Search results for " stem"

showing 10 items of 2170 documents

Contribution of sinusoidal endothelial liver cells to liver fibrosis: expression of transforming growth factor-beta 1 receptors and modulation of pla…

1993

Transforming growth factor-beta 1 is an important cytokine in the pathophysiology of liver fibrosis, stimulating the production of extracellular matrix. Whether this cytokine can also control the degradation of matrix proteins in liver cells has not been investigated. Because plasmin is an important protease for the degradation of matrix glycoproteins, we investigated whether sinusoidal endothelial liver cells could contribute to fibrosing liver disease through the modulation of plasmin-generating enzymes in response to transforming growth factor-beta 1. Sinusoidal endothelial cells from guinea pig liver were investigated in pure monolayer culture. Using 125I-labelled transforming growth fa…

PlasminGuinea PigsBiologyLiver Cirrhosis Experimental03 medical and health sciencesPlasminogen Activators0302 clinical medicineCell surface receptorTransforming Growth Factor betaPlasminogen Activator Inhibitor 1medicineAnimalsFibrinolysinCells Cultured030304 developmental biology0303 health sciencesHepatology3. Good healthCell biologyFibronectinEndothelial stem cellBiochemistryLiverTransforming growth factor beta 3Cell culturebiology.protein030211 gastroenterology & hepatologyFemaleEndothelium VascularPlasminogen activatorReceptors Transforming Growth Factor betamedicine.drugTransforming growth factorHepatology (Baltimore, Md.)
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Polycomb-like 2 Associates with PRC2 and Regulates Transcriptional Networks during Mouse Embryonic Stem Cell Self-Renewal and Differentiation

2010

SummaryPolycomb group (PcG) proteins are conserved epigenetic transcriptional repressors that control numerous developmental gene expression programs and have recently been implicated in modulating embryonic stem cell (ESC) fate. We identified the PcG protein PCL2 (polycomb-like 2) in a genome-wide screen for regulators of self-renewal and pluripotency and predicted that it would play an important role in mouse ESC-fate determination. Using multiple biochemical strategies, we provide evidence that PCL2 is a Polycomb Repressive Complex 2 (PRC2)-associated protein in mouse ESCs. Knockdown of Pcl2 in ESCs resulted in heightened self-renewal characteristics, defects in differentiation, and alte…

Pluripotent Stem CellsCellular differentiationGene regulatory networkDown-RegulationPolycomb-Group Proteinsmacromolecular substancesMethylationBiochemistryArticleCell LineHistonesSelf-RenewalMice03 medical and health sciences0302 clinical medicineEmbryonic Stem CellHistone methylationPolycomb-group proteinsGeneticsAnimalsGene Regulatory NetworksEpigeneticsInduced pluripotent stem cellEmbryonic Stem Cells030304 developmental biologyGenetics0303 health sciencesbiologyurogenital systemGene Expression ProfilingPolycomb Repressive Complex 2Cell DifferentiationCell BiologyCellular ReprogrammingSTEMCELLPRC2Embryonic stem cellRepressor ProteinsOncologyDifferentiation030220 oncology & carcinogenesisembryonic structuresbiology.proteinMolecular MedicineTranscriptional NetworkPRC2Genome-Wide Association StudyProtein BindingCell Stem Cell
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Colorectal Cancer Stem Cells: From the Crypt to the Clinic

2014

Since their first discovery, investigations of colorectal cancer stem cells (CSCs) have revealed some unexpected properties, including a high degree of heterogeneity and plasticity. By exploiting a combination of genetic, epigenetic, and microenvironmental factors, colorectal CSCs metastasize, resist chemotherapy, and continually adapt to a changing microenvironment, representing a formidable challenge to cancer eradication. Here, we review the current understanding of colorectal CSCs, including their origin, relationship to stem cells of the intestine, phenotypic characterization, and underlying regulatory mechanisms. We also discuss limitations to current preclinical models of colorectal …

Pluripotent Stem CellsColorectal cancerAnimals; Colonic Neoplasms; Colorectal Neoplasms; Disease Models Animal; Gene Expression Regulation Neoplastic; Humans; Intestines; Neoplastic Stem Cells; Pluripotent Stem Cells; Tumor EscapeCryptAnimals; Colonic Neoplasms; Colorectal Neoplasms; Disease Models Animal; Gene Expression Regulation Neoplastic; Humans; Intestines; Neoplastic Stem Cells; Pluripotent Stem Cells; Tumor Escape; Molecular Medicine; Genetics; Cell BiologyBiologySettore MED/04 - PATOLOGIA GENERALEmedicineGeneticsAnimalsHumansEpigeneticsRegulation of gene expressionNeoplasticAnimalCancerCell Biologymedicine.diseasePhenotypeGene Expression Regulation NeoplasticIntestinesDisease Models AnimalTumor EscapeGene Expression RegulationImmunologyColonic NeoplasmsDisease ModelsCancer researchNeoplastic Stem CellsMolecular MedicineTumor EscapeStem cellColorectal Neoplasmscolorectal cancer stem cells CSCsCell Stem Cell
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Upregulated acetylcholine synthesis during early differentiation in the embryonic stem cell line CGR8

