Search results for " beta"

showing 10 items of 1438 documents

Prognostic Value of Immunohistochemical Expression of Beta-1 Integrin in Pancreatic Carcinoma

1999

<i>Background: </i>Prognostically relevant factors based on the histological assessment of the resected pancreas are known. However, the knowledge of additional factors associated with the prognosis is helpful in planning the therapy for an individual patient. β<sub>1</sub> Integrin expression is known to have a prognostic influence in some malignant tumors. No data are, however, available on the prognostic value of β<sub>1</sub> integrins in pancreatic carcinoma. <i>Method: </i>We investigated paraffin-embedded specimens of 19 patients undergoing surgical treatment for periampullary carcinoma and of 42 patients for ductal pancreatic carcinoma…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyPancreatic diseaseIntegrinFlow cytometryPredictive Value of TestsBeta 1 integrinHumansMedicineAgedNeoplasm StagingAged 80 and overmedicine.diagnostic_testbiologybusiness.industryCell adhesion moleculeIntegrin beta1CarcinomaGeneral MedicineMiddle AgedPrognosismedicine.diseaseImmunohistochemistryGene Expression Regulation NeoplasticPancreatic Neoplasmsmedicine.anatomical_structureOncologyLymphatic Metastasisbiology.proteinAdenocarcinomaImmunohistochemistryFemalebusinessPancreasOncology
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A Putatively Functional Haplotype in the Gene Encoding Transforming Growth Factor Beta-1 as a Potential Biomarker for Radiosensitivity

2011

Purpose To determine whether genetic variability in TGFB1 is related to circulating transforming growth factor-β1 (TGF-β1) plasma concentrations after radiotherapy and to radiosensitivity of lymphoid cells. Patients and Methods Transforming growth factor-β1 plasma concentrations ( n = 79) were measured in patients 1 year after radiotherapy and chromosomal aberrations ( n = 71) ex vivo before therapy start. Furthermore, TGF-β1 secretion and apoptosis were measured in isolated peripheral blood mononuclear cells of 55 healthy volunteers. These phenotypes were analyzed in relation to five germline polymorphisms in the 5′ region of the TGFB1 gene. Because of high linkage disequilibrium, these fi…

AdultMaleCancer ResearchSomatic cellDNA damageApoptosisPolymorphism Single NucleotideRadiation TolerancePeripheral blood mononuclear cellLinkage DisequilibriumCell LineTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineHumansMedicineRadiology Nuclear Medicine and imagingRadiosensitivityGeneMicronuclei Chromosome-DefectiveAged030304 developmental biologyAged 80 and over0303 health sciencesRadiationbiologybusiness.industryHaplotypeTransforming growth factor betaMiddle AgedMolecular biology3. Good healthHaplotypesOncology030220 oncology & carcinogenesisMicronucleus testImmunologyLeukocytes Mononuclearbiology.proteinFemalebusinessBiomarkersDNA DamageInternational Journal of Radiation Oncology*Biology*Physics
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Transforming growth factor-β1 in autoimmune hepatitis: correlation of liver tissue expression and serum levels with disease activity

1998

Abstract Background/Aims: Transforming growth factor-β 1 (TGF-β 1 ) is considered the most important mediator of hepatic fibrogenesis. At the same time, TGF-β 1 is an immunosuppressive cytokine. Development of fibrosis, often rapid, is a characteristic of autoimmune hepatitis, as is spontaneous systemic immunosuppression. The aim of our study was therefore to define the role of TGF-β 1 in autoimmune hepatitis. Methods/Results: Using the MV 1Lu bioassay, we found markedly elevated serum levels of TGF-β 1 (median 109 ng/ml) in active autoimmune hepatitis, which normalised when patients reached biochemical remission following immunosuppressive therapy (median 34 ng/ml; p =0.0001 compared to ac…

AdultMaleCirrhosisAdolescentmedicine.medical_treatmentInflammationAutoimmune hepatitismedicine.disease_causeMonocytesAutoimmunityTransforming Growth Factor betaFibrosisHumansMedicineIn Situ HybridizationAgedInflammationHepatitisHepatologybusiness.industryImmunosuppressionMiddle Agedmedicine.diseaseImmunohistochemistryHepatitis AutoimmuneCytokineLiverImmunologyBiological AssayFemalemedicine.symptombusinessBiomarkersJournal of Hepatology
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Lithium and GSK3-β promoter gene variants influence white matter microstructure in bipolar disorder

2013

Lithium is the mainstay for the treatment of bipolar disorder (BD) and inhibits glycogen synthase kinase 3-beta (GSK3-beta). The less active GSK3-beta promoter gene variants have been associated with less detrimental clinical features of BD. GSK3-beta gene variants and lithium can influence brain gray matter structure in psychiatric conditions. Diffusion tensor imaging (DTI) measures of white matter (WM) integrity showed widespred disruption of WM structure in BD. In a sample of 70 patients affected by a major depressive episode in course of BD, we investigated the effect of ongoing long-term lithium treatment and GSK3-beta promoter rs334558 polymorphism on WM microstructure, using DTI and …

