Search results for " peptide"

showing 10 items of 2395 documents

Dietary cholic acid lowers plasma levels of mouse and human apolipoprotein A-I primarily via a transcriptional mechanism

2000

To induce dietary atherosclerosis in mice, high-fat/high-cholesterol (HF) diets are frequently supplemented with cholic acid (CA). This diet produces low plasma levels of high-density lipoprotein (HDL) and high levels of low-density lipoprotein (LDL). However, HF diets without any added CA, which more closely resemble human diets, increase levels of both HDL and LDL, suggesting that CA may be responsible for the lowering of HDL. Our aim was to examine the potential mechanism responsible for the lowering of HDL. Nontransgenic (NTg) C57BL mice and apoA-I-transgenic (apoAI-Tg) mice, with greatly increased basal apoA-I and HDL levels, were used. Mice were fed the following four diets: control (…

medicine.medical_specialtyBile acidmedicine.drug_classCholesterolResponse elementCholic acidnutritional and metabolic diseasesBiologyBiochemistrychemistry.chemical_compoundEndocrinologyHigh-density lipoproteinchemistryInternal medicineLow-density lipoproteinpolycyclic compoundsmedicinelipids (amino acids peptides and proteins)Hepatic lipaseLipoproteinEuropean Journal of Biochemistry
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Management of Dyslipidemia in the Metabolic Syndrome

2007

In order to characterize the metabolic syndrome it becomes necessary to establish a number of diagnostic criteria. Because of its impact on cardiovascular morbidity/mortality, considerable attention has been focussed on the dyslipidemia accompanying the metabolic syndrome. The aim of this review is to highlight the fundamental aspects of the pathophysiology, diagnosis, and the treatment of the metabolic syndrome dyslipidemia with recommendations to clinicians. The clinical expression of the metabolic syndrome dyslipidemia is characterized by hypertriglyceridemia and low levels of high-density lipoprotein-cholesterol (HDL-C). In addition, metabolic syndrome dyslipidemia is associated with hi…

medicine.medical_specialtyBioinformaticsClofibric Acidchemistry.chemical_compoundInsulin resistanceInternal medicineHyperlipidemiamedicineHumansPharmacology (medical)DyslipidemiasMetabolic Syndromemedicine.diagnostic_testCholesterolbusiness.industryCholesterol HDLHypertriglyceridemianutritional and metabolic diseasesGeneral Medicinemedicine.diseaseEndocrinologyPostprandialchemistrySpainPractice Guidelines as Topiclipids (amino acids peptides and proteins)Hydroxymethylglutaryl-CoA Reductase InhibitorsMetabolic syndromeCardiology and Cardiovascular MedicineLipid profilebusinessDyslipidemiaAmerican Journal of Cardiovascular Drugs
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Antiapoptotic effect of calcitonin gene-related peptide on oxidative stress-induced injury in H9c2 cardiomyocytes via the RAMP1/CRLR complex.

2005

Calcitonin gene-related peptide (CGRP) plays an important role in the mediation of protective effects observed in situations such as ischemic preconditioning in rat hearts. In this study, we investigated in H9c2 rat cardiomyoblasts if the protective effect of CGRP could be linked to an inhibitory effect on the apoptotic pathway. We also determined the specificity of observed effects by treatment with adrenomedullin (ADM) in stress conditions generated by 100 microM hydrogen peroxide. Using MTT assays, we demonstrate that a pretreatment with CGRP decreases by half the loss of cell viability induced by H(2)O(2). CGRP inhibits phosphatidylserine externalization, caspase 3 activation and DNA fr…

medicine.medical_specialtyCalcitonin Gene-Related PeptideCaspase 3DNA FragmentationCalcitonin gene-related peptideReceptor Activity-Modifying Protein 2Receptor Activity-Modifying Protein 3Receptor Activity-Modifying ProteinsCell LineReceptor Activity-Modifying Protein 1Internal medicinemedicineAnimalsMyocytes CardiacViability assayMolecular BiologyReceptor activity-modifying proteinintegumentary systemChemistryCalcitonin Receptor-Like ProteinIntracellular Signaling Peptides and ProteinsMembrane ProteinsReceptors CalcitoninPeptide FragmentsRatsAdrenomedullinOxidative StressEndocrinologyGene Expression RegulationRAMP2ApoptosisRAMP1Multiprotein ComplexesIschemic Preconditioning MyocardialCardiology and Cardiovascular MedicineMioticsSignal TransductionJournal of molecular and cellular cardiology
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Peptide neuroanatomy of adjuvant-induced arthritic inflammation in rat

