Search results for "Activation"

showing 10 items of 2079 documents

Glycoprotein 96-activated dendritic cells induce a CD8-biased T cell response.

2005

Heat shock proteins (Hsps) are able to induce protective immune responses against pathogens and tumors after injection into immunocompetent hosts. The activation of components of the adaptive immune system, including cytotoxic T lymphocytes specific for pathogen- or tumor-derived peptides, is crucial for the establishment of immuno- protection. Hsps acquire these peptides during intracellular protein degradation and when released during necrotic cell death, facilitate their uptake and Minor Histocompatibility Complex (MHC)-restricted representation by professional antigen-presenting cells (APCs). In addition, the interaction of Hsps with APCs, including the Endoplasmatic Reticulum (ER)-resi…

CD4-Positive T-LymphocytesLipopolysaccharidesAntigen-Presenting CellsBone Marrow CellsMice TransgenicReceptors Cell SurfaceBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexLymphocyte ActivationBiochemistryMiceImmune systemHeat shock proteinCytotoxic T cellAnimalsHumansAntigen-presenting cellCells CulturedMembrane GlycoproteinsToll-Like ReceptorsCell DifferentiationCell BiologyDendritic cellDendritic CellsOriginal ArticlesAcquired immune systemLymphocyte SubsetsCell biologyMice Inbred C57BLToll-Like Receptor 4biology.proteinInflammation MediatorsCD8Signal TransductionCell stresschaperones
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Blue light irradiation suppresses dendritic cells activationin vitro

2013

Blue light is a UV-free irradiation suitable for treating chronic skin inflammation, for example, atopic dermatitis, psoriasis, and hand- and foot eczema. However, a better understanding of the mode of action is still missing. For this reason, we investigated whether dendritic cells (DC) are directly affected by blue light irradiation in vitro. Here, we report that irradiation neither induced apoptosis nor maturation of monocyte-derived and myeloid DC. However, subsequent DC maturation upon LPS/IFNγ stimulation was impaired in a dose-dependent manner as assessed by maturation markers and cytokine release. Moreover, the potential of this DC to induce cytokine secretion from allogeneic CD4 T …

CD4-Positive T-LymphocytesLipopolysaccharidesLightUltraviolet Raysmedicine.medical_treatmentStimulationInflammationCell SeparationDermatologyLymphocyte ActivationBiochemistryInterferon-gammaPsoriasismedicineHumansIrradiationMolecular BiologyImmunosuppression TherapyInflammationChemistryDendritic Cellsmedicine.diseaseCoculture TechniquesIn vitroCell biologyCytokineApoptosisImmunologyCytokinesCytokine secretionmedicine.symptomExperimental Dermatology
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Induction of regulatory T cells by leflunomide in a murine model of contact allergen sensitivity.

2006

Allergic contact dermatitis and contact hypersensitivity (CHS) are characterized by allergen-specific activation of CD8 + and CD4 + T cells and the production of cytokines resulting in an inflammatory response and tissue damage. We show here that the immunosuppressive compound leflunomide ( N -[4-trifluoro-methylphenyl]-5-methylisoxazol-4 carboxamide, HWA 486) (LF) inhibited the contact allergic response induced in mice by epicutaneous application of the haptens dinitrofluorobenzene (DNFB) and oxazolone. The extent of ear swelling remained significantly reduced following repeated challenge with DNFB for up to 18 weeks. LF and DNFB had to be applied simultaneously for inhibition to occur. Th…

CD4-Positive T-LymphocytesMaleAdoptive cell transferDermatologyCD8-Positive T-Lymphocytesmedicine.disease_causeLymphocyte ActivationBiochemistryOxazolone03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineAllergenDinitrofluorobenzenemedicineAnimalsRNA MessengerAllergic contact dermatitisMolecular Biology030304 developmental biology0303 health sciencesMice Inbred BALB Cintegumentary systemChemistryOxazoloneCell BiologyIsoxazolesAllergensmedicine.diseaseMolecular biology3. Good healthInterleukin-10Disease Models AnimalAllergic responseImmunologyDermatitis Allergic ContactCytokinesDinitrofluorobenzeneFemaleHaptenCD8Immunosuppressive AgentsLeflunomide030215 immunologyThe Journal of investigative dermatology
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Co-activation of naive CD4+ T cells and bone marrow-derived mast cells results in the development of Th2 cells

