Search results for "212-2"

showing 9 items of 9 documents

Adolescent Exposure to the Synthetic Cannabinoid WIN 55212-2 Modifies Cocaine Withdrawal Symptoms in Adult Mice

2017

Chronic cannabinoid consumption is an increasingly common behavior among teenagers and has been shown to cause long-lasting neurobehavioral alterations. Besides, it has been demonstrated that cocaine addiction in adulthood is highly correlated with cannabis abuse during adolescence. Cocaine consumption and subsequent abstinence from it can cause psychiatric symptoms, such as psychosis, cognitive impairment, anxiety, and depression. The aim of the present research was to study the consequences of adolescent exposure to cannabis on the psychiatric-like effects promoted by cocaine withdrawal in adult mice. We pre-treated juvenile mice with the cannabinoid CB1 receptor agonist WIN 55212-2 (WIN)…

MalecannabisCannabinoid receptormedicine.medical_treatment0302 clinical medicineCocaineDopamine Uptake InhibitorsReceptor Cannabinoid CB1Spectroscopymedia_commoncocaine withdrawalBehavior Animalbiologyadolescence; cannabis; WIN 55212-2; cocaine withdrawal; miceGeneral MedicineSubstance Withdrawal SyndromeComputer Science ApplicationsHindlimb SuspensionElevated plus mazePsychosismedicine.medical_specialtymiceMorpholinesmedia_common.quotation_subjectNaphthalenesArticleCatalysisInorganic ChemistryCocaine-Related Disorders03 medical and health sciencesMemoryInternal medicinemedicineAnimalsWIN 55212-2Physical and Theoretical ChemistryPsychiatryMolecular BiologyCannabinoid Receptor Agonistsbusiness.industryAddictionOrganic ChemistryAbstinencemedicine.diseasebiology.organism_classificationBenzoxazines030227 psychiatryEndocrinologyAnxiogenicExploratory BehavioradolescenceCannabisCannabinoidbusiness030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Prenatal exposure to the CB1 receptor agonist WIN 55,212-2 causes learning disruption associated with impaired cortical NMDA receptor function and em…

2005

The aim of this study was to investigate whether prenatal exposure to the cannabinoid CB1 receptor agonist WIN 55,212-2 (WIN) at a daily dose devoid of overt signs of toxicity and/or gross malformations (0.5 mg/kg, gestation days 5-20), influences cortical glutamatergic neurotransmission, learning and emotional reactivity in rat offspring. Basal and K+-evoked extracellular glutamate levels were significantly lower in cortical cell cultures obtained from pups exposed to WIN during gestation with respect to those measured in cultures obtained from neonates born from vehicle-treated dams. The addition of NMDA to cortical cell cultures from neonates born from vehicle-treated dams concentration-…

MaleMarijuana AbuseCannabinoid receptoractive avoidance behaviour; basal and K+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationEmotionsReceptor Cannabinoid CB1Pregnancyactive avoidance behaviourWIN 55212-2Cells CulturedCerebral CortexBehavior AnimalGlutamate receptorBraincortical cell culturesCalcium Channel Blockersactive avoidance behaviour; basal and k plus -evoked glutamate levels; basal and k+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationPrenatal Exposure Delayed EffectsChloratesNMDA receptorbasal and K+-evoked glutamate levelsFemaleMicrotubule-Associated Proteinsmedicine.drugAgonistmedicine.medical_specialtyOffspringmedicine.drug_classCognitive NeuroscienceMorpholinesGlutamic Acidmaternal marijuana consumptionNeurotransmissionBiologyNaphthalenesReceptors N-Methyl-D-AspartateCellular and Molecular NeuroscienceGlutamatergicInternal medicinemedicineAvoidance LearningAnimalsRats WistarBenzoxazinesRatsultrasonic vocalizationEndocrinologyAnimals Newbornhoming behaviourVocalization AnimalExtracellular SpaceNeuroscience
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Involvement of TRPV1 channels in the activity of the cannabinoid WIN 55,212-2 in an acute rat model of temporal lobe epilepsy

2016

The exogenous cannabinoid agonist WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4-morpholinylmethyl) pyrrolo[1,2,3-de]-1,4-benzoxazin-6-Yl]-1-naphthalenylmethanone (WIN), has revealed to play a role on modulating the hyperexcitability phenomena in the hippocampus. Cannabinoid-mediated mechanisms of neuroprotection have recently been found to imply the modulation of transient receptor potential vanilloid 1 (TRPV1), a cationic channel subfamily that regulate synaptic excitation. In our study, we assessed the influence of pharmacological manipulation of TRPV1 function, alone and on WIN antiepileptic activity, in the Maximal Dentate Activation (MDA) acute model of temporal lobe epilepsy. Our r…

