Search results for " jun"

showing 10 items of 611 documents

Effect of verapamil and diltiazem on isolated gastro-oesophageal sphincter of the rat

1985

Abstract The effect of verapamil and diltiazem on the contraction induced by agonists on the rat lower oesophageal sphincter in-vitro has been studied. Both calcium entry blockers inhibited the contractile response to acetylcholine, carbachol and KCl. The potency of the inhibitory action was diltiazem > verapamil. The results give substance to the use of calcium entry blockers in the treatment of oesophageal spasm.

Malemedicine.medical_specialtyContraction (grammar)CarbacholPharmaceutical ScienceIn Vitro TechniquesInhibitory postsynaptic potentialPotassium ChlorideDiltiazemInternal medicineAnimalsPotencyMedicineDiltiazemPharmacologybusiness.industryMuscle SmoothBenzazepinesRatsEndocrinologyVerapamilcardiovascular systemVerapamilCarbacholFemaleEsophagogastric Junctionmedicine.symptombusinessAcetylcholineMuscle ContractionMuscle contractionmedicine.drugJournal of Pharmacy and Pharmacology
researchProduct

First-line nivolumab plus chemotherapy versus chemotherapy alone for advanced gastric, gastro-oesophageal junction, and oesophageal adenocarcinoma (C…

2021

First-line chemotherapy for advanced or metastatic human epidermal growth factor receptor 2 (HER2)-negative gastric or gastro-oesophageal junction adenocarcinoma has a median overall survival (OS) of less than 1 year. We aimed to evaluate first-line programmed cell death (PD)-1 inhibitor-based therapies in gastric, gastro-oesophageal junction, and oesophageal adenocarcinoma. We report the first results for nivolumab plus chemotherapy versus chemotherapy alone.In this multicentre, randomised, open-label, phase 3 trial (CheckMate 649), we enrolled adults (≥18 years) with previously untreated, unresectable, non-HER2-positive gastric, gastro-oesophageal junction, or oesophageal adenocarcinoma, …

Malemedicine.medical_specialtyEsophageal Neoplasmsmedicine.medical_treatmentIpilimumabAdenocarcinoma030204 cardiovascular system & hematologyGastroenterologyCapecitabine03 medical and health sciences0302 clinical medicineStomach NeoplasmsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumans030212 general & internal medicineProgression-free survivalImmune Checkpoint InhibitorsAgedChemotherapybusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseProgression-Free SurvivalOxaliplatinNivolumabFluorouracilAdenocarcinomaDrug Therapy CombinationFemaleEsophagogastric JunctionNivolumabbusinessmedicine.drugThe Lancet
researchProduct

Regulation of cell-to-cell communication in non-tumorigenic and malignant human prostate epithelial cells.

2002

BACKGROUND Gap-junction-mediated intercellular communication (GJIC) is required for normal development and tissue homeostasis. However, the role of GJIC in human prostate carcinogenesis and progression remains ill-defined. METHODS The ability of hormones, anti-hormones, and the anti-hypertensive drug, forskolin, to restore GJIC in non-tumorigenic (RWPE-1 and PWR-1E) and malignant (RWPE-2, LNCaP, DU-145) human prostate epithelial cell lines, was examined by Scrape-Loading/Dye Transfer (SL/DT) and Fluorescence Recovery After Photobleaching (FRAP) methods using an Ultima laser cytometer. RESULTS Results from both assays show that PWR-1E, RWPE-2, LNCaP, and DU-145 cells have weak or absent GJIC…

Malemedicine.medical_specialtyEstroneUrologyCell CommunicationBiologyurologic and male genital diseasesmedicine.disease_causeConnexinschemistry.chemical_compoundProstate cancerCell–cell interactionInternal medicineLNCaPmedicineTumor Cells CulturedHumansTissue homeostasisForskolinColforsinGap JunctionsProstatic NeoplasmsEpithelial Cellsmedicine.diseaseEndocrinologyCell Transformation NeoplasticOncologychemistryCell cultureCancer researchCarcinogenesisImmortalised cell lineThe Prostate
researchProduct

Uptake and metabolism of [3H]choline by the rat phrenic nerve-hemidiaphragm preparation

1987

A whole nerve-muscle preparation (about 160 mg) or an end-plate preparation (about 25 mg) of the rat phrenic nerve-hemidiaphragm were incubated with [3H]choline, to investigate choline uptake and choline metabolism. Choline uptake was measured from the disappearance of choline from the incubation medium during the loading period and from the retention of tritium in the tissue after the loading and washout period. Based on the results obtained with both methods the end-plate preparation takes up three times as much choline than the whole nerve-muscle preparation or a small muscle strip that was cut outside the end-plate region and had a similar size as the end-plate preparation. Choline upta…

