Search results for "ADENOCARCINOMA"

showing 10 items of 589 documents

Identification of a tumor-reactive T-cell repertoire in the immune infiltrate of patients with resectable pancreatic ductal adenocarcinoma

2016

The devastating prognosis of patients with resectable pancreatic ductal adenocarcinoma (PDA) presents an urgent need for the development of therapeutic strategies targeting disseminated tumor cells. Until now, T-cell therapy has been scarcely pursued in PDA, due to the prevailing view that it represents a poorly immunogenic tumor.We systematically analyzed T-cell infiltrates in tumor biopsies from 127 patients with resectable PDA by means of immunohistochemistry, flow cytometry, T-cell receptor (TCR) deep-sequencing and functional analysis ofProminent T-cell infiltrates, as well as tertiary lymphoid structures harboring proliferating T-cells, were detected in the vast majority of biopsies f…

0301 basic medicinePathologymedicine.medical_specialtyT cell repertoirePancreatic ductal adenocarcinomaTertiary Lymphoid StructuresTumor-infiltrating lymphocyteseducationImmunologyBiology03 medical and health sciences030104 developmental biology0302 clinical medicineOncology030220 oncology & carcinogenesisMIATA Compliant Research PapermedicineImmunology and AllergyImmune infiltrateOncoImmunology
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TTF-1/p63-Positive Poorly Differentiated NSCLC: A Histogenetic Hypothesis from the Basal Reserve Cell of the Terminal Respiratory Unit

2020

TTF-1 is expressed in the alveolar epithelium and in the basal cells of distal terminal bronchioles. It is considered the most sensitive and specific marker to define the adenocarcinoma arising from the terminal respiratory unit (TRU). TTF-1, CK7, CK5/6, p63 and p40 are useful for typifying the majority of non-small-cell lung cancers, with TTF and CK7 being typically expressed in adenocarcinomas and the latter three being expressed in squamous cell carcinoma. As tumors with coexpression of both TTF-1 and p63 in the same cells are rare, we describe different cases that coexpress them, suggesting a histogenetic hypothesis of their origin. We report 10 cases of poorly differentiated non-small-…

0301 basic medicinePathologymedicine.medical_specialtyendocrine systemAlveolar EpitheliumClinical Biochemistryhistogenetic hypothesisBiologyNSCLCArticle03 medical and health sciencesBasal (phylogenetics)0302 clinical medicineterminal respiratory unitmedicineCarcinomabasal reserve cellslcsh:R5-920p63LungBasal reserve cellCancerrespiratory systemmedicine.disease030104 developmental biologymedicine.anatomical_structurenon-small-cell lung cancerTTF-1030220 oncology & carcinogenesisAdenocarcinomaImmunohistochemistrylcsh:Medicine (General)ImmunostainingHistogenetic hypothesiDiagnostics
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Non-interventional LUME-BioNIS study of nintedanib plus docetaxel after chemotherapy in adenocarcinoma non-small cell lung cancer: A subgroup analysi…

2020

Abstract Objectives To evaluate the effectiveness and safety of nintedanib plus docetaxel in patients with advanced adenocarcinoma non-small cell lung cancer (NSCLC) previously treated with both chemo- and immunotherapy. Materials and methods LUME-BioNIS is a European, prospective, multicenter, non-interventional study of patients with advanced adenocarcinoma NSCLC, who initiated nintedanib plus docetaxel after first-line chemotherapy in routine practice according to the approved nintedanib EU label. The primary objective is to explore whether molecular biomarkers can predict overall survival (OS). Information on clinical or radiologic progression and death, and adverse drug reactions (ADRs…

0301 basic medicinePulmonary and Respiratory MedicineOncologyCancer Researchmedicine.medical_specialtyIndolesLung Neoplasmsmedicine.medical_treatmentSubgroup analysisPembrolizumabDocetaxelAdenocarcinoma03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAtezolizumabInternal medicineCarcinoma Non-Small-Cell LungAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansProspective StudiesLung cancerChemotherapybusiness.industrymedicine.disease030104 developmental biologyTreatment OutcomeOncologychemistryDocetaxel030220 oncology & carcinogenesisNintedanibTaxoidsImmunotherapyNivolumabbusinessmedicine.drugLung cancer (Amsterdam, Netherlands)
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Genome-Wide DNA Methylation Profiling in Early Stage I Lung Adenocarcinoma Reveals Predictive Aberrant Methylation in the Promoter Region of the Long…

2020

Introduction: Surgical procedure is the treatment of choice in early stage I lung adenocarcinoma. However, a considerable number of patients experience recurrence within the first 2 years after complete resection. Suitable prognostic biomarkers that identify patients at high risk of recurrence (who may probably benefit from adjuvant treatment) are still not available. This study aimed at identifying methylation markers for early recurrence that may become important tools for the development of new treatment modalities. Methods: Genome-wide DNA methylation profiling was performed on 30 stage I lung adenocarcinomas, comparing 14 patients with early metastatic recurrence with 16 patients with …

