Search results for " viruses"

showing 10 items of 230 documents

Detection of tick-borne encephalitis virus in I. ricinus ticks collected from autumn migratory birds in Latvia.

2014

Birds have a potential of spreading ticks via bird migration routes. In this study, we screened 170 ticks removed during autumn 2010 from 55 birds belonging to 10 species for the presence of tick-borne encephalitis virus (TBEV). In total, TBEV RNA was detected in 14% of I. ricinus tick samples obtained from different birds species. The results of this study indicate the possible role of migrating birds in the dispersal of TBEV-infected ticks along the southward migration route.

Bird migrationBiologyTickMicrobiologyVirusEncephalitis Viruses Tick-BorneBirdsZoonosesparasitic diseasesmedicineAnimalsHumansIxodesBird DiseasesRicinusbacterial infections and mycosesmedicine.diseasebiology.organism_classificationVirologyLatviaTick-borne encephalitis virusInfectious DiseasesInsect ScienceBiological dispersalRNA ViralParasitologyAnimal MigrationArachnid VectorsSeasonsEncephalitisEncephalitis Tick-BorneTicks and tick-borne diseases
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GIV noroviruses and other enteric viruses in bivalves

2011

The impact of sewage discharge on the microbiological quality of shellfish is well known. Bivalves filter large volumes of water as part of their feeding activities, and thus accumulate and concentrate different types of pathogens from human fecal pollution. The aim of the study was to evaluate the presence, in these molluscs, of six different groups of enteric viruses: norovirus (genogroups I, II, and GIV), adenovirus, enterovirus, astrovirus, hepatitis A virus, and hepatitis E virus. The present study is the first to document the presence of GIV noroviruses in shellfish.

Bivalves norovirus GIV enteric viruses
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A Ciliary Motility Index for Activity Measurement in Cell Cultures With Respiratory Syncytial Virus

2018

[EN] Background: The respiratory epithelium is frequently infected by the respiratory syncytial virus, resulting in inflammation, a reduction in cilia activity and an increase in the production of mucus. Methods: In this study, an automatic method has been proposed to characterize the ciliary motility from cell cultures by means of a motility index using a dense optical flow algorithm. This method allows us to determine the ciliary beat frequency (CBF) together with a ciliary motility index of the cells in the cultures. The object of this analysis is to automatically distinguish between normal and infected cells in a culture. Results: The method was applied in 2 stages. It was concluded fro…

BronchiInflammationRespiratory syncytial virusVirusMicrobiologyTECNOLOGIA ELECTRONICA03 medical and health sciences0302 clinical medicineCell MovementMotility indexImage Processing Computer-AssistedHumansCiliary motility: Cell culturesImmunology and AllergyMedicineCiliaRespiratory system030223 otorhinolaryngologyCells Culturedbusiness.industryCell cultures [Ciliary motility]CiliumOptical ImagingCiliary beat frequencyEpithelial CellsGeneral MedicineCiliary motilityhumanitiesRespiratory Syncytial VirusesActivity measurements030228 respiratory systemOtorhinolaryngologyCell cultureFISICA APLICADARespiratory epitheliumRespiratory epitheliumCiliary beat patternmedicine.symptomDense optical flowbusinessMATEMATICA APLICADAAlgorithms
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Effect of female genital schistosomiasis and anti-schistosomal treatment on monocytes, CD4+ T-cells and CCR5 expression in the female genital tract

2014

Published version of an article from the journal: PLoS One. Also available from the publisher: http://dx.doi.org/10.1371/journal.pone.0098593 BACKGROUND: Schistosoma haematobium is a waterborne parasite that may cause female genital schistosomiasis (FGS), characterized by genital mucosal lesions. There is clinical and epidemiological evidence for a relationship between FGS and HIV. We investigated the impact of FGS on HIV target cell density and expression of the HIV co-receptor CCR5 in blood and cervical cytobrush samples. Furthermore we evaluated the effect of anti-schistosomal treatment on these cell populations. DESIGN: The study followed a case-control design with post treatment follow…