AdultMaleCorpus callosumNerve Fibers MyelinatedWhite matterGlycogen Synthase Kinase 3GSK3-β03 medical and health sciences0302 clinical medicineCorona radiataFasciculusmedicineHumansInferior longitudinal fasciculusPromoter Regions GeneticGSK3-β; lithium; bipolar disorder; white matter; cingulum bundle030304 developmental biologybipolar disorderPharmacology0303 health sciencesGlycogen Synthase Kinase 3 betabiologyGenetic VariationMiddle Agedbiology.organism_classification3. Good healthPsychiatry and Mental healthmedicine.anatomical_structurenervous systemlithiumCorticospinal tractSettore BIO/14 - FarmacologiaGSK3-beta lithium bipolar disorder white matter cingulum bundleFemaleOriginal ArticleBrain Gray Mattercingulum bundlePsychologywhite matterNeuroscience030217 neurology & neurosurgeryDiffusion MRI
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Longitudinal analysis of the T-cell receptor (TCR)-VA and -VB repertoire in CD8+T cells from individuals immunized with recombinant hepatitis B surfa…

2002

SUMMARYRecent studies have suggested that vaccination induces alterations in the T cell receptor (TCR) repertoire. We investigate the diversity of the TCR repertoire after immunization with a recombinant hepatitis B surface vaccine in seven healthy subjects in CD8+ T cells in peripheral blood lymphocytes. Cellular immune responses were monitored over time by sorting CD8 T cells followed by TCR-VA and -VB complementarity determining region 3 (CDR3) analysis. Frequency of individual VB families was determined by flow cytometry. TCR-VA/VB repertoires obtained from CD8+ T cells drawn after vaccination were compared to the TCR repertoire determined prior to vaccination. Monoclonal TCR transcript…

AdultMaleDNA ComplementaryReceptors Antigen T-Cell alpha-betaT cellMolecular Sequence DataImmunologychemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyEpitopeInterleukin 21AntigenAntibody SpecificityClinical StudiesmedicineHumansImmunology and AllergyCytotoxic T cellHepatitis B VaccinesAmino Acid SequenceLongitudinal StudiesGene Rearrangement beta-Chain T-Cell Antigen ReceptorImmunity CellularVaccines SyntheticBase SequenceT-cell receptorAntibodies Monoclonalhemic and immune systemsT lymphocyteMiddle AgedComplementarity Determining RegionsVirologymedicine.anatomical_structureImmunologyFemaleImmunizationGene Rearrangement alpha-Chain T-Cell Antigen ReceptorCD8Clinical and Experimental Immunology
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Temporal lobe grey matter volume in schizophrenia is associated with a genetic polymorphism influencing glycogen synthase kinase 3-beta activity

2010

At the crossroad of multiple pathways regulating trophism and metabolism, glycogen synthase kinase (GSK)3 is considered a key factor in influencing the susceptibility of neurons to harmful stimuli (neuronal resilience) and is a target for several psychiatric drugs that directly inhibit it or increase its inhibitory phosphorylation. Inhibition of GSK3 prevents apoptosis and could protect against the neuropathological processes associated with psychiatric disorders. A GSK3-beta promoter single-nucleotide polymorphism (rs334558) influences transcriptional strength, and the less active form was associated with less detrimental clinical features of mood disorders. Here we studied the effect of r…

AdultMaleGenotypeApoptosisNeuropathologyBiologyGrey matterGene Expression Regulation EnzymologicTemporal lobe03 medical and health sciencesBehavioral NeuroscienceSuperior temporal gyrusGlycogen Synthase Kinase 30302 clinical medicineGSK-3GeneticsmedicineHumansGenetic Predisposition to DiseasePromoter Regions GeneticGSK3B030304 developmental biology0303 health sciencesGlycogen Synthase Kinase 3 betaPolymorphism GeneticGenetic VariationBrodmann area 21medicine.diseaseTemporal LobeEnzyme Activationmedicine.anatomical_structureNeurologySchizophreniaChronic DiseaseNerve DegenerationSchizophreniaFemaleAtrophyNeuroscience030217 neurology & neurosurgeryGenes, Brain and Behavior
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Hepatitis B and C virus variants in long-term immunosuppressed renal transplant patients in Latvia.

2004

The incidence of genome variants of hepatitis B and hepatitis C viruses among 38 long-term (2–15 years) immunosuppressed patients after renal transplantation and 10 patients undergoing dialysis was investigated. Twelve patients had only HBV infection, 9 had only HCV infection and 14 were co-infected. Regions corresponding to the HBV X/EnII/BCP, preC/C, preS/S and to the HCV core were sequenced for molecular characterization of the HBV and HCV genomes. Fifty-seven percent of HBV DNA isolates belonged to genotype D and 42% to genotype A, whereas 77% of HCV RNA isolates belonged to genotype 1b and only 17% to genotype 3a. One sample (6%) was of genotype 2c. Detailed analysis of the above-menti…

