Search results for "ALPHA"

showing 10 items of 3228 documents

Changes in different salivary biomarkers related to physiologic stress in elite handball players: the case of females

2019

AbstractThis study evaluates pre- and post-match concentrations of salivary cortisol, alpha-amylase (AA) and immunoglobulin A (lgA) in a group of 21 elite female handball players in the Spanish national women’s league. The players’ mean age was 23.0 ± 5.4 years. The concentration of the biomarkers was determined using ELISA. Mean playing time was 25.2 min. The players’ cortisol concentration increased significantly (p < 0.05) whereas the IgA concentration fell significantly (p < 0.01) at the end of the match. There were no significant changes in the concentrations of AA between pre and post-match timepoints. The changes observed in the study also depended on the position played; defen…

AdultImmunoglobulin AAdolescentHydrocortisonelcsh:MedicineAdrenergicPhysiologyAthletic PerformanceArticleYoung Adult03 medical and health sciencesApplied immunology0302 clinical medicineStress PhysiologicalHumansMedicinelcsh:ScienceSalivaSalivary biomarkersSalivary cortisolMultidisciplinarybiologybusiness.industrylcsh:RDiagnostic markersMean age030229 sport sciencesImmunoglobulin Abiology.proteinlcsh:QFemalealpha-AmylasesImmunocompetencebusinessBiomarkers030217 neurology & neurosurgeryScientific Reports
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High prevalence of polycystic ovary syndrome in women with mild hirsutism and no other significant clinical symptoms

2010

Objective To verify the conclusions of the Endocrine Society Guidelines that patients with mild hirsutism and no other important clinical signs (menstrual irregularities, infertility, central obesity, acanthosis nigricans, rapid progression of the hirsutism, clitoromegaly) should not be further studied. Design Retrospective study in patients referred because of mild hirsutism and no other clinical signs. Setting Department of Clinical Medicine of the University of Palermo. Patient(s) One hundred fifty-two patients with mild hirsutism. Intervention(s) Measurement of serum testosterone, dehydroepiandrosterone sulfate, 17-OH-Progesterone, assessment of ovulation by measurement of progesterone …

AdultInfertilityHirsutismmedicine.medical_specialtyPediatricsSettore MED/09 - Medicina Internaendocrine system diseasesmedia_common.quotation_subjectClitoromegalyYoung Adultchemistry.chemical_compoundDehydroepiandrosterone sulfatePrevalenceHumansMedicineOvulationAcanthosis nigricanshirsutismRetrospective Studiesmedia_commonGynecologybusiness.industry17-alpha-HydroxyprogesteroneHyperandrogenismObstetrics and Gynecologymedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsReproductive Medicinechemistrypolycystic ovary syndromeFemalemedicine.symptombusiness
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ICAM-1 and α3β1 expression by bronchial epithelial cells and theirin vitromodulation by inflammatory and anti-inflammatory mediators

2000

Background: Adhesion molecules are involved in inflammatory and repair processes of the bronchial epithelium. ICAM-1 is mainly involved in inflammatory reactions, whereas integrins, such as α3β1, are mainly involved in repair processes. Methods: Using bronchial biopsies from 10 asthmatics and eight controls, we first evaluated by immunohistochemistry expression of α3β1 and ICAM-1 in intact and damaged epithelium. Then, using the human pulmonary epithelial cell line WI-26 VA, we studied, by flow-cytometry, the modulation of ICAM-1 and α3β1 expression, and, by ELISA, the release of fibronectin by proinflammatory cytokines, such as IL-5, and anti-inflammatory cytokines, such as IL-4, TGF-β, an…

AdultIntegrinsAdolescentBiopsyImmunologyIntegrinIntercellular Adhesion Molecule-1BronchiEnzyme-Linked Immunosorbent AssayInflammationRespiratory MucosaCell LineProinflammatory cytokineTransforming Growth Factor betamedicineHumansImmunology and AllergyAgedInflammationICAM-1Epidermal Growth FactorbiologyCell adhesion moleculeIntegrin alpha3beta1Epithelial CellsMiddle AgedFlow CytometryIntercellular Adhesion Molecule-1Molecular biologyAsthmaEpitheliumFibronectinsFibronectinmedicine.anatomical_structureImmunologybiology.proteinCytokinesInterleukin-4medicine.symptomAllergy
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Activation of cutaneous immune responses in complex regional pain syndrome

2014

The pathogenesis of complex regional pain syndrome (CRPS) is unresolved, but tumor ne- crosis factor alpha (TNF-a) and interleukin-6 (IL-6) are elevated in experimental skin blister fluid from CRPS-affected limbs, as is tryptase, a marker for mast cells. In the rat fracture model of CRPS, exag- gerated sensory and sympathetic neural signaling stimulate keratinocyte and mast cell proliferation, causing the local production of high levels of inflammatory cytokines leading to pain behavior. The current investigation used CRPS patient skin biopsies to determine whether keratinocyte and mast cell proliferation occur in CRPS skin and to identify the cellular source of the up-regulated TNF-a, IL-6…

