Search results for " jun"

showing 10 items of 611 documents

Identification and functional expression of HCx31.9, a novel gap junction gene

2002

By combining in silico and bench molecular biology methods we have identified a novel human gap junction gene that encodes a protein designated HCx31.9. We have determined its human chromosomal location and gene structure, and we have identified a putative mouse ortholog, mCx30.2. We have observed the presence of HCx31.9 in human cerebral cortex, liver, heart, spleen, lung, and kidney and the presence of mCx30.2 in mouse cerebral cortex, liver and lung. Moreover, preliminary data on the electrophysiological properties of HCx31.9 have been obtained by functional expression in paired Xenopus oocytes and in transfected N2A cells.

Patch-Clamp TechniquesIn silicoMolecular Sequence DataClinical BiochemistryXenopuscloningGene ExpressionConnexinConnexinsCell Linegap junctionMiceXenopus laevisGene expressionmedicineAnimalsHumansTissue DistributionAmino Acid SequenceCloning MolecularGenePhylogenybiologycloning; CNS; gap junctionGap junctionGap JunctionsCell BiologyGeneral MedicineTransfectionbiology.organism_classificationMolecular biologymedicine.anatomical_structureCerebral cortexOocytesCNSSequence Alignment
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Perlecan is critical for heart stability

2008

Aims Perlecan is a heparansulfate proteoglycan found in basement membranes, cartilage, and several mesenchymal tissues that form during development, tumour growth, and tissue repair. Loss-of-function mutations in the perlecan gene in mice are associated with embryonic lethality caused primarily by cardiac abnormalities probably due to hemopericards. The aim of the present study was to investigate the mechanism underlying the early embryonic lethality and the pathophysiological relevance of perlecan for heart function. Methods and results Perlecan-deficient murine embryonic stem cells were used to investigate the myofibrillar network and the electrophysiological properties of single cardiomy…

Patch-Clamp TechniquesPhysiologyMyocardial InfarctionMice TransgenicCell CommunicationPerlecanSarcomereBasement MembraneVentricular Function LeftAdherens junctionExtracellular matrixMicePhysiology (medical)medicineAnimalsMyocytes CardiacCells CulturedEmbryonic Stem CellsBasement membranebiologyCartilageCell DifferentiationHeartAnatomyEmbryonic stem cellCell biologyMice Inbred C57BLcarbohydrates (lipids)Disease Models Animalmedicine.anatomical_structurebiology.proteinFemaleCardiology and Cardiovascular MedicineMyofibrilHeparan Sulfate ProteoglycansCardiovascular Research
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Ultramicro-trauma in the endometrial-myometrial junctional zone and pale cell migration in adenomyosis

2015

Objective To determine if ultrastructural tissue trauma occurs in the junctional zone in uteri in adenomyosis. Design A case-control experimental study. Setting Endometriosis research center. Patient(s) Twelve uteri with adenomyosis, and 9 uteri without adenomyosis, were gained during laparoscopy-assisted vaginal hysterectomy. Intervention(s) Transmission electron microscopic study of the junctional zone, as well as immunohistochemical staining for epithelial cadherin, and van Gieson staining and immunofluorescence for CD45 and CD68. Main Outcome Measure(s) Analysis of the electron microscopy photos and the immunoreactive scores of the staining. Result(s) The inner myometrial muscle fibers …

PathologyEndometriosisFluorescent Antibody TechniqueEndometriumBasal (phylogenetics)EndometriumMyofibrilsCell MovementVan Gieson's stainHysterectomy VaginalMedicine (all)MyometriumObstetrics and GynecologyAntigens CD45AnatomyMiddle AgedCadherinsultrastructureImmunohistochemistrymedicine.anatomical_structurePhenotypeMyometriumImmunohistochemistryFemaleAdenomyosis pathogenesiCase-Control StudieAdenomyosisHumanAdultmedicine.medical_specialtyAntigens Differentiation MyelomonocyticBiologypale cellMicroscopy Electron TransmissionAntigens CDmedicineHumansAdenomyosisendometrial-myometrial junctional zoneEpithelial CellCadherinEpithelial CellsBiomarkermedicine.diseaseReproductive MedicineCase-Control StudiesCadherinLeukocyte Common AntigensLaparoscopyAdenomyosiBiomarkersMyofibril
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Gastric cancer: epidemiology, pathology and treatment.

