Search results for "2723 Immunology and Allergy"

showing 10 items of 25 documents

The Cytokine GM-CSF Drives the Inflammatory Signature of CCR2+ Monocytes and Licenses Autoimmunity.

2015

Granulocyte-macrophage colony-stimulating factor (GM-CSF) has emerged as a crucial cytokine produced by auto-reactive T helper (Th) cells that initiate tissue inflammation. Multiple cell types can sense GM-CSF, but the identity of the pathogenic GM-CSF-responsive cells is unclear. By using conditional gene targeting, we systematically deleted the GM-CSF receptor (Csf2rb) in specific subpopulations throughout the myeloid lineages. Experimental autoimmune encephalomyelitis (EAE) progressed normally when either classical dendritic cells (cDCs) or neutrophils lacked GM-CSF responsiveness. The development of tissue-invading monocyte-derived dendritic cells (moDCs) was also unperturbed upon Csf2r…

CCR2Myeloidmedicine.medical_treatmentInterleukin-1betaAutoimmunitymedicine.disease_causeMonocytesAutoimmunityCytokine Receptor Common beta Subunit0302 clinical medicineSTAT5 Transcription FactorImmunology and AllergyAntigens LyMyeloid CellsPhosphorylationMice Knockout0303 health sciencesReverse Transcriptase Polymerase Chain ReactionExperimental autoimmune encephalomyelitisGene targetingFlow CytometryInfectious DiseasesCytokinemedicine.anatomical_structureGranulocyte macrophage colony-stimulating factor2723 Immunology and Allergymedicine.symptommedicine.drugSignal TransductionEncephalomyelitis Autoimmune ExperimentalReceptors CCR2Immunology610 Medicine & healthInflammationMice TransgenicBiology03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologyInflammation2403 ImmunologyGranulocyte-Macrophage Colony-Stimulating Factor2725 Infectious DiseasesDendritic Cellsmedicine.disease10040 Clinic for NeurologyImmunologyTranscriptome030217 neurology & neurosurgery
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Exclusive transduction of human CD4+ T Cells upon systemic delivery of CD4-targeted lentiviral vectors

2015

Abstract Playing a central role in both innate and adaptive immunity, CD4+ T cells are a key target for genetic modifications in basic research and immunotherapy. In this article, we describe novel lentiviral vectors (CD4-LV) that have been rendered selective for human or simian CD4+ cells by surface engineering. When applied to PBMCs, CD4-LV transduced CD4+ but not CD4− cells. Notably, also unstimulated T cells were stably genetically modified. Upon systemic or intrasplenic administration into mice reconstituted with human PBMCs or hematopoietic stem cells, reporter gene expression was predominantly detected in lymphoid organs. Evaluation of GFP expression in organ-derived cells and blood …

CD4-Positive T-Lymphocytes10028 Institute of Medical VirologyCell TransplantationGenetic enhancementAdoptiveMice SCIDImmunotherapy AdoptiveInterleukin 21MiceMice Inbred NODTransduction GeneticBone MarrowLeukocytesImmunology and AllergyCytotoxic T cellIL-2 receptorLuciferasesCells CulturedMice KnockoutHeterologousTumorCulturedForkhead Transcription FactorsAcquired immune systemFlow Cytometry3. Good healthCell biologymedicine.anatomical_structure[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology2723 Immunology and Allergy[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapyRegulatory T cellCellsKnockoutTransplantation HeterologousImmunologyMononuclearGenetic VectorsGreen Fluorescent Proteins610 Medicine & healthStreptamerThymus GlandBiologySCIDCell LineTransductionGeneticCell Line TumormedicineAnimalsHumansInterleukin 3Transplantation2403 ImmunologyLentivirusGenetic TherapyMolecular biology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyHEK293 CellsLeukocytes MononuclearInbred NOD570 Life sciences; biologySpleen
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Identification of NY-ESO-1 epitopes presented by human histocompatibility antigen (HLA)-DRB4*0101-0103 and recognized by CD4(+) T lymphocytes of pati…

2000

NY-ESO-1 is a member of the cancer-testis family of tumor antigens that elicits strong humoral and cellular immune responses in patients with NY-ESO-1–expressing cancers. Since CD4+ T lymphocytes play a critical role in generating antigen-specific cytotoxic T lymphocyte and antibody responses, we searched for NY-ESO-1 epitopes presented by histocompatibility leukocyte antigen (HLA) class II molecules. Autologous monocyte-derived dendritic cells of cancer patients were incubated with recombinant NY-ESO-1 protein and used in enzyme-linked immunospot (ELISPOT) assays to detect NY-ESO-1–specific CD4+ T lymphocyte responses. To identify possible epitopes presented by distinct HLA class II allele…