AdultKeratinocytesMaleBiopsyTryptaseArticleMast cell proliferationProinflammatory cytokineYoung AdultSkin Physiological PhenomenamedicineHumansMast CellsAgedCell ProliferationSkinSkin Physiological Phenomenabiologyintegumentary systembusiness.industryInterleukin-6Tumor Necrosis Factor-alphaOrgan SizeMiddle Agedmedicine.diseaseMast cellAnesthesiology and Pain MedicineComplex regional pain syndromemedicine.anatomical_structureNeurologyImmunologybiology.proteinTumor necrosis factor alphaFemaleNeurology (clinical)EpidermisKeratinocytebusinessComplex Regional Pain Syndromes
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Lack of Hypoxic Response in Uterine Leiomyomas despite Severe Tissue Hypoxia

2008

Abstract Hypoxia is now established as a key factor influencing the pathophysiology of malignant growth. Among other effects, hypoxia modulates the expression of a multitude of genes through the induction of hypoxia-inducible transcription factors. This differential gene expression favors angiogenesis, cell survival, an invasive/metastatic phenotype, and resistance to anticancer therapies. Because benign tumors do not exhibit these traits, one might expect these entities to be neither hypoxic nor to induce the genetic hypoxia response program. To test this hypothesis, an investigation of the oxygenation status of 17 leiomyomas and 1 leiomyosarcoma of the uterus using polarographic needle el…

AdultLeiomyosarcomaLeiomyosarcomaCancer ResearchPathologymedicine.medical_specialtyAngiogenesisUterusUterine Cervical NeoplasmsApoptosisBiologyImmunoenzyme TechniquesAntigens NeoplasmBiopsyBasic Helix-Loop-Helix Transcription FactorsIn Situ Nick-End LabelingmedicineHumansProspective StudiesCarbonic Anhydrase IXHypoxiaCarbonic AnhydrasesCell ProliferationGlucose Transporter Type 1Uterine leiomyomaLeiomyomamedicine.diagnostic_testMiddle AgedHypoxia (medical)Hypoxia-Inducible Factor 1 alpha Subunitmedicine.diseasePhenotypeOxygenmedicine.anatomical_structureLeiomyomaOncologyMyometriumFemalemedicine.symptomCancer Research
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Expression of the obesity hormone leptin and its receptor correlates with hypoxia-inducible factor-1 alpha in human colorectal cancer.

2007

Background: The obesity hormone, leptin, has been found to play a role in development and proliferation of normal and malignant tissues. Leptin activity is mediated through the leptin receptor (ObR) that is often expressed in different human cancer cells. Previously, we found that the expression of leptin and ObR can be stimulated by hypoxia-mimetic agents. The aim of this study was to analyze the abundance of and relationships among leptin, ObR and hypoxia-inducible factor-1α (HIF-1α, transcriptional regulator) in human colorectal cancer. Materials and methods: We investigated the expression of leptin, ObR and HIF-1 a in colorectal cancer specimens from 135 patients who underwent curative …

AdultLeptinMalemedicine.medical_specialtyColorectal cancerAdipokineReceptors Cell SurfaceInternal medicinemedicineHumansObesityReceptorAgedAged 80 and overLeptin receptorbusiness.industryLeptindigestive oral and skin physiologyHematologyMiddle Agedmedicine.diseaseHypoxia-Inducible Factor 1 alpha SubunitObesityUp-RegulationGene Expression Regulation NeoplasticEndocrinologyOncologyHypoxia-inducible factorsDisease ProgressionReceptors LeptinFemalebusinessColorectal Neoplasmshormones hormone substitutes and hormone antagonistsHormoneAnnals of oncology : official journal of the European Society for Medical Oncology
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Leptin and leptin receptor expression in asthma.

2009

Background The adipokine leptin is a potential new mediator for bronchial epithelial homeostasis. Asthma is a chronic inflammatory disease characterized by airway remodeling that might affect disease chronicity and severity. TGF-β is a tissue growth factor the dysregulation of which is associated with airway remodeling. Objective We sought to determine whether a bronchial epithelial dysfunction of the leptin/leptin receptor pathway contributes to asthma pathogenesis and severity. Methods We investigated in vitro the presence of leptin/leptin receptor on human bronchial epithelial cells. Then we studied the effect of TGF-β and fluticasone propionate on leptin receptor expression. Finally, th…

