Search results for " oxidation"

showing 10 items of 675 documents

Oxidative stress in patients with Alzheimer's disease: Effect of extracts of fermented papaya powder

2015

Brain tissue is particularly susceptible to oxidative stress (OS). Increased production of reactive oxygen species (ROS), reduced antioxidant systems, and decreased efficiency in repairing mechanisms have been linked to Alzheimer’s disease (AD). Postmortem studies in AD patients’ brains have shown oxidative damage markers (i.e., lipid peroxidation, protein oxidative damage, and glycoxidation). Fermented papaya (FPP, a product ofCarica papaya Linnfermentation with yeast) is a nutraceutical supplement with favorable effects on immunological, hematological, inflammatory, and OS parameters in chronic/degenerative diseases. We studied 40 patients (age 78.2 ± 1.1 years), 28 AD patients, and 12 co…

MaleAntioxidantSettore MED/09 - Medicina Internamedicine.medical_treatmentReview Articlemedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundchemistry.chemical_classificationAged 80 and overbiologyCaricaBrainBiochemistry8-Hydroxy-2'-DeoxyguanosineFemalePowdersCaricaAlzheimer's diseaseAntioxidantCase-Control StudieReactive Oxygen SpecieOxidation-Reductionlcsh:RB1-214Humanmedicine.medical_specialtyUrinary systemImmunologyPowderAlzheimer DiseaseInternal medicinemedicinelcsh:PathologyHumansAgedDietary SupplementReactive oxygen speciesCase-control studyDeoxyguanosineOxidative StrePlant PreparationCell Biologybiology.organism_classificationmedicine.diseaseAged; Aged 80 and over; Alzheimer Disease; Antioxidants; Brain; Carica; Case-Control Studies; Deoxyguanosine; Dietary Supplements; Female; Fermentation; Humans; Lipid Peroxidation; Male; Oxidation-Reduction; Oxygen; Plant Preparations; Powders; Reactive Oxygen Species; Oxidative Stress; Immunology; Cell BiologyOxygenOxidative StressEndocrinologychemistryCase-Control StudiesDietary SupplementsFermentationPlant PreparationsLipid PeroxidationReactive Oxygen SpeciesOxidative stress
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Application of hull, bur and leaf chestnut extracts on the shelf-life of beef patties stored under MAP: Evaluation of their impact on physicochemical…

2018

The impact of chestnut extracts (Castanea sativa) from leaf, bur and hull at different concentrations on the shelf-life of beef patties during 18 days of refrigerated storage at (2 ± 1 °C) was studied and compared to control and synthetic antioxidant (BHT) samples. Total phenolics and in vitro antioxidant capacity of extracts were evaluated by using DPPH, FRAP, reducing power and oil accelerated oxidation test (peroxide value, conjugated dienes, p-anisidine and fatty acid profile). The microbial spoilage, colour parameters, lipid oxidation and sensorial properties were used to assess antioxidant activity in beef patties. The highest total phenolic content was found in bur extracts (43.68 ± …

MaleAntioxidantTime FactorsDPPHmedicine.medical_treatmentFood spoilageAesculusColorShelf lifeAntioxidantschemistry.chemical_compound0404 agricultural biotechnologyLipid oxidationAnti-Infective AgentsRefrigerationFood PreservationmedicineAnimalsHumansFood sciencePeroxide valuechemistry.chemical_classificationDose-Response Relationship DrugChemistryPlant ExtractsFatty acidTaste Perception04 agricultural and veterinary sciencesAntimicrobialOlfactory Perception040401 food scienceLipidsCold TemperaturePlant LeavesSmellRed MeatFood StorageTasteOdorantsSeedsFood MicrobiologyFood PreservativesCattleFemaleLipid PeroxidationOxidation-ReductionFood ScienceFood research international (Ottawa, Ont.)
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Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome

2010

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome BACKGROUND & AIMS: Oxidative stress has a key role in atherosclerosis, cancer and other chronic diseases. Some bioactive compounds in nuts have been implicated in antioxidant activities. OBJECTIVE: We assessed how nut consumption affected several markers of oxidation and endothelial function (EF) in metabolic syndrome (MetS) patients. PATIENTS AND METHODS: A randomized, controlled, parallel feeding trial was conducted on 50 MetS adults who were recommended a healthy diet supplemented or not with 30 g of mixed nuts (Nut and Control groups, respectively) every day for 12 weeks. The plasma anti…