1988

The influence of adjuvant-induced arthritis of the rat on central and peripheral peptide neuroanatomy was investigated by immunohistochemistry. The most striking feature of arthritic rats was the differential intensification of neuronal proenkephalin- and prodynorphin-related staining in dorsal horn. Changes were ipsilateral in monoarthritic and bilateral in polyarthritic rats as compared to controls. Opioid responsive neurons were target of substance P (SP) and calcitonin gene-related peptide (CGRP) fibers. Changes of SP and CGRP predominated in peripheral inflamed tissue and consisted of intensified immunostaining and an apparent sprouting of sensory fibers particularly around venules, in…

medicine.medical_specialtyCalcitonin Gene-Related PeptideImmunologyInflammationSubstance PSubstance PCalcitonin gene-related peptideToxicologychemistry.chemical_compoundNerve FibersNeuroimmune systemGanglia SpinalInternal medicinemedicineAnimalsPharmacology (medical)Protein PrecursorsSkinPharmacologybusiness.industryArthritisNeuropeptidesRats Inbred StrainsEnkephalinsArthritis ExperimentalImmunohistochemistryRatsProenkephalinEndocrinologyNociceptionSpinal CordchemistryCalcitoninmedicine.symptombusinessImmunostainingAgents and Actions
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Coexpression of vasoactive intestinal peptide, calcitonin gene-related peptide and substance P immunoreactivity in parasympathetic neurons of the rhe…

1995

Abstract By the use of light microscopic immunohistochemistry, the present study investigates whether substance P (SP) and calcitonin generelated peptide (CGRP), which are well documented neurotransmitter candidates in primary sensory fibers, are also expressed in parasympathetic neurons of the rhesus monkey lung. A combination of double fluorescence immunohistochemistry and staining of adjacent sections revealed triple coexistence of SP, CGRP and the cholinergic co-transmitter vasoactive intestinal peptide (VIP) in a large number of neuronal cell bodies in intrinsic peribronchial ganglia. In addition, there was co-localization of SP and CGRP in choline acetyltransferase (ChAT)-positive neu…

medicine.medical_specialtyCalcitonin Gene-Related PeptideVasoactive intestinal peptideNeuropeptideSubstance PSubstance PCalcitonin gene-related peptideBiologyCholine O-Acetyltransferasechemistry.chemical_compoundParasympathetic Nervous SystemInternal medicinemedicineAnimalsLungNeuronsGeneral NeuroscienceImmunohistochemistryMacaca mulattaCholine acetyltransferaseEndocrinologynervous systemchemistryFluorescent Antibody Technique DirectCalcitoninCholinergicAcetylcholineVasoactive Intestinal Peptidemedicine.drug
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Head-to-head comparison of contemporary heart failure risk scores.

2021

Altres ajuts: acords transformatius de la UAB Aims: Several heart failure (HF) web-based risk scores are currently used in clinical practice. Currently, we lack head-to-head comparison of the accuracy of risk scores. This study aimed to assess correlation and mortality prediction performance of Meta-Analysis Global Group in Chronic Heart Failure (MAGGIC-HF) risk score, which includes clinical variables + medications; Seattle Heart Failure Model (SHFM), which includes clinical variables + treatments + analytes; PARADIGM Risk of Events and Death in the Contemporary Treatment of Heart Failure (PREDICT-HF) and Barcelona Bio-Heart Failure (BCN-Bio-HF) risk calculator, which also include biomarke…

medicine.medical_specialtyCalibration (statistics)Head to headHeart failureSpearman's rank correlation coefficientRisk AssessmentCorrelationRisk FactorsInternal medicineNatriuretic Peptide BrainmedicineRisk modelsHumansOverall performanceMortalityNatriuretic PeptidesHeart FailureFramingham Risk Scorebusiness.industrymedicine.diseasePrognosisRisk predictionPeptide FragmentsClinical PracticeHeart failureCardiology and Cardiovascular MedicinebusinessBiomarkersEuropean journal of heart failureReferences
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Genetic bases of urinary albumin excretion and related traits in hypertension

2010

Epidemiological as well as animal studies have recognized the potential role of genetic factors in the development of microalbuminuria and related traits (renal insufficiency, end-stage renal disease and nephroangiosclerosis) in hypertension. To unravel genetic variants of susceptibility, candidate gene, linkage and genome wide scan analysis has been used. In spite of the great efforts that have been made in the field, sound knowledge about the major genetic variants causing the susceptibility to develop renal damage in hypertension is scarce, since many associations were not replicated or only showed association in a certain subgroup of patients. Looking initially at genes of the most impo…

medicine.medical_specialtyCandidate geneGenetic LinkagePhysiologyGenome-wide association studyDiseaseBioinformaticsGenomeRenin-Angiotensin SystemGenetic linkageInternal medicineInternal MedicinemedicineAlbuminuriaAnimalsHumansGenetic Predisposition to DiseaseNatriuretic PeptidesGenebusiness.industrymedicine.diseaseReceptors AdrenergicOxidative StressEndocrinologyHypertensionAlbuminuriaKallikreinsMicroalbuminuriamedicine.symptomCardiology and Cardiovascular MedicinebusinessGenome-Wide Association StudyJournal of Hypertension
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Hardwiring the Brain: Endocannabinoids Shape Neuronal Connectivity