1995

Activation of naive dense CD4+ T cells by plate-bound anti-CD3 antibodies favors the development of Th1 cells which, upon re-stimulation, produce significant amounts of IFN-gamma but no IL-4. However, co-activation of such naive T cells in the presence of IgE [anti-dinitrophenyl (DNP)]-loaded bone marrow-derived mast cells (BMMC) on plates coated with anti-CD3 antibodies and DNP-BSA led to the development of IL-4-producing Th2 cells. The same result could be observed if irradiated (800 rad) BMMC were applied as co-stimulators. Moreover, BMMC could be replaced by the supernatant of IgE-activated BMMC suggesting that a soluble mediator, presumably IL-4, was responsible for this effect. This a…

CD4-Positive T-LymphocytesMaleCD3 ComplexT cellImmunologyBone Marrow CellsLymphocyte ActivationMiceInterleukin 21Th2 CellsmedicineAnimalsImmunology and AllergyCytotoxic T cellMast CellsIL-2 receptorCells CulturedInterleukin 3Mice Inbred BALB CReceptors IgEChemistryIonomycinDegranulationGeneral MedicineMolecular biologyInterleukin 33medicine.anatomical_structureImmunologyInterleukin 12CytokinesFemaleInterleukin-4International Immunology
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Potential involvement of IL-9 and Th9 cells in the pathogenesis of rheumatoid arthritis

2015

Objective IL-9 has been shown to be upregulated before the clinical onset of articular disease in RA. The exact role of IL-9 and Th9 cells in RA, however, has not yet been adequately studied. The aim of this study was to evaluate the expression of IL-9 and IL-9-expressing cells in RA patients. Methods IL-9, IL-9R, PU.1, IL-9, thymic stromal lymphopoietin (TSLP), IL-4 and TGF-β expression was assessed by real-time-PCR in the synovial tissues of RA and OA patients. IL-9, IL-9R, IL-4, TSLP and TGF-β were also investigated by immunohistochemistry. Peripheral CD4(+) T cell subsets were studied by flow cytometry analysis before and after incubation with citrullinated peptides. Results IL-9 was ov…

CD4-Positive T-LymphocytesMaleCitrullinated peptide; IL-9; Rheumatoid arthritis; Th9 cells; Adolescent; Adult; Arthritis Rheumatoid; CD4-Positive T-Lymphocytes; Cells Cultured; Cytokines; Female; Gene Expression Regulation; Humans; Interleukin-4; Interleukin-9; Lymphocyte Activation; Male; Middle Aged; RNA Messenger; Synovial Membrane; T-Lymphocyte Subsets; Transforming Growth Factor beta; Young Adult; Rheumatology; Medicine (all); Pharmacology (medical)MessengerLymphocyte ActivationArthritis RheumatoidT-Lymphocyte SubsetsTransforming Growth Factor betaRheumatoidTh9 cellPharmacology (medical)Cells CulturedCulturedmedicine.diagnostic_testbiologyMedicine (all)Synovial MembraneMiddle Agedmedicine.anatomical_structureCD4-Positive T-LymphocyteCytokinesFemaleArthritiHumanAdultThymic stromal lymphopoietinAdolescentT cellCD3T-Lymphocyte SubsetCitrullinated peptidePeripheral blood mononuclear cellFlow cytometryYoung AdultRheumatologyThymic Stromal LymphopoietinmedicineHumansInterleukin 9RNA MessengerCytokineInterleukin 4Rheumatoid arthritibusiness.industryInterleukin-9IL-9Settore MED/16 - ReumatologiaGene Expression RegulationImmunologybiology.proteinRNACellInterleukin-4Synovial membranebusiness
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Prospective Study of the Evolution of Blood Lymphoid Immune Parameters during Dacarbazine Chemotherapy in Metastatic and Locally Advanced Melanoma Pa…