MaleAntioxidantmedicine.medical_treatmentantioxidant capacityIsoprostanesCritical Care and Intensive Care Medicinemedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundendothelial functionMedicineNutschemistry.chemical_classificationMetabolic SyndromeBioquímica y tecnologíaNutrition and Dieteticsdigestive oral and skin physiologyfood and beveragesArteriesMiddle Aged0261-5614Lipoproteins LDLBiochemistry and technologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineFatty Acids UnsaturatedPolyunsaturated fatty acidAdultmedicine.medical_specialtyEndotheliumAdolescentDNA damageEstrès oxidatiuFruita seca -- Aspectes nutritiusBioquímica i biotecnologiaYoung AdultLipid oxidationInternal medicineHumansAgedbusiness.industryDeoxyguanosineEndoteli vascularmedicine.diseaseDietOxidative StressEndocrinologychemistryDNA damageVascular ResistanceEndothelium VascularMetabolic syndromebusinessOxidative stressBiomarkers
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Redox state alteration modulates astrocyte glucuronidation.

2004

We have investigated the effects of mild oxidative conditions on drug-metabolizing enzyme activity in rat cultured astrocytes. These experimental conditions promoting an oxidative environment were obtained by short exposure to a low concentration of menadione (5 microM) for a short duration (15 min). This resulted in the rapid and transient production of reactive oxygen species (+130%), associated with a decrease in GSH cellular content (-24%), and an increase in total protein oxidation (+26%), but promoted neither PGE(2) nor NO production. This treatment induced a rapid and persistent decrease in astrocyte glucuronidation activities, which was totally prevented by N-acetyl-l-cysteine. Thes…

MaleCell SurvivalGlucuronidationApoptosisGlucuronatesOxidative phosphorylationmedicine.disease_causeProtein oxidationBiochemistryRedoxchemistry.chemical_compoundMenadionePhysiology (medical)CricetinaemedicineAnimalsProtein IsoformsRNA MessengerGlucuronosyltransferaseRats WistarPromoter Regions GeneticCells Culturedchemistry.chemical_classificationInflammationReactive oxygen speciesBase SequenceVitamin K 3GlutathioneHydrogen PeroxideMolecular biologyGlutathioneCell biologyRatschemistryAstrocytesFemaleReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree radical biologymedicine
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Mandarin Juice Improves the Antioxidant Status of Hypercholesterolemic Children

2008

Background: Oxidative stress has been linked to such degenerative diseases as atherosclerosis, and it has been suggested that increased dietary intake of antioxidants may reduce its progression. Objective: To determine the effect of mandarin juice consumption on biomarkers related to oxidative stress in hypercholesterolemic children. Materials and Methods: The diet of 48 children with plasma cholesterol >200 mg/dL and low-density lipoprotein cholesterol >130 mg/dL was supplemented for 28 days with 500 mL/day of pure (100%) mandarin juice (Citrus clementina Hort. ex Tan.). The composition of the mandarin juice was analyzed, and its antioxidant antiradical activity was evaluated in vitro. Mal…

MaleCitrusmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentHypercholesterolemiaNutritional StatusAscorbic AcidProtein oxidationmedicine.disease_causeAntioxidantsBeverageschemistry.chemical_compoundMalondialdehydeInternal medicinemedicineHumansVitamin EChildbusiness.industryCholesterolVitamin EGastroenterologyFree Radical ScavengersGlutathioneMalondialdehydeLipidsOxidative StressCholesterolEndocrinologychemistryDietary SupplementsPediatrics Perinatology and Child HealthFemaleLipid PeroxidationbusinessOxidation-ReductionBiomarkersOxidative stressLipoproteinJournal of Pediatric Gastroenterology & Nutrition
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Fatty acid oxidation and related gene expression in heart depleted of carnitine by mildronate treatment in the rat.

2004

The metabolic and genic effects induced by a 20-fold lowering of carnitine content in the heart were studied in mildronate-treated rats. In the perfused heart, the proportion of palmitate taken up then oxidized was 5-10% lower, while the triacylglycerol (TAG) formation was 100% greater than in controls. The treatment was shown to increase the maximal capacity of heart homogenates to oxidize palmitate, the mRNA level of carnitine palmitoyltransferase I (CPT-I) isoforms, the specific activity of CPT-I in subsarcolemmal mitochondria and the total carnitine content of isolated mitochondria. Concomitantly, the increased mRNA expression of lipoprotein lipase, fatty acid translocase and enzymes of…

MaleClinical BiochemistryPalmitic AcidBlood lipidsBiologyMitochondrionIn Vitro TechniquesMitochondria HeartOxygen ConsumptionCarnitinemedicineAnimalsCarnitineRNA MessengerRats WistarMolecular BiologyBeta oxidationHeart metabolismTriglycerideschemistry.chemical_classificationLipoprotein lipaseCarnitine O-PalmitoyltransferaseEsterificationMyocardiumFatty AcidsFatty acidBiological TransportCardiovascular AgentsCell BiologyGeneral MedicineRatsPerfusionLipoprotein LipasechemistryBiochemistryGene Expression RegulationCarnitine palmitoyltransferase IOxidation-Reductionmedicine.drugMethylhydrazinesMolecular and cellular biochemistry
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Hypertensive status and lipoprotein oxidation in an elderly population at high cardiovascular risk.

