Search results for "ABNORMALITIES"

showing 10 items of 638 documents

Phenotypic analysis of individuals with Costello syndrome due to HRAS p.G13C.

2011

Costello syndrome is characterized by severe failure-to-thrive, short stature, cardiac abnormalities (heart defects, tachyarrhythmia, and hypertrophic cardiomyopathy (HCM)), distinctive facial features, a predisposition to papillomata and malignant tumors, postnatal cerebellar overgrowth resulting in Chiari 1 malformation, and cognitive disabilities. De novo germline mutations in the proto-oncogene HRAS cause Costello syndrome. Most mutations affect the glycine residues in position 12 or 13, and more than 80% of patients share p.G12S. To test the hypothesis that subtle genotype-phenotype differences exist, we report the first cohort comparison between 12 Costello syndrome individuals with p…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyAdolescentrasopathy.RASopathyShort statureProto-Oncogene MasArticleProto-Oncogene Proteins p21(ras)Young AdultGermline mutationSettore MED/38 - Pediatria Generale E SpecialisticaCostello syndromePregnancyInternal medicineNeoplasmsGeneticsMedicineHumansHRASChildGenetics (clinical)business.industryloose anagen hairCostello SyndromeMacrocephalyHypertrophic cardiomyopathyBrainInfantgenotype–phenotype correlationmedicine.diseaseDermatologyMagnetic Resonance ImagingMusculoskeletal AbnormalitiesEndocrinologyPhenotypeChild PreschoolFaceMutationFemalemedicine.symptombusinessMultifocal atrial tachycardiaAmerican journal of medical genetics. Part A
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Photoplethysmography Analysis of Artery Properties in Patients with Cardiovascular Diseases

2008

In this study arterial parameters of healthy subjects were compared to those of patients with cardiovascular diseases. The photoplethysmography (PPG) measurements of blood volume pulsations have been performed. Using a novel algorithm for analysis of simultaneously measured ear and finger PPG signals, arterial parameters were evaluated in representative groups of healthy subjects and patients with cardiovascular diseases. Digital volume pulse (DVP), pulse cycle duration (T), augmentation index (AIx), reflection index (RI) and transit time of reflected wave (RTT) were evaluated in every heartbeat cycle. Correlations between the AIx and RI, T and RTT, AIx and standard deviation of AIx, RTT an…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyHeartbeatPulse (signal processing)business.industryTransit timeBlood volumemedicine.diseasemedicine.anatomical_structureInternal medicinePhotoplethysmogrammedicineArterial stiffnessCardiologyIn patientbusinessArtery
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Correction: Daunorubicin reduces MBNL1 sequestration caused by CUG-repeat expansion and rescues cardiac dysfunctions in a Drosophila model of myotoni…

2018

ABSTRACT Myotonic dystrophy (DM) is a dominantly inherited neuromuscular disorder caused by expression of mutant myotonin-protein kinase (DMPK) transcripts containing expanded CUG repeats. Pathogenic DMPK RNA sequesters the muscleblind-like (MBNL) proteins, causing alterations in metabolism of various RNAs. Cardiac dysfunction represents the second most common cause of death in DM type 1 (DM1) patients. However, the contribution of MBNL sequestration in DM1 cardiac dysfunction is unclear. We overexpressed Muscleblind (Mbl), the Drosophila MBNL orthologue, in cardiomyocytes of DM1 model flies and observed a rescue of heart dysfunctions, which are characteristic of these model flies and resem…

congenital hereditary and neonatal diseases and abnormalitiesRNA StabilityNeuroscience (miscellaneous)Medicine (miscellaneous)MuscleblindGeneral Biochemistry Genetics and Molecular BiologyImmunology and Microbiology (miscellaneous)AnimalsDrosophila ProteinsMyotonic DystrophyMyocytes CardiacRNA MessengerDaunorubicinCorrectionNuclear ProteinsReproducibility of ResultsHeartSurvival AnalysisAlternative SplicingDisease Models AnimalDrosophila melanogasterTrinucleotide repeat disorderDrosophilaTrinucleotide Repeat ExpansionResearch ArticleProtein BindingDisease Models & Mechanisms
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The aristaless (Arx) gene: one gene for many "interneuronopathies".