2007

The roles of endocannabinoid signaling during central nervous system development are unknown. We report that CB 1 cannabinoid receptors (CB 1 Rs) are enriched in the axonal growth cones of γ-aminobutyric acid–containing (GABAergic) interneurons in the rodent cortex during late gestation. Endocannabinoids trigger CB 1 R internalization and elimination from filopodia and induce chemorepulsion and collapse of axonal growth cones of these GABAergic interneurons by activating RhoA. Similarly, endocannabinoids diminish the galvanotropism of Xenopus laevis spinal neurons. These findings, together with the impaired target selection of cortical GABAergic interneurons lacking CB 1 Rs, identify endoc…

medicine.medical_specialtyCannabinoid receptorGrowth ConesSynaptogenesisXenopus ProteinsBiologyRats Sprague-DawleyMiceXenopus laevisReceptor Cannabinoid CB1ChemorepulsionCell MovementInterneuronsInternal medicineCannabinoid Receptor ModulatorsmedicineAnimalsAxonGrowth coneCells CulturedIn Situ Hybridizationgamma-Aminobutyric AcidUltrasonographyCerebral CortexMicroscopy ConfocalMultidisciplinaryStem Cellsmusculoskeletal neural and ocular physiologyEndocannabinoid systemAxonsRatsMice Inbred C57BLEndocrinologymedicine.anatomical_structurenervous systemSynapsesGABAergiclipids (amino acids peptides and proteins)Axon guidanceNeuroscienceEndocannabinoidsSignal TransductionScience
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Exercise, the endocannabinoid system and metabolic health

2013

As obesity and associated metabolic disorders, such as type 2 diabetes and dyslipidemia, are becoming one of the most serious health problems worldwide, development of effective therapies is a high priority. In the search for treatments, the recently discovered endocannabinoid system (ECS) has begun to garner attention, and a wealth of research is now focusing on this unique neuromodulatory system named after the plant that led to its discovery. The ECS consists of G protein-coupled cannabinoid receptors (CB1 and CB2), their endogenous lipid-derived ligands (endocannabinoids, N-arachidonoylethanolamine, named anandamide (AEA) and 2-arachidonoylglycerol (2-AG)) and the enzymes for ligand syn…

medicine.medical_specialtyCannabinoid receptorbusiness.industryCalorie restrictionAdipose tissuePhysical Therapy Sports Therapy and RehabilitationType 2 diabetesmedicine.diseaseEndocannabinoid systemInsulin resistanceEndocrinologyInternal medicinemedicinelipids (amino acids peptides and proteins)Orthopedics and Sports MedicineReceptorbusinessDyslipidemiaJournal of Sport and Health Science
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Mucosa-dependent muscarinic liberation of prostaglandins from rat isolated trachea.

1995

1. The present study examined whether cholinoceptor stimulation modulates the release of arachidonic acid-derived mediators from rat isolate tracheae. 2. Tracheae were preincubated with [3H]-arachidonic acid and the outflow of 3H-compounds was determined. Acetylcholine and the muscarinic agonist, carbachol but not nicotine, increased the rate of tritium outflow maximally by about 30%. The M3 receptor-preferring antagonist rho-fluoro-hexahydrosiladiphenidol was more effective than pirenzepine and methoctramine in antagonizing the effect of acetylcholine. 3. High performance liquid chromatography analysis (methanol gradient) of the released 3H-compounds showed that one peak, co-eluting with […

medicine.medical_specialtyCarbacholAcetonitrilesMuscarinic AntagonistsIn Vitro TechniquesMuscarinic AgonistsMuscarinic agonistRats Sprague-Dawleychemistry.chemical_compoundPiperidinesInternal medicineMuscarinic acetylcholine receptormedicineMethoctramineAnimalsDrug InteractionsAcetylcholine receptorPharmacologyArachidonic AcidDose-Response Relationship DrugMuscarinic acetylcholine receptor M1PirenzepineAcetylcholineRatsTracheaEndocrinologychemistryProstaglandinslipids (amino acids peptides and proteins)FemaleAcetylcholinemedicine.drugResearch Article
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