Search results for " cells"

showing 10 items of 6636 documents

Consequence of Histoincompatibility beyond GvH-Reaction in Cytomegalovirus Disease Associated with Allogeneic Hematopoietic Cell Transplantation: Cha…

2021

Hematopoietic cell (HC) transplantation (HCT) is the last resort to cure hematopoietic malignancies that are refractory to standard therapies. Hematoablative treatment aims at wiping out tumor cells as completely as possible to avoid leukemia/lymphoma relapse. This treatment inevitably co-depletes cells of hematopoietic cell lineages, including differentiated cells that constitute the immune system. HCT reconstitutes hematopoiesis and thus, eventually, also antiviral effector cells. In cases of an unrelated donor, that is, in allogeneic HCT, HLA-matching is performed to minimize the risk of graft-versus-host reaction and disease (GvHR/D), but a mismatch in minor histocompatibility antigens …

0301 basic medicine030106 microbiologyCytomegalovirusGraft vs Host DiseaseCD8 T cellsReviewHuman leukocyte antigengraft-versus-host disease (GvHD)MicrobiologyMinor Histocompatibility AntigensMice03 medical and health sciencesImmune systemavidityVirologyMinor histocompatibility antigenmedicineAnimalsHumansTransplantation HomologousCytotoxic T cellImmunodeficiencybusiness.industryHematopoietic Stem Cell Transplantationcytomegalovirus diseasehematopoietic reconstitutionhematopoietic cell transplantation (HCT)medicine.diseaseQR1-502Transplantationantigen presentationLeukemia030104 developmental biologyInfectious DiseasesHematologic NeoplasmsCytomegalovirus InfectionsImmunologybusinessCD8Viruses
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Phosphorylation of CENP-A on serine 7 does not control centromere function.

2019

CENP-A is the histone H3 variant necessary to specify the location of all eukaryotic centromeres via its CENP-A targeting domain and either one of its terminal regions. In humans, several post-translational modifications occur on CENP-A, but their role in centromere function remains controversial. One of these modifications of CENP-A, phosphorylation on serine 7, has been proposed to control centromere assembly and function. Here, using gene targeting at both endogenous CENP-A alleles and gene replacement in human cells, we demonstrate that a CENP-A variant that cannot be phosphorylated at serine 7 maintains correct CENP-C recruitment, faithful chromosome segregation and long-term cell viab…

0301 basic medicine1.1 Normal biological development and functioningScience[SDV]Life Sciences [q-bio]CentromereGeneral Physics and Astronomy02 engineering and technology[SDV.BC]Life Sciences [q-bio]/Cellular Biologymacromolecular substancesBiologyGeneral Biochemistry Genetics and Molecular BiologyArticleSerineChromosome segregation03 medical and health sciencesHistone H3Underpinning researchCentromereGeneticsHumansViability assayPhosphorylationlcsh:ScienceComputingMilieux_MISCELLANEOUSCancerGene EditingMultidisciplinaryQGene targetingGeneral Chemistry021001 nanoscience & nanotechnologyCell biologySettore BIO/18 - Genetica030104 developmental biologyChromosome segragationHela CellsPhosphorylationEpigeneticslcsh:QGeneric health relevance0210 nano-technologyFunction (biology)Centromere Protein AHumanHeLa CellsNature communications
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Cognate HLA absence in trans diminishes human NK cell education

2016

NK cells are innate lymphocytes with protective functions against viral infections and tumor formation. Human NK cells carry inhibitory killer cell Ig-like receptors (KIRs), which recognize distinct HLAs. NK cells with KIRs for self-HLA molecules acquire superior cytotoxicity against HLA– tumor cells during education for improved missing-self recognition. Here, we reconstituted mice with human hematopoietic cells from donors with homozygous KIR ligands or with a mix of hematopoietic cells from these homozygous donors, allowing assessment of the resulting KIR repertoire and NK cell education. We found that co-reconstitution with 2 KIR ligand–mismatched compartments did not alter the frequenc…

