Search results for " plaque"

showing 10 items of 233 documents

The Role of Chemokines in Alzheimer's Disease

2019

Objective: The most common multifactorial neurodegenerative disorder occurring in old age is Alzheimer’s disease. The neuropathological hallmarks of that disorder are amyloid plaques with the presence of β -amyloid aggregates, intraneuronal tau protein tangles, and chronic inflammation. Brain cells such as microglia and astrocytes are inflammatory cells associated with Alzheimer’s disease and involved in the production of inflammatory mediators, such as cytokines and chemokines. Chemokines consist of a large family of protein mediators with low molecular weight, which able to control the migration and residence of all immune cells. In pathological conditions, such as Alzheimer’s disease, c…

0301 basic medicineAgingChemokineAmyloidEndocrinology Diabetes and MetabolismTau protein030209 endocrinology & metabolismInflammation03 medical and health sciences0302 clinical medicineImmune systemAlzheimer DiseaseAmyloid precursor proteinAnimalsHumansImmunology and AllergyMedicineSenile plaquesInflammationbiologyMicrogliabusiness.industryBrainOxidative Stress030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinReceptors ChemokineChemokinesmedicine.symptombusinessEndocrine, Metabolic & Immune Disorders - Drug Targets
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Microglia in Alzheimer’s Disease: Activated, Dysfunctional or Degenerative

2018

Microglial activation has been considered a crucial player in the pathological process of multiple human neurodegenerative diseases. In some of these pathologies, such as Amyotrophic Lateral Sclerosis or Multiple Sclerosis, the immune system and microglial cells (as part of the cerebral immunity) play a central role. In other degenerative processes, such as Alzheimer’s disease (AD), the role of microglia is far to be elucidated. In this “mini-review” article, we briefly highlight our recent data comparing the microglial response between amyloidogenic transgenic models, such as APP/PS1 and AD patients. Since the AD pathology could display regional heterogeneity, we focus our work at the hipp…

0301 basic medicineAgingMini ReviewCognitive NeuroscienceAPP modelsmicrogliainflamationDegeneration (medical)Hippocampal formationlcsh:RC321-57103 medical and health sciences0302 clinical medicineImmune systemmedicineAmyotrophic lateral sclerosislcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMicrogliabusiness.industryMultiple sclerosisDentate gyrusmedicine.disease030104 developmental biologymedicine.anatomical_structureAbeta plaquesMicrogliaAlzheimer diseaseAlzheimer's diseasebusinessInflamationNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Aging Neuroscience
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Differential preservation of endogenous human and microbial DNA in dental calculus and dentin.

2018

AbstractDental calculus (calcified dental plaque) is prevalent in archaeological skeletal collections and is a rich source of oral microbiome and host-derived ancient biomolecules. Recently, it has been proposed that dental calculus may provide a more robust environment for DNA preservation than other skeletal remains, but this has not been systematically tested. In this study, shotgun-sequenced data from paired dental calculus and dentin samples from 48 globally distributed individuals are compared using a metagenomic approach. Overall, we find DNA from dental calculus is consistently more abundant and less contaminated than DNA from dentin. The majority of DNA in dental calculus is microb…

0301 basic medicineDNA BacterialMicrobial DNAPreservation Biologicallcsh:MedicineBiologyDental plaqueArticle03 medical and health scienceschemistry.chemical_compoundstomatognathic systemCalculusDentinmedicineHumansDental CalculusFragmentation (cell biology)lcsh:ScienceCromosomes humansMultidisciplinaryBacteriaCalculus (dental)Microbiotalcsh:Rmedicine.diseasestomatognathic diseases030104 developmental biologymedicine.anatomical_structurechemistryMetagenomicsDentinlcsh:QOral MicrobiomeMetagenomicsDNAScientific reports
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ADAM10 in Alzheimer's disease: Pharmacological modulation by natural compounds and its role as a peripheral marker.

2019

Abstract Alzheimer’s disease (AD) represents a global burden in the economics of healthcare systems. Amyloid-β (Aβ) peptides are formed by amyloid-β precursor protein (AβPP) cleavage, which can be processed by two pathways. The cleavage by the α-secretase A Disintegrin And Metalloprotease 10 (ADAM10) releases the soluble portion (sAβPPα) and prevents senile plaques. This pathway remains largely unknown and ignored, mainly regarding pharmacological approaches that may act via different signaling cascades and thus stimulate non-amyloidogenic cleavage through ADAM10. This review emphasizes the effects of natural compounds on ADAM10 modulation, which eventuates in a neuroprotective mechanism. M…