2008

BACKGROUND: In elderly individuals, hypertension is a main risk factor for cardiovascular disease and oxidative damage is increased. Our aim was to assess the relationship between the degree of in vivo low-density lipoprotein (oxLDL) oxidation and the hypertensive status in a elderly population at high cardiovascular risk. METHODS: Cross-sectional study with baseline data from the PREDIMED (PREvención con DIeta MEDiterránea) trial, an intervention study directed at testing the efficacy of the Mediterranean diet on the primary prevention of cardiovascular disease. Participants were 1,130 subjects at high cardiovascular risk aged 55-80. At baseline, in vivo circulating oxLDL was measured, and…

MaleCoronary DiseaseDiet MediterraneanSeverity of Illness IndexRisk groupsRisk FactorsMedicineAged 80 and overCoronary diseaseBioquímica y tecnologíaIncidence0895-7061Follow up studiesAge FactorsMiddle AgedPrognosisBiochemistry and technologyHypertensionChristian ministrylipids (amino acids peptides and proteins)HipertensióFemaleHypertensive StatusLypoproteinsmedicine.medical_specialtyLipoproteinsLipid peroxidationEnzyme-Linked Immunosorbent AssayBioquímica i biotecnologiaEnvironmental healthElderly populationInternal medicineInternal MedicineHumansLipoprotein oxidationLipoprotein OxidationAgedRetrospective Studiesbusiness.industryLipoproteïnes -- OxidacióUnited StatesPersones grans -- NutricióElderly PopulationOxidative StressEndocrinologyCross-Sectional StudiesMulticenter studyOxidative stressHypertension complicationsLipid PeroxidationbusinessOlive oilFollow-Up StudiesAmerican journal of hypertension
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Protein oxidation in chronic kidney disease.

2013

An imbalance between oxidative processes and antioxidant systems has been widely demonstrated in chronic kidney diseases (CKD). In this study we enrolled 26 healthy subjects, 27 patients with CKD on conservative treatment (CT-CKD) with various degrees of renal failure, and 31 CKD subjects in haemodialysis treatment (HD-CKD), evaluated before and after a standard haemodialysis session. In each group we measured protein carbonyl groups (PC) as an index of protein oxidation, lipid peroxidation (TBARS) and two plasma markers of leukocyte activation, elastase and myeloperoxidase (MPO). In CT-CKD subjects the PC level was significantly higher than in normal controls, and it was negatively correla…

MaleHEMODIALYSISmedicine.medical_specialtySettore MED/09 - Medicina InternaPhysiologyBIOMARKERSRenal functionurologic and male genital diseasesProtein oxidationThiobarbituric Acid Reactive SubstancesLipid peroxidationDiabetes Complicationschemistry.chemical_compoundCARBONYL STRESSMARKERSINFLAMMATIONGlycationRenal DialysisPhysiology (medical)Internal medicinemedicineTBARSHumansRenal Insufficiency ChronicPeroxidasebiologyPancreatic Elastasebusiness.industryNITRIC-OXIDE METABOLITESElastaseHematologyMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsOxidative StressEndocrinologychemistryMyeloperoxidaseNITRIC-OXIDE METABOLITES; CHRONIC-RENAL-FAILURE; CARBONYL STRESS; HEMODIALYSIS; BIOMARKERS; MARKERS; INFLAMMATIONImmunologybiology.proteinFemaleCHRONIC-RENAL-FAILURELipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidation-ReductionKidney diseaseClinical hemorheology and microcirculation
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Measurement of substrate-induced oxygen uptake during microsomal drug oxidation using a gold micro-electrode.