0301 basic medicine10028 Institute of Medical VirologyEpstein-Barr Virus InfectionsHerpesvirus 4 HumanCellchemical and pharmacologic phenomena610 Medicine & healthMice SCIDHuman leukocyte antigen2700 General MedicineAdaptive ImmunityBiology10263 Institute of Experimental Immunology03 medical and health sciencesMice Inbred NOD10049 Institute of Pathology and Molecular PathologymedicineAnimalsHumansCytotoxicityReceptorHistocompatibility Antigens Class IHEK 293 cellsGeneral MedicineAcquired immune systemKiller Cells NaturalHaematopoiesisHEK293 Cells030104 developmental biologymedicine.anatomical_structureNatural-Killer-Cells Cord Blood Transplantation Cytomegalovirus-Infection Class-I Inhibitory receptors Pediatric Patients TumorsImmunologyK562 CellsResearch ArticleK562 cells
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2,3-Dihydrobenzofuran privileged structures as new bioinspired lead compounds for the design of mPGES-1 inhibitors

2016

International audience; 2,3-Dihydrobenzofurans are proposed as privileged structures and used as chemical platform to design small compound libraries. By combining molecular docking calculations and experimental verification of biochemical interference, we selected some potential inhibitors of microsomal prostaglandin E2 synthase (mPGES)-1. Starting from low affinity natural product 1, by our combined approach we identified the compounds 19 and 20 with biological activity in the low micromolar range. Our data suggest that the 2,3-dihydrobenzofuran derivatives might be suitable bioinspired lead compounds for development of new generation mPGES-1 inhibitors with increased affinity.

0301 basic medicine300323-Dihydrobenzofuran privileged structure; Cancer; Inflammation; Molecular docking; mPGES-1 inhibitors; Biochemistry; Clinical Biochemistry; Molecular Biology; Molecular Medicine; Organic Chemistry; Drug Discovery3003 Pharmaceutical Science; 3003Amino Acid MotifsClinical BiochemistryGene ExpressionPharmaceutical Science01 natural sciencesClinical biochemistryBiochemistry[ CHIM ] Chemical SciencesProtein Structure Secondary[ SDV.CAN ] Life Sciences [q-bio]/Cancerchemistry.chemical_compoundLow affinityDrug DiscoveryEnzyme Inhibitors23-Dihydrobenzofuran privileged structure; Molecular docking; mPGES-1 inhibitors; Cancer; InflammationProstaglandin-E SynthasesCancerAnti-Inflammatory Agents Non-SteroidalBiological activityProto-Oncogene Proteins c-metIntramolecular OxidoreductasesMolecular Docking SimulationMolecular dockingMolecular Medicinelipids (amino acids peptides and proteins)Cell SurvivalStereochemistryMolecular Sequence Data2Antineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/Cancer3-Dihydrobenzofuran privileged structureInhibitory Concentration 50Structure-Activity Relationship03 medical and health sciencesCell Line TumorMicrosomesHumans[CHIM]Chemical SciencesMolecular BiologyBenzofuransInflammationNatural product010405 organic chemistryDrug Discovery3003 Pharmaceutical ScienceOrganic ChemistryEpithelial CellsmPGES-1 inhibitorsCombinatorial chemistryCombined approach0104 chemical sciences030104 developmental biologychemistryDrug DesignDrug Screening Assays Antitumor
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Functions of histone modifications and histone modifiers in Schwann cells.

2019

Schwann cells (SCs) are the main glial cells present in the peripheral nervous system (PNS). Their primary functions are to insulate peripheral axons to protect them from the environment and to enable fast conduction of electric signals along big caliber axons by enwrapping them in a thick myelin sheath rich in lipids. In addition, SCs have the peculiar ability to foster axonal regrowth after a lesion by demyelinating and converting into repair cells that secrete neurotrophic factors and guide axons back to their former target to finally remyelinate regenerated axons. The different steps of SC development and their role in the maintenance of PNS integrity and regeneration after lesion are c…

0301 basic medicine570 Life sciencesLesionHistones03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeurotrophic factorsPeripheral Nerve InjuriesmedicineAnimalsHumansSecretionTranscription factorMyelin SheathbiologyRegeneration (biology)AxonsCell biologyNerve Regeneration030104 developmental biologyHistonemedicine.anatomical_structurenervous systemNeurologyMyelin sheathPeripheral nervous systembiology.proteinSchwann Cellsmedicine.symptom030217 neurology & neurosurgery570 BiowissenschaftenGliaREFERENCES
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Melatonin Targets Metabolism in Head and Neck Cancer Cells by Regulating Mitochondrial Structure and Function.

