Search results for " vivo"

showing 10 items of 1661 documents

Implanted neonatal human dermal fibroblasts influence the recruitment of endothelial cells in mice

2012

The vascularization of new tissue within a reasonable time is a crucial prerequisite for the success of different cell- and material-based strategies. Considering that angiogenesis is a multi-step process involving humoral and cellular regulatory components, only in vivo assays provide the adequate information about vessel formation and the recruitment of endothelial cells. The present study aimed to investigate if neonatal human dermal fibroblasts could influence in vivo neovascularization. Results obtained showed that fibroblasts were able to recruit endothelial cells to vascularize the implanted matrix, which was further colonized by murine functional blood vessels after one week. The ve…

CD31MalePathologymedicine.medical_specialtyAngiogenesisCell TransplantationBiomedical EngineeringCD34Medicine (miscellaneous)Neovascularization PhysiologicInflammationAntigens CD34BiologyNitric OxideRegenerative MedicineBiomaterialsNeovascularizationHemoglobinsMiceTissue engineeringMicroscopy Electron TransmissionIn vivoReportmedicineAnimalsHumansRegenerationSkinInflammationMatrigelNeovascularization PathologicTissue EngineeringEndothelial CellsGeneral MedicineFibroblastsMice Inbred C57BLPlatelet Endothelial Cell Adhesion Molecule-1Drug CombinationsPhenotypeProteoglycansCollagenLamininmedicine.symptom
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Antibodies to PECAM-1 and glucocorticoids reduce leukocyte adhesion in adjuvant arthritis of the rat knee synovium in vivo.

2002

Objective and design: To demonstrate the effect of monoclonal antibodies to the adhesion-molecule PECAM-1 (CD31) and of prednisolone on leukocyte adhesion in rat adjuvant arthritis.¶Material: Adjuvant arthritis was induced in male CD®-rats (five groups of n=6) 18 days prior to measurements.¶Treatment: Mouse-monoclonal antibody to rat CD-31 at 200 μg/kg or prednisolone at 24 mg/kg were administered i.v. 15 minutes prior to measurements.¶Methods: Venules within the intact rat-knee synovium were focused by confocal laser scanning microscopy. Numbers of rolling and adherent leukocytes were assessed in vivo.¶Results: Induction of arthritis significantly increased rolling and adherent leukocytes …

CD31Malemedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentPrednisoloneImmunologyAnti-Inflammatory AgentsArthritisMonoclonal antibodyLeukocyte CountIn vivoInternal medicineSynovial FluidmedicineCell AdhesionLeukocytesAnimalsAntibodies BlockingGlucocorticoidsPharmacologyMicroscopy Confocalbusiness.industryAntibodies Monoclonalmedicine.diseaseArthritis ExperimentalRheumatologyRatsPlatelet Endothelial Cell Adhesion Molecule-1Rheumatoid arthritisImmunologyPrednisoloneJointsbusinessAdjuvantmedicine.drugInflammation research : official journal of the European Histamine Research Society ... [et al.]
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Intestinal Scavenger Receptors Are Involved in Vitamin K 1 Absorption

2014

International audience; Vitamin K-1 (phylloquinone) intestinal absorption is thought to be mediated by a carrier protein that still remains to be identified. Apical transport of vitamin K-1 was examined using Caco-2 TC-7 cell monolayers as a model of human intestinal epithelium and in transfected HEK cells. Phylloquinone uptake was then measured ex vivo using mouse intestinal explants. Finally, vitamin K-1 absorption was compared between wild-type mice and mice overexpressing scavenger receptor class B type I (SR-BI) in the intestine and mice deficient in cluster determinant 36 (CD36). Phylloquinone uptake by Caco-2 cells was saturable and was significantly impaired by co-incubation with al…