2003

Gastric cancer incidence and mortality has fallen dramatically over the last 50 years in many regions, but remains the second most common cancer worldwide. Despite a marked decline in fundic and distal tumors, there is a rising incidence of adenocarcinomas of the gastroesophageal junction and gastric cardia, particularly in Western nations. This may imply that there are in fact two diseases differing from each other in epidemiology, etiology, pathology and clinical expression. While surgical resection remains the cornerstone of gastric cancer treatment, the optimum extent of nodal resection remains controversial, with randomized studies failing to show that the D2 procedure improves surviva…

Pathologymedicine.medical_specialtyChemotherapybusiness.industrymedicine.medical_treatmentCancerHematologyGastroesophageal Junction Adenocarcinomamedicine.diseaseChemotherapy regimenRadiation therapyOncologyChemotherapy AdjuvantRisk FactorsStomach NeoplasmsEpidemiology of cancerMedicineHumansRadiotherapy AdjuvantbusinessAdjuvantEpirubicinmedicine.drugNeoplasm StagingAnnals of oncology : official journal of the European Society for Medical Oncology
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Lung epithelial cell lines in coculture with human pulmonary microvascular endothelial cells: development of an alveolo-capillary barrier in vitro.

2004

We have established a coculture system of human distal lung epithelial cells and human microvascular endothelial cells in order to study the cellular interactions of epithelium and endothelium at the alveolocapillary barrier in both pathogenesis and recovery from acute lung injury. The aim was to determine conditions for the development of functional cellular junctions and the formation of a tight epithelial barrier similar to that observed in vivo. The in vitro coculture system consisted of monolayers of human lung epithelial cell lines (A549 or NCI H441) and primary human pulmonary microvascular endothelial cells (HPMEC) on opposite sides of a permeable filter membrane. A549 failed to sho…

Pathologymedicine.medical_specialtyEndotheliummedicine.medical_treatmentBiologyLung injuryCell junctionDexamethasonePathology and Forensic MedicineCell LineTight JunctionsAdherens junctionmedicineElectric ImpedanceHumansMolecular BiologyLungLungBlood-Air BarrierTumor Necrosis Factor-alphaEpithelial CellsCell BiologyAdherens JunctionsEpitheliumCoculture TechniquesCell biologyEndothelial stem cellPulmonary AlveoliMicroscopy Electronmedicine.anatomical_structureCytokineEndothelium VascularInflammation MediatorsLaboratory investigation; a journal of technical methods and pathology
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Neuronal and BBB damage induced by sera from patients with secondary progressive multiple sclerosis.

2009

An important component of the pathogenic process of multiple sclerosis (MS) is the blood-brain barrier (BBB) damage. We recently set an in vitro model of BBB, based on a three-cell-type co-culture system, in which rat neurons and astrocytes synergistically induce brain capillary endothelial cells to form a monolayer with permeability properties resembling those of the physiological BBB. Herein we report that the serum from patients with secondary progressive multiple sclerosis (SPMS) has a damaging effect on isolated neurons. This finding suggests that neuronal damaging in MS could be a primary event and not only secondary to myelin damage, as generally assumed. SPMS serum affects the perme…

Pathologymedicine.medical_specialtyProgrammed cell deathBlotting WesternBiologyImmunofluorescenceOccludinModels BiologicalMyelinWestern blotOccludinGeneticsmedicineElectric ImpedanceAnimalsmultiple sclerosis brain cell cultures in vitro models of blood-brain barrier neuronal cell death transendothelial electrical resistanceMicroscopy Phase-ContrastRats WistarCells CulturedNeuronsmedicine.diagnostic_testTight junctionCell DeathMultiple sclerosisMembrane ProteinsGeneral MedicineMultiple Sclerosis Chronic Progressivemedicine.diseaseImmunohistochemistryRatsBlotmedicine.anatomical_structurenervous systemBlood-Brain BarrierAstrocytescardiovascular systemInternational journal of molecular medicine
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The “mode” of lymphocyte extravasation through HEV of Peyer's patches and its role in normal homing and inflammation

2007

The mode of lymphocyte transendothelial migration in the postcapillary high endothelial venules (HEVs) of Peyer's patches during normal homing and acute inflammation in the guinea pig was studied. It is common opinion that the lymphocyte transendothelial passage from the blood stream into the extravasal lymphoid tissue calls for a multistep process of endothelial and lymphocyte molecules favoring tethering, rolling, activation, arrest and its firm adhesion to the endothelial luminal surface. Ultrastructural serial pictures and the three-dimensional reconstruction of HEVs with lymphocytes during different moments of their transmigration through the endothelial wall enabled us to demonstrate …

Pathologymedicine.medical_specialtyTime FactorsLymphocyteHigh endothelial venulesGuinea PigsHomingLeukocyte RollingBiologyBiochemistryHigh endothelial venulePeyer's PatchesMicroscopy Electron TransmissionmedicineAnimalsReplica TechniquesLeukocyte RollingLymphocytesLymphocyte homing receptorTransendothelial migrationLymphatic VesselsInflammationICAM-1Endothelial CellsCell BiologyExtravasationEnteritisPeyer PatchChemotaxis LeukocyteDisease Models Animalmedicine.anatomical_structureIntercellular JunctionsPeyer patcheLymphocyteDairy ProductsCardiology and Cardiovascular MedicineExtravasationHoming (hematopoietic)
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Barrier functions and paracellular integrity in human cell culture models of the proximal respiratory unit.