2021

This study was funded by grants from the Ministerio de Economia, Industria y Competitividad y por el Fondo de Desarrollo Regional FEDER, Spain nº SAF2013-49019, SAF2017-85903-P, and from the Consejería de Innovación, Ciencia y Empresa, Junta de Andalucía (P07- CTS- 03135, P10- CTS- 5784, and CTS- 101), Spain. J.F. and L.M. have FPU fellowships from the Ministerio de Educación Cultura y Deporte, Spain. C.R.S. was a schorlarship holder from the Plan Propio de Investigación of the University of Granada.

0301 basic medicine:Phenomena and Processes::Chemical Phenomena::Biochemical Phenomena::Biochemical Processes::Phosphorylation::Oxidative Phosphorylation [Medical Subject Headings]PhysiologyClinical BiochemistrymelatoninMitochondrionBiochemistryMelatonina:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]0302 clinical medicine:Anatomy::Cells::Cells Cultured::Cell Line [Medical Subject Headings]head and neck cancer cells:Phenomena and Processes::Physiological Phenomena::Pharmacological Phenomena::Drug Resistance::Drug Resistance Neoplasm [Medical Subject Headings]MitophagyMitocondriasChemistryapoptosisglycolysisOXPHOSmitochondria030220 oncology & carcinogenesishormones hormone substitutes and hormone antagonistsmedicine.drug:Phenomena and Processes::Chemical Phenomena::Biochemical Phenomena::Biochemical Processes::Carbohydrate Metabolism::Glycolysis [Medical Subject Headings]Neoplasias de cabeza y cuello:Diseases::Neoplasms::Neoplasms by Site::Head and Neck Neoplasms [Medical Subject Headings]:Chemicals and Drugs::Inorganic Chemicals::Free Radicals::Reactive Oxygen Species [Medical Subject Headings]Mitofagiafree radicalsOxidative phosphorylationArticleMelatonin03 medical and health sciencesmedicine:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Growth Processes::Cell Proliferation [Medical Subject Headings]Molecular BiologyRadicales libresCell growth:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::DNA-Binding Proteins::Receptors Cytoplasmic and Nuclear::Receptors Melatonin [Medical Subject Headings]:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Therapeutic Uses::Antineoplastic Agents [Medical Subject Headings]lcsh:RM1-950:Anatomy::Cells::Cellular Structures::Subcellular Fractions::Mitochondria [Medical Subject Headings]Cell Biologymedicine.diseaseHead and neck squamous-cell carcinoma:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Death::Apoptosis [Medical Subject Headings]Glucólisis030104 developmental biologylcsh:Therapeutics. PharmacologymitophagyApoptosisCancer cellCancer research:Chemicals and Drugs::Hormones Hormone Substitutes and Hormone Antagonists::Hormones::Melatonin [Medical Subject Headings]
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Mechanics Insights of Alpha-Lipoic Acid against Cardiovascular Diseases during COVID-19 Infection

2021

Coronavirus disease 2019 (COVID-19) was first reported in Wuhan, China, in late December 2019. Since then, COVID-19 has spread rapidly worldwide and was declared a global pandemic on 20 March 2020. Cardiovascular complications are rapidly emerging as a major peril in COVID-19 in addition to respiratory disease. The mechanisms underlying the excessive effect of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection on patients with cardiovascular comorbidities remain only partly understood. SARS-CoV-2 infection is caused by binding of the viral surface spike (S) protein to the human angiotensin-converting enzyme 2 (ACE2), followed by the activation of the S protein by transme…

0301 basic medicineARDSEndotheliumQH301-705.5InflammationReviewmedicine.disease_causeCatalysisAntioxidantsProinflammatory cytokineInorganic Chemistry03 medical and health sciences0302 clinical medicinecardiovascular diseasemedicineoxidative stressAnimalsHumansPhysical and Theoretical ChemistryEndothelial dysfunctionBiology (General)Molecular BiologyQD1-999SpectroscopyThioctic Acidbusiness.industrySARS-CoV-2alpha-lipoic acidOrganic ChemistryRespiratory diseaseCOVID-19Endothelial CellsGeneral Medicinemedicine.diseaseComputer Science ApplicationsCOVID-19 Drug TreatmentChemistry030104 developmental biologymedicine.anatomical_structureinflammationCardiovascular Diseases030220 oncology & carcinogenesisImmunologyAngiotensin-Converting Enzyme 2medicine.symptombusinessCytokine stormCytokine Release SyndromeOxidative stressInternational Journal of Molecular Sciences
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Polyphosphate as a donor of high-energy phosphate for the synthesis of ADP and ATP.

