Search results for " muscular dystrophy"

showing 10 items of 63 documents

Deflazacort vs. prednisone in Duchenne muscular dystrophy: trends of an ongoing study

1995

Several studies have demonstrated the slowing effect of corticosteroids on the decline of muscle strength in Duchenne muscular dystrophy (DMD). Deflazacort (DFC) is supposed to have fewer side effects than prednisone (PRED). An ongoing double blind multicenter study is comparing the effects and side effects of deflazacort (0.9 mg/kg/day) and prednisone (0.75 mg/kg/day) in DMD. This interim report includes data for 67 boys between age 5 years and loss of ambulation. Besides the common clinical and laboratory data for chronic corticoid treatment, motor performance has been tested. Interim results, 3-15 months after starting the medication, show some scattering but no grouping of data for all …

Malemedicine.medical_specialtyDuchenne muscular dystrophyAnti-Inflammatory AgentsMuscular DystrophiesDouble-Blind MethodDevelopmental NeurosciencePregnenedionesPrednisoneInternal medicinemedicineHumansChildCreatine KinaseDose-Response Relationship Drugbiologybusiness.industryBody WeightGeneral MedicineAlkaline Phosphatasemedicine.diseaseClinical trialDeflazacortDose–response relationshipEndocrinologyNeurologyMulticenter studyChild PreschoolAnesthesiaPediatrics Perinatology and Child HealthOsteocalcinbiology.proteinPrednisoneNeurology (clinical)medicine.symptombusinessWeight gainMuscle Contractionmedicine.drugBrain and Development
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Treatment of Duchenne muscular dystrophy with ciclosporin A: a randomised, double-blind, placebo-controlled multicentre trial.

2010

Summary Background Duchenne muscular dystrophy is a rare X-linked progressive disease characterised by loss of ambulation at about age 10 years, with death in early adulthood due to respiratory and cardiac insufficiency. Steroids are effective at slowing the progression of muscle weakness; however, their use is limited by side-effects, prompting the search for alternatives. We assessed the effect of ciclosporin A as monotherapy and in combination with intermittent prednisone for the treatment of ambulant patients with this disorder. Methods Our study was a parallel-group, placebo-controlled, double-blind, multicentre trial at trial sites of the German muscular dystrophy network, MD-NET, ove…

Malemedicine.medical_specialtyDuchenne muscular dystrophyMedizinPlacebolaw.invention03 medical and health sciences0302 clinical medicineRandomized controlled trialDouble-Blind MethodlawPrednisoneInternal medicinemedicineHumansMuscular dystrophyChild030304 developmental biology0303 health sciencesbusiness.industryMuscle weaknessmedicine.diseaseCiclosporin3. Good healthSurgeryClinical trialMuscular Dystrophy DuchenneReview Literature as TopicTreatment OutcomeCyclosporineNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Lancet. Neurology
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Muscle degeneration in inguinal hernia specimens.

2012

BACKGROUND: There are few articles in the literature reporting the histological changes of groin structures affected by inguinal hernia. A deeper knowledge of this matter could represent an important step forward in the identification of the causes of hernia protrusion. This study aimed to recognise the pathological modifications of muscular structures in autopsy specimens excised from tissues surrounding the hernia orifice. METHODS: Inguinal hernia was identified in 30 autopsied cadavers, which presented different varieties of hernia, including indirect, direct and mixed. Tissue specimens were resected for histological study from structures of the inguinal area surrounding the hernia openi…

Malemedicine.medical_specialtyHyalinMuscle Fibers SkeletalAutopsyContext (language use)Hernia InguinalGroinherniaVeinsBiopsymedicineHumansHerniaMuscular dystrophyHyalineAgedAged 80 and overGroinmedicine.diagnostic_testbusiness.industryAnatomyArteriesMiddle Agedmedicine.diseaseFibrosisdigestive system diseasesAxonsSurgerystomatognathic diseasesInguinal herniaSettore MED/18 - Chirurgia Generalesurgical procedures operativemedicine.anatomical_structureSurgeryInguinal hernia Etiology Muscles Fibrosis Hyalin Muscular dystrophy IntAtrophybusiness
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Myogenic NOS and endogenous NO production are defective in colon from dystrophic (mdx) mice.