2014

BackgroundThe importance of immune responses in the control of melanoma growth is well known. However, the implication of these antitumor immune responses in the efficacy of dacarbazine, a cytotoxic drug classically used in the treatment of melanoma, remains poorly understood in humans.MethodsIn this prospective observational study, we performed an immunomonitoring of eleven metastatic or locally advanced patients treated with dacarbazine as a first line of treatment. We assessed by flow cytometry lymphoid populations and their activation state; we also isolated NK cells to perform in vitro cytotoxicity tests.ResultsWe found that chemotherapy induces lymphopenia and that a significantly hig…

CD4-Positive T-LymphocytesMaleSkin Neoplasmsmedicine.medical_treatmentCancer TreatmentGene ExpressionNK cellsLymphocyte ActivationT-Lymphocytes RegulatoryLeukocyte CountCellular typesMedicine and Health SciencesCytotoxic T cellProspective StudiesNeoplasm MetastasisProspective cohort studyImmune ResponseMelanomaAged 80 and overMultidisciplinarymedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionMelanomaQRMiddle AgedFlow CytometryPrognosis3. Good healthDacarbazineKiller Cells NaturalTreatment OutcomeOncologyCutaneous MelanomaMedicineWhite blood cellsFemaleImmunotherapymedicine.drugResearch ArticleTumor ImmunologyAdultCell biologyBlood cellsCell SurvivalDacarbazineScienceImmune CellsImmunologyLocally advancedT cellsMalignant Skin NeoplasmsDermatologyCancer ImmunotherapyFlow cytometryImmune systemCell Line TumormedicineHumansAntineoplastic Agents AlkylatingAgedChemotherapyBiology and life sciencesbusiness.industrymedicine.diseaseAnimal cellsImmunologyCancer researchClinical ImmunologybusinessTranscription FactorsPLoS ONE
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Cellular and humoral immune responses against autoreactive T cells in multiple sclerosis patients after T cell vaccination.

1999

Myelin basic protein (MBP)-reactive T cells may play an important role in the autoimmune pathogenesis of multiple sclerosis (MS). MBP-reactive T cells can be specifically targeted by T cell vaccination, a procedure whereby MS patients are immunized with attenuated autologous MBP reactive T cells. T cell vaccination induces immune responses to the vaccine cells together with a depletion of MBP reactive T cells. Forty-nine MS patients were treated with T cell vaccination in an extended phase I trial to study the safety, immune responses and clinical effects of T cell vaccination. In the present paper the immune responses towards the vaccine cells were characterized. Substantial long-term in v…

CD4-Positive T-LymphocytesMultiple SclerosisT-LymphocytesImmunologyT-cell vaccinationLymphocyte ActivationInterleukin 21Immunology and AllergyMedicineCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellImmunity CellularVaccinesCD40biologyClinical Trials Phase I as Topicbusiness.industryVaccinationMyelin Basic ProteinNatural killer T cellLymphocyte SubsetsVaccines InactivatedCTLA-4ImmunologyAntibody Formationbiology.proteinCytokinesImmunotherapybusinessJournal of autoimmunity
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Induction of CD4+/CD25+ regulatory T cells by targeting of antigens to immature dendritic cells

2003

AbstractCoupling of ovalbumin (OVA) to anti–DEC-205 monoclonal antibody (mAb) (αDEC) induced the proliferation of OVA-specific T cells in vivo. Expansion was short-lived, caused by dendritic cells (DCs), and rendered T cells anergic thereafter. Phenotypic analysis revealed the induction of CD25+/CTLA-4+ T cells suppressing proliferation and interleukin-2 (IL-2) production of effector CD4+ T cells. The findings were supported by 2 disease models: (1) CD4+ T-cell–mediated hypersensitivity reactions were suppressed by the injection of αDEC-OVA and (2) the application of hapten-coupled αDEC-205 reduced CD8+ T-cell–mediated allergic reactions. Thus, targeting of antigens to immature DCs through …

