Search results for " aging."

showing 10 items of 556 documents

Sarcopenia reduces quality of life in the long-term: Longitudinal analyses from the English Longitudinal Study of Ageing

2022

Abstract Purpose Mixed findings exist for sarcopenia/quality of life (QoL) relationship. Moreover, the majority of studies in this area have utilized a cross-sectional design or specific clinical populations. Therefore, the aim of the present study was to examine the association between sarcopenia at baseline and QoL at 10 years follow-up in a large representative sample of older English adults. Methods Sarcopenia was diagnosed as having low handgrip strength and low skeletal muscle mass index. QoL was measured using the CASP (control, autonomy, self-realisation and pleasure)-19, with higher values reflecting higher QoL. Multivariable logistic regression analysis was conducted to assess pro…

Quality of lifeMaleAgingSarcopeniaHand StrengthEpidemiologyELSAAgeingCross-Sectional StudiesAgeing; ELSA; Epidemiology; Longitudinal; Older adults; Quality of life; Sarcopenia; Aged; Aging; Cross-Sectional Studies; Female; Hand Strength; Humans; Longitudinal Studies; Male; Quality of Life; SarcopeniaOlder adultsLongitudinalQuality of LifeHumansFemaleQuality of life Sarcopenia ELSA Older adults Epidemiology Longitudinal AgeingLongitudinal Studieshuman activitiesAged
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Timed up-and-go performance is associated with objectively measured life space in patients 3 months after ischemic stroke: a cross-sectional observat…

2023

Background Stroke is a common cause of mobility limitation, including a reduction in life space. Life space is defined as the spatial extent in which a person moves within a specified period of time. We aimed to analyze patients' objective and self-reported life space and clinical stroke characteristics. Methods MOBITEC-Stroke is a prospective observational cohort study addressing poststroke mobility. This cross-sectional analysis refers to 3-month data. Life space was assessed by a portable tracking device (7 consecutive days) and by self-report (Life-Space Assessment; LSA). We analysed the timed up-and-go (TUG) test, stroke severity (National Institutes of Health Stroke Scale; NIHSS), and…

Quality of lifeMobility limitation; Spatial behaviour; Quality of life; GPS; Mobility capacityGPSspatial behaviourUFSP13-4 Dynamics of Healthy AgingliikuntarajoitteetelämänlaatuSpatial behaviourMobility limitationmobility capacityaivohalvausmobility limitation10122 Institute of Geography2728 Neurology (clinical)liikkuvuusNeurologyquality of life2808 NeurologyliikuntakykyelinympäristökuntoutusNeurology (clinical)910 Geography & travelMobility capacitykohorttitutkimus
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RISK FACTORS, VASCULAR AGING AND ATHEROSCLEROSIS: 3rd ANTONIO STRANO LECTURE

2006

RISK FACTORS, VASCULAR AGING AND ATHEROSCLEROSIS: 3rd ANTONIO STRANO LECTURE

RISK FACTORS VASCULAR AGING AND ATHEROSCLEROSIS: 3rd ANTONIO STRANO LECTURE
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Differential associations of age with volume and microstructure of hippocampal subfields in healthy older adults

2015

Hippocampal atrophy in advanced healthy aging has frequently been reported. However, the vulnerability of different hippocampal subfields to age-related atrophy is still a source of debate. Moreover, the association of age with the microstructural integrity of subfields is largely unknown. In this study, we investigated the associations between age and volume as well as microstructural integrity of hippocampal subfields using a three-dimensional (3D) surface mapping approach. Forty-three healthy older adults spanning the age range from 60 to 85 years underwent T1-weighted and diffusion-tensor imaging. Analyses demonstrated an association of age with hippocampal volume predominantly in the m…

Radiological and Ultrasound TechnologySubiculumHippocampal formationmedicine.diseaseHippocampal atrophySurface mappingAtrophynervous systemNeurologymedicineHippocampal volumeRadiology Nuclear Medicine and imagingNeurology (clinical)AnatomyHealthy agingPsychologyNeuroscienceDiffusion MRIHuman Brain Mapping
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Comparison of Disease Clusters in Two Elderly Populations Hospitalized in 2008 and 2010

