Search results for " glutamate"

showing 10 items of 97 documents

Nrg1 haploinsufficiency alters inhibitory cortical circuits

2021

Neuregulin 1 (NRG1) and its receptor ERBB4 are schizophrenia (SZ) risk genes that control the development of both excitatory and inhibitory cortical circuits. Most studies focused on the characterization ErbB4 deficient mice. However, ErbB4 deletion concurrently perturbs the signaling of Nrg1 and Neuregulin 3 (Nrg3), another ligand expressed in the cortex. In addition, NRG1 polymorphisms linked to SZ locate mainly in non-coding regions and they may partially reduce Nrg1 expression. Here, to study the relevance of Nrg1 partial loss-of-function in cortical circuits we characterized a recently developed haploinsufficient mouse model of Nrg1 (Nrg1tm1Lex). These mice display SZ-like behavioral d…

Cortical neuronsReceptor ErbB-4Neuregulin-1Gene ExpressionneuronsNeurosciences. Biological psychiatry. NeuropsychiatryHaploinsufficiencyBiologyInhibitory postsynaptic potentialHippocampusMagnetic&nbspMiceInterneuronsNeuregulin 3mental disordersMagnetic resonance spectroscopyAnimalsRNA MessengerneurotransmissionNeuregulin 1GABAergic Neuronsgamma-Aminobutyric AcidInhibitory&nbspCerebral CortexNrg1resonance spectroscopyNeural InhibitionMagnetic Resonance ImagingCortex (botany)Inhibitory neurotransmissionParvalbuminsNeurologyInhibitory Postsynaptic PotentialsCalbindin 2Vesicular Glutamate Transport Protein 1biology.proteinExcitatory postsynaptic potentialSchizophreniaCalretininHaploinsufficiencyCortical&nbspNeuroscienceParvalbuminRC321-571Neurobiology of Disease
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Nitric oxide and brain hyperexcitability.

2004

Nitric oxide (NO) is a gaseous messenger involved in atypical forms of intercellular communications, able to exert a strong functional modulation of several neurotransmitter systems. In particular, NO heavily influences the excitatory neurotransmitter glutamate, mainly through NMDA receptors, and the inhibitory neurotransmitter GABA, mainly through GABA A receptors. Due to the involvement of glutamate and GABA in a delicate balance conditioning the functional status of the neural cells, this interaction suggests a role for NO in regulating neuronal excitability and its transition towards hyperexcitability phenomena. This article reviews the main knowledge about the relationships existing be…

Disease Models AnimalEpilepsyNG-Nitroarginine Methyl EsterAnimalsBrainGlutamic AcidHumansNitric oxide glutamate GABA epilepsy reviewNervous System DiseasesNitric OxideSettore BIO/09 - Fisiologiagamma-Aminobutyric AcidIn vivo (Athens, Greece)
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Chronic fluoxetine treatment in middle-aged rats induces changes in the expression of plasticity-related molecules and in neurogenesis

2012

Abstract Background Antidepressants promote neuronal structural plasticity in young-adult rodents, but little is known of their effects on older animals. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) may mediate these structural changes through its anti-adhesive properties. PSA-NCAM is expressed in immature neurons and in a subpopulation of mature interneurons and its expression is modulated by antidepressants in the telencephalon of young-adult rodents. Results We have analyzed the effects of 14 days of fluoxetine treatment on the density of puncta expressing PSA-NCAM and different presynaptic markers in the medial prefrontal cortex, hippocampus and amygdala of mi…

