Search results for "53"

showing 10 items of 2908 documents

Role of Gadd45a in Wip1-dependent regulation of intestinal tumorigenesis.

2012

Conversion of intestinal stem cells into tumor-initiating cells is an early step in Apc(Min)-induced polyposis. Wild-type p53-induced phosphatase 1 (Wip1)-dependent activation of a DNA damage response and p53 has a permanent role in suppression of stem cell conversion, and deletion of Wip1 lowers the tumor burden in Apc(Min) mice. Here we show that cyclin-dependent kinase inhibitor 2a, checkpoint kinase 2, and growth arrest and DNA damage gene 45a (Gadd45a) exert critical functions in the tumor-resistant phenotype of Wip1-deficient mice. We further identified Gadd45a as a haploinsufficient gene in the regulation of Wip1-dependent tumor resistance in mice. Gadd45a appears to function through…

Genes APCDNA RepairDNA repairDNA damageApoptosisCell Cycle ProteinsBiologyProtein Serine-Threonine KinasesReceptors G-Protein-CoupledMicePhosphoprotein PhosphatasesGene silencingAnimalsMolecular BiologyCheckpoint Kinase 2Cyclin-Dependent Kinase Inhibitor p16beta CateninMice KnockoutOriginal PaperKinaseIntestinal PolyposisStem CellsJNK Mitogen-Activated Protein KinasesNuclear ProteinsCell BiologyCell biologyProtein Phosphatase 2CCheckpoint Kinase 2Cell Transformation NeoplasticCancer researchSignal transductionStem cellTumor Suppressor Protein p53GADD45ASignal TransductionCell death and differentiation
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Biological characterization of laryngeal squamous-cell carcinoma

1996

The traditional prognostic factors, including stage of disease and tumour grade, have shown a limited prognostic significance and an inability to predict clinical response to specific treatment in patients with laryngeal squamous-cell carcinoma. More recent data suggest that cell kinetics indices, DNA-ploidy, lysosomal cysteine proteinase expression and genetic changes of both tumour suppressor genes and proto-oncogenes may be considered as reliable and reproducible indicators of biological aggressiveness in these patients. Moreover, the frequency of different genetic alterations suggests that several pathways are involved in the genesis of these neoplasias and, in particular, it is very pr…

Genes rasChromosomes Human Pair 11Settore BIO/14 - FarmacologiaHumansDNA NeoplasmFlow CytometryGenes p53Laryngeal NeoplasmsLaryngeal squamous-cell carcinomaCell Division
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A Potent Tumor-Reactive p53-Specific Single-Chain TCR without On- or Off-Target Autoimmunity In Vivo

2018

Genetic engineering of T cells with a T cell receptor (TCR) targeting tumor antigen is a promising strategy for cancer immunotherapy. Inefficient expression of the introduced TCR due to TCR mispairing may limit the efficacy and adversely affect the safety of TCR gene therapy. Here, we evaluated the safety and therapeutic efficiency of an optimized single-chain TCR (scTCR) specific for an HLA-A2.1-restricted (non-mutated) p53(264–272) peptide in adoptive T cell transfer (ACT) models using our unique transgenic mice expressing human p53 and HLA-A2.1 that closely mimic the human setting. Specifically, we showed that adoptive transfer of optimized scTCR-redirected T cells does not induce on-tar…

Genetically modified mouseAdoptive cell transfermedicine.medical_treatmentGenetic enhancementT-LymphocytesReceptors Antigen T-CellAutoimmunityBiology03 medical and health sciencesMice0302 clinical medicineAntigenCancer immunotherapyDrug DiscoveryHLA-A2 AntigenGeneticsmedicineAnimalsHumansMolecular Biology030304 developmental biologyPharmacology0303 health sciencesT-cell receptorImmunotherapyGenetic TherapyTumor antigen030220 oncology & carcinogenesisCancer researchMolecular MedicineOriginal ArticleTumor Suppressor Protein p53
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Strategies for tumor elimination by cytotoxic T lymphocytes.

