Search results for "Active oxygen"

showing 10 items of 884 documents

Detection of oxidative mutagenesis by isoniazid and other hydrazine derivatives in Escherichia coli WP2 tester strain IC203, deficient in OxyR: stron…

1998

Abstract Strain IC203, deficient in the OxyR function, was sensitive to both cytotoxic and mutagenic effects of isoniazid (INH) whereas its parent, WP2 uvrA /pKM101, was resistant to these effects. Four other hydrazine compounds, hydrazine hydrate (HZH), phenylhydrazine (PHZ), hydralazine (HLZ) and nialamide (NLD), were mutagenic in WP2 uvrA /pKM101. Increases in mutagenicity were observed in IC203 for HZH and PHZ but not for HLZ and NLD. Growth inhibition zones by HZH, PHZ and NLD were larger in IC203 than in WP2 uvrA /pKM101. The enhancements in the effects of INH, HZH and PHZ in IC203 with respect to its oxyR + parent are considered to be caused by the production of reactive oxygen speci…

Health Toxicology and Mutagenesismedicine.disease_causechemistry.chemical_compoundSpecies SpecificityEscherichia coliIsoniazidGeneticsmedicineAnimalsEscherichia coliPhenylhydrazinechemistry.chemical_classificationReactive oxygen speciesbiologyMutagenicity TestsEscherichia coli ProteinsMutagenesisbiology.organism_classificationEnterobacteriaceaeRatsDNA-Binding ProteinsRepressor ProteinsLiverBiochemistrychemistryMutagenesisCatalasebiology.proteinbacteriaGrowth inhibitionReactive Oxygen SpeciesOxidation-ReductionCytosineMutagensTranscription FactorsMutation Research/Genetic Toxicology and Environmental Mutagenesis
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Nitrate tolerance as a model of vascular dysfunction: Roles for mitochondrial aldehyde dehydrogenase and mitochondrial oxidative stress

2008

Organic nitrates are a group of very effective anti-ischemic drugs. They are used for the treatment of patients with stable angina, acute myocardial infarction and chronic congestive heart failure. A major therapeutic limitation inherent to organic nitrates is the development of tolerance, which occurs during chronic treatment with these agents. The mechanisms underlying nitrate tolerance remain incompletely defined and are likely multifactorial. One mechanism seems to be a diminished bioconversion of nitroglycerin, another seems to be the induction of vascular oxidative stress, and a third may include neurohumoral adaptations. Recent studies have revealed that mitochondrial reactive oxygen…

Heart DiseasesAldehyde dehydrogenaseOxidative phosphorylationBiologymedicine.disease_causeNitrate reductaseNitroglycerinchemistry.chemical_compoundmedicineAnimalsHumansEndothelial dysfunctionPharmacologychemistry.chemical_classificationReactive oxygen speciesNitratesSuperoxideAldehyde Dehydrogenase MitochondrialDrug ToleranceGeneral MedicineAldehyde Dehydrogenasemedicine.diseaseMitochondriaOxidative StressBiochemistrychemistrybiology.proteinEndothelium VascularOxidative stressPeroxynitritePharmacological Reports
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Mitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stress.

2014

Significance: Oxidative stress is a well established hallmark of cardiovascular disease and there is strong evidence for a causal role of reactive oxygen and nitrogen species (RONS) therein. Recent Advances: Improvement of cardiovascular complications by genetic deletion of RONS producing enzymes and overexpression of RONS degrading enzymes proved the involvement of these species in cardiovascular disease at a molecular level. Vice versa, overexpression of RONS producing enzymes as well as deletion of antioxidant enzymes was demonstrated to aggravate cardiovascular complications. Critical Issues: With the present overview we present and discuss different pathways how mitochondrial RONS inte…

Heart DiseasesPhysiologyClinical BiochemistryMitochondrionmedicine.disease_causeBiochemistrychemistry.chemical_compoundmedicineAnimalsHumansXanthine oxidaseMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationInflammationReactive oxygen speciesNADPH oxidasebiologyNADPH OxidasesCell BiologyForum Review ArticlesAngiotensin IICell biologyMitochondriaNitric oxide synthaseCrosstalk (biology)Oxidative StressBiochemistrychemistrybiology.proteinGeneral Earth and Planetary SciencesReactive Oxygen SpeciesOxidation-ReductionOxidative stressSignal TransductionAntioxidantsredox signaling
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Role of endothelial cell stress in the pathogenesis of chronic heart failure.

