Search results for " mutation"

showing 10 items of 1212 documents

The Suppressor of fused Gene Encodes a Novel PEST Protein Involved in Drosophila Segment Polarity Establishment

1995

Abstract Suppressor of fused, Su(fu), was identified as a semi-dominant suppressor of the putative serine/threonine kinase encoded by the segment polarity gene fused in Drosophila melanogaster. The amorphic Su(fu) mutation is viable, shows a maternal effect and displays no phenotype by itself. Su(fu) mutations are often found associated to karmoisin (kar) mutations but two complementation groups can be clearly identified. By using a differential hybridization screening method, we have cloned the Su(fu) region and identified chromosomal rearrangements associated with Su(fu) mutations. Two classes of cDNAs with similar developmental patterns, including a maternal contribution, are detectable …

Untranslated regionDNA Complementary[SDV]Life Sciences [q-bio]Recombinant Fusion ProteinsMolecular Sequence DataRestriction MappingInvestigations03 medical and health sciencesPEST sequence0302 clinical medicineTranscription (biology)GeneticsAnimalsDrosophila ProteinsAmino Acid SequenceCloning MolecularGenes SuppressorPeptide sequenceGeneGerm-Line MutationIn Situ Hybridization030304 developmental biologyGenetics0303 health sciencesBase SequencebiologyBlotting Northernbiology.organism_classificationMolecular biology[SDV] Life Sciences [q-bio]Repressor ProteinsComplementationDrosophila melanogasterPhenotypeSegment polarity geneDrosophila melanogaster030217 neurology & neurosurgery
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A029 Identification of polymorphisms in the gene encoding secreted phospholipase A2 group X and study of their role in coronary artery disease. The a…

2009

Human secreted phospholipases A2 (sPLA2s) represent novel attractive therapeutic targets and biomarkers in coronary artery diseases (CAD). We have shown that human Group X sPLA2 (hGX sPLA2) is present in atherosclerotic lesions and that hGX sPLA2 modified LDL induces foam cell formation. To elucidate whether hGX sPLA2 has a causative role in CAD we have screened the human PLA2G10 gene to identify frequent polymorphisms, and we have examined their possible association with cardiovascular end-points and intermediate inflammatory phenotypes in a large prospective study of patients with CAD (the AtheroGene study). Although no significant association was found between the various polymorphisms i…

Untranslated regionPathologymedicine.medical_specialtybiologybusiness.industryGeneral Medicinemedicine.diseasePhenotypeCoronary artery diseasePhospholipase A2Cancer researchbiology.proteinMedicineMissense mutationAlleleCardiology and Cardiovascular MedicinebusinessGeneFoam cellArchives of Cardiovascular Diseases
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Giustizia ambientale e recupero delle aree verdi: il caso di Palermo

2021

L’urban shrinkage è diventata una questione centrale tanto nel dibattito scientifico quanto nell’agenda politica di diverse città europee. Secondo i dati EUROSTAT (2019), circa il 40% delle città europee con più di 200.000 abitanti stanno perdendo popolazione; una contrazione che si esplicita maggiormente, sotto il profilo spaziale, in una progressiva sottoutilizzazione del patrimonio edificato, così come nel contestuale incremento di aree e terreni in stato di abbandono. In questo quadro controverso di mutazione strutturale della città, così come l’abbiamo conosciuta, le politiche urbane alla prova in Europa tentano di declinare la decrescita quale occasione strategica di investimento sull…

Urban shrinkage became a central issue in both the scientific debate and the political agenda of several European cities. According to EUROSTAT data (2019) about 40% of European cities with more than 200000 inhabitants are losing population. A shrinkage that is more explicit in a progressive underutilization of the built heritage as well as in the concomitant increase of abandoned areas. In this controversial framework of structural mutation of the city urban policies in Europe try to decline the degrowth as a strategic opportunity to invest in green areas in order to increase urban quality and to act simultaneously on the development of local economies. With respect to the above framework this contribution focuses on the evaluation of the effects that the project of new public green spaces have generated on some sample areas on the outskirts of the city of Palermo. An urban context where according to the latest Svimez Report (2019) the phenomenon of urban shrinkage represents a constantly growing trend and on which the new Master Plan operates through a revival of interventions on environmental capital and urban green areas of the city.Settore ICAR/21 - Urbanistica
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Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene

1997

We previously isolated a mutant cell that is the only mammalian cell reported to have a persistently low level of UDP-glucose. In this work we obtained a spontaneous revertant whose UDP-glucose level lies between those found in the wild type and the mutant cell. The activity of UDP-glucose pyrophosphorylase (UDPG:PP), the enzyme that catalyzes the formation of UDP-glucose, was in the mutant 4% and in the revertant 56% of the activity found in the wild type cell. Sequence analysis of UDPG: PP cDNAs from the mutant cell showed one missense mutation, which changes amino acid residue 115 from glycine to aspartic acid. The substituted glycine is located within the largest stretch of strictly con…

Uridine Diphosphate GlucoseDNA ComplementaryMagnetic Resonance SpectroscopyUTP-Glucose-1-Phosphate UridylyltransferaseMolecular Sequence DataMutantDeoxyglucoseBiologymedicine.disease_causeBiochemistryProtein Structure SecondaryCell LineCricetulusCricetinaeAspartic acidmedicineAnimalsPoint MutationMissense mutationAmino Acid SequenceMolecular Biologychemistry.chemical_classificationMutationSequence Homology Amino AcidPoint mutationWild typeCell BiologyMolecular biologyEnzymeBiochemistrychemistryGlycineJournal of Biological Chemistry
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Study of USH1 Splicing Variants through Minigenes and Transcript Analysis from Nasal Epithelial Cells

2012

Usher syndrome type I (USH1) is an autosomal recessive disorder characterized by congenital profound deafness, vestibular areflexia and prepubertal retinitis pigmentosa. The first purpose of this study was to determine the pathologic nature of eighteen USH1 putative splicing variants found in our series and their effect in the splicing process by minigene assays. These variants were selected according to bioinformatic analysis. The second aim was to analyze the USH1 transcripts, obtained from nasal epithelial cells samples of our patients, in order to corroborate the observed effect of mutations by minigenes in patient’s tissues. The last objective was to evaluate the nasal ciliary beat fre…

Usher syndromelcsh:Medicinemedicine.disease_causeGene SplicingMolecular cell biologyAutosomal Recessivelcsh:ScienceGeneticsMutationMultidisciplinaryCadherinsMyosin VIIaRNA splicingSensory PerceptionUsher SyndromesResearch ArticleRNA SplicingCadherin Related ProteinsBiologyMyosinsNoseGenetic MutationRetinitis pigmentosamedicineGeneticsotorhinolaryngologic diseasesHumansCiliaBiologyMessenger RNAlcsh:RIntronMutation TypesComputational BiologyGenetic VariationEpithelial CellsHuman Geneticsmedicine.diseaseMolecular biologyRNA processingMutagenesisCase-Control StudiesMutationGenetics of Diseaselcsh:QGene expressionSensory DeprivationPCDH15MinigeneCloningNeuroscience
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VDR MUTATION IN TWO SISTERS: PHENOTYPE VARIABILITY AND CLINICAL OUTCOME

2017

Objectives: Hereditary vitamin D-resistant rickets (HVDRR) is an autosomal recessive disease secondary to the mutation of vitamin D receptor (VDR) gene. These children show an early onset of rickets and in some of them alopecia is associated. Methods: We describe clinical features and laboratory findings in two sisters affected by HVDRR, as well as their response to treatment. Results: The first born is now 4 years old and had a severe and resistant hypocalcaemia, with low response to high doses of calcium per os, the requirement of intravenous infusion of calcium for a prolonged period, hypocalcaemic seizures resolved with high doses of intravenous calcium and high doses of vitamin D. Clin…

VDR MUTATION PHENOTYPE VARIABILITY CLINICAL OUTCOME
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Spatial and temporal distribution of SARS-CoV-2 diversity circulating in wastewater

2022

Wastewater-based epidemiology (WBE) has proven to be an effective tool for epidemiological surveillance of SARS-CoV-2 during the current COVID-19 pandemic. Furthermore, combining WBE together with high-throughput sequencing techniques can be useful for the analysis of SARS-CoV-2 viral diversity present in a given sample. The present study focuses on the genomic analysis of SARS-CoV-2 in 76 sewage samples collected during the three epidemiological waves that occurred in Spain from 14 wastewater treatment plants distributed throughout the country. The results obtained demonstrate that the metagenomic analysis of SARS-CoV-2 in wastewater allows the detection of mutations that define the B.1.1.…

Variants of concernEnvironmental EngineeringSARS-CoV-2Ecological ModelingfungiVariants of interestCOVID-19MicrobiologiaWastewaterGenome sequencingvariants of concernPollutionArticlespike mutationsEcologiagenome sequencingAigües residuals Microbiologiavariants of interestHumansSpike mutationsPandemicswastewaterWaste Management and DisposalWater Science and TechnologyCivil and Structural EngineeringWater Research
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Tbx1 regulates Vegfr3 and is required for lymphatic vessel development

2010

Defects in lymphangiogenesis are added to the broad clinical manifestations of DiGeorge syndrome, caused by deletion of the T box transcription factor Tbx1.