Doublecortin Domain ProteinsMaleTelencephalonmedicine.medical_specialtyDoublecortin ProteinVesicular glutamate transporter 1NeurogenesisGlutamate decarboxylaseSynaptophysinHippocampusSubventricular zoneCell CountNeural Cell Adhesion Molecule L1Hippocampal formationSubgranular zonelcsh:RC321-571Cellular and Molecular NeuroscienceInternal medicineFluoxetineLateral VentriclesmedicineAnimalsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCell ProliferationbiologyGlutamate DecarboxylaseGeneral NeuroscienceNeurogenesisBody WeightNeuropeptideslcsh:QP351-495DoublecortinRatsEndocrinologymedicine.anatomical_structureKi-67 Antigenlcsh:Neurophysiology and neuropsychologyGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1biology.proteinSialic AcidsAntidepressive Agents Second-GenerationNeuroscienceMicrotubule-Associated ProteinsResearch ArticleBMC Neuroscience
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Separation of presynaptic Cav2 and Cav1 channel function in synaptic vesicle exo- and endocytosis by the membrane anchored Ca2+ pump PMCA

2021

Significance Synaptic vesicle (SV) release from presynaptic terminals requires nanometer precise control of action potential (AP)–triggered calcium influx through voltage-gated calcium channels (VGCCs). SV recycling also depends on calcium signals, though in different spatiotemporal domains. Mechanisms for separate control of SV release and recycling by AP-triggered calcium influx remain elusive. Here, we demonstrate largely independent regulation of release and recycling by two different populations of VGCCs (Cav2, Cav1), identify Cav1 as one of potentially multiple calcium entry routes for endocytosis regulation, and show functional separation of simultaneous calcium signals in the nanome…

Drosophila ; Dmca1D ; cacophony ; PMCA ; synapse0301 basic medicine570ATPasecacophonyPresynaptic TerminalsAction PotentialsEndocytosisDmca1DSynaptic vesicleExocytosis03 medical and health scienceschemistry.chemical_compoundGlutamatergicPlasma Membrane Calcium-Transporting ATPases0302 clinical medicinePMCAsynapsemedicineAnimalsDrosophila ProteinsAxonNeurotransmitterProbabilityMotor NeuronsMultidisciplinaryVoltage-dependent calcium channelbiologyCell Membrane424500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; BiologieBiological SciencesEndocytosisCell biologyElectrophysiology030104 developmental biologymedicine.anatomical_structureDrosophila melanogasterchemistryReceptors Glutamatebiology.proteinDrosophilaCalciumCalcium ChannelsSynaptic Vesicles030217 neurology & neurosurgeryNeuroscienceProceedings of the National Academy of Sciences of the United States of America
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Modulation of Neurological Deficits and Expression of Glutamate Receptors during Experimental Autoimmune Encephalomyelitis after Treatment with Selec…

2013

The aim of our investigation was to characterize the role of group I mGluRs and NMDA receptors in pathomechanisms of experimental autoimmune encephalomyelitis (EAE), the rodent model of MS. We tested the effects of LY 367385 (S-2-methyl-4-carboxyphenylglycine, a competitive antagonist of mGluR1), MPEP (2-methyl-6-(phenylethynyl)-pyridine, an antagonist of mGluR5), and the uncompetitive NMDA receptor antagonists amantadine and memantine on modulation of neurological deficits observed in rats with EAE. The neurological symptoms of EAE started at 10-11 days post-injection (d.p.i.) and peaked after 12-13 d.p.i. The protein levels of mGluRs and NMDA did not increase in early phases of EAE (4 d.p…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisArticle SubjectHydrolasesEncephalomyelitislcsh:MedicineBiologyPharmacologyReceptors N-Methyl-D-AspartateGeneral Biochemistry Genetics and Molecular Biologymental disordersmedicineAmantadineAnimalsHumansRNA MessengerGeneral Immunology and MicrobiologyMetabotropic glutamate receptor 5Experimental autoimmune encephalomyelitislcsh:RGlutamate receptorMemantineGeneral Medicinemedicine.diseaseRatsDisease Models AnimalGene Expression RegulationReceptors Glutamatenervous systemCompetitive antagonistImmunologyNMDA receptorMetabotropic glutamate receptor 1FemaleExcitatory Amino Acid Antagonistsmedicine.drugResearch ArticleBioMed Research International
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Origin of neuronal-like receptors in Metazoa: cloning of a metabotropic glutamate/GABA-like receptor from the marine sponge Geodia cydonium.