CD4-Positive T-LymphocytesViral DiseasesGynecologic InfectionsVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Tropical medicine: 761Gene Expressionlcsh:MedicineGlobal HealthMonocytesPraziquantelWhite Blood CellsImmunodeficiency VirusesAnimal CellsMedicine and Health SciencesSchistosomiasisPublic and Occupational Healthlcsh:ScienceT CellsCoinfectionObstetrics and GynecologyGenitalia FemaleAIDSInfectious DiseasesPhenotypeMedical MicrobiologyHelminth InfectionsViral PathogensSchistosoma haematobiumFemaleCellular TypesResearch ArticleNeglected Tropical DiseasesAdultAdolescentReceptors CCR5Immune CellsUrologyImmunologySexually Transmitted DiseasesMicrobiologyImmunophenotypingYoung AdultParasitic DiseasesAnimalsHumansMicrobial PathogensBlood CellsGenitourinary Infectionslcsh:RBiology and Life SciencesHIVCell BiologyTropical DiseasesCase-Control StudiesWomen's HealthClinical Immunologylcsh:QGenital Diseases Female
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TP53 mutations and hepatocellular carcinoma: insights into the etiology and pathogenesis of liver cancer.

2007

Hepatocellular carcinoma (HCC) is one of the most common malignancies worldwide and the major risk factors include chronic infections with the hepatitis B (HBV) or C (HCV) virus, and exposure to dietary aflatoxin B(1) (AFB(1)) or alcohol consumption. Multiple genetic and epigenetic changes are involved in the molecular pathogenesis of HCC, for example, somatic mutations in the p53 tumor suppressor gene (TP53) and the activation of the WNT signal transduction pathway. AFB(1) frequently induces G:C to T:A transversions at the third base in codon 249 of TP53 and cooperates with HBV in causing p53 mutations in HCC. The detection of TP53 mutant DNA in plasma is a biomarker of both AFB(1) exposur…

Cancer ResearchAflatoxin B1Carcinoma HepatocellularTumor suppressor geneDNA damageDNA repairBiologymedicine.disease_causeHepatitis VirusesGeneticsmedicineHumansGenetic Predisposition to DiseaseEpigeneticsMolecular BiologyGeneHepatitis ChronicIncidenceLiver Neoplasmsmedicine.diseaseVirologydigestive system diseasesHBxMutagenesisHepatocellular carcinomaMutationCancer researchTumor Suppressor Protein p53CarcinogenesisOncogene
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The effect of genetic complementation on the fitness and diversity of viruses spreading as collective infectious units

2019

Viruses can spread collectively using different types of structures such as extracellular vesicles, virion aggregates, polyploid capsids, occlusion bodies, and even cells that accumulate virions at their surface, such as bacteria and dendritic cells. Despite the mounting evidence for collective spread, its implications for viral fitness and diversity remain poorly understood. It has been postulated that, by increasing the cellular multiplicity of infection, collective spread could enable mutually beneficial interactions among different viral genetic variants. One such interaction is genetic complementation, whereby deleterious mutations carried by different genomes are compensated. Here, we…

Cancer ResearchMutation rateViral diversityEvolutionPopulationViral transmissionGenome ViralBiologyVirus ReplicationGenomeEvolution Molecular03 medical and health sciencesMultiplicity of infectionPolyploidVirologyeducation030304 developmental biologyGenetics0303 health scienceseducation.field_of_study030306 microbiologyVirionDefective VirusesGenetic VariationDendritic cellGenetic complementationMutation AccumulationModels TheoreticalCollective spread3. Good healthComplementationInfectious DiseasesMutationGenetic FitnessVirus Research
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Experimental evolution of an oncolytic vesicular stomatitis virus with increased selectivity for p53-deficient cells

2014

Experimental evolution has been used for various biotechnological applications including protein and microbial cell engineering, but less commonly in the field of oncolytic virotherapy. Here, we sought to adapt a rapidly evolving RNA virus to cells deficient for the tumor suppressor gene p53, a hallmark of cancer cells. To achieve this goal, we established four independent evolution lines of the vesicular stomatitis virus (VSV) in p53-knockout mouse embryonic fibroblasts (p53-/- MEFs) under conditions favoring the action of natural selection. We found that some evolved viruses showed increased fitness and cytotoxicity in p53-/- cells but not in isogenic p53+/+ cells, indicating gene-specifi…

