Search results for " inflammation."

showing 10 items of 623 documents

Influence of HMG-CoA reductase inhibitors on markers of coagulation, systemic inflammation and soluble cell adhesion.

2002

Abstract Background: Beneath its lipid-lowering properties additional non-lipid effects of statin therapy are discussed. We therefore examined the impact of statins on laboratory markers of coagulation, inflammation and soluble cell adhesion to further explore these effects in 950 hospitalised patients with angiographically proven CAD. Methods and results: Although no significant differences were found in total cholesterol, LDL and HDL and triglyceride levels a statistically lower value in 277 statin-treated patients was found for von Willebrand factor [162(130/224) vs. 208(154/283)%, P =0.0001], leukocyte count [6.9(5.8/8.4) vs. 7.3(6.1/9.4)/nl, P =0.0005], high sensitive CRP [4.3(1.8/10.8…

Malemedicine.medical_specialtyStatinmedicine.drug_classInflammationCoronary Artery DiseaseSystemic inflammationchemistry.chemical_compoundVon Willebrand factorInternal medicinevon Willebrand FactormedicineCell AdhesionHumansAgedbiologyTriglycerideCholesterolbusiness.industryAnticholesteremic AgentsCholesterol LDLMiddle AgedEndocrinologyCoagulationchemistryHMG-CoA reductasebiology.proteinFemalemedicine.symptomHydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessBiomarkersInternational journal of cardiology
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Insulin resistance as common molecular denominator linking obesity to Alzheimer’s disease

2015

Alzheimer’s disease (AD) is an aging-related multi-factorial disorder to which metabolic factors contribute at what has canonically been considered a centrally mediated process. Although the exact underlying mechanisms are still unknown, obesity is recognized as a risk factor for AD and the condition of insulin resistance seems to be the link between the two pathologies. Using mice with high fat diet (HFD) obesity we dissected the molecular mechanisms shared by the two disorders. Brains of HFD fed mice showed elevated levels of APP and Aβ 40 /Aβ 42 together with BACE, GSK3β and Tau proteins involved in APP processing and Aβ accumulation. Immunofluorescence, Thioflavin T staining experiments…

Malemedicine.medical_specialtyTime FactorsAdipokineAmyloidogenic ProteinsInflammationBiologyDiet High-Fatmedicine.disease_causeAdipokines Alzheimer’s disease gene expression inflammation insulin resistance mitochondrial dysfunction obesity.Settore BIO/09 - FisiologiaGlycogen Synthase Kinase 3MiceInsulin resistanceAlzheimer DiseaseInternal medicinemedicineAnimalsInsulinObesityReceptorGSK3BGlycogen Synthase Kinase 3 betaSettore BIO/16 - Anatomia UmanaNeurodegenerationBrainmedicine.diseaseReceptor InsulinMice Inbred C57BLDisease Models AnimalOxidative StressInsulin receptorEndocrinologyGene Expression RegulationNeurologyCase-Control Studiesbiology.proteinCytokinesNeurology (clinical)Amyloid Precursor Protein SecretasesInsulin Resistancemedicine.symptomOxidative stressSignal Transduction
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Prognostic Value of White Blood Cell Count in Acute Myocardial Infarction: Long-Term Mortality

2005

Although traditionally an elevated white blood cell count (WBC), an indicator of systemic inflammation, has been accepted as part of the healing response following acute myocardial infarction (AMI), it has frequently been shown to be a predictor of adverse cardiovascular events. The present study was designed to assess the association between WBC and long-term mortality in AMI patients either with ST-segment elevation (STEMI) or without ST-segment elevation (non-STEMI). Patients and method. The study included 1118 consecutive patients who were admitted with the diagnosis of AMI: 569 non-STEMI and 549 STEMI. The WBC was measured in the 24 hours following admission. Patients were divided into…

Malemedicine.medical_specialtyTime FactorsMyocardial InfarctionSystemic inflammationElectrocardiographyLeukocyte CountRisk FactorsInternal medicineWhite blood cellMyocardial RevascularizationmedicineHumansHospital Mortalitycardiovascular diseasesMyocardial infarctionAngioplasty Balloon CoronarySurvival analysisAgedProportional Hazards Modelsmedicine.diagnostic_testbusiness.industryProportional hazards modelHazard ratioGeneral MedicinePrognosismedicine.diseaseSurvival AnalysisSurgerysurgical procedures operativemedicine.anatomical_structureCardiologyRegression AnalysisFemaleLong term mortalitymedicine.symptombusinessElectrocardiographyFollow-Up StudiesRevista Española de Cardiología (English Edition)
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Ocular ductions after rectus muscle recession and resection in thyroid eye disease.