2012

Stem cells are used to generate differentiated somatic cells including neuronal cells. Synthesis and release of acetylcholine, a neurotransmitter and widely expressed signaling molecule, were investigated in the murine embryonic stem cell line CGR8 during early differentiation, i.e. in the presence of leukemia inhibitory factor (LIF) to maintain pluripotency and in the absence of LIF to induce early differentiation. CGR8 cells express choline acetyltransferase (ChAT) as demonstrated by measurement of enzyme activity and substantial inhibition by bromoacetylcholine. Pluripotent CGR8 cells showed a ChAT activity of 250 pmol acetylcholine/mg/h, contained 1.1 pmol acetylcholine/10⁶ cells and re…

Pluripotent Stem CellsHomeobox protein NANOGSomatic cellGeneral NeuroscienceCell DifferentiationOct-4BiologyMolecular biologyCholine acetyltransferaseAcetylcholineCell LineCholine O-AcetyltransferaseUp-RegulationMiceCell culturemedicineAnimalsStem cellLeukemia inhibitory factorEmbryonic Stem CellsAcetylcholinemedicine.drugNeuroscience Letters
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Mouse embryonic stem cells are hypersensitive to apoptosis triggered by the DNA damage O(6)-methylguanine due to high E2F1 regulated mismatch repair.

2007

Exposure of stem cells to genotoxins may lead to embryonic lethality or teratogenic effects. This can be prevented by efficient DNA repair or by eliminating genetically damaged cells. Using undifferentiated mouse embryonic stem (ES) cells as a pluripotent model system, we compared ES cells with differentiated cells, with regard to apoptosis induction by alkylating agents forming the highly mutagenic and killing DNA adduct O(6)-methylguanine. Upon treatment with N-methyl-N'-nitro-N-nitrosoguanidine (MNNG), ES cells undergo apoptosis at much higher frequency than differentiated cells, although they express a high level of the repair protein O(6)-methylguanine-DNA methyltransferase (MGMT). Apo…

Pluripotent Stem CellsMethylnitronitrosoguanidineDNA ComplementaryGuanineDNA damageDNA repairCellular differentiationApoptosisBiologyDNA Mismatch RepairModels BiologicalDNA AdductsMiceO(6)-Methylguanine-DNA MethyltransferaseDNA adductAnimalsMolecular BiologyEmbryonic Stem CellsSwiss 3T3 CellsBase SequenceCell DifferentiationCell BiologyDNA MethylationFibroblastsEmbryonic stem cellMolecular biologyDNA-Binding ProteinsMutS Homolog 2 ProteinDNA methylationDNA mismatch repairStem cellE2F1 Transcription FactorDNA DamageCell death and differentiation
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Stem cells, cancer stem-like cells, and natural products.

2012

Somatic stem cells can be found in many rapidly regenerating tissues, e.g., the skin, gastrointestinal mucosa, and hematopoietic system, but are also present at low numbers in non-regenerative organs such as the heart and brain. In these organs, somatic stem cells aid in normal tissue homeostasis and repair after injury as well as self-renewal and the generation of specific progenitor cells during differentiation. Cancer stem-like cells are a small subpopulation of self-renewing cells that are able to proliferate upon appropriate stimulation and differentiate into heterogeneous lineages in tumors. Modulation of the behavior of normal tissue stem cells and cancer stem-like cells is an emergi…

Pluripotent Stem CellsPathologymedicine.medical_specialtyCell SurvivalStem cell theory of agingPharmaceutical ScienceClinical uses of mesenchymal stem cellsTretinoinBiologyAnalytical ChemistryCancer stem cellNeoplasmsDrug DiscoverymedicineHumansCell LineageProgenitor cellEmbryonic Stem CellsStem cell transplantation for articular cartilage repairCell ProliferationPharmacologyBiological ProductsOrganic ChemistryCell DifferentiationCell Cycle CheckpointsAntineoplastic Agents PhytogenicCell biologyComplementary and alternative medicineAmniotic epithelial cellsNeoplastic Stem CellsMolecular MedicineStem cellAdult stem cellSignal TransductionPlanta medica
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mRNA-based therapeutics — developing a new class of drugs