0301 basic medicinePulmonary and Respiratory MedicineOncologyLung adenocarcinomamedicine.medical_specialtyLung NeoplasmsADNAdenocarcinoma of LungMethylation profilingmedicine.disease_causeMethylation03 medical and health sciences0302 clinical medicinePrognostic markerPLUTInternal medicineBiomarkers TumorHumansMedicineddc:610Promoter Regions Geneticbusiness.industryHazard ratioPromoterMethylationDNADNA MethylationPrognosismedicine.diseaseLong non-coding RNA030104 developmental biologyDifferentially methylated regionsOncology030220 oncology & carcinogenesisIncRNACàncer de pulmóBiomarker (medicine)AdenocarcinomaRNA Long NoncodingNeoplasm Recurrence LocalLung cancerbusinessCarcinogenesisMetilació
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SNVSniffer: an integrated caller for germline and somatic single-nucleotide and indel mutations

2016

Various approaches to calling single-nucleotide variants (SNVs) or insertion-or-deletion (indel) mutations have been developed based on next-generation sequencing (NGS). However, most of them are dedicated to a particular type of mutation, e.g. germline SNVs in normal cells, somatic SNVs in cancer/tumor cells, or indels only. In the literature, efficient and integrated callers for both germline and somatic SNVs/indels have not yet been extensively investigated. We present SNVSniffer, an efficient and integrated caller identifying both germline and somatic SNVs/indels from NGS data. In this algorithm, we propose the use of Bayesian probabilistic models to identify SNVs and investigate a mult…

0301 basic medicineSomatic cellBayesian probabilityBiologyPolymorphism Single NucleotideGermline03 medical and health sciencesGene FrequencyINDEL MutationStructural BiologyModelling and SimulationIndel callingGenetic variationHumansAlleleIndelMolecular BiologyOvarian NeoplasmsGeneticsResearchApplied MathematicsComputational BiologyHigh-Throughput Nucleotide SequencingSNP callingSomatic SNV callingCystadenocarcinoma SerousComputer Science ApplicationsGerm Cells030104 developmental biologyBayesian modelModeling and SimulationMutation (genetic algorithm)FemaleMultinomial distributionAlgorithmsBMC Systems Biology
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Characterizing diversity in the tumor-immune microenvironment of distinct subclasses of gastroesophageal adenocarcinomas

2020

Background Gastroesophageal adenocarcinomas (GEAs) are heterogeneous cancers where immune checkpoint inhibitors have robust efficacy in heavily inflamed microsatellite instability (MSI) or Epstein-Barr virus (EBV)-positive subtypes. Immune checkpoint inhibitor responses are markedly lower in diffuse/genome stable (GS) and chromosomal instable (CIN) GEAs. In contrast to EBV and MSI subtypes, the tumor microenvironment of CIN and GS GEAs have not been fully characterized to date, which limits our ability to improve immunotherapeutic strategies. Patients and methods Here we aimed to identify tumor-immune cell association across GEA subclasses using data from The Cancer Genome Atlas (N = 453 GE…

0301 basic medicineT cellmedicine.medical_treatmentAdenocarcinomaArticle03 medical and health sciences0302 clinical medicineImmune systemStomach NeoplasmsTumor MicroenvironmentMedicineHumansTumor microenvironmentbusiness.industryMicrosatellite instabilityHematologyImmunotherapyCell cyclemedicine.diseaseImmunohistochemistry030104 developmental biologymedicine.anatomical_structureOncology030220 oncology & carcinogenesisCancer researchAdenocarcinomaMicrosatellite InstabilitybusinessCD8
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An in vitro investigation on the cytotoxic and nuclear receptor transcriptional activity of the mycotoxins fumonisin B1 and beauvericin.

2016

Fumonisin B1 (FB1) and beauvericin (BEA) are secondary metabolites of filamentous fungi, which under appropriate temperature and humidity conditions may develop on various foods and feeds. To date few studies have been performed to evaluate the toxicological and endocrine disrupting effects of FB1 and BEA. The present study makes use of various in vitro bioassays including; oestrogen, androgen, progestagen and glucocorticoid reporter gene assays (RGAs) for the study of nuclear receptor transcriptional activity, the thiazolyl blue tetrazolium bromide (MTT) assay to monitor cytotoxicity and high content analysis (HCA) for the detection of pre-lethal toxicity in the RGA and Caco-2 human colon …

0301 basic medicineTranscription GeneticCell SurvivalBiologyAdenocarcinomaEndocrine DisruptorsToxicologyFumonisins03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyGlucocorticoid receptorReceptors GlucocorticoidGenes ReporterDepsipeptidesmedicineHumansCytotoxicityReceptorCell NucleusFumonisin B1Dose-Response Relationship Drug04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceBeauvericin030104 developmental biologychemistryNuclear receptorBiochemistryReceptors AndrogenToxicityColonic NeoplasmsCaco-2 CellsReceptors ProgesteroneGlucocorticoidmedicine.drugToxicology letters
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Chloroquine plays a cell-dependent role in the response to treatment of pancreatic adenocarcinoma

