Search results for " type 2"

showing 10 items of 761 documents

Trans-limb embolization for treatment of Type 2 endoleak post EVAR: Case report

2021

Introduction Type 2 endoleaks (T2EL) occur after 10%–25% of endovascular abdominal aortic aneurysm repairs and increase the risk factor of endograft repair failure and rupture. Herein we report a case of endovascular treatment of T2EL where we performed a trans-limb embolization. Presentation of case A 63-years-old male previously treated for AAA with endovascular aortic aneurysms repair (EVAR), showed an angio-CT scan followup with a type 2 endoleak fed from inferior mesenteric artery (AMI) with growth of AAA greater of 1 cm than preoperative CT-scan and increase of chronic lumbar pain. Due to high risk of rupture was performed a trans-limb embolization with complete sealing. The 6 months …

medicine.medical_specialtyEndoleak type 2business.industrymedicine.medical_treatmentCoil embolizationmedicine.diseaseSize increaseInferior mesenteric arteryAneurysmSettore MED/22 - Chirurgia VascolareAbdominal aortic aneurysmSurgeryAneurysm ruptureLumbarAneurysmmedicine.arteryCase reportcardiovascular systemMedicineSurgerycardiovascular diseasesEmbolizationRisk factorbusiness
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Angiotensin II induces leukocyte-endothelial cell interactions in vivo via AT(1) and AT(2) receptor-mediated P-selectin upregulation.

2000

Background —Angiotensin II (Ang II) plays a critical role in the development of vascular lesions in hypertension, atherosclerosis, and several renal diseases. Because Ang II may contribute to the leukocyte recruitment associated with these pathological states, the aim of the present study was to assess the role of Ang II in leukocyte–endothelial cell interactions in vivo. Methods and Results —Intravital microscopy of the rat mesenteric postcapillary venules was used. Sixty minutes of superfusion with 1 nmol/L Ang II induced a significant increase in leukocyte rolling flux (83.8±20.7 versus 16.4±3.1 cells/min), adhesion (11.4±1.0 versus 0.8±0.5 cells/100 μm), and emigration (4.0±0.7 versus …

medicine.medical_specialtyEndotheliumPyridinesLeukocyte RollingCell CommunicationReceptor Angiotensin Type 2LosartanReceptor Angiotensin Type 1Rats Sprague-DawleyDownregulation and upregulationPhysiology (medical)Internal medicineCromolyn SodiummedicineLeukocytesAnimalsEndotheliumReceptorAngiotensin II receptor type 1Receptors Angiotensinbusiness.industryAngiotensin IIImidazolesFlow CytometryAngiotensin IIRatsUp-RegulationEndothelial stem cellP-Selectinmedicine.anatomical_structureEndocrinologyLosartanCardiology and Cardiovascular Medicinebusinessmedicine.drugCirculation
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Cardiometabolic non-response to aerobic exercise: Identifying subclinical ischaemic coronary disease

2019

Sin financiación 5.864 JCR (2019) Q1, 18/138 Cardiac & Cardiovascular Systems 1.459 SJR (2019) Q1, 58/362 Cardiology and Cardiovascular Medicine; Q2, 36/104 Epidemiology No data IDR 2019 UEM

medicine.medical_specialtyEpidemiologyEnfermedad cardiovascularCardiologyMEDLINECardiometabolic responseCoronary Artery DiseaseCoronary diseaseCoronary artery diseaseInternal medicineDiabetes mellitusmedicineHumansAerobic exerciseExerciseCardiometabolic response aerobic exercise coronary disease.Subclinical infectionMetabolismobusiness.industrymedicine.diseaseaerobic exerciseDiabetes Mellitus Type 2coronary disease.Cardiovascular DiseasesCardiologyCardiopatía coronariaCardiology and Cardiovascular MedicinebusinessEuropean Journal of Preventive Cardiology
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Type 2 diabetes mellitus and osteoarthritis