2014

In vitro transcribed (IVT) mRNA has recently come into focus as a potential new drug class to deliver genetic information. Such synthetic mRNA can be engineered to transiently express proteins by structurally resembling natural mRNA. Advances in addressing the inherent challenges of this drug class, particularly related to controlling the translational efficacy and immunogenicity of the IVTmRNA, provide the basis for a broad range of potential applications. mRNA-based cancer immunotherapies and infectious disease vaccines have entered clinical development. Meanwhile, emerging novel approaches include in vivo delivery of IVT mRNA to replace or supplement proteins, IVT mRNA-based generation o…

Pluripotent Stem CellsPharmacologyDrugImmunogenicitymedia_common.quotation_subjectProteinsGeneral MedicineComputational biologyBiologyPharmacologyGenome engineeringDrug Delivery SystemsDrug classProtein replacement therapyDrug developmentInfectious disease (medical specialty)Drug DesignDrug DiscoveryAnimalsHumansImmunotherapyRNA MessengerInduced pluripotent stem cellmedia_commonNature Reviews Drug Discovery
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TRIC: an automated alignment strategy for reproducible protein quantification in targeted proteomics

2016

Nature Methods, 13 (9)

Pluripotent Stem CellsProteomics0301 basic medicineAnalyteStreptococcus pyogenesSoftware toolQuantitative proteomicsProteomic analysisComputational biologyBiologyProteome informaticsProteomicsBioinformaticsBiochemistryArticleMass Spectrometry03 medical and health sciencesSequence Analysis ProteinProtein methodsHumansProtein PrecursorsHuman Induced Pluripotent Stem CellsMolecular BiologyElectronic Data ProcessingReproducibility of ResultsCell BiologyMass spectrometricTargeted proteomics030104 developmental biologyProteolysissense organsPeptidesSequence AlignmentAlgorithmsSoftwareBiotechnologyNature Methods
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In Vitro Generation of Pancreatic Endocrine Cells from Human Adult Fibroblast-Like Limbal Stem Cells

2012

Stem cells might provide unlimited supply of transplantable cells for β-cell replacement therapy in diabetes. The human limbus is a highly specialized region hosting a well-recognized population of epithelial stem cells, which sustain the continuous renewal of the cornea, and the recently identified stromal fibroblast-like stem cells (f-LSCs), with apparent broader plasticity. However, the lack of specific molecular markers for the identification of the multipotent limbal subpopulation has so far limited the investigation of their differentiation potential. In this study we show that the human limbus contains uncommitted cells that could be potentially harnessed for the treatment of diabete…

Pluripotent Stem CellsStromal cellCellular differentiationPopulationBiomedical Engineeringlcsh:MedicineEnteroendocrine cellLimbus CorneaeBiologyLimbus; β-CellsSettore MED/13 - EndocrinologiaLimbuInsulin-Secreting CellsInsulin SecretionDiabetes MellitusHumansInsulineducationInduced pluripotent stem cellCells CulturedProinsulinTransplantationeducation.field_of_studyDiabeteslcsh:RCell DifferentiationCell Biologyβ-CellsCell biologyAdult Stem CellsStem cellFibroblast-like stem cellsBiomarkersAdult stem cellCell Transplantation
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Gata4 Blocks Somatic Cell Reprogramming By Directly Repressing Nanog

2012

Abstract Somatic cells can be reprogrammed to induced pluripotent stem (iPS) cells by ectopic expression of the four factors Oct4, Klf4, Sox2, and Myc. Here, we investigated the role of Gata4 in the reprogramming process and present evidence for a negative role of this family of transcription factors in the induction of pluripotency. Coexpression of Gata4 with Oct4, Klf4, and Sox2 with or without Myc in mouse embryonic fibroblasts greatly impaired reprogramming and endogenous Nanog expression. The lack of Nanog upregulation was associated with a blockade in the transition from the initiation phase of reprogramming to the full pluripotent state characteristic of iPS cells. Addition of Nanog …

Pluripotent Stem CellsTranscriptional ActivationHomeobox protein NANOGChromatin ImmunoprecipitationTranscription GeneticRex1Kruppel-Like Transcription FactorsDown-RegulationElectrophoretic Mobility Shift AssayBiologyCell LineProto-Oncogene Proteins c-mycKruppel-Like Factor 4MiceSOX2AnimalsRNA MessengerRNA Small InterferingInduced pluripotent stem cellEmbryonic Stem Cellsreproductive and urinary physiologyHomeodomain ProteinsSOXB1 Transcription FactorsNanog Homeobox ProteinCell DifferentiationNanog Homeobox ProteinCell BiologyCellular ReprogrammingEmbryonic stem cellGATA4 Transcription FactorKLF4embryonic structuresHepatocyte Nuclear Factor 3-betaCancer researchMolecular MedicineRNA Interferencebiological phenomena cell phenomena and immunityOctamer Transcription Factor-3ReprogrammingDevelopmental BiologyStem Cells
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