2019

Background: Recession and resection of rectus muscles for correction of strabismus in Thyroid Eye Disease (TED) is relatively unpopular as it is assumed to enhance the restriction of ocular ductions. Therefore, the purpose of this study was to compare ductions of recession only and recess/resect procedures. Methods: We retrospectively reviewed the charts of 119 patients who underwent strabismus surgery for TED from 1991 to 2015, of which 102 were included in the present study. Forty-six interventions were performed on horizontal, 56 on vertical rectus muscles; comprising 41 recess/resect surgeries for horizontal deviations, and 7 in patients with vertical strabismus. Ocular ductions and ali…

Malemedicine.medical_specialtygenetic structuresEye MovementsEye diseaseeducationOphthalmologic Surgical ProceduresResection03 medical and health sciences0302 clinical medicinemedicineHumansIn patient030212 general & internal medicineStrabismusRetrospective Studiesbusiness.industryThyroidRectus muscleMiddle Agedmusculoskeletal systemmedicine.diseaseeye diseasesSurgeryGraves OphthalmopathyStrabismusOphthalmologymedicine.anatomical_structureTreatment OutcomeOculomotor Muscles030221 ophthalmology & optometryFemalesense organsPostoperative inflammationbusinessStrabismus surgeryStrabismus
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A Multiplex Panel of Plasma Markers of Immunity and Inflammation in Classical Kaposi Sarcoma

2014

Kaposi sarcoma (KS) risk is affected by perturbed immunity. Herein, we compared plasma from 15 human immunodeficiency virus (HIV)–negative classic KS cases to plasma from 29 matched controls, using a multiplex panel of immunity markers. Of 70 markers, CXCL10 (IP-10), sIL-1RII, sIL-2RA, and CCL3 (MIP-1A) were strongly and significantly associated with KS, after adjustment for age and smoking status. These and previous observations are consistent with a tumor-promoting role for these cytokines, particularly CXCL10, but the small sample size and case-control design preclude firm conclusions on KS risk or pathogenesis. Larger, well-designed prospective studies are needed to better assess the as…

Malemedicine.medical_treatmentInflammationBiologySettore MED/42 - Igiene Generale E ApplicataPathogenesisKaposi SarcomaPlasma Markers of InflammationMajor Articles and Brief ReportsImmunitymedicineHumansImmunology and AllergyProspective cohort studySarcoma KaposiInflammationCase-control studyvirus diseasesPlasma Markers of Immunitymedicine.diseaseSquamous intraepithelial lesionInfectious DiseasesCytokineItalyCase-Control StudiesImmunologySarcomamedicine.symptomBiomarkers
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Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice

2011

Polymorphisms in the transcription factor Stat4 gene have been implicated as risk factors for systemic lupus erythematosus. Although some polymorphisms have a strong association with autoantibodies and nephritis, their impact on pathophysiology is still unknown. To explore this further we used signal transducers and activators of transcription 4 (Stat4) knockout MRL/MpJ-Fas(lpr)/Fas(lpr) (MRL-Fas(lpr)) mice and found that they did not differ in survival or renal function from Stat4-intact MRL-Fas(lpr) mice. Circulating interleukin (IL)-18 levels, however, were elevated in Stat4-deficient compared to Stat4-intact mice, suggesting that this interleukin might contribute to the progression of l…