2009

Endothelial cells are key modulators of diverse physiological processes, and their impaired function is a cause of numerous cardiovascular diseases. Under physiologic condition, the reactive oxygen and nitrogen mediators in endothelia lead to the signal propagation of the initial stimulus, by forming molecules with a longer half-life like hydrogen peroxide. Hydrogen peroxide is the focus of growing attention in endothelial biology, and consequently the enzymes involved in its generation and clearance are viewed as novel mediators of great importance. In particular, among peroxidases, myeloperoxidase is recognized as a key enzyme, capable of impairing intracellular NO reservoirs as well as p…

Heart FailureEndotheliumbiologyEndothelial cells Myeloperoxidase Hydrogen Peroxide Oxidative Stress Enos Nitric Oxide Superoxide ROS RNS 3-Chlorotyrosine 3-Nitrotyrosine Nitrosylaton ReviewSuperoxideSettore BIO/16 - Anatomia UmanaOxidative phosphorylationmedicine.disease_causeNitric oxideCell biologyEndothelial stem cellchemistry.chemical_compoundOxidative Stressmedicine.anatomical_structurechemistryMyeloperoxidaseChronic Diseasemedicinebiology.proteinHumansEndothelium VascularReactive Oxygen SpeciesOxidative stressIntracellularPeroxidaseFrontiers in bioscience (Landmark edition)
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Chemistry in Crime Investigation: Sodium Percarbonate Effects on Bloodstains Detection

2011

Chemistry plays a leading role in crime investigation. In the study of bloodstains, chemical reactions provide the means for the detection. All these procedures have been thoroughly studied. However, recently, a new source of error has been found: washing stains with "active oxygen" detergents abrogates presumptive and human hemoglobin tests for bloodstains (although visible). The aim of this investigation was to evaluate the ability of pure sodium percarbonate-main component of detergents-to abrogate presumptive and human hemoglobin tests. Then, a solution to this problem could be found. The results demonstrate that pure sodium percarbonate-itself-is able to abrogate all tests, as well as …

Hemoglobin testsChromatographyForensic chemistryBlood StainsPoison controlSodium percarbonateComputer securitycomputer.software_genreCrime investigationStainPathology and Forensic MedicineActive oxygenchemistry.chemical_compoundchemistryGeneticscomputerJournal of Forensic Sciences
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Functional assays of oxidative stress using genetically engineered Escherichia coli strains.

2003

Oxidative stress may be induced in bacteria by exogenous biocidal agents and is involved in endogenous metabolism. The oxyR operon is a main sensor of oxidative stress and oxyR-deficient bacteria show enhanced sensitivity to oxidative stress and increased accumulation of intracellular reactive oxygen species (ROS). Flow cytometric functional assays in bacteria are limited by the impaired penetration of vital dyes trough the cell wall. Escherichia coli B WP2 strains possess an altered cell-wall lipopolysaccharide that leads to increased membrane permeability. Flow cytometric analysis of WP2 strains is a convenient alternative for cytometric assays of bacterial function. This unit presents pr…

HistologyMembrane permeabilityLipopolysaccharideOperonBiologymedicine.disease_causeBiochemistryCell wallchemistry.chemical_compoundmedicineEscherichia coliEscherichia coliFluorescent DyesEscherichia coli ProteinsGeneral Medicinebiology.organism_classificationFlow CytometryDNA-Binding ProteinsRepressor ProteinsMedical Laboratory TechnologyOxidative StressBiochemistrychemistrybacteriaGenetic EngineeringReactive Oxygen SpeciesIntracellularBacteriaOxidative stressCurrent protocols in cytometry
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Hyperhomocysteinemia and Cardiovascular Risk: Effect of Vitamin Supplementation in Risk Reduction

2010

Abstract Homocysteine is a sulfur-containing aminoacid produced during metabolism of methionine. Since 1969 the relationship between altered homocysteine metabolism and both coronary and peripheral atherotrombosis has been known; in recent years experimental evidences have shown that elevated plasma levels of homocysteine are associated with an increased risk of atherosclerosis and cardiovascular ischemic events. Several mechanisms by which elevated homocysteine impairs vascular function have been proposed, including impairment of endothelial function, production of Reactive Oxygen Species (ROS) and consequent oxidation of low-density lipids. Folic acid and B vitamins, required for remethyl…

Hyperhomocysteinemiamedicine.medical_specialtyHomocysteineHyperhomocysteinemiachemistry.chemical_compoundFolic AcidRisk FactorsInternal medicinehomocysteine MTHFR cardiovascular disease folate B vitaminsmedicineAnimalsHumansPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsVitamin supplementationchemistry.chemical_classificationReactive oxygen speciesMethioninebusiness.industryRisk effectGeneral MedicineMetabolismmedicine.diseaseVitamin B 6Vitamin B 12B vitaminsEndocrinologychemistryCardiovascular DiseasesVitamin B ComplexbusinessRisk Reduction BehaviorCurrent Clinical Pharmacology
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Impact of therapeutically induced reactive oxygen species and radical scavenging by α-tocopherol on tumor cell adhesion