Vascular Endothelial Growth Factor ATBX1Cellular differentiationBiologyMice03 medical and health sciences0302 clinical medicinestomatognathic systemReportLymphatic vesselmedicineAnimalsHumansLymphangiogenesisEnhancerCells CulturedResearch ArticlesLymphatic Vessels030304 developmental biology0303 health sciencesABNORMAL CAROTID ARTERIES; TRANSGENIC MICE; VELOCARDIOFACIAL SYNDROME; CARDIOVASCULAR DEFECTS; LYMPHANGIOGENESIS; LYMPHEDEMA; MOUSE; RECEPTOR-3; MUTATION; SYSTEMEndothelial CellsGene Expression Regulation DevelopmentalCell DifferentiationCell BiologyEmbryo Mammalian3. Good healthLymphangiogenesisCell biologyVascular endothelial growth factor ALymphatic systemmedicine.anatomical_structureVascular endothelial growth factor Cembryonic structuresImmunologyT-Box Domain Proteins030217 neurology & neurosurgeryJournal of Cell Biology
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Mosaic activating mutations in GNA11 and GNAQ are associated with phakomatosis pigmentovascularis and extensive dermal melanocytosis

2016

Common birthmarks can be an indicator of underlying genetic disease but are often overlooked. Mongolian blue spots (dermal melanocytosis) are usually localized and transient, but they can be extensive, permanent, and associated with extracutaneous abnormalities. Co-occurrence with vascular birthmarks defines a subtype of phakomatosis pigmentovascularis, a group of syndromes associated with neurovascular, ophthalmological, overgrowth, and malignant complications. Here, we discover that extensive dermal melanocytosis and phakomatosis pigmentovascularis are associated with activating mutations in GNA11 and GNAQ, genes that encode Gα subunits of heterotrimeric G proteins. The mutations were det…

WT wild typeDNA Mutational AnalysisMolecular Sequence Datapostzygotic mutationsMutation MissenseSWS Sturge-Weber syndromeDermatologycesioflammeagermlineBiochemistrySkin DiseasesAnimals Genetically Modifiedg-proteinDNA deoxyribonucleic acidMongolian Spotoculodermal melanocytosis[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyGeneticsAnimalsHumansddc:610Phosphorylationchoroidal melanomaMolecular BiologyAllelesZebrafishdiseaseBase SequenceNeurocutaneous Syndromessturge-weberInfantCell Biologymongolian spotPPV phakomatosis pigmentovascularisGTP-Binding Protein alpha SubunitsHEK293 CellsPhenotypeMutationGTP-Binding Protein alpha Subunits Gq-G11Original Articleuveal melanoma[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologySignal Transduction
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WWOX-related encephalopathies: delineation of the phenotypical spectrum and emerging genotype-phenotype correlation

2014

International audience; BACKGROUND:Homozygous mutations in WWOX were reported in eight individuals of two families with autosomal recessive spinocerebellar ataxia type 12 and in two siblings with infantile epileptic encephalopathy (IEE), including one who deceased prior to DNA sampling.METHODS:By combining array comparative genomic hybridisation, targeted Sanger sequencing and next generation sequencing, we identified five further patients from four families with IEE due to biallelic alterations of WWOX.RESULTS:We identified eight deleterious WWOX alleles consisting in four deletions, a four base-pair frameshifting deletion, one missense and two nonsense mutations. Genotype-phenotype correl…

WWOXMicrocephaly[SDV]Life Sciences [q-bio]Nonsense mutationMutation MissenseBiology03 medical and health sciences0302 clinical medicineGeneticsmedicineHumansSpinocerebellar AtaxiasMissense mutationAlleleGenetics (clinical)infantile030304 developmental biologyGeneticsComparative Genomic Hybridization0303 health sciences[ SDV ] Life Sciences [q-bio]Tumor Suppressor ProteinsChromosomal fragile siteHigh-Throughput Nucleotide Sequencinggenotype/phenotype correlationsmedicine.diseaseNull allele3. Good healthPhenotypeWW Domain-Containing OxidoreductaseCodon Nonsenseintellectual disabilitySpinocerebellar ataxiaOxidoreductasesSpasms Infantilehigh throughput data mining030217 neurology & neurosurgeryJournal of Medical Genetics
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