Malemusculoskeletal diseasesMice Inbred MRL lprchronic inflammationLupus nephritisKidneyInterleukin-23ArticleProinflammatory cytokineOligodeoxyribonucleotides AntisenseGene Knockout TechniquesInterferon-gammaMiceimmune system diseasesmedicineAnimalsskin and connective tissue diseasesSTAT4DNA PrimersAutoimmune diseaseMice Knockoutlupus nephritisMice Inbred BALB CBase Sequencebusiness.industryGene Transfer TechniquesInterleukin-18InterleukinGlomerulonephritishemic and immune systemsSTAT4 Transcription Factormedicine.diseaseInterleukin-12chronic glomerulonephritisNephrologyImmunologyInterleukin 18FemalebusinessNephritisKidney International
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Cardiac Injury in COVID-19–Echoing Prognostication∗

2020

Background Myocardial injury is frequent among patients hospitalized with coronavirus disease-2019 (COVID-19) and is associated with a poor prognosis. However, the mechanisms of myocardial injury remain unclear and prior studies have not reported cardiovascular imaging data. Objectives This study sought to characterize the echocardiographic abnormalities associated with myocardial injury and their prognostic impact in patients with COVID-19. Methods We conducted an international, multicenter cohort study including 7 hospitals in New York City and Milan of hospitalized patients with laboratory-confirmed COVID-19 who had undergone transthoracic echocardiographic (TTE) and electrocardiographic…

Malemyocaardial injuryCoronary AngiographyElectrocardiographyPandemicVentricular Dysfunctionechocardiographymyocardial injuryOriginal Investigationechocardiography; inflammation; myocaardial injury; SARS-CoV-2; troponin; COVID-19COVID-19 coronavirus disease-2019biologyViral EpidemiologytroponinHeartMiddle AgedRV right ventriclemyocardial infarctionItalyTTE transthoracic echocardiographyFemaleCardiology and Cardiovascular MedicineCoronavirus InfectionsEditorial Comment2019-20 coronavirus outbreakCoronavirus disease 2019 (COVID-19)Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)Pneumonia ViralAKI acute kidney injuryECG electrocardiographyBetacoronavirusmedicineHumansPandemicsARDS acute respiratory distress syndromeIQR interquartile rangeAgedRetrospective Studiesbusiness.industrySARS-CoV-2MyocardiumCOVID-19medicine.diseasebiology.organism_classificationVirologyCOVID-19 Drug TreatmentCI confidence intervalOR odds ratioPneumoniaLV left ventricleinflammationACS acute coronary syndromeNew York CitybusinessCoronavirus InfectionsBetacoronavirusBiomarkersJournal of the American College of Cardiology
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Monocytes from patients with rheumatoid arthritis and type 2 diabetes mellitus display an increased production of interleukin (IL)-1β via the nucleot…

2015

Summary A better understanding about the mechanisms involved in the pathogenesis of type 2 diabetes mellitus (T2D) showed that inflammatory cytokines such as tumour necrosis factor (TNF) and interleukin (IL)-1β play a pivotal role, mirroring data largely reported in rheumatoid arthritis (RA). IL-1β is produced mainly by monocytes (MO), and hyperglycaemia may be able to modulate, in the cytoplasm of these cells, the assembly of a nucleotide-binding domain and leucine-rich repeat containing family pyrin (NLRP3)-inflammosome, a cytosolic multi-protein platform where the inactive pro-IL-1β is cleaved into active form, via caspase-1 activity. In this paper, we evaluated the production of IL-1 β …