2019

Objectives: Type 2 diabetes mellitus (T2DM) and osteoarthritis (OA) are common diseases that frequently co-exist, along with overweight/obesity. While the mechanical impact of excess body weight on joints may explain lower limb OA, we sought to explore whether T2DM is linked to OA outside of excess weight and whether T2DM may play a role in OA pathophysiology. The consequence of T2DM on OA outcomes is a question of research interest. Methods: We conducted a critical review of the literature to explore the association between T2DM and OA, whether any association is site-specific for OA, and whether the presence of T2DM impacts on OA outcomes. We also reviewed the literature to assess the saf…

medicine.medical_specialtyEvidence-based practicetype 2 diabetes mellituendocrine system diseasesMedicinaOsteoarthritisOverweightPathophysiologyArticle03 medical and health sciences0302 clinical medicineInsulin resistanceRheumatologyInternal medicineOsteoarthritisType 2 diabetes mellitusMedicineHumans030212 general & internal medicineObesityRisk factor030203 arthritis & rheumatologyddc:616[SDV.MHEP.EM] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.RSOA] Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal systembusiness.industryType 2 Diabetes Mellitusnutritional and metabolic diseases[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismmedicine.diseaseObesity3. Good healthInstitutional repositoryAnesthesiology and Pain MedicineDiabetes Mellitus Type 2[SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal systemDisease Progressionosteoarthritimedicine.symptomInsulin ResistanceSafetybusiness
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Role of exercise-induced hepatokines in metabolic disorders.

2019

International audience; The health-promoting effects of physical activity to prevent and treat metabolic disorders are numerous. However, the underlying molecular mechanisms are not yet completely deciphered. In recent years, studies have referred to the liver as an endocrine organ, since it releases specific proteins called hepatokines. Some of these hepatokines are involved in whole body metabolic homeostasis and are theorized to participate in the development of metabolic disease. In this regard, the present review describes the role of Fibroblast Growth Factor 21, Fetuin-A, Angiopoietin-like protein 4, and Follistatin in metabolic disease and their production in response to acute exerci…

medicine.medical_specialtyFGF21PhysiologyEndocrinology Diabetes and MetabolismPhysical activity030209 endocrinology & metabolism03 medical and health sciences0302 clinical medicineMetabolic DiseasesRegular exerciseNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineEndocrine systemMedicineAnimalsHumansObesityMetabolic diseaseBeneficial effectsExercise030304 developmental biology0303 health sciencesbiologybusiness.industry[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthEndocrinologyDiabetes Mellitus Type 2Liverbiology.proteinCytokinesInsulin ResistanceWhole bodybusinessFollistatinAmerican journal of physiology. Endocrinology and metabolism
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Hyperuricaemia and gout in cardiovascular, metabolic and kidney disease

2020

During the last century, there has been an increasing prevalence of hyperuricaemia noted in many populations. While uric acid is usually discussed in the context of gout, hyperuricaemia is also associated with hypertension, chronic kidney disease, hypertriglyceridaemia, obesity, atherosclerotic heart disease, metabolic syndrome, and type 2 diabetes. Here we review the connection between hyperuricaemia and cardiovascular, kidney and metabolic diseases. Contrary to the popular view that uric acid is an inert metabolite of purine metabolism, recent studies suggest serum uric acid may have a variety of pro-inflammatory, pro-oxidative and vasoconstrictive actions that may contribute to cardiomet…

medicine.medical_specialtyGoutHeart diseaseAllopurinolAllopurinolHyperuricemiaType 2 diabetes030204 cardiovascular system & hematologyurologic and male genital diseasesCoronary artery disease03 medical and health scienceschemistry.chemical_compoundFebuxostat0302 clinical medicineInternal medicineInternal MedicinemedicineHumans030212 general & internal medicineMetabolic SyndromeType 2 diabetes.business.industrynutritional and metabolic diseasesmedicine.diseaseUric AcidGoutDiabetes Mellitus Type 2chemistryHypertensionCardiologyUric acidMetabolic syndromebusinessHyperuricaemiaKidney diseasemedicine.drugEuropean Journal of Internal Medicine
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Disturbed Lipid Metabolism in Diabetic Patients with Manifest Coronary Artery Disease Is Associated with Enhanced Inflammation

2021

Background: Diabetic vasculopathy plays an important role in the pathophysiology of coronary artery disease (CAD) with oxidative stress as a strong mediator. This study aims to elucidate the underlying pathomechanisms of diabetic cardiac vasculopathy leading to coronary disease with an emphasis on the role of oxidative stress. Therefore, novel insights into antioxidant pathways might contribute to new strategies in the treatment and prevention of diabetic CAD. Methods: In 20 patients with insulin-dependent or non-insulin dependent diabetes mellitus (IDDM/NIDDM) and 39 non-diabetic (CTR) patients, myocardial markers of oxidative stress, vasoactive proteins, endothelial nitric oxide synthase …

medicine.medical_specialtyHealth Toxicology and MutagenesisInflammationmedicine.disease_causeArticleCoronary artery diseaseEnosDiabetes mellitusInternal medicinemedicineHumansoxidative stressbiologybusiness.industrydyslipidemiaPublic Health Environmental and Occupational HealthRLipid metabolismLipid Metabolismmedicine.diseasebiology.organism_classificationHeme oxygenaseEndocrinologynutritionDiabetes Mellitus Type 2inflammationdiabetes mellitusMedicinemedicine.symptombusinesschronic diseaseDiabetic AngiopathiesOxidative stressDyslipidemiacoronary artery diseaseInternational Journal of Environmental Research and Public Health
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Lysosomal trafficking in rat cardiac myocytes.