2009

International audience; Airway epithelial cells provide a barrier to the translocation of inhaled materials. Tight (TJ) and adherens junctions (AJ) play a key role in maintaining barrier functions, and are responsible for the selective transport of various substances through the paracellular pathway. In this study we compared a bronchial cell line (16HBE14o-) and primary bronchial cells (HBEC), both cocultivated with the fibroblast cell line Wi-38, with respect to their structural differentiation and their reaction to cytokine stimulation. HBEC formed a pseudostratified epithelial layer and expressed TJ and AJ proteins after 2 weeks in coculture. Mucus-producing and ciliated cells were foun…

Pathologymedicine.medical_specialty[SDV]Life Sciences [q-bio]Blotting WesternCell Culture TechniquesPharmaceutical ScienceBronchi[SDV.BC]Life Sciences [q-bio]/Cellular Biology[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]BiologyModels BiologicalTight JunctionsAdherens junctionInterferon-gammaMicroscopy Electron Transmission[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]medicineHumansBarrier functionLungTumor Necrosis Factor-alphaEpithelial CellsAdherens JunctionsGeneral MedicineImmunohistochemistryCoculture TechniquesIn vitroCell biologyBlotmedicine.anatomical_structureCell cultureParacellular transportMicroscopy Electron ScanningRespiratory epitheliumBiotechnology
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Acute Morphological and Toxicological Effects in a Human Bronchial Coculture Model after Sulfur Mustard Exposure

2009

International audience; Sulfur mustard (SM) is a strong alkylating agent. Inhalation of SM causes acute lung injury accompanied by severe disruption of the airway barrier. In our study, we tested the acute effects after mustard exposure in an in vitro coculture bronchial model of the proximal barrier. To achieve this, we seeded normal human bronchial epithelial explant-outgrowth cells (HBEC) together with lung fibroblasts as a bilayer on filter plates and exposed the bronchial model after 31 days of differentiation to various concentrations of SM (30, 100, 300, and 500mM). The HBEC formed confluent layers, expressing functional tight junctions as measured by transepithelial electrical resis…

Pathologymedicine.medical_specialtysulfur mustard[SDV]Life Sciences [q-bio]ApoptosisBronchiEnzyme-Linked Immunosorbent Assay[SDV.BC]Life Sciences [q-bio]/Cellular BiologyBiologyLung injuryToxicologyCell LinelungProinflammatory cytokinechemistry.chemical_compoundIn vivoMustard GasmedicineHumansChemical Warfare AgentsInterleukin 8Tight junctionInterleukinSulfur mustardprimary bronchial cellsMolecular biologyCoculture TechniqueschemistryApoptosis[SDV.TOX]Life Sciences [q-bio]/ToxicologyMicroscopy Electron ScanningbarriercocultureToxicological Sciences
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Fingolimod (FTY720-P) Does Not Stabilize the Blood–Brain Barrier under Inflammatory Conditions in an in Vitro Model

2015

Breakdown of the blood-brain barrier (BBB) is an early hallmark of multiple sclerosis (MS), a progressive inflammatory disease of the central nervous system. Cell adhesion in the BBB is modulated by sphingosine-1-phosphate (S1P), a signaling protein, via S1P receptors (S1P\(_1\)). Fingolimod phosphate (FTY720-P) a functional S1P\(_1\) antagonist has been shown to improve the relapse rate in relapsing-remitting MS by preventing the egress of lymphocytes from lymph nodes. However, its role in modulating BBB permeabilityin particular, on the tight junction proteins occludin, claudin 5 and ZO-1has not been well elucidated to date. In the present study, FTY720-P did not change the transendotheli…

Pathologytight junctionsDrug Evaluation PreclinicalApoptosisVascular permeabilityOccludinlcsh:ChemistryMedicinelcsh:QH301-705.5Cells CulturedSpectroscopyTight junctionrat brain microvascular endothelial cell cultureGeneral MedicineFingolimodComputer Science ApplicationsCell biologyEndothelial stem cellmedicine.anatomical_structureMatrix Metalloproteinase 2Immunosuppressive AgentsFTY720-P; blood-brain barrier; rat brain microvascular endothelial cell culture; inflammation; tight junctionsmedicine.drugmedicine.medical_specialtyMultiple SclerosisMAP Kinase Signaling SystemBlood–brain barrierArticleCatalysisCapillary PermeabilityInorganic ChemistryOccludinFingolimod HydrochlorideAnimalsFTY720-Pddc:610Physical and Theoretical ChemistryClaudinMolecular BiologyFingolimod Hydrochloridebusiness.industryOrganic ChemistryEndothelial Cellsblood-brain barrierRatslcsh:Biology (General)lcsh:QD1-999inflammationMicrovesselsbusinessInternational Journal of Molecular Sciences
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