CD4-Positive T-LymphocytesImmunologyMolecular Sequence DataAntigen-Presenting Cells10050 Institute of Pharmacology and Toxicology610 Medicine & healthHuman leukocyte antigenBiologyEpitopeCell LineAntigenAntigens NeoplasmImmunology and AllergyCytotoxic T cellHumansAmino Acid SequenceAntigen-presenting cellMelanomaHLA-DRB4Alleles2403 ImmunologyHLA class II–restricted NY-ESO-1 epitopesMembrane ProteinsProteinsT lymphocyteDendritic CellsHLA-DR AntigensVirologyRecombinant ProteinsHistocompatibilityImmunologyCD4+ T cell recognition2723 Immunology and Allergy570 Life sciences; biologyOriginal ArticleHLA-DRB4 Chains
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Impact of body weight on virological and immunological responses to efavirenz-containing regimens in HIV-infected, treatment-naive adults

2015

Objective: The prevalence of overweight and obesity is increasing among HIV-infected patients. Whether standard antiretroviral drug dosage is adequate in heavy individuals remains unresolved. We assessed the virological and immunological responses to initial efavirenz (EFV)-containing regimens in heavy compared to normal-weight HIV-infected patients. Design: Observational European cohort collaboration study. Methods: Eligible patients were antiretroviral-naive with documented weight prior to EFV start and follow-up viral loads after treatment initiation. Cox regression analyses evaluated the association between weight and time to first undetectable viral load ( 50 copies/ml) after initial s…

CyclopropanesMaleEfavirenz; HIV; Immunological response; Virological response; Weight; Adult; Antiretroviral Therapy Highly Active; Benzoxazines; CD4 Lymphocyte Count; Cohort Studies; Female; HIV Infections; Humans; Male; Middle Aged; Obesity; Regression Analysis; Reverse Transcriptase Inhibitors; Treatment Outcome; Viral Load; Body WeightHIV InfectionsOverweight10234 Clinic for Infectious DiseasesCohort Studieschemistry.chemical_compoundAntiretroviral Therapy Highly ActiveImmunology and AllergyHIV InfectionImmunological responsevirus diseasesVirological responseMiddle AgedViral LoadReverse Transcriptase InhibitorTreatment OutcomeInfectious DiseasesAlkynesCohort2723 Immunology and AllergyRegression AnalysisReverse Transcriptase InhibitorsFemalemedicine.symptomViral loadCohort studyHumanBenzoxazineAdultmedicine.medical_specialtyEfavirenzImmunologyAntiretroviral Therapy610 Medicine & healthRegression AnalysiPharmacokineticsInternal medicinemedicineHumansHighly ActiveObesity2403 Immunologybusiness.industryProportional hazards modelBody WeightHIV2725 Infectious DiseasesEfavirenz; HIV; Immunological response; Virological response; Weight; Adult; Antiretroviral Therapy Highly Active; Benzoxazines; CD4 Lymphocyte Count; Cohort Studies; Female; HIV Infections; Humans; Male; Middle Aged; Obesity; Regression Analysis; Reverse Transcriptase Inhibitors; Treatment Outcome; Viral Load; Body Weight; Immunology and Allergy; Immunology; Infectious Diseasesmedicine.diseaseWeightObesityBenzoxazinesCD4 Lymphocyte Countlnfectious Diseases and Global Health Radboud Institute for Health Sciences [Radboudumc 4]chemistryImmunologyCohort StudieEfavirenzbusiness
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Dendritic Cells Ameliorate Autoimmunity in the CNS by Controlling the Homeostasis of PD-1 Receptor+ Regulatory T Cells

2012

SummaryMature dendritic cells (DCs) are established as unrivaled antigen-presenting cells (APCs) in the initiation of immune responses, whereas steady-state DCs induce peripheral T cell tolerance. Using various genetic approaches, we depleted CD11c+ DCs in mice and induced autoimmune CNS inflammation. Unexpectedly, mice lacking DCs developed aggravated disease compared to control mice. Furthermore, when we engineered DCs to present a CNS-associated autoantigen in an induced manner, we found robust tolerance that prevented disease, which coincided with an upregulation of the PD-1 receptor on antigen-specific T cells. Additionally, we showed that PD-1 was necessary for DC-mediated induction o…

Encephalomyelitis Autoimmune ExperimentalT cellProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationCD11cAutoimmunity610 Medicine & healthchemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoantigensT-Lymphocytes RegulatoryB7-H1 AntigenAutoimmunityImmune toleranceMiceImmune systemDownregulation and upregulationImmune TolerancemedicineAnimalsImmunology and AllergyReceptorMice KnockoutAntigen Presentation2403 Immunologyhemic and immune systemsDendritic Cells2725 Infectious DiseasesTh1 CellsCD11c AntigenMice Inbred C57BLInfectious Diseasesmedicine.anatomical_structure10032 Clinic for Oncology and HematologyImmunology2723 Immunology and AllergyTh17 CellsImmunity
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BRAF-V600E expression in precursor versus differentiated dendritic cells defines clinically distinct LCH risk groups.

2014

BRAF-V600E expression is identified in hematopoietic progenitor and precursor myeloid dendritic cells in patients with high-risk LCH, and enforced expression of BRAF-V600E in CD11c+ cells recapitulates a high-risk LCH-like phenotype in mice.

