Search results for "3-d"

showing 10 items of 1132 documents

Mild systemic inflammation and moderate hypoxia transiently alter neuronal excitability in mouse somatosensory cortex

2016

During the perinatal period, the brain is highly vulnerable to hypoxia and inflammation, which often cause white matter injury and long-term neuronal dysfunction such as motor and cognitive deficits or epileptic seizures. We studied the effects of moderate hypoxia (HYPO), mild systemic inflammation (INFL), or the combination of both (HYPO + INFL) in mouse somatosensory cortex induced during the first postnatal week on network activity and compared it to activity in SHAM control animals. By performing in vitro electrophysiological recordings with multi-electrode arrays from slices prepared directly after injury (P8–10), one week after injury (P13–16), or in young adults (P28–30), we investig…

Male0301 basic medicineAction PotentialsKainate receptorStimulationPotassium ChlorideMicechemistry.chemical_compound0302 clinical medicineHypoxia6-Cyano-7-nitroquinoxaline-23-dioneNeuronsAge FactorsInterleukin-1βElectrophysiologyEpileptiform activityNeurologyAnesthesiaCNQXNMDA receptorFemalemedicine.symptommedicine.drugmedicine.medical_specialtyAMPA receptorIn Vitro TechniquesBiologyBicucullineMulti-electrode arrayArticlelcsh:RC321-57103 medical and health sciencesInternal medicinemedicineAnimalsGABA-A Receptor Antagonistslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryInflammationSystemic inflammationSomatosensory CortexHypoxia (medical)BicucullineBarrel cortexMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinology2-Amino-5-phosphonovalerateGene Expression Regulationchemistrynervous systemExploratory BehaviorExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryNeurobiology of Disease
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Mast cells' involvement in inflammation pathways linked to depression: evidence in mastocytosis

2016

International audience; Converging sources of evidence point to a role for inflammation in the development of depression, fatigue and cognitive dysfunction. More precisely, the tryptophan (TRP) catabolism is thought to play a major role in inflammation-induced depression. Mastocytosis is a rare disease in which chronic symptoms, including depression, are related to mast cell accumulation and activation. Our objectives were to study the correlations between neuropsychiatric features and the TRP catabolism pathway in mastocytosis in order to demonstrate mast cells' potential involvement in inflammation-induced depression. Fifty-four patients with mastocytosis and a mean age of 50.1 years were…

Male0301 basic medicine[SHS.PSY]Humanities and Social Sciences/PsychologyKynurenic Acidchemistry.chemical_compound0302 clinical medicineKynurenic acidMast CellsIndoleamine 23-dioxygenaseAcute stressQuinolinic acidKynurenineDepression (differential diagnoses)DepressionTryptophanMiddle AgedMast cellRat-brain3. Good healthPsychiatry and Mental healthmedicine.anatomical_structure[ SCCO.NEUR ] Cognitive science/NeuroscienceFemalemedicine.symptomMastocytosisSerotoninmedicine.medical_specialtyInflammationAryl-hydrocarbon receptorCentral-nervous-system[ SHS.PSY ] Humanities and Social Sciences/Psychology03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicinemedicineHumansIndoleamine-Pyrrole 23-Dioxygenase[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyMolecular BiologyInflammationPsychiatric Status Rating ScalesDepressive Disorder Majorbusiness.industry[SCCO.NEUR]Cognitive science/NeuroscienceBeck Depression InventoryInterferon-alphaMammalian brain030104 developmental biologyEndocrinologyImmune-systemchemistryImmunologyIndoleamine 2?3-dioxygenasebusinessStress Psychological030217 neurology & neurosurgeryKynurenineQuinolinic acid
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Non-uniform displacement and strain between the soleus and gastrocnemius subtendons of rat Achilles tendon