0301 basic medicineFarmacologiaADAM10DiseaseRM1-950Natural compoundsCleavage (embryo)NeuroprotectionCatechin03 medical and health sciencesADAM10 ProteinAmyloid beta-Protein Precursor0302 clinical medicineAlzheimer DiseaseDisintegrinHumansSenile plaquesPharmacological modulationPharmacologyMetalloproteinaseAmyloid beta-PeptidesbiologyChemistryPlant ExtractsADAM10ProteinsGinkgo bilobaMembrane ProteinsGeneral Medicineα-SecretaseAlzheimer's disease030104 developmental biologyMalaltia d'AlzheimerNeuroprotective Agents030220 oncology & carcinogenesisPharmaceuticalbiology.proteinTherapeutics. PharmacologyAmyloid Precursor Protein SecretasesNeuroscienceAlzheimer’s diseaseProteïnesBiomarkersBiomedicinepharmacotherapy = Biomedecinepharmacotherapie
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Expression of endogenous mouse APP modulates β-amyloid deposition in hAPP-transgenic mice

2017

Amyloid-β (Aβ) deposition is one of the hallmarks of the amyloid hypothesis in Alzheimer’s disease (AD). Mouse models using APP-transgene overexpression to generate amyloid plaques have shown to model only certain parts of the disease. The extent to which the data from mice can be transferred to man remains controversial. Several studies have shown convincing treatment results in reducing Aβ and enhancing cognition in mice but failed totally in human. One model-dependent factor has so far been almost completely neglected: the endogenous expression of mouse APP and its effects on the transgenic models and the readout for therapeutic approaches. Here, we report that hAPP-transgenic models of …

0301 basic medicineGenetically modified mouseMaleMurine amyloid-betaBACE1-ASMice TransgenicPlaque Amyloidlcsh:RC346-429Pathology and Forensic Medicine03 medical and health sciencesCellular and Molecular NeuroscienceAmyloid beta-Protein Precursor0302 clinical medicineMeningesAmyloid precursor proteinMedicineAnimalsHumansTransgenic miceSenile plaqueslcsh:Neurology. Diseases of the nervous systemNeuronsAmyloid beta-Peptidesbiologybusiness.industryAmyloidosisResearchP3 peptideBrainAmyloidosismedicine.diseasePeptide FragmentsBiochemistry of Alzheimer's diseaseAstrogliosisCell biologyMice Inbred C57BL030104 developmental biologyCaspasesAmyloid precursor proteinMutationbiology.proteinAbetaFemaleNeurology (clinical)businessNeuroscienceAlzheimer’s disease030217 neurology & neurosurgery
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Plasma phospholipid transfer protein (PLTP) modulates adaptive immune functions through alternation of T helper cell polarization

2016

International audience; Objective: Plasma phospholipid transfer protein (PLTP) is a key determinant of lipoprotein metabolism, and both animal and human studies converge to indicate that PLTP promotes atherogenesis and its thromboembolic complications. Moreover, it has recently been reported that PLTP modulates inflammation and immune responses. Although earlier studies from our group demonstrated that PLTP can modify macrophage activation, the implication of PLTP in the modulation of T-cell-mediated immune responses has never been investigated and was therefore addressed in the present study. Approach and results: In the present study, we demonstrated that PLTP deficiency in mice has a pro…

0301 basic medicineLymphocyteIpid Transfer ProteinAdaptive ImmunityCardiovascular-DiseaseT-Lymphocytes RegulatoryLipoprotein MetabolismLeukocyte CountPhospholipid transfer proteinPolarizationImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyHypersensitivity DelayedPhospholipid Transfer ProteinsCell PolarityCell DifferentiationT-Lymphocytes Helper-InducerT helper cellFlow CytometryAcquired immune systemCell biologyInfectious Diseasesmedicine.anatomical_structureEndothelial-CellsCytokines[SDV.IMM]Life Sciences [q-bio]/ImmunologyLymphocytemedicine.symptomResearch ArticleDensity-Lipoprotein[SDV.IMM] Life Sciences [q-bio]/ImmunologyHuman Atherosclerotic PlaquesT cellCirculating Interleukin-18ImmunologyT CellAntigen-Presenting CellsInflammationAcute Myocardial-InfarctionGATA3 Transcription FactorBiology03 medical and health sciencesImmune systemmedicineAnimalsAntigen-presenting cellDeficient MiceAlpha-TocopherolMice Inbred C57BL030104 developmental biologyImmunologyVitamin-ET-Box Domain ProteinsBiomarkersSpleen
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Cytotoxic activity of Holothuria tubulosa (Echinodermata) coelomocytes.

2017

Abstract The immune system of marine invertebrates, in particular that of holothurians, still requires further study. Our research showed that coelomocyte cells contained in the coelomic fluid of the sea cucumber, Holothuria tubulosa, are able to lyse, in vitro, red blood cells in rabbits and sheep. A plaque-forming assay showed spherule cells to be the effector cells, able to release cytotoxic molecules after xenogenic cell contact. The coelomocyte lysate supernatant, analysed by polyacrylamide gel electrophoresis overlay technique, using rabbit and sheep erythrocytes, showed two different haemolytic protein patterns: one calcium dependent and the other calcium independent. The fractions o…

0301 basic medicineLysisErythrocytesOverlay assayAquatic ScienceMicrobiologyLysis plaque assay03 medical and health sciencesSea cucumber0302 clinical medicineImmune systemLeukocytesEnvironmental ChemistryCytotoxic T cellAnimalsHolothuriaPolyacrylamide gel electrophoresisCoelomocyteSheepbiologyHolothuria tubulosaGeneral Medicinebiology.organism_classificationHolothuria tubulosaIn vitroImmunity InnateHaemolytic activity030104 developmental biologyBiochemistryElectrophoresis Polyacrylamide GelCoelomocyteRabbits030215 immunologyFishshellfish immunology
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Autoantibodies Profile in Matching CSF and Serum from AD and aMCI patients: Potential Pathogenic Role and Link to Oxidative Damage.