Malemedicine.medical_specialtyMetaboliteDiaphragmNeuromuscular JunctionPhospholipidIn Vitro TechniquesBiologyMotor EndplateNeuromuscular junctionCholinechemistry.chemical_compoundPhosphatidylcholineInternal medicinemedicineAnimalsCholinePhrenic nervePharmacologyRats Inbred StrainsHemicholinium 3General MedicineMetabolismMuscle DenervationRatsPhrenic NerveLysophosphatidylcholinemedicine.anatomical_structureEndocrinologychemistryNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct

Presynaptic nicotine receptors mediating a positive feed-back on transmitter release from the rat phrenic nerve.

1986

The effects of 1,1-dimethyl-4-phenylpiperazinium (DMPP) and of nicotine receptor antagonists on [3H]acetylcholine release from the rat phrenic nerve preincubated with [3H]choline were investigated in the absence and presence of cholinesterase inhibitors (presynaptic effects). Additionally, the effects of hexamethonium and tubocurarine on the muscle contraction of the indirectly stimulated diaphragm were examined (postsynaptic effects). DMPP (1-30 microM) increased (76-92%), whereas hexamethonium (0.001-1 mM) and tubocurarine (1-10 microM) decreased (52-60%) the release of [3H]acetylcholine following a train of 100 pulses at 5 Hz. The release caused by a longer train (750 pulses at 5 Hz) was…

Malemedicine.medical_specialtyMotor nerveTubocurarineHexamethonium CompoundsIn Vitro TechniquesReceptors NicotinicNeuromuscular junctionFeedbackchemistry.chemical_compoundPostsynaptic potentialInternal medicinemedicineAnimalsCholinesterasePhrenic nervePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsGeneral Medicinemusculoskeletal systemElectric StimulationRatsPhrenic NerveEndocrinologymedicine.anatomical_structurechemistrybiology.proteinHexamethoniummedicine.symptomDimethylphenylpiperazinium IodideAcetylcholineMuscle contractionmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Myasthenia-like syndrome induced by cardiovascular agents. Report of a case

1987

The case of a myasthenia-like syndrome induced by cardiovascular drugs is reported. The clinical and electrophysiological features of the case are discussed. © 1987 Masson Italia Periodici S.r.l.

Malemedicine.medical_specialtyNeurologyNeuromuscular JunctionCoronary DiseaseDermatologyPeruvosideMyasthenia-like syndromeSynaptic TransmissionPropafenoneimmune system diseasesMyasthenia GravisHumansMedicineperuvosideEtafenone hydrochlorideAgedNeuroradiologyOphthalmoplegiaNeuroscience (all)business.industryGeneral NeuroscienceGeneral Medicinenervous system diseasesCardenolidesPsychiatry and Mental healthAnesthesiaCardiovascular agentetafenone hydrochlorideDrug Therapy CombinationSettore MED/26 - NeurologiaNeurosurgeryNeurology (clinical)businessmedicine.drug
researchProduct

Impaired neuromuscular transmission during partial inhibition of acetycholinest-erase: The of stimulus-induced antiromic backfiring in the generation…

1992

Neuromuscular transmission was studied in the rat phrenic nerve-hemidiaphragm preparation with acetylcholinesterase (AChE) partially inactivated. Enzyme inhibition resulted in (1) increased single-twitch tension of the diaphragm; (2) compound muscle action potential (CMAP) containing repetitive discharges; (3) stimulus-induced antidromic backfiring (SIAB) seen in the phrenic nerve; and (4) repetitive nerve stimulation (RNS) eliciting a decrement-increment (D-I) phenomenon (i.e., amplitude reduction maximal with the second CMAP). Using a high-calcium and low-magnesium solution, SIAB and the decrement of the second CMAP during RNS were intensified, whereas closely spaced trains and (+)-tubocu…

Malemedicine.medical_specialtyPhysiologyDiaphragmNeuromuscular JunctionNeuromuscular transmissionAction PotentialsReceptors NicotinicSynaptic TransmissionRats Sprague-DawleyCellular and Molecular NeurosciencePostsynaptic potentialPhysiology (medical)Internal medicinemedicineAnimalsRepetitive nerve stimulationEvoked PotentialsPhrenic nerveChemistrymusculoskeletal systemElectric StimulationNeostigmineRatsAntidromicCompound muscle action potentialPhrenic NerveEndocrinologymedicine.anatomical_structurePeripheral nervous systemAcetylcholinesteraseNeurology (clinical)NeuroscienceAcetylcholinemedicine.drugMuscle & Nerve
researchProduct