1998

Despite differences in their tissue of origin, many tumors share high level expression of certain tumor-associated proteins. Our laboratory has focused on the possibility of utilizing antigenic components of these proteins as a focus for T-cell immunotherapy of cancer. The advantage of targeting such commonly expressed proteins is the fact that such therapy could be of value in eliminating many different types of tumors. A potential barrier in the identification of T-cell epitopes derived from these proteins and presented by tumor cells is the fact that these proteins are also expressed at low levels in some normal tissues, and therefore, self-tolerance may eliminate T cells that are capabl…

Genetically modified mousePolymers and Plasticsmedicine.medical_treatmentTransgeneHemagglutinin (influenza)ImmunotherapyBiologyEpitopeCell biologyMiceImmune systemAntigenAntigens NeoplasmNeoplasmsbiology.proteinmedicineImmune ToleranceCytotoxic T cellAnimalsHumansTumor Suppressor Protein p53General Environmental ScienceT-Lymphocytes Cytotoxic
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Laser Pressure Catapulting (LPC): Optimization LPC-System and Genotyping of Colorectal Carcinomas

2005

Genotype analysis is becoming more and more useful in clinical practice, since specific mutations in tumors often correlate with prognosis and/or therapeutic response. Unfortunately, current molecular analytical techniques often require time-consuming and costly steps of analysis, thus making their routine clinical use difficult. Moreover, one of the most difficult problems arising during tumor research is that of their cell heterogeneity, which depends on their clear molecular heterogeneity. SSCP analysis discriminates by means of aberrant electrophoresis migration bands, mutated alleles which may represent as little as 15-20% of their total number. Nevertheless, in order to identify by se…

GeneticsGenotypePhysiologyLasersCarcinomaDNA Mutational AnalysisClinical BiochemistrySingle-strand conformation polymorphismCell BiologyDNA Genotype MicrodissectionGene mutationBiologyGenes rasGenotypeDNA Mutational AnalysisHumansProspective StudiesTumor Suppressor Protein p53AlleleColorectal NeoplasmsMicrodissectionGenotypingPolymorphism Single-Stranded ConformationalMicrodissectionLaser capture microdissection
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TP53 mutations and S-phase fraction but not DNA-ploidy are independent prognostic indicators in laryngeal squamous cell carcinoma

2005

ToprospectivelyevaluatetheprognosticsignificanceofTP53,H-,K-,andN-Rasmutations,DNA-ploidyandS-phasefraction(SPF) in patients affected by locally advanced laryngeal squamous cell carcinoma (LSCC). Eight-one patients (median follow-up was 71 months) who underwent resective surgery for primary operable locally advanced LSCC were analyzed. Tumor DNA was screened for mutational analysis by PCR/SSCP and sequencing. DNA-ploidy and SPF were performed byflow cytometric analyses. Thirty-six patients (44%) had, at least, a mutation in the TP53 gene. Of them, 22% (8/36) had double mutations and 3% (1/36) had triplemutations.Intotal,46TP53mutationswereobserved.Themajority(41%)oftheseoccurinexon5(19/46),…

Geneticsmedicine.medical_specialtyMutationPhysiologyClinical BiochemistrySingle-strand conformation polymorphismCell BiologyBiologyTp53 mutationLaryngeal squamous cell carcinomamedicine.disease_causeGastroenterologyExonInternal medicinemedicineS-Phase FractionGeneDna ploidyJournal of Cellular Physiology
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DNA double-strand breaks trigger apoptosis in p53-deficient fibroblasts

2001

DNA double-strand breaks (DSBs) are induced by ionizing radiation (IR) and various radiomimetic agents directly, or indirectly as a consequence of DNA repair, recombination and replication of damaged DNA. They are ultimately involved in the generation of chromosomal aberrations and were reported to cause genomic instability, gene amplification and reproductive cell death. To address the question of whether DSBs act as a trigger of apoptosis, we induced DSBs by means of restriction enzyme electroporation and compared the effect with IR in mouse fibroblasts that differ in p53 status [wild-type (+/+) versus p53-deficient (-/-) cells]. We show that (i) electroporation of PVU:II is highly effici…

Genome instabilityCancer ResearchProgrammed cell deathTime FactorsDNA RepairDNA repairBlotting WesternApoptosisBiologymedicine.disease_causeCell LineMiceNecrosischemistry.chemical_compoundProto-Oncogene ProteinsRadiation IonizingmedicineAnimalsDeoxyribonucleases Type II Site-SpecificCells Culturedbcl-2-Associated X ProteinMice KnockoutRecombination GeneticMutationElectroporationDose-Response Relationship RadiationDNAGeneral MedicineTransfectionFibroblastsGenes p53Molecular biologyElectroporationProto-Oncogene Proteins c-bcl-2chemistryGamma RaysApoptosisComet AssayTumor Suppressor Protein p53DNADNA DamageCarcinogenesis
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MAD2 depletion triggers premature cellular senescence in human primary fibroblasts by activating a P53 pathway preventing aneuploid cells propagation.