Male0301 basic medicineAgonistCannabinoid Receptor Modulatorsmedicine.drug_classMorpholinesmedicine.medical_treatmentTRPV1TRPV Cation ChannelsHippocampusNaphthalenesPharmacologySettore BIO/09 - FisiologiaNeuroprotection03 medical and health scienceschemistry.chemical_compound0302 clinical medicineReceptor Cannabinoid CB1Hippocampus Temporal lobe epilepsy Cannabinoids TRPV1 Capsaicin ElectrophysiologyMembrane Transport ModulatorsCannabinoid Receptor ModulatorsmedicineAnimalsRats WistarWIN 55212-2ChemistryElectric StimulationBenzoxazinesDisease Models Animal030104 developmental biologyEpilepsy Temporal LobeNeurologyAcute DiseaseAnticonvulsantslipids (amino acids peptides and proteins)Neurology (clinical)CannabinoidCapsaicinCapsazepineNeurosciencepsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugEpilepsy Research
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Cannabinoid control of brain bioenergetics: Exploring the subcellular localization of the CB1 receptor

2014

Brain mitochondrial activity is centrally involved in the central control of energy balance. When studying mitochondrial functions in the brain, however, discrepant results might be obtained, depending on the experimental approaches. For instance, immunostaining experiments and biochemical isolation of organelles expose investigators to risks of false positive and/or false negative results. As an example, the functional presence of cannabinoid type 1 (CB1) receptors on brain mitochondrial membranes (mtCB1) was recently reported and rapidly challenged, claiming that the original observation was likely due to artifact results. Here, we addressed this issue by directly comparing the procedures…

CB1 receptorWIN WIN55212-2Cannabinoid receptorBrain bioenergeticsLactate dehydrogenase Amedicine.medical_treatmentSDHADMSO dimethyl sulfoxideMitochondrionBiologySlp2 stomatin-like protein 2SDHA succinate dehydrogenase aTechnical ReportmedicineantibodieseducationReceptorKO knock-outMolecular Biologyeducation.field_of_studyelectron microscopyLDHa lactate dehydrogenase aDAB–Ni Ni-intensified 33ʹ-diaminobenzidine–4HClCell BiologySubcellular localizationWT wild-typemitochondriaBiochemistryCB1 cannabinoid type 1 receptorBSA bovine serum albuminCannabinoidorganelle purificationNeuroscienceImmunostainingMolecular Metabolism
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The effect of cannabinoid receptor agonist WIN 55,212-2 on anxiety-like behavior and locomotion in a genetic model of absence seizures in the elevate…

2022

GAERS and NEC rats were treated with cannabinoid 1/2 receptor agonist WIN 55,212-2 2 mg/kg and tested on the Elevated Plus-Maze

PharmacologyCannabinoid Receptor AgonistsModels GeneticMorpholinesNECWIN 55212-2 2AnxietyNaphthalenesSettore BIO/09 - FisiologiaBenzoxazinesRatsElevated Plus Maze TestPsychiatry and Mental healthEpilepsy AbsenceSeizuresPhysiology (medical)GAERSAnimalsPharmacology (medical)Elevated Plus-MazeRats WistarLocomotionCNS neurosciencetherapeutics
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Simulación de Plataformas Robóticas de Movimiento para Aplicaciones de Realidad Virtual Mediante Filtros Digitales

2017

[ES] El uso de plataformas robóticas de movimiento en simuladores de vehículos y aplicaciones de Realidad Virtual es relativamente habitual. Sin embargo, el ajuste de los algoritmos que controlan su funcionamiento, denominados algoritmos de washout, no es sencillo y requiere de numerosas pruebas hasta obtener una apropiada fidelidad de movimiento. Disponer de herramientas que permitan simular plataformas de movimiento puede permitir simplificar esta tarea. Es por ello que este trabajo presenta un método para la caracterización y simulación de manipuladores robóticos mediante filtros digitales de segundo orden, sencillo de implementar y ajustar a partir de una caracterización previa. El simu…

0209 industrial biotechnologyEngineeringGeneral Computer Sciencemedia_common.quotation_subjectTiempo reallcsh:Control engineering systems. Automatic machinery (General)Fidelity02 engineering and technologyVirtual realitySimulation systemMotion (physics)Filtros digitalesRealidad virtuallcsh:TJ212-225020901 industrial engineering & automation0203 mechanical engineeringSimulationmedia_commonMotion platforms020301 aerospace & aeronauticsbusiness.industryDigital filtersProcess (computing)Virtual RealityRoboticsReal timeControl and Systems EngineeringbusinessRobóticaSimuladoresSimulationPlataformas de movimientoRevista Iberoamericana de Automática e Informática Industrial RIAI
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WIN 55,212-2, agonist of cannabinoid receptors, prevents amyloid β1-42 effects on astrocytes in primary culture