Suppression by cholinesterase inhibition of a Ca(2+)-independent efflux of [3H]acetylcholine from the neuromuscular junction of the isolated rat diap…

1992

Abstract Endplate preparations of the left rat hemidiaphragm were incubated with [ 3 H]choline to label neuronal acetylcholine stores. Elevation of the concentration (13.5–135 mmol/l) of extracellular potassium chloride (KC1) stimulated the release of [ 3 H]acetylcholine in a concentration-dependent manner. KC1 (27 mmol/l) still caused a significant efflux of [ 3 H]acetylcholine in a Ca 2+ -free medium. Inhibitors of cholinesterase (physostigmine, diisopropylfluorophosphate) suppressed by 80% this Ca 2+ -independent efflux of [ 3 H]acetylcholine. Vesamicol (10 μmol/l), the blocker of the vesicular acetylcholine carrier, also suppressed the stimulated, Ca 2+ -independent efflux of [ 3 H]acet…

Malemedicine.medical_specialtyPhysostigmineVesamicolPhysostigmineDiaphragmNeuromuscular JunctionIn Vitro TechniquesTritiumNeuromuscular junctionCholinePotassium ChlorideRats Sprague-Dawleychemistry.chemical_compoundPiperidinesInternal medicinemedicineCholineAnimalsCholinesterasePharmacologyMuscarinebiologyAcetylcholineRatsEndocrinologymedicine.anatomical_structurechemistrybiology.proteinCalciumFemaleCholinesterase Inhibitorsmedicine.symptomAcetylcholinemedicine.drugMuscle contractionEuropean journal of pharmacology
researchProduct

Some pharmacological properties of the false cholinergic transmitter acetylpyrrolidinecholine and its precursor pyrrolidinecholine

1976

The acetylchline analogue acetylpyrrolidinecholine as well as the choline analogue pyrrolidinecholine were synthesized and the cholinergic properties of both substances were investigated on the guinea-pig ileum, rat blood pressure and frog rectus abdominis muscle. Acetylpyrrolidinecholine was 3-5 times less potent than acetylcholine on the three preparations tested. The dose-response curves to acetylpyrrolidinecholine were shifted to the right in a parallel manner by atropine and (+)-tubocurarine. The dissociation constants for atropine and (+)-tubocurarine obtained with acetylpyrrolidinecholine as agonist were not different from those obtained with acetylcholine. This indicates that acetyl…

Malemedicine.medical_specialtyPyrrolidinesGuinea PigsRana temporariaNeuromuscular JunctionNeuromuscular transmissionBlood PressureReceptors NicotinicSynaptic TransmissionCholinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsAcetylcholine receptorPharmacologyMuscarineMuscarinic acetylcholine receptor M3Muscle SmoothGeneral MedicineReceptors MuscarinicAcetylcholineRatsNicotinic agonistEndocrinologyParasympathomimeticschemistryCholinergicAcetylcholineMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct

Antiabsence effects of carbenoxolone in two genetic animal models of absence epilepsy (WAG/Rij rats and lh/lh mice).

2005

Carbenoxolone (CBX), the succinyl ester of glycyrrhetinic acid, is an inhibitor of gap junctional intercellular communication. We have tested its possible effects upon two genetic animal models of epilepsy (WAG/Rij rats and lethargic (lh/lh) mice). Systemic administration of CBX was unable to significantly affect the occurrence of absence seizures in WAG/Rij rats. In particular, intravenous (5-40 mg/kg) or intraperitoneal (i.p.; 10-80 mg/kg) administration of CBX was unable to significantly modify the number and duration of spike-wave discharges (SWDs) in WAG/Rij rats, whereas the bilateral microinjection (0.05, 0.1, 0.5 and 1 microg/0.5 microl) of CBX into nucleus reticularis thalami (NRT)…

Malemedicine.medical_specialtyTime FactorsCarbenoxoloneConnexinConnexinsCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsyMiceMice Neurologic MutantsInternal medicinemedicineAnimalsGlycyrrhizinMicroinjectionGap junctionsPharmacologyDose-Response Relationship DrugGap junctionElectroencephalographyRats Inbred StrainsEpilepsy Carbenoxolone WAG/Rij rat Lethargic mouse Gap junction Connexin Absence seizuresmedicine.diseaseRatsDisease Models AnimalEndocrinologymedicine.anatomical_structurechemistryEpilepsy AbsenceGene Expression RegulationThalamic NucleiSystemic administrationCarbenoxoloneepilepsyAutoradiographyNucleusmedicine.drugGap junctions; Carbenoxolone ; epilepsyNeuropharmacology
researchProduct