Cancer TreatmentVirus OncolíticosProtein EngineeringMiceMedicine and Health SciencesMacromolecular EngineeringMice KnockoutOncolytic VirotherapyMultidisciplinaryQProteína p53 Supresora de TumorRNeoplasias de la Mama3. Good healthOncolytic VirusesOncologyVesicular stomatitis virusColonic NeoplasmsMedicineFemaleVesicular StomatitisResearch ArticleBiotechnologyDirected EvolutionEvolutionary ProcessesTumor suppressor geneScienceBioengineeringBreast NeoplasmsBiologyMicrobiologyViral EvolutionVirusVesicular StomatitisVirologyCell Line TumorGeneticsAnimalsHumansEvolutionary BiologyNeoplasias del ColonBiology and Life SciencesRNA virusVesiculovirusbiology.organism_classificationVirologyOrganismal EvolutionOncolytic virusAnimal Models of InfectionArtificial SelectionSynthetic BioengineeringViruses and CancerCell cultureMicrobial EvolutionCancer cellCancer researchDirected Molecular EvolutionTumor Suppressor Protein p53
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Evolution of RNA virus in spatially structured heterogeneous environments

2003

A hallmark of the infectious cycle for many RNA viruses parasitizing multicellular hosts is the need to invade and successfully replicate in tissues that comprise a variety of cell types. Thus, multicellular hosts represent a heterogeneous environment to evolving viral populations. To understand viral adaptation to multicellular hosts, we took a double approach. First, we developed a mathematical model that served to make predictions concerning the dynamics of viral populations evolving in heterogeneous environments. Second, the predictions were tested by evolving vesicular stomatitis virus in vitro on a spatially structured environment formed by three different cell types. In the absence o…

Cell typeeducation.field_of_studyPopulation DynamicsPopulationAdaptation BiologicalRNARNA virusEnvironmentIn Vitro TechniquesModels TheoreticalBiologybiology.organism_classificationBiological EvolutionVirologyMulticellular organismEvolutionary biologyVesicular stomatitis virusViral evolutionRNA VirusesAdaptationeducationEcology Evolution Behavior and SystematicsJournal of Evolutionary Biology
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Collective Viral Spread Mediated by Virion Aggregates Promotes the Evolution of Defective Interfering Particles

2020

Recent insights have revealed that viruses use a highly diverse set of strategies to release multiple viral genomes into the same target cells, allowing the emergence of beneficial, but also detrimental, interactions among viruses inside infected cells. This has prompted interest among microbial ecologists and evolutionary biologists in studying how collective dispersal impacts the outcome of viral infections. Here, we have used vesicular stomatitis virus as a model system to study the evolutionary implications of collective dissemination mediated by viral aggregates, since this virus can spontaneously aggregate in the presence of saliva. We find that saliva-driven aggregation has a dual ef…

Cell typevirusesGene ExpressionEcological and Evolutionary ScienceGenome ViralBiologyVirus ReplicationMicrobiologyDeep sequencingVirusCell Linedefective interfering particles03 medical and health sciencesMultiplicity of infectionGenes ReporterVirologyAnimalsHumansexperimental evolutioncollective infectious unitssocial evolution030304 developmental biologyInfectivity0303 health sciencesExperimental evolution030306 microbiologyVirionDefective VirusesVesiculovirusbiology.organism_classificationBiological EvolutionVirologyQR1-5023. Good healthVirus DiseasesVesicular stomatitis virusBiological dispersalGenetic Fitnessvesicular stomatitis virusResearch ArticlemBio
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Roles of a conserved proline in the internal fusion peptide of Ebola glycoprotein

2004

AbstractThe structural determinants underlying the functionality of viral internal fusion peptides (IFPs) are not well understood. We have compared EBOwt (GAAIGLAWIPYFGPAAE), representing the IFP of the Ebola fusion protein GP, and EBOmut (GAAIGLAWIPYFGRAAE) derived from a non-functional mutant with conserved Pro537 substituted by Arg. P537R substitution did not abrogate peptide-membrane association, but interfered with the ability to induce bilayer destabilization. Structural determinations suggest that Pro537 is required to preserve a membrane-perturbing local conformation in apolar environments.

Circular dichroismEbola glycoproteinProtein insertion into membranesProlinePeptide conformationMutantMolecular Sequence DataBiophysicsBiochemistrySendai viruschemistry.chemical_compoundStructural BiologyGeneticsProlineAmino Acid SequenceMolecular BiologyPeptide sequencePOPCchemistry.chemical_classificationChemistryProteïnes de membranaCell BiologyEbolavirusFusion proteinPeptide FragmentsPeptide ConformationViral fusion peptideBiochemistryAvian Sarcoma VirusesLiposomesHIV-1PèptidsGlycoproteinPeptide–lipid interactionViral Fusion ProteinsFEBS Letters
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