2018

In this study, our aim is to assess the role played by autophagy and its inhibition in the different PDAC cellular compartments, and its involvement in chemo-resistance using primary human pancreatic cancer-derived cells (PCC) and Cancer Associated Fibroblasts (CAF). Autophagy flux, as measured by LC3-I and -II in the presence of Chloroquine, showed a variable level in PCC and CAFs. We found no correlation between autophagy level and degree of tumor differentiation. Association of Chloroquine with gemcitabine, 5FU, oxaliplatin, irinotecan and docetaxel revealed that its effect on survival is cell- and drug-dependent in vitro and in vivo. In addition, we demonstrated that autophagy in CAFs c…

0301 basic medicineautophagyCIENCIAS MÉDICAS Y DE LA SALUDCiencias de la Salud//purl.org/becyt/ford/3.3 [https]03 medical and health sciences0302 clinical medicinepancreas cancerChloroquineMedicineCHLOROQUINEbusiness.industryAutophagygemcitabineCancerChloroquinemedicine.diseaseGemcitabineOxaliplatinOtras Ciencias de la SaludIrinotecanPANCREAS CANCER030104 developmental biologyGEMCITABINEOncologyDocetaxel030220 oncology & carcinogenesisCancer researchAdenocarcinomaAUTOPHAGY//purl.org/becyt/ford/3 [https]businessResearch Papermedicine.drugOncotarget
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Extracellular Superoxide Dismutase Expression in Papillary Thyroid Cancer Mesenchymal Stem/Stromal Cells Modulates Cancer Cell Growth and Migration

2017

AbstractTumor stroma-secreted growth factors, cytokines, and reactive oxygen species (ROS) influence tumor development from early stages to the metastasis phase. Previous studies have demonstrated downregulation of ROS-producing extracellular superoxide dismutase (SOD3) in thyroid cancer cell lines although according to recent data, the expression of SOD3 at physiological levels stimulates normal and cancer cell proliferation. Therefore, to analyze the expression of SOD3 in tumor stroma, we characterized stromal cells from the thyroid. We report mutually exclusive desmoplasia and inflammation in papillary and follicular thyroid cancers and the presence of multipotent mesenchymal stem/stroma…

0301 basic medicineendocrine systemPathologymedicine.medical_specialtyStromal cellendocrine system diseasesThyroid GlandBiologyArticleMetastasisPapillary thyroid cancer03 medical and health sciences0302 clinical medicineCell MovementExtracellular ;Thyroid ;Cancer ;Cell .Adenocarcinoma FollicularParacrine CommunicationBiomarkers TumormedicineHumansThyroid NeoplasmsThyroid cancerCell ProliferationMultidisciplinarySuperoxide DismutaseMesenchymal stem cellThyroidEpithelial CellsMesenchymal Stem Cellsmedicine.diseaseFibrosisCarcinoma PapillaryDesmoplasiaGene Expression Regulation NeoplasticPhenotype030104 developmental biologymedicine.anatomical_structureThyroid Cancer Papillary030220 oncology & carcinogenesisCancer cellmedicine.symptomExtracellular SpaceScientific Reports
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Proton Pump Inhibitors Display Antitumor Effects in Barrett's Adenocarcinoma Cells

2016

Recent evidence has reported that proton pump inhibitors (PPIs) can exert antineoplastic effects through the disruption of pH homeostasis by inhibiting vacuolar ATPase (H+-VATPase), a proton pump overexpressed in several tumor cells, but this aspect has not been deeply investigated in EAC yet. In the present study, the expression of H+-VATPase was assessed through the metaplasia-dysplasia-adenocarcinoma sequence in Barrett’s esophagus (BE) and the antineoplastic effects of PPIs and cellular mechanisms involved were evaluated in vitro. H+-VATPase expression was assessed by immunohistochemistry in paraffined-embedded samples or by immunofluorescence in cultured BE and EAC cell lines. Cells we…

0301 basic medicineesophageal adenocarcinomaIntracellular pHvacuolar ATPaseBiologymedicine.disease_causeBarrett's esophagus03 medical and health sciencesmedicineBarrett’s esophagusCytotoxic T cellPharmacology (medical)Original Researchreactive oxygen speciesPharmacologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950AutophagyProton Pump InhibitorsIn vitrolcsh:Therapeutics. Pharmacology030104 developmental biologyBiochemistrychemistryCell cultureApoptosisCancer researchEsophageal adenocarcinomaproton pump inhibitorsReactive Oxygen SpeciesOxidative stressFrontiers in Pharmacology
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