2018

Achilles tendon (AT) comprises of 3 subtendons arising from the soleus (SOL) and the lateral (LG) and medial (MG) heads of the gastrocnemius muscle. While recent human studies show differential displacement within AT, these displacements have not been attributed to specific subtendons. We tested the hypothesis that the SOL and LG subtendons show differential displacement and strain during various combinations of SOL, LG, and MG excitations. Movement of knots, sutured onto SOL and LG subtendons of 12 Wistar rats, was videotaped, while the muscles were stimulated intramuscularly and ankle torque was assessed. When SOL only was stimulated, the plantar flexion torque was the smallest among the …

Male0301 basic medicineanatomyPhysical Therapy Sports Therapy and RehabilitationAchilles TendonPlantar flexion03 medical and health sciencesGastrocnemius muscle0302 clinical medicinemedicineAnimalsOrthopedics and Sports MedicineDisplacement (orthopedic surgery)Active stateRats WistarMuscle Skeletalta315Achilles tendonStrain (chemistry)Human studiesChemistryankle torquedeformation030229 sport sciencesAnatomy3-dimensional modelRatsAchilles tendon030104 developmental biologymedicine.anatomical_structureTorquecalcaneal tendontriceps suraekantajänneAnkle JointAnkle torqueMuscle Contraction
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Acute in vivo administration of a fish oil-containing emulsion improves post-ischemic cardiac function in n-3-depleted rats

2006

International audience; A novel i.v. lipid preparation (MCT:FO) containing 80% medium chain-triacylglycerols and 20% fish oil was recently developed to rapidly replenish cell membrane phospholipids with omega 3 (n-3) polyunsaturated fatty acids (PUFA). In regard of this property, we investigated the effect of a single i.v. administration of MCT:FO on the recovery of cardiac function after ischemia in control and n-3-depleted rats. Results were compared with those obtained either with a control preparation, where FO was replaced by triolein (MCT:OO), or with saline. Saline (1 ml) or lipid preparation (also 1 ml) was injected as a bolus via the left saphenous vein. After 60 min the heart was …

MaleFat EmulsionsTime Factorsmedicine.medical_treatmentMyocardial IschemiaWistar030204 cardiovascular system & hematologyPharmacologyLIPID PREPARATIONchemistry.chemical_compound0302 clinical medicineBolus (medicine)MESH: Fatty Acids Omega-3Heart Rate[SDV.IDA]Life Sciences [q-bio]/Food engineeringMedicineMESH: AnimalsMESH: Oxygen ConsumptionTrioleinMESH: Heart RateSalineOMEGA3-DEFICIENCYOmega-3chemistry.chemical_classification0303 health sciencesFatty AcidsHeartGeneral Medicine[SDV.IDA] Life Sciences [q-bio]/Food engineeringFish oilMESH: Myocardial Reperfusion Injury3. Good healthLactatesMESH: Myocardial IschemiaIntravenousPolyunsaturated fatty acidCardiac function curveFat Emulsions Intravenousmedicine.medical_specialtyMESH: Rats[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringACIDE GRAS POLYINSATURE OMEGA3IschemiaMESH: Fish OilsMyocardial Reperfusion InjuryMESH: Coronary CirculationMESH: Lactates03 medical and health sciencesISCHEMIA-REPERFUSIONFish OilsOxygen ConsumptionIn vivoCoronary CirculationMESH: Analysis of VarianceFatty Acids Omega-3[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyGeneticsAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRats WistarLANGENDORFF030304 developmental biologyAnalysis of Variancebusiness.industryBody WeightMESH: Time FactorsMESH: Rats Wistarmedicine.diseaseMESH: MaleRatsMESH: Body WeightMESH: Fat Emulsions IntravenousSurgeryMESH: Heartchemistrybusiness
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Phospholipase D in rat myocardium: formation of lipid messengers and synergistic activation by G-protein and protein kinase C.