2017

Here, we studied the potential role of inorganic polyphosphate (polyP) as an energy source for ADP and ATP formation in the extracellular space. In SaOS-2 cells, we show that matrix vesicles are released into the extracellular space after incubation with polyP. These vesicles contain both alkaline phosphatase (ALP) and adenylate kinase (AK) activities (mediated by ALPL and AK1 enzymes). Both enzymes translocate to the cell membrane in response to polyP. To distinguish the process(es) of AMP and ADP formation during ALP hydrolysis from the ATP generated via the AK reaction, inhibition studies with the AK inhibitor A(5')P5(5')A were performed. We found that ADP formation in the extracellular …

0301 basic medicineAdenylate kinaseBiologydigestive systemExocytosisCatalysisCell membrane03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAdenosine TriphosphatePolyphosphatesExtracellularmedicineTumor Cells CulturedHumansPhosphorylationchemistry.chemical_classificationATP synthasePolyphosphateAdenylate KinaseCell BiologyAlkaline PhosphataseAdenosine DiphosphateKinetics030104 developmental biologyEnzymemedicine.anatomical_structurechemistryBiochemistry030220 oncology & carcinogenesisbiology.proteinEnergy sourceEnergy MetabolismExtracellular SpaceJournal of cell science
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Spheroids from adipose-derived stem cells exhibit an miRNA profile of highly undifferentiated cells

2017

Two-dimensional (2D) cell cultures have been extensively used to investigate stem cell biology, but new insights show that the 2D model may not properly represent the potential of the tissue of origin. Conversely, three-dimensional cultures exhibit protein expression patterns and intercellular junctions that are more representative of their in vivo condition. Multiclonal cells that grow in suspension are defined as "spheroids," and we have previously demonstrated that spheroids from adipose-derived stem cells (S-ASCs) displayed enhanced regenerative capability. With the current study, we further characterized S-ASCs to further understand the molecular mechanisms underlying their stemness pr…

0301 basic medicineAdipose stem cellPhysiologyCellular differentiationClinical BiochemistryCell Culture TechniquesAdipose tissueBiology03 medical and health sciences0302 clinical medicineOsteogenesisSpheroids CellularLong-term cultureMiR-142-3pmicroRNAAdipocytesHumansInduced pluripotent stem cellCell ProliferationAdipogenesisStem CellsGene Expression Regulation DevelopmentalCell DifferentiationCell BiologyIn vitroCell biologyMicroRNAs030104 developmental biologyMesenchymal differentiationCell cultureAdipogenesis030220 oncology & carcinogenesisStem cellMiRNA
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Tumor-priming converts NK cells to memory-like NK cells

2017

Fascinating earlier evidence suggests an intrinsic capacity of human natural killer (NK) cells to acquire adaptive immune features in the context of cytomegalovirus (CMV) infection or pro-inflammatory cytokine stimulation. Since the role of memory NK cells in cancer has so far remained elusive and adoptive NK cell transfer in relapsing pediatric acute B cell precursor leukemia (BCP-ALL) patients awaits improvement, we asked the question whether tumor-priming could promote the generation of memory NK cells with enhanced graft-vs.-leukemia (GvL) reactivity. Here, we provide substantial evidence that priming of naive human NK cells with pediatric acute B cell leukemia or acute myeloid leukemia…

0301 basic medicineAdoptive cell transferImmunology03 medical and health sciencesInterleukin 21NK-92childrenmedicineImmunology and Allergyddc:610B celladoptive cell transferRC254-282Original ResearchLymphokine-activated killer cellnatural killer cellsbiologyJanus kinase 3Neoplasms. Tumors. Oncology. Including cancer and carcinogensRC581-607030104 developmental biologymedicine.anatomical_structureacute b cell precursor leukemiaOncologyPerforinImmunologyInterleukin 12biology.proteinImmunologic diseases. AllergyOncoImmunology
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