limited. Here, we have shown that human and mouse ATMs express NPFFR2, a receptor for the appetite-reducing neuropeptide FF (NPFF), and that NPFFR2 expression is upregulated by IL-4, an M2-polarizing cytokine. Plasma levels of NPFF decreased in obese patients and high-fat diet-fed mice and increased following caloric restriction. NPFF promoted M2 activation and increased the proliferation of murine and human ATMs. Both M2 activation and increased ATM…

Male0301 basic medicinemedicine.medical_specialty[SDV.BIO]Life Sciences [q-bio]/BiotechnologyUbiquitin-Protein LigasesAdipose tissue macrophagesMafB Transcription FactorAdipose tissueMice TransgenicReceptors Cell SurfaceSelf renewalMice03 medical and health sciencesClinical investigationInternal medicinemedicineAnimalsNeuropeptide FFTranscription factorAdaptor Proteins Signal TransducingCell ProliferationSTAT62. Zero hungerArginasebiologybusiness.industryChemistryMacrophagesProteinsSciences du Vivant [q-bio]/BiotechnologiesGeneral MedicineMacrophage ActivationInterleukin-10Ubiquitin ligaseCell biologyEndocrinology030104 developmental biologyAdipose TissueMAFBbiology.proteinInterleukin-4CorrigendumbusinessOligopeptidesMacrophage proliferationResearch ArticleJournal of Clinical Investigation
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Elevated Testosterone Level and Urine Scent Marking in Male 5xFAD Alzheimer Model Mice

2019

Background:Function of the Amyloid Precursor Protein (AβPP) and its various cleavage products still is not unraveled down to the last detail. While its role as a source of the neurotoxic Amyloid beta (Aβ) peptides in Alzheimer’s Disease (AD) is undisputed and its property as a cell attachment protein is intriguing, while functions outside the neuronal context are scarcely investigated. This is particularly noteworthy because AβPP has a ubiquitous expression profile and its longer isoforms, AβPP750 and 770, are found in various tissues outside the brain and in non-neuronal cells.Objective:Here, we aimed at analyzing the 5xFAD Alzheimer’s disease mouse model in regard to male sexual function.…

Male0301 basic medicinemedicine.medical_specialtymiceAmyloid betaCentral nervous system610Mice Transgenicamyloid precursor proteinAmyloid beta-Protein PrecursorSexual Behavior Animal03 medical and health sciences0302 clinical medicinesexual behaviorAlzheimer DiseaseInternal medicineTestisPresenilin-1medicineAmyloid precursor proteinAnimalsSperm CountbiologyWild typeBrainOrgan SizeAlzheimer's diseaseSertoli cellSpermPhenotypeAndrogen receptorDisease Models Animal030104 developmental biologymedicine.anatomical_structureEndocrinologyNeurologytestosteronebiology.proteinNeurology (clinical)030217 neurology & neurosurgeryurine scent marking test
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Alteration of inhibitory circuits in the somatosensory cortex of Ts65Dn mice, a model for Down's syndrome.

2010

Down’s syndrome (DS), with an incidence of one in 800 live births, is the most common genetic disorder associated with mental retardation. This trisomy on chromosome 21 induces a variable phenotype in which the only common feature is the presence of mental retardation. The neural mechanisms underlying mental retardation might include defects in the formation of neuronal networks and neural plasticity. DS patients have alterations in the morphology, the density and the distribution of dendritic spines in the pyramidal neurons of the cortex. Our hypothesis is that the deficits in dendritic arborization observed in the principal neurons of DS patients and Ts65Dn mice (a model for DS that mimic…

MaleAgingDendritic spineFisiologia patològicaSynaptophysinCell CountMice TransgenicInhibitory postsynaptic potentialSomatosensory systemMiceInterneuronsCortex (anatomy)NeuroplasticityNeural PathwaysmedicineAnimalsBiological PsychiatrybiologyGlutamate DecarboxylaseCalcium-Binding ProteinsNeural InhibitionSomatosensory CortexImmunohistochemistryPsychiatry and Mental healthDisease Models Animalmedicine.anatomical_structureNeurologynervous systemSynapsesbiology.proteinSynaptophysinNeurology (clinical)CalretininDown SyndromeNeuroscienceParvalbuminJournal of neural transmission (Vienna, Austria : 1996)
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