1999

To date, no conclusive evidence has been presented for the existence of neuronal-like elements in Porifera (sponges). In the present study, isolated cells from the marine sponge Geodia cydonium are shown to react to the excitatory amino acid glutamate with an increase in the concentration of intracellular calcium [Ca2+]i. This effect can also be observed when the compounds L-quisqualic acid (L-QA) or L-(+)-2-amino-4-phosphonobutyric acid (L-AP-4) are used. The effect of L-QA and L-AP-4, both agonists for metabotropic glutamate receptors (mGluRs), can be abolished by the antagonist of group I mGluRs, (RS)-alpha-methyl-4-carboxyphenylglycine. These data suggest that sponge cells contain an mG…

HistologyMolecular Sequence DataGlutamic AcidClass C GPCRBiologyReceptors Metabotropic GlutamatePathology and Forensic MedicineMiceReceptors GABAAnimalsAmino Acid SequenceCloning MolecularSequence Homology Amino AcidMetabotropic glutamate receptor 4Metabotropic glutamate receptor 7Metabotropic glutamate receptor 6Cell BiologyRecombinant ProteinsPoriferaRatsKineticsDrosophila melanogasternervous systemBiochemistryMetabotropic glutamate receptorMetabotropic glutamate receptor 1CalciumMetabotropic glutamate receptor 3Metabotropic glutamate receptor 2Excitatory Amino Acid AntagonistsSequence AlignmentCell and tissue research
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Neurochemical correlates of brain atrophy in fibromyalgia syndrome: a magnetic resonance spectroscopy and cortical thickness study

2020

(1) Background: Recently, a series of clinical neuroimaging studies on fibromyalgia (FM) have shown a reduction in cortical volume and abnormally high glutamate (Glu) and glutamate + glutamine (Glx) levels in regions associated with pain modulation. However, it remains unclear whether the volumetric decreases and increased Glu levels in FM are related each other. We hypothesized that higher Glu levels are related to decreases in cortical thickness (CT) and volume in FM patients. (2) Methods: Twelve females with FM and 12 matched healthy controls participated in a session of combined 3.0 Tesla structural magnetic resonance imaging (MRI) and single-voxel MR spectroscopy focused on the thalami…

In vivo magnetic resonance spectroscopymedicine.medical_specialtybrain MRIArticlelcsh:RC321-57103 medical and health sciences0302 clinical medicineNeurochemicalAtrophyNeuroimagingGyrusFibromyalgiaInternal medicinemedicinecortical thicknelcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biology0303 health sciencesbusiness.industryGeneral NeuroscienceMR spectroscopyGlutamate receptorSettore MED/37 - Neuroradiologiacortical thicknessmedicine.diseaseSubcortical gray matterEndocrinologymedicine.anatomical_structurefibromyalgia; glutamate excitotoxicity; cortical thickness; brain MRI; chronic pain; MR spectroscopyMR spectroscopy; brain MRI; chronic pain; cortical thickness; fibromyalgia; glutamate excitotoxicity.fibromyalgiaSettore MED/36 - Diagnostica Per Immagini E Radioterapiabusinesschronic pain030217 neurology & neurosurgeryglutamate excitotoxicity
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mGluR control of interneuron output regulates feedforward tonic GABAA inhibition in the visual thalamus

2011

Metabotropic glutamate receptors (mGluRs) play a crucial role in regulation of phasic inhibition within the visual thalamus. Here we demonstrate that mGluR-dependent modulation of interneuron GABA release results in dynamic changes in extrasynaptic GABAA receptor (eGABAAR)-dependent tonic inhibition in thalamocortical (TC) neurons of the rat dorsal lateral geniculate nucleus (dLGN). Application of the group I selective mGluR agonist dihydroxyphenylglycine produces a concentration-dependent enhancement of both IPSC frequency and tonic GABAA current (IGABAtonic) that is due to activation of both mGluR1a and mGluR5 subtypes. In contrast, group II/III mGluR activation decreases both IPSC freque…