2015

Alzheimer's disease (AD), a neurodegenerative illness involving synaptic dysfunction with extracellular accumulation of Aβ1-42 toxic peptide, glial activation, inflammatory response and oxidative stress, can lead to neuronal death. Endogenous cannabinoid system is implicated in physiological and physiopathological events in central nervous system (CNS), and changes in this system are related to many human diseases, including AD. However, studies on the effects of cannabinoids on astrocytes functions are scarce. In primary cultured astrocytes we studied cellular viability using MTT assay. Inflammatory and oxidative stress mediators were determined by ELISA and Western-blot techniques both in…

Cannabinoid receptormedicine.medical_treatmentInterleukin-1betaNitric Oxide Synthase Type IIlcsh:Medicinemedicine.disease_causeReceptors CannabinoidWIN 55212-2Receptorlcsh:ScienceCerebral CortexMultidisciplinaryCalcium Channel BlockersSistema nerviós Malaltiesmedicine.symptomSignal transductionResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyCell SurvivalMorpholinesPrimary Cell CultureInflammationNaphthalenesBiologyNeurologiaFetusInternal medicinemedicineAnimalsViability assayCannabinoid Receptor AgonistsAmyloid beta-PeptidesSuperoxide DismutaseTumor Necrosis Factor-alphalcsh:RTranscription Factor RelAPeptide FragmentsBenzoxazinesRatsPPAR gammaOxidative StressEndocrinologyGene Expression RegulationCyclooxygenase 2Astrocyteslcsh:QFisiologia humanaCannabinoidOxidative stress
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Cannabinoid and nitric oxide signaling interplay in the modulation of hippocampal hyperexcitability: study on electrophysiological and behavioral mod…

2015

A growing bulk of evidence suggests that cannabinoid system plays a pivotal role in the control of hyperexcitability phenomena. Notwithstanding, the anticonvulsant action of cannabinoids has not been fully addressed, in particular the involvement of potential cellular neuromodulators, for instance nitric oxide. In the current study, we focused on two distinct rat models of temporal lobe epilepsy, the Maximal Dentate Activation and the pilocarpine-induced acute seizures, providing both electrophysiological and behavioral data on cannabinoid and nitrergic system interplay. We evaluated the antiepileptic effects of WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4- morpholinylmethyl) pyrrolo[1,…

AgonistAM251MaleCannabinoid receptorIndazolesmedicine.drug_classmedicine.medical_treatmentMorpholinesHippocampusPharmacologyNaphthalenesNitric OxideHippocampusSettore BIO/09 - FisiologiaEpilepsyPiperidinesReceptor Cannabinoid CB1medicineAnimalshippocampus temporal lobe epilepsy cannabinoids behavior percentage of protection electrophysiology.Rats WistarWIN 55212-2Cannabinoid Receptor AgonistsDose-Response Relationship DrugCannabinoidsGeneral NeurosciencePilocarpinemedicine.diseaseEndocannabinoid systemBenzoxazinesRatsDisease Models AnimalEpilepsy Temporal LobePyrazolesCannabinoidNitric Oxide SynthasePsychologyNeurosciencemedicine.drug
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The Synthetic Cannabinoid WIN 55,212-2 Sensitizes Hepatocellular Carcinoma Cells to Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)-I…

2010

In this article, we demonstrate that the synthetic cannabinoid R-(+)-(2,3-dihydro-5-methyl-3-[(4-morpholinyl)methyl]pyrol[1,2,3-de]-1,4-benzoxazin-6-yl)-(1-naphthalenyl) methanone mesylate (WIN 55,212-2) sensitizes human hepatocellular carcinoma (HCC) cells to apoptosis mediated by tumor necrosis-related apoptosis inducing ligand (TRAIL). The apoptotic mechanism induced by treatment with WIN/TRAIL combination involved the loss of the mitochondrial transmembrane potential and led to the activation of caspases. In HCC cells, WIN treatment induced the up-regulation of TRAIL death receptor DR5, an effect that seemed to be related to the increase in the level of p8 and CHOP, two factors implicat…

Carcinoma HepatocellularDNA ComplementaryMorpholinesApoptosisNaphthalenesCHOPMembrane PotentialsTNF-Related Apoptosis-Inducing LigandCell Line TumorSurvivinmedicineHumansWIN 55212-2Protein kinase BTranscription factorCaspaseDNA PrimersPharmacologybiologyCannabinoidsReverse Transcriptase Polymerase Chain ReactionLiver NeoplasmsGene AmplificationDNA NeoplasmFlow CytometryBenzoxazinesReceptors TNF-Related Apoptosis-Inducing LigandApoptosisMitochondrial MembranesImmunologybiology.proteinCancer researchMolecular MedicineTumor necrosis factor alphaTranscription Factor CHOPmedicine.drugMolecular Pharmacology
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