CD36 Antigens030309 nutrition & dietetics[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionCD36medicine.medical_treatmentBiochemistryIntestinal absorptionchemistry.chemical_compoundMiceVitamin EHUMAN PLASMACAROTENOIDSComputingMilieux_MISCELLANEOUSMicelles0303 health sciencesbiologyCELL-LINESR-BIVitamin K 1Scavenger Receptors Class BCD36 DEFICIENCYPostprandial PeriodIntestinal epitheliumLipidsCholesterolVitaminmedicine.medical_specialtyPHYLLOQUINONE VITAMIN-K-103 medical and health sciencesInternal medicinemedicineB TYPE-I;SR-BI;PHYLLOQUINONE VITAMIN-K-1;MENAQUINONE-4 VITAMIN-K-2;CD36 DEFICIENCY;HUMAN PLASMA;CELL-LINE;TRANSPORT;CACO-2;CAROTENOIDSAnimalsHumansScavenger receptorMolecular BiologyMENAQUINONE-4 VITAMIN-K-2030304 developmental biologyVitamin ECell MembraneCACO-2Cell BiologyTRANSPORT[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologyEnterocytesHEK293 CellschemistryIntestinal AbsorptionCaco-2B TYPE-Ibiology.proteinCaco-2 Cells[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEx vivo
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CD36 is involved in lycopene and lutein uptake by adipocytes and adipose tissue cultures

2011

International audience; Scope: Carotenoids are mainly stored in adipose tissue. However, nothing is known regarding the uptake of carotenoids by adipocytes. Thus, our study explored the mechanism by which lycopene and lutein, two major human plasma carotenoids, are transported. Methods and results: CD36 was a putative candidate for this uptake, 3T3-L1 cells were treated with sulfosuccinimidyl oleate, a CD36-specific inhibitor. sulfosuccinimidyl oleate-treated cells showed a significant decrease in both lycopene and lutein uptake as compared to control cells. Their uptake was also decreased by partial inhibition of CD36 expression using siRNA, whereas the overexpression of CD36 in Cos-1 cell…

CD36 AntigensMaleLutein030309 nutrition & dieteticsCD36[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionLYCOPENEAdipose tissueOleic Acidschemistry.chemical_compoundMiceChlorocebus aethiopsRNA Small InterferingCAROTENOIDSCarotenoidComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationMice KnockoutGENE CD360303 health sciencesbiologyCD 36food and beveragesLycopene3. Good healthADIPOCYTESADIPOSE TISSUEBiochemistryCOS CellsRNA InterferenceBiotechnologyAdipose tissue macrophagesAdipose Tissue WhiteSuccinimides03 medical and health sciencesOrgan Culture Techniques3T3-L1 CellsTRANSPORTEUR BIOLOGIQUEparasitic diseasesAnimalsHumans030304 developmental biologyBiological Transport[SDV.AEN] Life Sciences [q-bio]/Food and NutritionGLYCOPROTEINRchemistryLUTEINbiology.protein[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEx vivoFood ScienceExplant culture
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Fatty Acid Transporter CD36 Mediates Hypothalamic Effect of Fatty Acids on Food Intake in Rats

2013

Subject Areas: carotid arteries; emulsions; fatty acids; gene expression; heparin; hypothalamus; neurons; oxidation.; International audience; Variations in plasma fatty acid (FA) concentrations are detected by FA sensing neurons in specific brain areas such as the hypothalamus. These neurons play a physiological role in the control of food intake and the regulation of hepatic glucose production. Le Foll et al. previously showed in vitro that at least 50% of the FA sensing in ventromedial hypothalamic (VMH) neurons is attributable to the interaction of long chain FA with FA translocase/CD36 (CD36). The present work assessed whether in vivo effects of hypothalamic FA sensing might be partly m…