InterneuronReceptors metabotropic glutamateAction PotentialsMetabotropic glutamate receptors GABA dorsal geniculate neurons ratNeurotransmissionReceptors Metabotropic GlutamateQ1DihydroxyphenylglycineSynaptic TransmissionSettore BIO/09 - Fisiologiagamma-Aminobutyric acidArticlechemistry.chemical_compoundThalamusKidney glomerulusInterneuronsmedicineAnimalsVisual Pathwaysgamma-Aminobutyric AcidChemistryGABAA receptorGeneral Neurosciencemusculoskeletal neural and ocular physiologyNeural InhibitionReceptors GABA-ARatsElectrophysiologymedicine.anatomical_structureInhibitory Postsynaptic Potentialsnervous systemMetabotropic glutamate receptorRC0321GABAergicNeuronNeurosciencemedicine.drug
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mGluR2/3 agonist LY379268, by enhancing the production of GDNF, induces a time-related phosphorylation of RET receptor and intracellular signaling Er…

2011

In the present study we aimed to verify if the enhancement of glial cell line-derived neurotrophic factor (GDNF) production in mouse striatum following treatment with LY379268 may also induce in the nigrostriatal system a time-related activation of RET receptor and its specific intracellular signaling. For this purpose, we have investigated the effects of LY379268 treatment on RET phosphorylation at the Tyr1062 and on downstream signaling Erk1/2, Akt and PLCγ1 pathway activation. The results showed that treatment with LY379268 (3 mg/kg) induces a significant increase of GDNF levels and time-related RET and Erk1/2 phosphorylation in the striatum. These increases were detected at 24 h and 48 …

Intracellular FluidMalemedicine.medical_specialtyTime FactorsMAP Kinase Signaling SystemSubstantia nigraStriatumReceptors Metabotropic GlutamateSettore BIO/09 - FisiologiaCellular and Molecular NeuroscienceMiceErk1/2Neurotrophic factorsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsGlial Cell Line-Derived Neurotrophic FactorAmino AcidsPhosphorylationReceptormGluR2/3Protein kinase BPharmacologyMitogen-Activated Protein Kinase 3biologyChemistryProto-Oncogene Proteins c-retLY379268Bridged Bicyclo Compounds HeterocyclicGDNFCorpus StriatumUp-RegulationMice Inbred C57BLEndocrinologynervous systembiology.proteinPhosphorylationTrK phosphorylationRETGDNF family of ligandsNeuropharmacology
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Activation of metabotropic glutamate receptors induces propagating network oscillations in the intact cerebral cortex of the newborn mouse.

2006

Activation of metabotropic glutamate receptors (mGluRs) with (1S,3R)-1-aminocyclopentane-1,3-dicarboxylic acid (ACPD) elicited in the frontal or occipital pole of the intact cerebral cortex preparation of the newborn mouse (P0-P3) a transient oscillatory field potential activity in the frequency range of 11-14Hz. These oscillations propagated over the whole cortical hemisphere and were blocked by tetrodotoxin, indicating that action potentials are required for the generation of this activity. Blockade of GABA-A receptors with gabazine did not influence the ACPD-induced network activity, but the glycine antagonist strychnine caused a significant decrease in the frequency, amplitude and durat…

Kainate receptorCholinergic AgonistsReceptors Metabotropic GlutamateCellular and Molecular Neurosciencechemistry.chemical_compoundMiceKynurenic acidmedicineAnimalsLong-term depressionPharmacologyCerebral CortexDose-Response Relationship DrugDioxolanesEnzyme ActivationMice Inbred C57BLchemistryAnimals NewbornMetabotropic glutamate receptorPurinesCNQXGabazineACPDNMDA receptorCarbacholNerve NetNeuroscienceExcitatory Amino Acid Antagonistsmedicine.drugNeuropharmacology
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