2001

The aim of the present study was to evaluate whether alterations in the distribution and/or function of nitric oxide synthase (NOS) could be involved in the development of the spontaneous mechanical tone observed in colon from dystrophic ( mdx) mice. By recording the intraluminal pressure of isolated colon from normal mice, we showed that Nω-nitro- l-arginine methyl ester (l-NAME) increased the tone, even in the presence of tetrodotoxin. The effect was prevented by l-arginine, nifedipine, or Ca2+-free solution. In colon from mdx mice, l-NAME was ineffective. Immunohistochemistry revealed that the presence and distribution of neuronal (nNOS), endothelial, and inducible NOS isoforms in smooth…

Malemedicine.medical_specialtyPhysiologyRatónColonDuchenne muscular dystrophyEndogenyNitric OxideNitric oxidechemistry.chemical_compoundMiceReference ValuesPhysiology (medical)Internal medicineGene expressionmedicineAnimalsHepatologybiologyGastroenterologyMuscle Smoothmedicine.diseaseImmunohistochemistryPathophysiologyNitric oxide synthaseMice Inbred C57BLMuscular Dystrophy DuchenneEndocrinologychemistrybiology.proteinMice Inbred mdxImmunohistochemistryNitric Oxide SynthaseAmerican journal of physiology. Gastrointestinal and liver physiology
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Duchenne muscular dystrophy and idiopathic hyperCKemia segregating in a family

1995

A 7-month-old boy with gross motor delay and failure to thrive presented with rhabdomyolysis following an acute asthmatic episode. During hospitalization an electrocardiographic conversion to a Wolff-Parkinson-White type 1 (WPW) pattern took place. Duchenne muscular dystrophy (DMD) was suspected based on elevated creatine kinase (CK) serum levels, muscle biopsy, and family history. The diagnosis was confirmed by molecular analysis, which documented a deletion corresponding to cDNA probe 1-2a in the dystrophin gene, in the propositus and in an affected male cousin of his mother. "Idiopathic" hyperCKemia was found in the propositus, his father, and 5 of his relatives. We suggest that the unus…

Malemusculoskeletal diseasescongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtyDuchenne muscular dystrophyMolecular Sequence DataGene mutationPolymerase Chain ReactionMuscular DystrophiesGenomic ImprintingPrenatal DiagnosisInternal medicinemedicineHumansFamily historyCreatine KinaseGenetics (clinical)X-linked recessive inheritanceDNA PrimersGenes DominantMuscle biopsyBase Sequencebiologymedicine.diagnostic_testGenetic Carrier ScreeningInfantExonsmedicine.diseasePedigreeEndocrinologyMutationFailure to thrivebiology.proteinFemaleCreatine kinasemedicine.symptomDystrophinMetabolism Inborn ErrorsAmerican Journal of Medical Genetics
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Mehrsegmentale Fusion der Skoliose bei Duchenne-Muskeldystrophie

2008

Operations in scoliosis in patients suffering from an advanced stage of Duchenne muscular dystrophy are associated with a higher risk due to the extent of the curves, the respiratory insufficiency and frequent cardiomyopathia. Progressive scolioses in 20 wheelchair patients with an age between 10.5-18.3 years (mean 14.6 years) were treated by CDI. The mean preoperative angle in this group was 70.6 degrees, the postoperative angle 31.2 degrees (mean correction 39.4 degrees or 55.8%). The preoperative lordosis of the lumbar spine (mean angle 4.1 degrees) was corrected to 17.8 degrees postoperatively. The average intraoperative blood loss (2300 ccm) was evident more compared with idiopathic sc…

Mechanical ventilationmedicine.medical_specialtyLordosisCobb anglebusiness.industrymedicine.medical_treatmentArthrodesisDuchenne muscular dystrophyScoliosismedicine.diseaseSurgeryOrthopedic surgerymedicineOrthopedics and Sports MedicineSurgerybusinessRachisZeitschrift für Orthopädie und ihre Grenzgebiete
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FSHD muscular dystrophy region gene 1 binds Suv4-20h1 histone methyltransferase and impairs myogenesis.

2013

Facioscapulohumeral muscular dystrophy (FSHD) is an autosomal dominant myopathy with a strong epigenetic component. It is associated with deletion of a macrosatellite repeat leading to over-expression of the nearby genes. Among them, we focused on FSHD region gene 1 (FRG1) since its over-expression in mice, Xenopus laevis and Caenorhabditis elegans, leads to muscular dystrophy-like defects, suggesting that FRG1 plays a relevant role in muscle biology. Here we show that, when over-expressed, FRG1 binds and interferes with the activity of the histone methyltransferase Suv4-20h1 both in mammals and Drosophila. Accordingly, FRG1 over-expression or Suv4-20h1 knockdown inhibits myogenesis. Moreov…