1975

1. A resin-coated gold micro-electrode has been used for polarographic determination of oxygen concentration in liver microsomal suspensions from phenobarbital-pretreated rats. 2. The rate of oxygen uptake on addition of an NADPH-regenerating system and the rate after addition of various substrates of the mixed function oxidase system were measured. The rate of oxygen uptake was faster in the presence of substrate than in the presence of NADPH alone. 3. Kinetic constants (Km and V max) for biphenyl, hexobarbital, ethylmorphine, naphthalene and SKF 525-A measured by this technique compare favourably with those obtained either by measurements of NADPH oxidation, or chemical measurements of su…

MaleHealth Toxicology and MutagenesisInorganic chemistryHexobarbitalNaphthalenesToxicologyBiochemistryOxygen ConsumptionmedicineAnimalsPharmacologyPolarographyMorphine DerivativesCell-Free SystemMorphineChemistryProadifenBiphenyl CompoundsSubstrate (chemistry)General MedicineNADPH oxidationEthylmorphineRatsKineticsHexobarbitalMixed Function OxidaseMicrosomes LiverLimiting oxygen concentrationGoldOxidoreductasesMicroelectrodesOxidation-ReductionDrug metabolismNADPmedicine.drugPolarographyXenobiotica; the fate of foreign compounds in biological systems
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A role for the peroxisomal 3-ketoacyl-CoA thiolase B enzyme in the control of PPARα-mediated upregulation of SREBP-2 target genes in the liver.: ThB …

2011

International audience; Peroxisomal 3-ketoacyl-CoA thiolase B (Thb) catalyzes the final step in the peroxisomal β-oxidation of straight-chain acyl-CoAs and is under the transcription control of the nuclear hormone receptor PPARα. PPARα binds to and is activated by the synthetic compound Wy14,643 (Wy). Here, we show that the magnitude of Wy-mediated induction of peroxisomal β-oxidation of radiolabeled (1-(14)C) palmitate was significantly reduced in mice deficient for Thb. In contrast, mitochondrial β-oxidation was unaltered in Thb(-/-) mice. Given that Wy-treatment induced Acox1 and MFP-1/-2 activity at a similar level in both genotypes, we concluded that the thiolase step alone was respons…

MaleMESH: HepatomegalyPalmitatesMESH : PyrimidinesMESH : Gene DeletionBiochemistryelement-binding proteinsMESH : Acetyl-CoA C-AcyltransferaseMiceMESH: Up-RegulationMESH: AnimalsMESH : Up-RegulationMESH: Lipid Metabolism0303 health sciencesMESH : Gene Expression RegulationThiolase030302 biochemistry & molecular biologyGeneral MedicineMESH : HepatomegalyUp-Regulationzellweger-syndromePeroxisome ProliferatorsMESH: Peroxisome ProliferatorsHepatomegalySterol Regulatory Element Binding Protein 2peroxisomal 3-ketoacyl-CoA thiolase BMESH: Mitochondria3-oxoacyl-coa thiolaseLathosterolfatty-acid oxidationrat-liverMESH: Sterol Regulatory Element Binding Protein 203 medical and health sciencesMESH : Sterol Regulatory Element Binding Protein 2HumansPPAR alphaMESH : Peroxisome Proliferators[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyPPARaVLAGMESH : Oxidation-ReductionFatty Acid Oxidation.MESH: HumansCholesterolMESH : HumanscholesterolLipid MetabolismMESH: PeroxisomesSterol regulatory element-binding proteinchemistryMESH: PyrimidinesCholesterol; Micro-array analysis; Peroxisomal 3-ketoacyl-CoA thiolase B; PPARα and SREBP-2; Wy14643Fatty Acid OxidationGene DeletionMESH: LiverMESH: Oxidation-ReductionMESH: Signal TransductionMESH: Mice KnockoutVoeding Metabolisme en Genomicachemistry.chemical_compoundMESH: CholesterolMESH : Lipid MetabolismWy14MESH : PalmitatesMESH: PPAR alphaMESH : CholesterolMice Knockoutneuronal migration643PeroxisomeAcetyl-CoA C-AcyltransferaseMESH: Gene Expression RegulationMetabolism and GenomicsMitochondriaLiverBiochemistryMicro-array analysisMetabolisme en GenomicaACOX1Nutrition Metabolism and GenomicsMESH : MitochondriaOxidation-ReductionSignal Transductionacyl-coa oxidasecholesterol-synthesisMESH : MaleMESH : PPAR alphaPeroxisome ProliferationPPARα and SREBP-2Biologybeta-oxidationVoedingproliferator-activated receptorsMESH : MicePeroxisomesAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMESH: Mice030304 developmental biologySCP2NutritionMESH : Signal TransductionMESH : LiverMESH: PalmitatesMESH: MalePyrimidinesMESH: Acetyl-CoA C-AcyltransferaseGene Expression RegulationMESH: Gene DeletionMESH : Mice KnockoutMESH : AnimalsMESH : Peroxisomes
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