AdultMaleHepatitis B virusGenes ViralGenotypeHepatitis B virus DNA polymerasemedicine.medical_treatmentSequence HomologyHepacivirusVirusHepatitis B virus PRE betaVirologymedicineImmune ToleranceHumansPoint MutationDialysisPhylogenyAgedbusiness.industryIncidence (epidemiology)Hepatitis CHepatitis BMiddle Agedmedicine.diseaseHepatitis BVirologyHepatitis CKidney TransplantationLatviaTransplantationInfectious DiseasesAmino Acid SubstitutionFemalebusinessSequence AnalysisIntervirology
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Rare pre-core stop-codon mutant nt. 1897 predominates over wide-spread mutant nt. 1896 in an unusual course of chronic hepatitis B

1996

We present a patient with an unusual course of hepatitis B e antigen (HBeAg)-negative chronic hepatitis B who had repeated reactivations of his disease progressing to cirrhosis with terminal liver failure. Each flare up presented like an acute hepatitis with very high titres of hepatitis B virus (HBV) and high inflammatory activity followed by rapid clearance of viraemia. The pre-core genome of HBV isolated from sera during 5 years of follow up was analysed. Direct sequencing of polymerase chain reaction (PCR) products derived from consecutive sera showed a rare pre-core stop-codon mutation at nucleotide (nt.) 1897 G --> A with an accompanying mutation nt. 1857 C --> T as well as a stop-cod…

AdultMaleHepatitis B virusMolecular Sequence DataMutantBiologymedicine.disease_causePolymerase Chain ReactionHepatitis B virus PRE betaVirusVirologymedicineHumansHepatitis B e AntigensHepatitis B AntibodiesHepatitis B virusMutationHepatitis B Surface AntigensBase SequenceHepatologyHepatitis BHepatitis B Core AntigensVirologyMolecular biologyStop codonInfectious DiseasesLiverViral replicationHBeAgChronic DiseaseDNA ViralMutationCodon TerminatorLiver FailureSignal TransductionT-Lymphocytes Cytotoxic
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An Intergenic rs9275596 Polymorphism on Chr. 6p21 Is Associated with Multiple Sclerosis in Latvians

2020

Background and objectives: Multiple sclerosis (MS) is a chronic inflammatory disease of the central nervous system, leading to demyelination of neurons and potentially debilitating physical and mental symptoms. The disease is more prevalent in women than in men. The major histocompatibility complex (MHC) region has been identified as a major genetic determinant for autoimmune diseases, and its role in some neurological disorders including MS was evaluated. An intergenic single-nucleotide polymorphism (SNP), rs9275596, located between the HLA-DQB1 and HLA-DQA2 genes, is in significant association with various autoimmune diseases according to genome-wide association studies (GWASs). A cumulat…

AdultMaleMedicine (General)the major histocompatibility complex (MHC)PopulationDiseasemultiple sclerosisPolymorphism Single NucleotideArticleR5-920GenotypemedicineHLA-DQ beta-ChainsHumansSNPautoimmune diseasesAlleleeducationGenetic associationGeneticseducation.field_of_studybusiness.industryMultiple sclerosisOdds ratiors9275596; the major histocompatibility complex (MHC); Human leukocyte antigen (HLA); autoimmune diseases; multiple sclerosisMiddle Agedmedicine.diseaseLatviaHuman leukocyte antigen (HLA)Case-Control StudiesAutomotive Engineeringrs9275596FemalebusinessGenome-Wide Association StudyMedicina
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TpF1 from Treponema pallidum Activates Inflammasome and Promotes the Development of Regulatory T Cells

2011

Abstract Human syphilis is a multistage disease, with diverse and wide-ranging manifestations caused by Treponema pallidum. Despite the fact that a cell-mediated immune response takes part in the course of syphilis, T. pallidum often manages to evade host immunity and, in untreated individuals, may trigger chronic infection. With this study, we demonstrate for the first time, to our knowledge, that Treponema pallidum induces a regulatory T (Treg) response in patients with secondary syphilis and we found that the miniferritin TpF1, produced by the bacterium, is able to expand this response and promote the production of TGF-β. Accordingly, TpF1 stimulates monocytes to release IL-10 and TGF-β,…

AdultMaleMultiprotein complexInflammasomesVirulence FactorsCellsT-LymphocytesImmunologyAdult; Antigens Helminth; Cell Differentiation; Cells Cultured; Down-Regulation; Female; Humans; Inflammasomes; Inflammation Mediators; Male; Middle Aged; Monocytes; Syphilis; T-Lymphocytes Regulatory; Transforming Growth Factor beta; Treponema pallidum; Virulence FactorsDown-RegulationBiologyT-Lymphocytes RegulatoryMonocytesMicrobiologyProinflammatory cytokineImmune systemAntigenTransforming Growth Factor betaHelminthmedicineHumansImmunology and AllergySyphilisTreponema pallidumAntigensCells CulturedCulturedTreponemaCell DifferentiationInflammasomeMiddle Agedbiology.organism_classificationmedicine.diseaseRegulatoryChronic infectionAntigens HelminthImmunologyFemaleSyphilisInflammation Mediatorsmedicine.drugThe Journal of Immunology
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