Muscle DevelopmentEvolution Molecular03 medical and health sciencesMice0302 clinical medicineGeneticsmedicineFacioscapulohumeral muscular dystrophyMyocyteAnimalsHumansEpigeneticsMuscular dystrophyMyopathyMolecular Biology030304 developmental biologyCell NucleusMice Knockout0303 health sciencesMuscle CellsbiologyMyogenesisMicrofilament ProteinsNuclear ProteinsProteinsRNA-Binding ProteinsCell DifferentiationCell BiologyGeneral MedicineHistone-Lysine N-MethyltransferaseMuscular Dystrophy Animalmedicine.diseaseMolecular biologyHistoneDrosophila melanogasterHEK293 CellsPhenotypeOrgan SpecificityHistone methyltransferaseEpigenetic deregulation by FRG1Gene Knockdown Techniquesbiology.proteinmedicine.symptomCarrier Proteins030217 neurology & neurosurgeryProtein BindingJournal of molecular cell biology
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P.20.3 Targeting fibrosis and inflammation in Duchenne Muscular Dystrophy

2013

Duchenne Muscular Dystrophy is the most frequent genetic muscle disease worldwide affecting ∼1:5000 male births. It is caused by a defective DMD gene, which leads to reduced and defective dystrophin protein expression. The constant breakdown of fibres leads to focal necrosis, myophagocytosis and a considerable influx of inflammatory cells into the muscle tissue, which is followed by increasing endomysial fibrosis. Both, inflammation and fibrosis as well as a putative relation are not yet understood immunologically. Fibrosis directly correlates with adverse outcome and early loss of ambulation. We have studied how inflammation is linked to fibrosis in DMD, with an emphasis on the communicati…

Muscle biopsymedicine.diagnostic_testbiologyDuchenne muscular dystrophyInflammationDiseasemedicine.diseasePhenotypeImmune systemNeurologyFibrosisPediatrics Perinatology and Child HealthImmunologymedicinebiology.proteinNeurology (clinical)medicine.symptomDystrophinGenetics (clinical)Neuromuscular Disorders
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Pain Phenotypes in Rare Musculoskeletal and Neuromuscular Diseases

2020

For patients diagnosed with a rare musculoskeletal or neuromuscular disease, pain may transition from acute to chronic; the latter yielding additional challenges for both patients and care providers. We assessed the present understanding of pain across a set of ten rare, noninfectious, noncancerous disorders; Osteogenesis Imperfecta, Ehlers-Danlos Syndrome, Achondroplasia, Fibrodysplasia Ossificans Progressiva, Fibrous Dysplasia/McCune-Albright Syndrome, Complex Regional Pain Syndrome, Duchenne Muscular Dystrophy, Infantile- and Late-Onset Pompe disease, Charcot-Marie-Tooth Disease, and Amyotrophic Lateral Sclerosis. Through the integration of natural history, cross-sectional, retrospective…

Neuromuscular diseaseCognitive NeuroscienceDuchenne muscular dystrophyPainDiseaseBioinformaticsArticle03 medical and health sciencesBehavioral Neuroscience0302 clinical medicinemedicineHumans0501 psychology and cognitive sciences050102 behavioral science & comparative psychologyAmyotrophic lateral sclerosisRetrospective Studiesbusiness.industryFibrous dysplasia05 social sciencesNeuromuscular Diseasesmedicine.diseaseCross-Sectional StudiesPhenotypeNeuropsychology and Physiological PsychologyComplex regional pain syndromeOsteogenesis imperfectaFibrodysplasia ossificans progressivabusiness030217 neurology & neurosurgeryNeuroscience & Biobehavioral Reviews
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Value of insoluble PABPN1 accumulation in the diagnosis of oculopharyngeal muscular dystrophy.

2019

Background and purpose The aim was to assess the value of insoluble PABPN1 muscle fibre nuclei accumulation in the diagnosis of atypical cases of oculopharyngeal muscular dystrophy (OPMD). Methods Muscle biopsies from a selected cohort of 423 adult patients from several Italian neuromuscular centres were analysed by immunofluorescence: 30 muscle biopsies of genetically proven OPMD, 30 biopsies from patients not affected by neuromuscular disorders, 220 from genetically undiagnosed patients presenting ptosis or swallowing disturbances, progressive lower proximal weakness and/or isolated rimmed vacuoles at muscle biopsy and 143 muscle biopsies of patients affected by other neuromuscular diseas…

Pathologymedicine.medical_specialtyFluorescent Antibody TechniquePoly(A)-Binding Protein IOculopharyngeal muscular dystrophy03 medical and health sciences0302 clinical medicinePtosisMuscular Dystrophy OculopharyngealPABPN1 accumulations; PABPN1 immunofluorescence; oculopharyngeal musclular dystrophy; rimmed vacuoles; tubulofilamentous intranuclear inclusionsmedicineHumans030212 general & internal medicineMuscle fibreMuscle SkeletalCell NucleusMuscle biopsymedicine.diagnostic_testbusiness.industryExternal ophthalmoplegiaRimmed vacuolesmedicine.diseaseNeurologyCohortNeurology (clinical)Inclusion body myositismedicine.symptombusiness030217 neurology & neurosurgeryEuropean journal of neurologyReferences
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