1998

Activation of phospholipase D (PLD) and phosphoinositide-specific phospholipase C (PI-PLC) by fluoride, to stimulate heterotrimeric G-proteins, and by phorbol esters, to stimulate protein kinase C (PKC), was studied in rat atria. Fluoride and 4beta-phorbol-12beta,13alpha-dibutyrate (PDB), in contrast to 4beta-phorbol-13alpha-acetate (PAc), activated PLD, catalyzing the formation of [3H]-phosphatidylethanol ([3H]-PETH), [3H]-phosphatidic acid ([3H]-PA), choline and sn-1,2-diacylglycerol (DAG). Basal PLD activity was resistant to drastic changes in Ca2+ and to Ro 31-8220, a PKC inhibitor, but was decreased by genistein, an inhibitor of tyrosine kinase, and increased by vanadate, a tyrosine ph…

MaleG proteinProtein tyrosine phosphataseBiologyBiochemistrySecond Messenger Systemschemistry.chemical_compoundPhosphoinositide Phospholipase CGTP-Binding ProteinsPhorbol EstersPhospholipase DAnimalsRats WistarProtein kinase CPhorbol 1213-DibutyrateProtein Kinase CDiacylglycerol kinasePharmacologyPhospholipase CPhospholipase DMyocardiumPhosphatidylinositol Diacylglycerol-LyaseTyrosine phosphorylationDrug SynergismLipid MetabolismLipidsRatsEnzyme ActivationBiochemistrychemistryType C PhospholipasesSecond messenger systemlipids (amino acids peptides and proteins)Biochemical pharmacology
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Role of AMPA glutamate receptors in the conditioned rewarding effects of MDMA in mice

2018

Abstract Currently, there is not an effective treatment for 3,4-methylenedioxymethamphetamine (MDMA) dependence but pharmacotherapies targeting glutamate neurotransmission are a promising strategy. Previously, we showed that blockade of glutamate NMDA and AMPA receptors impairs the conditioned rewarding effects of MDMA and cocaine, respectively. In this study we evaluated the role of AMPA receptors in the rewarding effects of MDMA in mice using the conditioned place preference (CPP) paradigm. Mice were conditioned with MDMA (1.25 mg/kg) 60 min after the treatment with saline or different doses (0.25, 1 and 5 mg/kg) of the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dion…

MaleHallucinogenMDMAmiceN-Methyl-34-methylenedioxyamphetamineAmphetamine-Related DisordersSpatial BehaviorKainate receptorAMPA receptorPharmacologyMice03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineRewardConditioning Psychologicalmental disordersmedicineAnimalsReceptors AMPAAMPA receptorsreward6-Cyano-7-nitroquinoxaline-23-dioneDose-Response Relationship Drugbusiness.industryGlutamate receptorMDMACNQXconditioned place preferenceConditioned place preference030227 psychiatrynervous systemchemistryHallucinogensCNQXNMDA receptorbusinessExcitatory Amino Acid Antagonistspsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugBehavioural Brain Research
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Effect of memantine and CNQX in the acquisition, expression and reinstatement of cocaine-induced conditioned place preference

2006

The present study evaluates the effect of memantine, a non-competitive N-methyl-d-aspartate (NMDA) glutamate receptor antagonist and CNQX, an alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic acid (AMPA)/kainate receptor antagonist on the rewarding effects of cocaine in mice, using the conditioned place preference (CPP) paradigm. Cocaine-induced CPP was studied pairing this drug with different memantine or CNQX doses during either the acquisition or the expression phase of the procedure. Once CPP was established, and the preference extinguished, reinstatement was induced by a priming dose of cocaine. Both antagonists, which in themselves do not present motivational actions on the preferen…

MaleKainate receptorAMPA receptorPharmacologyExtinction PsychologicalMicechemistry.chemical_compoundCocaineDopamine Uptake InhibitorsMemantineAnimalsMedicineDrug InteractionsGlutamate receptor antagonistBiological Psychiatry6-Cyano-7-nitroquinoxaline-23-dionePharmacologyBehavior AnimalDose-Response Relationship Drugbusiness.industryGlutamate receptorMemantineConditioned place preferencenervous systemchemistryCNQXConditioning OperantNMDA receptorbusinessExcitatory Amino Acid AntagonistsReinforcement Psychologypsychological phenomena and processesmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Ceramide inhibits Kv currents and contributes to TP-receptor-induced vasoconstriction in rat and human pulmonary arteries

2011

et al.