2013

<b><i>Background:</i></b> As chronicity represents one of the major challenges in the healthcare of aging populations, the understanding of how chronic diseases distribute and co-occur in this part of the population is needed. <b><i>Objectives:</i></b> The aims of this study were to evaluate and compare patterns of diseases identified with cluster analysis in two samples of hospitalized elderly. <b><i>Methods:</i></b> Data were obtained from the multicenter ‘Registry Politerapie SIMI (REPOSI)' that included people aged 65 or older hospitalized in internal medicine and geriatric wards in Italy during 2008 and 2010. The s…

RegistrieMaleAgingCirrhosisSettore MED/09 - Medicina InternaTime FactorshispitalizationGerontology; aging populations;atterns of multimorbidity; diseases in the elderly population.Health care80 and overPrevalenceChronic diseases; Cluster analysis; Hospitalized elderlyRegistriesHospitalized elderlyaging populationAged 80 and overeducation.field_of_studySettore MED/45 - Scienze Infermieristiche Generali Cliniche E PediatricheHospitalizationItalyelderly; hispitalization; disease clustersFemaledisease clustershospitalizedHumanmedicine.medical_specialtyTime FactorAnemiaMULTIMORBIDITYPopulationMEDLINEMalignancyelderlyCluster analysisDiabetes mellitusInternal medicinedisease clusters; elderly; hospitalizedmedicinediseases in the elderly population.MultimorbidityHumansatterns of multimorbidityeducationAgedChronic diseases; Cluster analysis; Hospitalized elderly; Aged; Aged; 80 and over; Chronic Disease; Female; Hospitalization; Humans; Italy; Male; Prevalence; Registries; Time Factors; Cluster Analysis; Aging; Geriatrics and GerontologyCluster Analysibusiness.industrymedicine.diseaseChronic diseasesChronic DiseasePhysical therapydisease clusterGeriatrics and GerontologybusinessGerontologyChronic diseases; Cluster analysis; Hospitalized elderly; Aged; Aged 80 and over; Chronic Disease; Female; Hospitalization; Humans; Italy; Male; Prevalence; Registries; Time Factors; Cluster Analysis; Aging; Geriatrics and Gerontology
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Mothers' parenting stress and adolescents' emotional separation: The role of youngsters' self orientation

2015

The study examined the association among mothers’ parenting stress, adolescents’ emotional separation and self-orientation toward connectedness. Participants were 194 Italian adolescents, aged from 15 to 19 years (mean age = 17.39, SD = 1.18), and their mothers, aged from 33 to 64 years (mean age = 44.35, SD = 5.40). General findings showed that adolescents’ emotional separation may not necessarily be associated with their mothers’ parenting stress, but both of these variables may be related to adolescents’ personal characteristics, which may contribute to define parent-child relationship. Particularly, adolescents’ orientation towards a connected self was associated negatively with emotion…

Self orientationAgingChild rearingSocial PsychologySeparation (statistics)Parenting stressEmotional separationParent-adolescent relationshipStructural equation modelingDevelopmental psychologyParenting stressDevelopmental NeurosciencePARENTING STRESS PARENT-ADOLESCENT RELATIONSHIP EMOTIONAL SEPARATION SELF-ORIENTATIONDevelopmental and Educational PsychologyEmotional separation; Parent-adolescent relationship; Parenting stress; Self-orientation; Social Psychology; Aging; Developmental and Educational Psychology; Developmental Neuroscience; Life-span and Life-course StudiesPsychologySelf-orientationLife-span and Life-course StudiesSocial psychology
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Hsp60 and human aging: Les liaisons dangereuses 

2013

Stressors can cause abnormal intracellular accumulation of Hsp60 and its localization in extramitochondrial sites, secretion, and circulation, with immune system activation. Dysfunction of chaperones associated with their quantitative and qualitative decline with aging (chaperonopathies of aging) characterizes senescence and is a potential causal factor in the physiological deterioration that occurs with it. The role of Hsp60 in aging is not easy to elucidate, because aging is accompanied by pathologies (e.g., cardiovascular and neurodegenerative disorders, osteoporosis, diabetes, cancer, etc.) in which Hsp60 has been implicated but, although those disorders are more frequent in the elderly…

SenescenceAginganimal structuresOsteoporosischemical and pharmacologic phenomenaInflammationDiseaseBiologycomplex mixturesMitochondrial ProteinsPathogenesisImmune systemDiabetes mellitusmedicineHumansCellular SenescenceAutoantibodiesHeart FailurefungiHSP 60 AGING CHAPERONES.Neurodegenerative DiseasesChaperonin 60Atherosclerosismedicine.diseaseMitochondriaImmune SystemImmunologyHSP60Arthropathy Neurogenicmedicine.symptomFrontiers in Bioscience
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Overexpression of apolipoprotein J in human fibroblasts protects against cytotoxicity and premature senescence induced by ethanol and tert-butylhydro…