MalePathologyendocrine system diseasesCellular differentiationCD34Antigens CD34Mice0302 clinical medicineLangerhans cell histiocytosisBone MarrowRisk FactorsImmunology and Allergyskin and connective tissue diseasesChild0303 health sciencesCell Differentiation3. Good healthHistiocytosismedicine.anatomical_structurePhenotypeTreatment Outcome030220 oncology & carcinogenesisChild PreschoolAntigens Surface2723 Immunology and AllergyFemaleProto-Oncogene Proteins B-rafmedicine.medical_specialtyImmunologyCD11c610 Medicine & healthBiologyArticle03 medical and health sciencesGermline mutationmedicineAnimalsHumansCell LineageGenetic Predisposition to DiseaseLectins C-TypeProgenitor cellneoplasms030304 developmental biology2403 ImmunologyHistocompatibility Antigens Class II302InfantCorrectionDendritic Cellsmedicine.diseaseHematopoietic Stem Cellsdigestive system diseasesCD11c Antigenenzymes and coenzymes (carbohydrates)Histiocytosis Langerhans-CellMannose-Binding Lectins10032 Clinic for Oncology and HematologyMutationBone marrowThe Journal of experimental medicine
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Molecular evidence of HLA-B27 in a historical case of ankylosing spondylitis

2005

Malemedicine.medical_specialty10017 Institute of Anatomy2745 RheumatologyImmunologyMEDLINEMolecular evidence610 Medicine & healthPolymerase Chain ReactionHistory 17th CenturyRheumatologyInternal medicinemedicineHumans2736 Pharmacology (medical)Immunology and AllergySpondylitis AnkylosingPharmacology (medical)SpondylitisHLA-B27 AntigenHLA-B27Ankylosing spondylitis2403 Immunologybusiness.industrymedicine.diseaseDermatologyRheumatologyHistory 16th CenturyAnthropologyImmunology11294 Institute of Evolutionary Medicine2723 Immunology and Allergy570 Life sciences; biologybusiness
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Homeostasis of Microglia in the Adult Brain: Review of Novel Microglia Depletion Systems.

2015

Microglia are brain macrophages that emerge from early erythro-myeloid precursors in the embryonic yolk sac and migrate to the brain mesenchyme before the blood brain barrier is formed. They seed the brain, and proliferate until they have formed a grid-like distribution in the central nervous system that is maintained throughout lifespan. The mechanisms through which these embryonic-derived cells contribute to microglia homoeostasis at steady state and upon inflammation are still not entirely clear. Here we review recent studies that provided insight into the contribution of embryonically-derived microglia and of adult 'microglia-like' cells derived from monocytes during inflammation. We ex…

NeuroimmunomodulationCellular differentiationMesenchymeImmunologyCentral nervous systemEmbryonic DevelopmentInflammation610 Medicine & healthBiologyBlood–brain barrier10263 Institute of Experimental ImmunologymedicineImmunology and AllergyAnimalsHomeostasisHumansNeuroinflammationInflammation2403 ImmunologyMicrogliaMacrophagesBrainCell DifferentiationEmbryonic stem cellDisease Models Animalmedicine.anatomical_structureImmunologyModels Animal2723 Immunology and Allergy570 Life sciences; biologyMicrogliamedicine.symptomTrends in immunology
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Mast cell-derived mediators promote murine neutrophil effector functions

2013

Mast cells are able to trigger life-saving immune responses in murine models for acute inflammation. In such settings, several lines of evidence indicate that the rapid and protective recruitment of neutrophils initiated by the release of mast cell-derived pro-inflammatory mediators is a key element of innate immunity. Herein, we investigate the impact of mast cells on critical parameters of neutrophil effector function. In the presence of activated murine bone marrow-derived mast cells, neutrophils freshly isolated from bone marrow rapidly lose expression of CD62L and up-regulate CD11b, the latter being partly driven by mast cell-derived TNF and GM-CSF. Mast cells also strongly enhance neu…

PhagocytosisImmunologyApoptosisInflammation610 Medicine & healthmast cellsBiology142-005 142-005Neutrophil ActivationlungMiceImmune systemPhagocytosisneutrophilsmedicineAnimalsImmunology and AllergyCells CulturedMice Knockout2403 ImmunologyInnate immune systemTumor Necrosis Factor-alpharodentGranulocyte-Macrophage Colony-Stimulating FactorPneumoniaGeneral MedicineFlow CytometryMast cellMice Mutant StrainsCell biologycell activationMice Inbred C57BLInterleukin 33medicine.anatomical_structureinflammationImmunology2723 Immunology and AllergyTumor necrosis factor alphamedicine.symptomCell activation
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Cre-lox: Target Sensitivity Matters

2019

Recombination Genetic2403 ImmunologyIntegrasesImmunologyMice Transgenic610 Medicine & health2725 Infectious DiseasesBiology10263 Institute of Experimental ImmunologySubstrate SpecificityCell biologyProtein-Lysine 6-OxidaseMicePhenotypeInfectious DiseasesMutagenesis2723 Immunology and AllergyAnimalsHumans570 Life sciences; biologyImmunology and AllergySensitivity (control systems)Immunity
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