CD4-Positive T-LymphocytesOvalbuminT-LymphocytesImmunologyCD8-Positive T-LymphocytesDermatitis ContactLymphocyte ActivationBiochemistryMiceInterleukin 21Antigens CDHypersensitivityAnimalsCytotoxic T cellCTLA-4 AntigenHypersensitivity DelayedLymphocyte CountIL-2 receptorAntigensAntigen-presenting cellAntigen PresentationMice Inbred BALB CCD40biologyAntibodies MonoclonalReceptors Interleukin-2Dendritic CellsCell BiologyHematologyDendritic cellNatural killer T cellAntigens DifferentiationCell biologyImmunologyInterleukin 12biology.proteinInterleukin-2HaptensBlood
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PD-1 signalling in CD4+T cells restrains their clonal expansion to an immunogenic stimulus, but is not critically required for peptide-induced tolera…

2010

Summary The ultimate outcome of T-cell recognition of peptide–major histocompatibility complex (MHC) complexes is determined by the molecular context in which antigen presentation is provided. The paradigm is that, after exposure to peptides presented by steady-state dendritic cells (DCs), inhibitory signals dominate, leading to the deletion and/or functional inactivation of antigen-reactive T cells. This has been utilized in a variety of models providing peptide antigen in soluble form in the absence of adjuvant. A co-inhibitory molecule of considerable current interest is PD-1. Here we show that there is the opportunity for the PD-1/PD-L1 interaction to function in inhibiting the T-cell r…

CD4-Positive T-LymphocytesOvalbuminTransgeneProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationMice TransgenicCell SeparationCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMajor histocompatibility complexMiceImmune systemBlocking antibodyImmune ToleranceAnimalsImmunology and AllergyT-cell receptorOriginal ArticlesFlow CytometryAntigens DifferentiationPeptide FragmentsCell biologyMice Inbred C57BLTolerance inductionPhenotypeImmunologybiology.proteinCD8Signal TransductionImmunology
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TLR2 and Dectin-1 Signaling in Mouse Hematopoietic Stem and Progenitor Cells Impacts the Ability of the Antigen Presenting Cells They Produce to Acti…

2020

Microbial recognition by pattern recognition receptors (PRRs) expressed on hematopoietic stem and progenitor cells (HSPCs) not only activates myelopoiesis but also programs the function of the monocytes and macrophages they produce. For instance, changes in HSPC programming modify the ability of macrophages derived from them to produce inflammatory cytokines. While HSPCs exposed to a TLR2 agonist give rise to tolerized macrophages (lower proinflammatory cytokine production), HSPCs treated with Dectin-1 ligands produce trained macrophages (higher proinflammatory cytokine production). However, nothing is known about the impact of HSPC exposure to microbes on the function of antigen presenting…

CD4-Positive T-LymphocytesOvalbuminhematopoietic stem and progenitor cellsCD4 T cellsAntigen-Presenting CellsMice Transgenicantigen presenting cellsLymphocyte Activationinnate immune memoryProinflammatory cytokineLipopeptidesCandida albicansAnimalsTLR2Lectins C-TypeProgenitor cellAntigen-presenting celllcsh:QH301-705.5CD86CD40biologyChemistryCommunicationHistocompatibility Antigens Class IIZymosanGeneral MedicineTh1 CellsHematopoietic Stem CellsAcquired immune systemToll-Like Receptor 2Cell biologyMice Inbred C57BLlcsh:Biology (General)biology.proteinCytokinesTh17 CellsMyelopoiesisCD80Dectin-1Signal TransductionCells
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