2012

The spindle assembly checkpoint (SAC) is a cellular surveillance mechanism that ensures faithful chromosome segregation during mitosis and its failure can result in aneuploidy. Previously, it was suggested that reduction of the MAD2 gene, encoding a major component of the SAC, induced aneuploidy in human tumor cells. However, tumor cell lines contain multiple mutations that might affect or exacerbate the cellular response to Mad2 depletion. Thus, the scenario resulting by Mad2 depletion in primary human cells could be different and more complex that the one depicted so far. We used primary human fibroblasts (IMR90) and epithelial breast cells (MCF10A) to gain further insight on the effects …

Genome instabilityCyclin-Dependent Kinase Inhibitor p21Cell cycle checkpointMad2PhysiologyClinical BiochemistryMAD2 depletion Aneuploidy Premature cellular senescence TP53Cell Cycle ProteinsBiologyCyclin-dependent kinaseChromosome instabilityChromosomal InstabilityTumor Suppressor Protein p14ARFHumansGene SilencingRNA Small InterferingMitosisCells CulturedCellular SenescenceCell ProliferationCalcium-Binding ProteinsCell BiologyCell Cycle CheckpointsFibroblastsAneuploidybeta-GalactosidaseCell biologyRepressor ProteinsSpindle checkpointSettore BIO/18 - GeneticaGene Expression RegulationMad2 Proteinsbiology.proteinM Phase Cell Cycle CheckpointsTumor Suppressor Protein p53Cell agingSignal Transduction
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Sarcopenia and health-related outcomes: an umbrella review of observational studies

2019

Key summary pointsAimTo investigate associations of sarcopenia with adverse health-related outcomes, through an umbrella review method.FindingsSarcopenia appears to be significantly associated with several adverse outcomes in older people, with a strong evidence for increased risk of mortality, disability, and falls.MessageSarcopenia is associated with several adverse health-related outcomes in older people, indicating the need of assessing this condition in daily practice. AbstractBackgroundThe clinical relevance of sarcopenia has increasingly been recognized. However, whether it is associated with the development of other medical conditions is still unclear. Therefore, we aimed to capture…

GerontologySarcopeniaan umbrella review of observational studies- EUROPEAN GERIATRIC MEDICINE cilt.10 ss.853-862 2019 [Veronese N. Demurtas J. SOYSAL P. Smith L. Torbahn G. Schoene D. Schwingshackl L. Sieber C. Bauer J. Cesari M. et al. -Sarcopenia and health-related outcomes]Psychological intervention03 medical and health sciencesUmbrella review0302 clinical medicinemedicineFallClinical significanceMeta-analysi030212 general & internal medicineMortalityDisability030214 geriatricsbusiness.industryHealth relatedOdds ratiomedicine.diseaseConfidence intervalIncreased riskHealthSarcopeniaObservational studyRisk factorbusinesshuman activities
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Loredana Foti, Rita: Giudici e Corsari nel Mediterraneo. Il Tribunale delle prede di Sicilia 1808-1813

2018

Giulia 299 301Revista de historia moderna 500918 2018 44 6623964 Loredana Foti [0210-9093 553 Estudis]UNESCO::HISTORIA0210-9093 553 Estudis: Revista de historia moderna 500918 2018 44 6623964 Loredana FotiRita: Giudici e Corsari nel Mediterraneo. Il Tribunale delle prede di Sicilia 1808-1813 BonazzaGiudici e Corsari nel Mediterraneo. Il Tribunale delle prede di Sicilia 1808-1813 Bonazza [Rita]:HISTORIA [UNESCO]
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