Maletype 2 diabetes mellituInflammasomesMessengerIL-1β; NLRP3-inflammasome; rheumatoid arthritis; type 2 diabetes mellitus; Adult; Arthritis Rheumatoid; Carrier Proteins; Caspase 1; Cells Cultured; Diabetes Mellitus Type 2; Enzyme Activation; Female; Glucose; Humans; Hyperglycemia; Inflammasomes; Inflammation; Interleukin-1beta; Leukocytes Mononuclear; Male; Middle Aged; RNA Messenger; Tumor Necrosis Factor-alphaInterleukin-1betaArthritisPyrin domainInflammasomeArthritis RheumatoidRheumatoidImmunology and AllergyCells CulturedCulturedCaspase 1InterleukinDiabetes MellituMiddle AgedIL-1βTumor necrosis factor alphaNLRP3-inflammasomeFemalemedicine.symptomType 2ArthritiHumanAdultmedicine.medical_specialtyMononuclearImmunologyCaspase 1InflammationProinflammatory cytokineInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineHumansRNA MessengerInflammationbusiness.industryTumor Necrosis Factor-alphaType 2 Diabetes MellitusOriginal Articlesrheumatoid arthritiLeukocytemedicine.diseaseEnzyme ActivationEndocrinologyGlucoseDiabetes Mellitus Type 2HyperglycemiaImmunologyLeukocytes MononuclearRNACellbusinessCarrier ProteinsCarrier Protein
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Functional response of novel bioprotective poloxamer-structured vesicles on inflamed skin

2017

[EN] Resveratrol and gallic acid, a lipophilic and a hydrophilic phenol, were co-loaded in innovative, biocompatible nanovesicles conceived for ensuring the protection of the skin from oxidative-and inflammatory-related affections. The basic vesicles, liposomes and glycerosomes, were produced by a simple, one-step method involving the dispersion of phospholipid and phenols in water or water/glycerol blend, respectively. Liposomes and glycerosomes were modified by the addition of poloxamer, a stabilizer and viscosity enhancer, thus obtaining viscous or semisolid dispersions of structured vesicles. The vesicles were spherical, unilamellar and small in size (similar to 70 nm in diameter). The …

Materials scienceCell SurvivalSwineSkin AbsorptionBiomedical EngineeringPhospholipidPharmaceutical ScienceMedicine (miscellaneous)Bioengineering02 engineering and technologyPoloxamerResveratrol010402 general chemistry01 natural sciencesCell Linechemistry.chemical_compoundMiceIn vivoGallic AcidStilbenesGlycerolAnimalsEdemaGeneral Materials SciencePhenolsSkinLiposomePhenolVesicleAnti-Inflammatory Agents Non-SteroidalSkin inflammationPoloxamerFibroblasts021001 nanoscience & nanotechnology0104 chemical sciencesOxidative StresschemistryBiochemistryResveratrolLiposomesPhospholipid vesicleBiophysicsMolecular MedicineFemale0210 nano-technology
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Influence of heme oxygenase 1 modulation on the progression of murine collagen-induced arthritis.

2005

Contains fulltext : 48023.pdf (Publisher’s version ) (Closed access) OBJECTIVE: Heme oxygenase 1 (HO-1) can be induced by inflammatory mediators as an adaptive response. The objective of the present study was to determine the consequences of HO-1 modulation in the murine collagen-induced arthritis (CIA) model. METHODS: DBA/1J mice were treated with an inhibitor of HO-1, tin protoporphyrin IX (SnPP), or with an inducer of HO-1, cobalt protoporphyrin IX (CoPP), from day 22 to day 29 after CIA induction. The clinical evolution of disease was monitored visually. At the end of the experiment, joints were examined for histopathologic changes. Cytokine levels in paws were measured by enzyme-linked…

Metalloporphyrinsmedicine.medical_treatmentImmunologyArthritisProtoporphyrinsInflammationPharmacologyAuto-immunity transplantation and immunotherapy [N4i 4]MiceRheumatologyFibrosismedicinePerception and Action [DCN 1]Immunology and AllergyAnimalsPharmacology (medical)Enzyme InhibitorsChronic inflammation and autoimmunity [UMCN 4.2]biologybusiness.industryMembrane Proteinsmedicine.diseaseCOPPArthritis ExperimentalHeme oxygenaseEnzyme ActivationPathogenesis and modulation of inflammation [N4i 1]Disease Models AnimalCytokineCyclooxygenase 2Mice Inbred DBAProstaglandin-Endoperoxide SynthasesImmunologyChronic DiseaseHeme Oxygenase (Decyclizing)biology.proteinDisease ProgressionTumor necrosis factor alphaJointsCyclooxygenasemedicine.symptombusinessInfection and autoimmunity [NCMLS 1]Heme Oxygenase-1
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