2008

Human diploid fibroblasts (HDFs) exposed to subcytotoxic stresses under H2O2, tert-butylhydroperoxide (t-BHP), and ethanol (EtOH) undergo stress-induced premature senescence (SIPS) characterized by many biomarkers of HDFs replicative senescence. Among these biomarkers are a growth arrest, an increase in the senescence-associated β-galactosidase activity, a senescent morphology, an overexpression of p21waf-1 and the subsequent inability to phosphorylate pRb, the presence of the common 4977-bp mitochondrial deletion, and an increase in the steady-state level of several senescence-associated genes such as apolipoprotein J (apo J). Apo J has been described as a survival gene against cytotoxic s…

SenescenceCell SurvivalGene ExpressionSimian virus 40Biologymedicine.disease_causeTritiumBiochemistrytert-ButylhydroperoxideGene expressionmedicineHumansOsteonectinRNA MessengerCytotoxicityCells CulturedCellular SenescenceCell Line TransformedGlycoproteinsClusterinEthanolCentral Nervous System DepressantsCell BiologyTransfectionOriginal ArticlesFibroblastsbeta-GalactosidaseMolecular biologyRecombinant ProteinsFibronectinsOxidative StressClusterinbiology.proteinPhosphorylationMitogensCell agingOxidative stressMolecular ChaperonesThymidineCell Stress and Chaperones
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Senescence-associated HSP60 expression in normal human skin fibroblasts

2005

Normal mammalian fibroblasts cultured in vitro undergo a limited number of divisions before entering a senescent phase in which they can be maintained for long periods but cannot be induced to divide. Senescent cells become unresponsive to growth-promoting signals and exhibit senescent cell morphology with flattened and enlarged cell shape. Several chaperones have a direct effect on cellular senescence. HSP60 has been largely studied in our laboratories and it has been associated with uncontrolled cell proliferation in tumor cells. Since senescence is firmly regulated during cell cycle progression, we wanted to investigate HSP60 protein level during cellular senescence. Our data show that H…

SenescenceCell divisionCell growthfungiVimentinMitochondrionCell cycleBiologyAgricultural and Biological Sciences (miscellaneous)Cell biologybiology.proteinAnatomyCell agingCellular compartmentThe Anatomical Record Part A: Discoveries in Molecular, Cellular, and Evolutionary Biology
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DNA damage causes TP53-dependent coupling of self-renewal and senescence pathways in embryonal carcinoma cells.

2013

Recent studies have highlighted an apparently paradoxical link between self-renewal and senescence triggered by DNA damage in certain cell types. In addition, the finding that TP53 can suppress senescence has caused a re-evaluation of its functional role in regulating these outcomes. To investigate these phenomena and their relationship to pluripotency and senescence, we examined the response of the TP53-competent embryonal carcinoma (EC) cell line PA-1 to etoposide-induced DNA damage. Nuclear POU5F1/OCT4A and P21CIP1 were upregulated in the same cells following etoposide-induced G 2M arrest. However, while accumulating in the karyosol, the amount of OCT4A was reduced in the chromatin fract…

SenescenceCyclin-Dependent Kinase Inhibitor p21OCT4A/POU5F1Embryonal Carcinoma Stem CellssenescenceDNA RepairDNA repairDNA damagetumor cellsBiologyProtein Serine-Threonine Kinasesself-renewalHistonesAurora KinasesCell Line TumorReportAutophagyAurora Kinase BHumansTP53PhosphorylationRNA Small InterferingMolecular BiologyMitosisCellular SenescenceCyclin-Dependent Kinase Inhibitor p16EtoposideOvarian NeoplasmsEmbryonal Carcinoma Stem CellsCell BiologyG2-M DNA damage checkpointbeta-GalactosidasepluripotencyAntineoplastic Agents PhytogenicChromatinUp-RegulationG2 Phase Cell Cycle CheckpointsCheckpoint Kinase 2Cancer researchDNA damageFemaleRNA InterferenceRad51 RecombinaseTumor Suppressor Protein p53Cell agingOctamer Transcription Factor-3Developmental BiologyCell cycle (Georgetown, Tex.)
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