CD36 AntigensMaleMicrodialysismedicine.medical_specialty[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyCD36HypothalamusGene Expressionlcsh:MedicineModels BiologicalEating03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivoInternal medicinemedicineAnimalslcsh:SciencePhospholipids030304 developmental biology2. Zero hungerchemistry.chemical_classification0303 health sciencesMultidisciplinaryTriglyceridebiologyFatty Acidslcsh:RNeurosciences[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyFatty acidFeeding BehaviorFatty Acid Transport ProteinsRatsSoybean OilTriacsin CEndocrinologychemistryHypothalamus[ SDV.NEU.NB ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyNeurons and Cognitionbiology.proteinEmulsionslcsh:QProto-Oncogene Proteins c-fos030217 neurology & neurosurgeryEtomoxirResearch ArticlePLoS ONE
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Adoptive transfer of ex vivo expanded SARS‐CoV‐2‐specific cytotoxic lymphocytes: A viable strategy for COVID‐19 immunosuppressed patients?

2021

Cellular and humoral response to acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2) infections is on focus of research. We evaluate herein the feasibility of expanding virus‐specific T cells (VST) against SARS‐CoV‐2 ex vivo through a standard protocol proven effective for other viruses. The experiment was performed in three different donors' scenarios: (a) SARS‐CoV‐2 asymptomatic infection/negative serology, (b) SARS‐CoV‐2 symptomatic infection/positive serology, and (c) no history of SARS‐CoV‐2 infection/negative serology. We were able to obtain an expanded VST product from donors 1 and 2 (1.6x and 1.8x increase of baseline VST count, respectively) consisting in CD3 + cells (80.3% and 6…

CD4-Positive T-LymphocytesAdoptive cell transferviruses030230 surgerymedicine.disease_causevirus-specific T cellsAsymptomaticSARS‐CoV‐2Serology03 medical and health sciences0302 clinical medicineCOVID‐19medicineCytotoxic T cellHumansRespiratory systemthird‐party donorsCoronavirusTransplantationbusiness.industrySARS-CoV-2COVID-19Original Articlesvirus‐specific T cellsAdoptive Transferlymphocyte expansionrespiratory virusInfectious DiseasesImmunologyRespiratory virus030211 gastroenterology & hepatologyOriginal Articlethird-party donorsmedicine.symptombusinessadoptive immunotherapyEx vivo
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Cross-recognition of a myelin peptide by CD8+ T cells in the CNS is not sufficient to promote neuronal damage.

2015

Multiple sclerosis (MS) is an inflammatory disease of the CNS thought to be driven by CNS-specific T lymphocytes. Although CD8+T cells are frequently found in multiple sclerosis lesions, their distinct role remains controversial because direct signs of cytotoxicity have not been confirmedin vivo. In the present work, we determined that murine ovalbumin-transgenic (OT-1) CD8+T cells recognize the myelin peptide myelin oligodendrocyte glycoprotein 40–54 (MOG40–54) bothin vitroandin vivo. The aim of this study was to investigate whether such cross-recognizing CD8+T cells are capable of inducing CNS damagein vivo. Using intravital two-photon microscopy in the mouse model of multiple sclerosis, …

CD4-Positive T-LymphocytesCentral Nervous SystemMaleEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisAutoimmunityMice TransgenicCD8-Positive T-Lymphocytesmedicine.disease_causeMyelin oligodendrocyte glycoproteinMyelinMiceIn vivomedicineCytotoxic T cellAnimalsCells CulturedCell ProliferationbiologyCell DeathGeneral NeuroscienceMultiple sclerosisArticlesmedicine.diseaseMolecular mimicrymedicine.anatomical_structureImmunologyNerve Degenerationbiology.proteinFemaleMyelin-Oligodendrocyte GlycoproteinCD8Intravital microscopyThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Reconstitution of CD8 T cells is essential for the prevention of multiple-organ cytomegalovirus histopathology after bone marrow transplantation.