2009

The ARX (Aristaless-related (X-linked) homeobox) gene is not only present in arthropods and their ancestors, but also in vertebrates including humans (ARX orthologs). The gene is composed of 5 coding exons and it is expressed predominantly in foetal and adult brain and skeletal muscle. In this review we report on our experience and review the existing literature on the genotype and phenotype heterogeneity associated with ARX abnormalities in humans ranging from severe neuronal migration defects (e.g., lissencephaly), to mild forms of X-linked mental retardation without apparent brain abnormalities. The ARX-related disorders are reviewed focusing on their clinical features and on the role of…

Doublecortin ProteinGenotypeLissencephalyBiologyNeuronal migration defectsGeneral Biochemistry Genetics and Molecular BiologyExonMiceGenotype-phenotype distinctionSettore MED/38 - Pediatria Generale E SpecialisticaInterneuronsmedicineAnimalsHumansAbnormalities MultipleGeneZebrafishGeneticsHomeodomain ProteinsGeneral Immunology and MicrobiologyARX homeoboxmedicine.diseasePhenotypeCranial Nerve DiseasesPhenotypeMultigene FamilyMental Retardation X-LinkedHomeoboxAbnormalityTranscription FactorsFrontiers in bioscience (Elite edition)
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Subclinical executive function impairment in children with asymptomatic, treated phenylketonuria: A comparison with children with immunodeficiency vi…

2018

In this study we compared the neuropsychological profile of phenylketonuria (PKU) and human immunodeficiency virus (HIV) to examine the specificity of the executive function (EF) impairment reported in these two patologies. A total of 55 age-matched children and adolescents were assessed, including 11 patients with PKU, 16 patients with HIV and 28 healthy controls, underwent a neuropsychological assessment. Although neither the PKU nor the HIV group scored below the normative ranges, both groups showed lower scores in neuropsychological tests engaging EFs than controls. In addition, compared to patients with PKU the HIV group performed significantly worse in the Trail-Making Test A, Corsi S…

Malecongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtyAdolescentPhenylketonuriasprefrontal lobeCognitive NeurosciencephenylketonuriaExperimental and Cognitive PsychologyNeuropsychological TestsAsymptomatic050105 experimental psychologyDevelopmental psychologySettore M-PSI/04 - Psicologia Dello Sviluppo E Psicologia Dell'Educazione03 medical and health sciencesExecutive Function0302 clinical medicineArts and Humanities (miscellaneous)PhenylketonuriasmedicineDevelopmental and Educational PsychologyVerbal fluency testHumans0501 psychology and cognitive sciencesNeuropsychological assessmentChildSubclinical infectionSettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicamedicine.diagnostic_testWorking memory05 social sciencesNeuropsychologynutritional and metabolic diseasesHIVHIV phenylketonuria executive functions prefrontal lobe.Executive functionsexecutive functionsNeuropsychology and Physiological PsychologyFemalemedicine.symptomexecutive functions; HIV; phenylketonuria; prefrontal lobe; Adolescent; Child; Executive Function; Female; Humans; Male; Neuropsychological Tests; Phenylketonurias; Neuropsychology and Physiological Psychology; Experimental and Cognitive Psychology; Developmental and Educational Psychology; Arts and Humanities (miscellaneous); Cognitive NeurosciencePsychology030217 neurology & neurosurgery
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Psychiatric and neurological symptoms in patients with Niemann-Pick disease type C (NP-C): Findings from the International NPC Registry

2017

Objectives: Niemann-Pick disease type C (NP-C) is a rare inherited neurovisceral disease that should be recognised by psychiatrists as a possible underlying cause of psychiatric abnormalities. This...

AdultMalePsychosismedicine.medical_specialtyPediatricsInternationalityAdolescentDiseaseYoung Adult03 medical and health sciences0302 clinical medicinehemic and lymphatic diseasesmedicinePsychiatric abnormalitiesHumansIn patientProspective StudiesRegistriesAge of OnsetChildPsychiatryBiological PsychiatryAgedNiemann–Pick disease type Cbusiness.industryMental Disordersnutritional and metabolic diseasesNiemann-Pick Disease Type CMiddle Agedmedicine.disease030227 psychiatryPsychiatry and Mental healthSchizophreniaChild PreschoolFemalebusinessThe World Journal of Biological Psychiatry
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Efficacy and safety of Wilate in paediatric VWD patients under 6 years of age - results of a prospective multicentre clinical study including recover…

2013

Treatment with exogenous von Willebrand factor (VWF) is indicated in patients with von Willebrand disease (VWD) in whom treatment with 1-deamino-8-d-arginine vasopressin/desmopressin is contraindicated. Wilate is a new generation plasma-derived concentrate of native VWF and coagulation factor VIII (FVIII) (in a physiological 1:1 ratio) developed for the treatment of VWD. This is the first study to report safety, efficacy and in vivo recovery (IVR) data from 15 paediatric patients less than 6 years of age who received Wilate for either prophylaxis, on-demand treatment or for treatment in surgical procedures during a prospective open-label trial (VWD type 1: 5, type 2A: 1, type 2B: 2, type 3:…

Malecongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtyHemorrhageClinical studyVon Willebrand factorhemic and lymphatic diseasesvon Willebrand FactormedicineVon Willebrand diseaseHumansProspective StudiesChildDesmopressinGenetics (clinical)Paediatric patientsBleeding episodesFactor VIIIbiologyCoagulantsbusiness.industryInfantHematologyGeneral Medicinemedicine.diseasevon Willebrand DiseasesPhenotypeCoagulationTolerabilityChild Preschoolbiology.proteinFemalebusinessHalf-Lifemedicine.drugHaemophilia
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Abnormal Somatosensory Evoked Potentials Indicate Compressive Cervical Myelopathy in Mucopolysaccharidoses

2000

Compressive myelopathy at the cranio-cervical junction is a complication of mucopolysaccharidoses (MPS). To detect cervical myelopathy we recorded median and posterior tibial nerve SEPs in 15 patients aged 2.4 - 33.4 years (median 8.8 years) with MPS I-S (n = 3), MPS IVA (n = 8) and MPS VI (n = 4). In addition to the cortical waveforms we recorded the subcortical median nerve SEPs N13b and P13 generated near the cranio-cervical junction and the lemniscal P30 after posterior tibial nerve stimulation. MRI studies in 13 subjects revealed spinal cord compression at the cranio-cervical junction in 10 patients; 5 patients had an increased signal intensity on the T2-weighted initial MRI indicating…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAdolescentSensitivity and SpecificityCentral nervous system diseaseMyelopathySpinal cord compressionEvoked Potentials SomatosensorymedicineHumansChildbusiness.industryGeneral MedicineCervical cord compressionMucopolysaccharidosesmedicine.diseaseSpinal cordMagnetic Resonance ImagingMedian nerveMedian NerveSurgerybody regionsmedicine.anatomical_structureSpinal CordSomatosensory evoked potentialChild PreschoolPediatrics Perinatology and Child HealthFemaleNeurology (clinical)RadiologyTibial NervebusinessSpinal Cord CompressionMyelomalaciaNeckNeuropediatrics
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Novel <b><i>VANGL1</i></b> Gene Mutations in 144 Slovakian, Romanian and German Patients with Neural Tube Defects

2012

Neural tube defects (NTDs) are a group of congenital malformations of the central nervous system occurring at an average rate of 1 per 1,000 human pregnancies worldwide. Numerous genetic and environmental factors are discussed to be relevant in their etiology. In mice, mutants in >200 genes including the planar cell polarity (PCP) pathway are known to cause NTDs, and recently, heterozygous mutations in the human <i>VANGL1</i> gene have been described in a small subset of patients with NTDs. We performed a <i>VANGL1</i> mutation analysis in 144 unrelated individuals with NTDs from Slovakia, Romania and Germany and identified 3 heterozygous missense mutations: c.613…

Geneticscongenital hereditary and neonatal diseases and abnormalitiesMutationbusiness.industryMutantCentral nervous systemNeural tubeGene mutationmedicine.disease_causemedicine.anatomical_structureGeneticsMutation testingmedicineMissense mutationbusinessGeneGenetics (clinical)Molecular Syndromology
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The severe phenotype of Diamond-Blackfan anemia is modulated by heat shock protein 70.

2017

International audience; Diamond-Blackfan anemia (DBA) is a rare congenital bone marrow failure syndrome that exhibits an erythroid-specific phenotype. In at least 70% of cases, DBA is related to a haploinsufficient germ line mutation in a ribosomal protein (RP) gene. Additional cases have been associated with mutations in GATA1. We have previously established that the RPL11+/Mut phenotype is more severe than RPS19+/Mut phenotype because of delayed erythroid differentiation and increased apoptosis of RPL11+/Mut erythroid progenitors. The HSP70 protein is known to protect GATA1, the major erythroid transcription factor, from caspase-3 mediated cleavage during normal erythroid differentiation.…

0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesIdentificationApoptosis-Inducing FactorGata1 MutationsInhibits ApoptosisBiologyHsp7003 medical and health sciencesGermline mutationRed Cells Iron and Erythropoiesishemic and lymphatic diseasesmedicine[ SDV.MHEP.HEM ] Life Sciences [q-bio]/Human health and pathology/HematologyNuclear ImportErythropoiesisDiamond–Blackfan anemiaHuman ErythroblastsBone marrow failure[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/HematologyGATA1Hematologymedicine.diseasePhenotypeMolecular biology3. Good healthHsp70030104 developmental biologyRibosomal-ProteinsProtein Gene DeletionsErythropoiesisHaploinsufficiencyBlood advances
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