AdultLeptinMalemedicine.medical_specialtyPyridinesMorpholinesImmunologyAdipokineBronchiRespiratory MucosaSettore MED/10 - Malattie Dell'Apparato RespiratorioSettore BIO/09 - FisiologiaCell LinePathogenesisTransforming Growth Factor beta1Leptin leptin receptor severe asthma epithelium TGF-b remodelingInternal medicineImmunology and AllergyMedicineHumansEnzyme InhibitorsReceptorCell ProliferationLeptin receptorbusiness.industryTumor Necrosis Factor-alphaLeptindigestive oral and skin physiologyImidazolesMiddle AgedEpitheliumAsthmaRecombinant Proteinsrespiratory tract diseasesAndrostadienesmedicine.anatomical_structureEndocrinologyChromonesImmunologyFluticasoneReceptors LeptinFemalebusinesshormones hormone substitutes and hormone antagonistsEx vivoTransforming growth factorThe Journal of allergy and clinical immunology
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Combined aerobic and resistance training decreases inflammation markers in healthy men

2017

Our primary aim was to study the effects of 24 weeks of combined aerobic and resistance training performed on the same day or on different days on inflammation markers. Physically active, healthy young men were randomly divided into three groups that performed: aerobic and resistance training consecutively in the same training session (SS) 2-3 days wk-1 or on alternating days (AD) 4-6 days wk-1 as well as control (C). The total training volume was matched in the training groups. The control group was asked to maintain their habitual physical activity and exercise level. Maximal leg press strength (1RM) and peak oxygen uptake (VO2peak ) were measured. Abdominal fat mass was estimated with du…

AdultLeptinMalemedicine.medical_specialtytulehdusarvotAdipokinePhysical Therapy Sports Therapy and RehabilitationPhysical exercise030204 cardiovascular system & hematologyliikunta03 medical and health sciences0302 clinical medicineOxygen Consumptionphysical exerciseInternal medicinemedicinelow-grade inflammationHumansOrthopedics and Sports MedicineResistinLeg pressta315ExerciseadipokinesChemokine CCL2InflammationbiologyAdiponectinbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaLeptinC-reactive proteinabdominal fatVO2 maxResistance Training030229 sport sciencesEndocrinologyC-Reactive Proteinbiology.proteinBody CompositionResistinAdiponectinbusinessBiomarkersScandinavian Journal of Medicine and Science in Sports
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Inflammatory markers and cardiovascular risk in the metabolic syndrome

2011

Elevated blood glucose, obesity, high blood pressure, elevated triglycerides and low high density lipoprotein (HDL) cholesterol are well accepted risk factors in the development of coronary artery disease. Clustering of at least three of these factors in an individual is defined as metabolic syndrome (MetS). Obesity is a central pathological mechanism in the disease and it is expected that the incidence of this condition will increase dramatically within the next years. The visceral adipose tissue is not only an energy depot but also an endocrine organ which produces a large number of bioactive molecules, the so called adipokines. In the setting of obesity, the over-production of proinflamm…

AdultLeptinRiskAdolescentAngiotensinogenAdipokineAdipose tissueDiseaseIntra-Abdominal FatBioinformaticsCoronary artery diseasePathogenesisInsulin resistancePlasminogen Activator Inhibitor 1medicineHumansResistinObesityLife StyleChemokine CCL2InflammationMetabolic SyndromeInterleukin-6Tumor Necrosis Factor-alphabusiness.industryAtherosclerosisPrognosismedicine.diseaseObesityPlaque AtheroscleroticCardiovascular DiseasesAdiponectinEndothelium VascularInsulin ResistanceMetabolic syndromebusinessBiomarkersFrontiers in Bioscience
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Expression of properdin in human monocytes

1994

Properdin is the only known positive regulator of the alternative pathway of complement activation. Northern blot analysis of cell lines derived from fibroblasts, B-cells, hepatoma cells, and cells of the monocyte-macrophage lineage revealed properdin expression only in the myelomonocytic cell line HL-60, in the monoblastic cell line U-937 and in the monocytic line Mono Mac 6. Culture of Mono Mac 6 cells for 24 h with phorbol 12-myristate 13-acetate, bacterial lipopolysaccharide and the cytokines interleukin-1 beta and tumour necrosis factor-alpha enhanced mRNA abundance, with the strongest effect (tenfold) being observed with the lipopolysaccharide. In contrast, recombinant interferon-gamm…

AdultLipopolysaccharidesLipopolysaccharidemedicine.medical_treatmentMolecular Sequence DataEnzyme-Linked Immunosorbent AssayBiologyurologic and male genital diseasesBiochemistryMonocytesCell LineInterferon-gammachemistry.chemical_compoundTumor Cells CulturedmedicineHumansRNA MessengerNorthern blotBase SequenceProperdinTumor Necrosis Factor-alphaMacrophagesMonocyteBlotting NorthernMolecular biologyRecombinant ProteinsComplement systemCytokinemedicine.anatomical_structurechemistryCell cultureImmunologyAlternative complement pathwayCytokinesTetradecanoylphorbol AcetateProperdinInterleukin-1European Journal of Biochemistry
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