2015

Abstract Alzheimer disease (AD) is the most common form of dementia among the elderly and is characterized by progressive loss of memory and cognition. Amyloid-s-peptide (As) forms senile plaques, which, together with hyperphosphorylated tau-based neurofibrillary tangles, are the hallmarks of AD neuropathology. Evidence support the involvement of immune system in AD progression and current concepts regarding its pathogenesis include the participation of inflammatory and autoimmune components in the neurodegenerative process. Pathologically, immune system components have been detected in the brain, cerebrospinal fluid (CSF) and in serum of AD subjects and their trend of variation correlates …

0301 basic medicineMalePathologymedicine.medical_specialtyBlotting WesternNeuropathologyaged; aged 80 and over; alzheimer disease; autoantibodies; biomarkers; blotting western; cognitive dysfunction; female; frontal lobe; humans; male; mass spectrometry; oxidative stressmedicine.disease_causeMass SpectrometryAutoimmunity03 medical and health sciences0302 clinical medicineCerebrospinal fluidImmune systemAlzheimer DiseasemedicineDementiaHumansCognitive DysfunctionSenile plaquesAgedAutoantibodiesAged 80 and overAutoantibodymedicine.diseaseFrontal LobeOxidative Stress030104 developmental biologyNeurologyImmunologyFemaleNeurology (clinical)Alzheimer's diseasePsychology030217 neurology & neurosurgeryBiomarkersCurrent Alzheimer research
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The predictive value of microbiological findings on teeth, internal and external implant portions in clinical decision making

2017

International audience; Aim: The primary aim of this study was to evaluate 23 pathogens associated with peri-implantitis at inner part of implant connections, in peri-implant and periodontal pockets between patients suffering peri-implantitis and participants with healthy peri-implant tissues; the secondary aim was to estimate the predictive value of microbiological profile in patients wearing dental implants using data mining methods.Material and Methods: Fifty participants included in the present case─control study were scheduled for collection of plaque samples from the peri-implant pockets, internal connection, and periodontal pocket. Real-time polymerase chain reaction was performed to…

0301 basic medicineMalePeri-implantitisGingival and periodontal pocketAlveolar Bone LossDentistry0302 clinical medicineRadiography DentalMedicinePeri-implantitisperiodontitis[SDV.MHEP.RSOA] Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal system[SDV.MHEP.GEG] Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontology[SDV.MHEP.GEG]Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyMiddle AgedPredictive value3. Good healthDental Implantation[SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal systemFemaleOral SurgeryInfectionperi-implantitisAdultDecision treesClinical Decision-MakingDental PlaqueDental plaqueReal-Time Polymerase Chain Reaction03 medical and health sciencesHumansPeriodontal PocketPeriodontitisParvimonas micraData miningAgedPeriodontitisDental Implantsdecision treesbusiness.industryCase-control studydata mining; decision trees; infection; peri-implantitis; periodontitis030206 dentistrydata miningmedicine.diseaseinfection030104 developmental biologyCase-Control StudiesImplantbusinessTooth
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Development of an in vitro system to study oral biofilms in real time through impedance technology: validation and potential applications

2019

ABSTRACT Background and objectives: We have developed a standardized, easy-to-use in vitro model to study single- and multiple-species oral biofilms in real time through impedance technology, which elucidates the kinetics of biofilm formation in 96-well plates, without the requirement for any further manipulation. Design and Results: Using this system, biofilms of Streptococcus mutans appear to be sugar-dependent and highly resistant to amoxicilin, an antibiotic to which this oral pathogen is highly sensitive in a planktonic state. Saliva, tongue and dental plaque samples were also used as inocula to form multiple-species biofilms. DNA isolation and Illumina sequencing of the biofilms showe…

0301 basic medicineMicrobiology (medical)Fastidious organismSalivamultiple-species biofilmdental plaquemedicine.drug_classMicroorganismAntibioticslcsh:QR1-502real-timeDental plaquebiofilm dynamicslcsh:Microbiologylcsh:Infectious and parasitic diseasesMicrobiologyStreptococcus mutans03 medical and health sciences0302 clinical medicinetongueantibioticmedicinelcsh:RC109-216Dentistry (miscellaneous)PathogenbiologyOral biofilmsChemistryoral biofilmsBiofilm030206 dentistrybiochemical phenomena metabolism and nutritionmedicine.diseasebiology.organism_classificationStreptococcus mutansstreptococcus mutans030104 developmental biologyInfectious Diseasesin vitro modelimpedanceOriginal ArticleJournal of Oral Microbiology
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