2007

Many tumor treatment modalities such as ionizing radiation or some chemotherapy induce reactive oxygen species (ROS) resulting in therapeutic cell damage. The aim of this study was to analyze whether such ROS induction may affect the mechanical stability of solid tumor tissue by degradation of the extracellular matrix proteins or by a loss of cell adhesion molecules. Additionally, the protective impact of alpha-tocopherol treatment on these processes was studied. Experimental DS-sarcomas in rats were treated with a combination of localized 44 degrees C hyperthermia, inspiratory hyperoxia and xanthine oxidase in order to induce pronounced oxidative stress. A second group of animals were pret…

Hyperoxiachemistry.chemical_classificationCancer ResearchReactive oxygen speciesbiologyCell adhesion moleculeIntegrinGeneral Medicinemedicine.diseasemedicine.disease_causeExtracellular matrixchemistry.chemical_compoundOncologyBiochemistrychemistrymedicinebiology.proteinCancer researchmedicine.symptomXanthine oxidaseCell damageOxidative stressOncology Reports
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Role of glutathione in plant signaling under biotic stress

2012

International audience; Glutathione (GSH) is a non-protein thiol compound which has been repeatedly reported to play an important role in plant responses during biotic stresses. However, our knowledge of glutathione-related molecular mechanisms underlying plant defense responses still remains limited. We first discovered that the Arabidopsis thaliana phytoalexin deficient 2-1 (pad2-1) mutant was linked to glutathione deficiency since the mutation was identified in the GSH1 gene encoding the first enzyme of glutathione biosynthesis: Glutamate Cysteine Ligase (GCL). Interestingly, this glutathione-deficient mutant pad2-1 also displays a high susceptibility to a wide range of invaders. We rece…

Hypersensitive responsePhytophthoraMini Reviewredox environment[SDV]Life Sciences [q-bio]ArabidopsisPlant ScienceBiologymedicine.disease_causeGenes Plantphytophthora brassicaechemistry.chemical_compoundpad2-1 mutantGene Expression Regulation PlantStress PhysiologicalArabidopsismedicineArabidopsis thalianaPlant ImmunityglutathionePlant Diseaseschemistry.chemical_classificationReactive oxygen speciesArabidopsis ProteinsPhytoalexinarabidopsis thalianaGlutathioneBiotic stressbiology.organism_classificationdefence responseOxidative StressBiochemistrychemistryMutation[SDE]Environmental SciencesOxidative stressSignal Transduction
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Characterization of a new, nonpathogenic mutant of Botrytis cinerea with impaired plant colonization capacity.

2006

International audience; Botrytis cinerea is a necrotrophic pathogen that attacks more than 200 plant species.Here, the nonpathogenic mutant A336, obtained via insertional mutagenesis, was characterized.Mutant A336 was nonpathogenic on leaves and fruits, on intact and wounded tissue, while still able to penetrate the host plant. It grew normally in vitro on rich media but its conidiation pattern was altered. The mutant did not produce oxalic acid and exhibited a modified regulation of the production of some secreted proteins (acid protease 1 and endopolygalacturonase 1). Culture filtrates of the mutant triggered an important oxidative burst in grapevine ( Vitis vinifera ) suspension cells, a…

Hypersensitive responsehypersensitive responsePhysiology[SDV]Life Sciences [q-bio]colonisationMutantArabidopsisConidiationPlant ScienceMicrobiologyInsertional mutagenesisFungal Proteins03 medical and health sciencesPlasmidGene Expression Regulation FungalOnionsBotrytis cinerea; endopolygalacturonase BcPG1; hypersensitive responseVitismutantoxidative burstPathogenGene030304 developmental biologyBotrytis cinereaPhaseolus0303 health sciencesoxalateendopolygalacturonase BcPG1biology030306 microbiologyOxalic Acidfungifood and beveragesHydrogen-Ion Concentrationbiology.organism_classificationImmunity Innatenonpathogenic mutantEnzymesPlant LeavesMutagenesis Insertionalnonaspartyl acid protease ACP1Mutationbotrytis cinerea;endopolygalacturonase BcPG1;hypersensitive response;nonaspartyl acid protease ACP1;nonpathogenic mutant;oxalate;oxidative burstBotrytisbotrytis cinereaReactive Oxygen SpeciesThe New phytologist
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