1990

By immunolabeling of cryosections, we have characterized in rat cardiac myocytes the cation-independent mannose-6-phosphate receptor (MPR), a lysosomal membrane glycoprotein, lgp120, and a lysosomal enzyme, MEP (homologous to cathepsin L). Most of the MPR label was located in large membrane-filled structures (MPR structures) in large clusters of mitochondria adjacent to but distinct from the Golgi complex. Lpg120 and MEP showed typical lysosomal localization throughout the cell, often associated with regions that appeared to contain autophagosome-like structures. In addition, MEP and lgp120 co-localized within MPR structures. MEP and MPR were localized inside the lumen of MPR structures. M…

medicine.medical_specialtyHistologyCathepsin LImmunoblottingFluorescent Antibody TechniqueReceptors Cell SurfaceMitochondrionMitochondria HeartReceptor IGF Type 2Cathepsin LImmunolabelingsymbols.namesakeAntigens CDLysosomal-Associated Membrane Protein 1Internal medicineLysosomeEndopeptidasesmedicineAnimalsFrozen SectionsMyocyteReceptorchemistry.chemical_classificationMembrane GlycoproteinsbiologyMyocardiumLysosome-Associated Membrane GlycoproteinsIntracellular MembranesGolgi apparatusCathepsinsRatsCell biologyCysteine EndopeptidasesMicroscopy ElectronEndocrinologymedicine.anatomical_structureAnimals NewbornLiverchemistrybiology.proteinsymbolsCattleAnatomyLysosomesGlycoproteinJournal of Histochemistry & Cytochemistry
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Impaired border zone formation and adverse remodeling after reperfused myocardial infarction in cannabinoid CB2 receptor deficient mice.

2014

Abstract Aims Reperfusion of myocardial infarction is associated with inflammatory reaction and subsequent myocardial remodeling with a rapid scar formation in mice. The cannabinoid receptor CB2 has been associated with cardioprotection and regulation of macrophage function. We investigated its role in remodeling of reperfused infarction. Main methods One hour LAD-occlusion was followed by reperfusion over 6 h and 1, 3 and 7 days in wild-type C57/BL6J (WT) and CB2 receptor-deficient (Cnr2 −/− ) mice (n = 8/group). Hearts were processed for functional, morphological and mRNA/protein analysis, and tissue concentration of endocannabinoids was determined using liquid chromatography-multiple rea…

medicine.medical_specialtyIschemiaMyocardial InfarctionInfarctionMyocardial Reperfusion InjuryGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceInternal medicinemedicineCannabinoid receptor type 2AnimalsMyocytes CardiacMyocardial infarctionGeneral Pharmacology Toxicology and PharmaceuticsCardioprotectionInflammationMice KnockoutbiologyChemistryMyocardiumTenascin CHemodynamicsGranulation tissueGeneral Medicinemedicine.diseaseEndocannabinoid systemMice Inbred C57BLEndocrinologymedicine.anatomical_structureCardiologybiology.proteinGranulation TissueCytokinesLife sciences
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Association of ACACB polymorphisms with obesity and diabetes

2011

El pdf del artículo es la versión pre-print.-- et al.

medicine.medical_specialtyLinkage disequilibriumEndocrinology Diabetes and MetabolismPopulationSingle-nucleotide polymorphismType 2 diabetesBiologyBiochemistryPolymorphism Single NucleotideLinkage DisequilibriumCohort StudiesEndocrinologyInternal medicineGeneticsmedicineHumansObesityAlleleeducationMolecular BiologyAllelesGenetic Association StudiesAgedGeneticsACACBAged 80 and overeducation.field_of_studyHaplotypeAcetyl-CoA carboxylaseType 2 diabetesMiddle Agedmedicine.diseasePostmenopauseEndocrinologyDiabetes Mellitus Type 2HaplotypesACACBCase-Control StudiesFemalePolymorphismsAcetyl-CoA Carboxylase
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