1998

Cytomegalovirus (CMV) infection in the period of temporary immunodeficiency after haematoablative treatment and bone marrow transplantation (BMT) is associated with a risk of graft failure and multiple-organ CMV disease. The efficacy of immune system reconstitution is decisive for the prevention of CMV pathogenesis after BMT. Previous data in murine model systems have documented a redundancy in the immune effector mechanisms controlling CMV. CD8 T cells proved to be relevant but not irreplaceable as antiviral effectors. Specifically, in a state of long-term in vivo depletion of the CD8 T-cell subset, CD4 T cells were educed to become deputy effectors controlling CMV by a mechanism involving…

CD4-Positive T-LymphocytesCongenital cytomegalovirus infectionCytomegalovirusGraft vs Host DiseaseCD8-Positive T-LymphocytesBiologyVirus ReplicationLymphocyte DepletionPathogenesisMiceImmune systemRisk FactorsIn vivoVirologymedicineAnimalsHumansCytotoxic T cellImmunodeficiencyBone Marrow TransplantationMice Inbred BALB CEffectorvirus diseasesmedicine.diseaseVirologyDisease Models AnimalTransplantation IsogeneicCytomegalovirus InfectionsImmunologyCD8Journal of General Virology
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Mage-3 and influenza-matrix peptide-specific cytotoxic T cells are inducible in terminal stage HLA-A2.1+ melanoma patients by mature monocyte-derived…

2000

Abstract Dendritic cell (DC) vaccination, albeit still in an early stage, is a promising strategy to induce immunity to cancer. We explored whether DC can expand Ag-specific CD8+ T cells even in far-advanced stage IV melanoma patients. We found that three to five biweekly vaccinations of mature, monocyte-derived DC (three vaccinations of 6 × 106 s.c. followed by two i.v. ones of 6 and 12 × 106, respectively) pulsed with Mage-3A2.1 tumor and influenza matrix A2.1-positive control peptides as well as the recall Ag tetanus toxoid (in three of eight patients) generated in all eight patients Ag-specific effector CD8+ T cells that were detectable in blood directly ex vivo. This is the first time …

CD4-Positive T-LymphocytesCytotoxicity Immunologicmedicine.medical_treatmentInjections SubcutaneousImmunologyImmunization SecondaryEpitopes T-LymphocyteCD8-Positive T-LymphocytesLymphocyte ActivationCancer VaccinesMonocytesViral Matrix ProteinsAntigens NeoplasmTetanus ToxoidImmunology and AllergyMedicineCytotoxic T cellHumansMelanomaCells Culturedbusiness.industryMelanomaToxoidCell DifferentiationDendritic cellDendritic Cellsmedicine.diseaseNeoplasm ProteinsImmunizationImmunologyInjections IntravenousIntercellular Signaling Peptides and ProteinsbusinessPeptidesAdjuvantCD8Ex vivoT-Lymphocytes Cytotoxic
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CD4 monoclonal antibody VIT4 in human alloimmune response in vitro and in vivo.

1996

In the present report the immunosuppressive effects of the murine anti-human CD4 monoclonal antibody (mAb) VIT4 on human alloimmune response in vitro were analyzed. Moreover, the antibody was tested for its activity to prolong allograft survival in seven patients with steroid-refractory allograft rejection. VIT4 inhibited the proliferative response to alloantigens in the mixed lymphocyte reaction (MLR) in a dose-dependent manner. At concentrations of 1 and 10 micrograms/ml VIT4 blocked MLR by 55 +/- 11% and 77 +/- 1%, respectively. Also alloantigen-specific proliferation of in vitro- generated memory T cells was dose-dependently reduced to 23 +/- 1% at a VIT4 concentration of 100 micrograms…

CD4-Positive T-LymphocytesGraft Rejectionmedicine.drug_classImmunologyDose-Response Relationship ImmunologicPilot ProjectsPharmacologyMonoclonal antibodyMiceIn vivoT-Lymphocyte SubsetsmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansCells CulturedImmunosuppression Therapybiologybusiness.industryAntibodies MonoclonalHematologyMixed lymphocyte reactionKidney TransplantationIn vitroCTL*Cell cultureImmunologybiology.proteinPancreas TransplantationAntibodyLymphocyte Culture Test MixedbusinessImmunologic MemoryT-Lymphocytes CytotoxicImmunobiology
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