MalePatch-Clamp TechniquesPhysiologyReceptors ThromboxaneSpider Venoms030204 cardiovascular system & hematologyMuscle Smooth VascularMembrane Potentialschemistry.chemical_compound0302 clinical medicineHypoxic pulmonary vasoconstrictionVasoconstrictor AgentsProtein Kinase C0303 health sciencesAniline Compounds3. Good healthSphingomyelin Phosphodiesterasemedicine.anatomical_structurePotassium Channels Voltage-GatedCirculatory systemmedicine.symptomSphingomyelinSignal TransductionBlood vesselmedicine.medical_specialtyCeramidePhosphinesMyocytes Smooth MusclePulmonary ArteryBiologyCeramidesBenzylidene Compounds03 medical and health sciencesInternal medicinemedicineAnimalsHumansRats Wistar030304 developmental biologyCell BiologySphingolipidRatsHEK293 CellsEndocrinologychemistryVasoconstriction15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidVascular resistanceVascular ResistancePeptidesVasoconstrictionAmerican Journal of Physiology-Cell Physiology
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The effects of phorbol esters on choline phospholipid hydrolysis in heart and brain

1990

The efflux of choline was determined in rat striatal slices, incubated chicken atria and perfused chicken hearts. 4 beta-Phorbol-12 beta,13 alpha-dibutyrate (PDB) and 4 beta-phorbol-12 beta-myristate, 13 alpha-acetate (PMA) were used to stimulate protein kinase C. The other phorbol esters, 4 beta-phorbol-13 alpha-acetate (PAc) and 4 alpha-phorbol-12 beta,13 alpha-didecanoate (4 alpha PDD), known to be inactive, were tested to evaluate the specificity of the responses. PDB markedly enhanced the efflux of choline in all of the three preparations. The PDB-evoked efflux of choline in incubated chicken atria was equal to the net production of choline and, therefore, was not caused by translocati…

MalePhospholipidIn Vitro TechniquesBiologychemistry.chemical_compoundPhosphatidylcholinePhorbol EstersmedicineAnimalsCholinePhorbol 1213-DibutyrateProtein kinase CCholinesteraseBrain ChemistryPharmacologyHydrolysisMyocardiumHeartRats Inbred StrainsRatsEGTAchemistryBiochemistryPhosphatidylcholinesbiology.proteinTetradecanoylphorbol AcetateEffluxChickensAcetylcholinemedicine.drugEuropean Journal of Pharmacology: Molecular Pharmacology
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Effects of CNQX and MPEP on sensitization to the rewarding effects of morphine

2011

The present study was conducted to evaluate the influence of the glutamatergic receptors α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) and metabotropic glutamate 5 (mGlu5) receptors on sensitization to the rewarding effects of morphine. The effects of pre-treatment with saline or 20 mg/kg morphine plus the AMPA receptor antagonist 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX) (5 or 10 mg/kg) or the metabotropic Glu5 receptor antagonist 6-methyl-2-(phenylethynyl)-pyridine (MPEP) (5 or 10 mg/kg) on the place conditioning induced by a low dose of morphine (2 mg/kg) were assessed. The 2 mg/kg dose of morphine was ineffective in animals pre-treated with saline but induced a clear con…

MalePyridinesmedicine.drug_classReceptor Metabotropic Glutamate 5AMPA receptorPharmacologyReceptors Metabotropic GlutamateMicechemistry.chemical_compoundRewardmedicineAnimalsReceptors AMPASensitization6-Cyano-7-nitroquinoxaline-23-dionePharmacologyDose-Response Relationship DrugMorphineAntagonistReceptor antagonistConditioned place preferencemedicine.anatomical_structureMetabotropic receptorchemistryAnesthesiaCNQXMorphineConditioning OperantExcitatory Amino Acid Antagonistsmedicine.drugEuropean Journal of Pharmacology
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