Search results for "Active oxygen"

showing 10 items of 884 documents

Non-Hemodynamic Effects of Organic Nitrates and the Distinctive Characteristics of Pentaerithrityl Tetranitrate

2009

Organic nitrates are among the oldest and yet most commonly employed drugs in the long-term therapy of coronary artery disease and congestive heart failure. While they have long been used in clinical practice, our understanding of their mechanism of action and side effects remains incomplete. For instance, recent findings provide evidence of previously unanticipated, non-hemodynamic properties that include potentially beneficial mechanisms (such as the induction of a protective phenotype that mimics ischemic preconditioning), but also toxic effects (such as endothelial and autonomic dysfunction, rebound angina, tolerance). To date, the most commonly employed organic nitrates are isosorbide …

medicine.medical_specialtyHeart DiseasesVasodilator AgentsIsosorbide DinitratePharmacologyDrug Administration ScheduleAnginaCoronary artery diseaseNitroglycerinmedicineIsosorbide mononitrateAnimalsHumansPentaerythritol TetranitratePharmacology (medical)NitroglycerinNitratesbusiness.industryDrug ToleranceGeneral Medicinemedicine.diseaseSurgeryEastern europeanOxidative StressHeart failureIschemic preconditioningIsosorbide dinitrateReactive Oxygen SpeciesCardiology and Cardiovascular Medicinebusinessmedicine.drugAmerican Journal of Cardiovascular Drugs
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Therapeutical approach to plasma homocysteine and cardiovascular risk reduction

2008

Marcello Ciaccio, Giulia Bivona, Chiara BelliaDepartment of Medical Biotechnologies and Forensic Medicine, Faculty of Medicine, University of Palermo, ItalyAbstract: Homocysteine is a sulfur-containing aminoacid produced during metabolism of methionine. Since 1969 the relationship between altered homocysteine metabolism and both coronary and peripheral atherotrombosis is known; in recent years experimental evidences have shown that elevated plasma levels of homocysteine are associated with an increased risk of atherosclerosis and cardiovascular ischemic events. Several mechanisms by which elevated homocysteine impairs vascular function have been proposed, including impairment of endothelial…

medicine.medical_specialtyHyperhomocysteinemiaHomocysteineReviewRM1-950folatehomocysteine MTHFR cardiovascular disease folate B vitaminchemistry.chemical_compoundcardiovascular diseaseInternal medicineMedicinePharmacology (medical)General Pharmacology Toxicology and PharmaceuticsEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesChemical Health and SafetyMethioninebiologybusiness.industryhomocysteineGeneral MedicineMetabolismmedicine.diseaseB vitaminB vitaminsEndocrinologychemistryMethylenetetrahydrofolate reductaseMTHFRbiology.proteinTherapeutics. PharmacologybusinessSafety ResearchTherapeutics and Clinical Risk Management
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Phosphodiesterase-4 inhibition improves corticosteroid insensitivity in pulmonary endothelial cells under oxidative stress.

2012

Several clinical studies have shown that smoking in asthmatics and chronic obstructive pulmonary disease patients is closely associated with corticosteroid refractoriness. In this work, we have analyzed glucocorticoid insensitivity in human pulmonary artery endothelial cells (HPAECs) under cigarette smoke extract (CSE) exposure as well as the possible additive effects of the combination therapy with a phosphodiesterase (PDE)-4 inhibitor. Interleukin (IL)-8 was measured in cell supernatants by ELISA. Histone deacetylase (HDAC), histone acetylase (HAT), and intracellular cAMP levels were measured by colorimetric assays and enzyme immunoassay, respectively. PDE4 isotypes and glucocorticoid rec…

medicine.medical_specialtyImmunologyApoptosisDexamethasoneHistone DeacetylasesGlucocorticoid receptorReceptors GlucocorticoidAdrenal Cortex HormonesInternal medicinemedicineCyclic AMPImmunology and AllergyHumansReceptorLungDexamethasoneRolipramCell ProliferationHistone AcetyltransferasesChemistryTumor Necrosis Factor-alphaInterleukin-8InterleukinPhosphodiesteraseEndothelial CellsAparato respiratorioCyclic Nucleotide Phosphodiesterases Type 4Enzyme ActivationOxidative StressEndocrinologyHistone deacetylasePhosphodiesterase 4 InhibitorsPulmonesReactive Oxygen SpeciesRolipramGlucocorticoidmedicine.drugAllergy
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CRP-induced levels of oxidative stress are higher in brain than aortic endothelial cells

2010

C-reactive protein (CRP) has been demonstrated to induce blood-brain barrier disruption (BBB) involving NAD(P)H-oxidase dependent oxidative stress. It is unclear why CRP affects the BBB and not other vascular beds following stroke. Therefore we examined CRP receptor and NAD(P)H-oxidase expression levels in bovine brain- (BEC) and aortic endothelial cells. Dichlorodihydrofluorescein measurements revealed significantly higher CRP-induced reactive oxygen species (ROS) levels in BEC. Protein expression of the CRP-receptors CD16, CD32 and of the NAD(P)H-oxidase subunit p22phox were also significantly higher in BEC. In conclusion BEC show a higher vulnerability to CRP due to increased levels of C…

medicine.medical_specialtyImmunologyBlood–brain barriermedicine.disease_causeBiochemistryInternal medicinemedicineAnimalsImmunology and AllergyReceptorMolecular BiologyAortachemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologyChemistryReceptors IgGBrainEndothelial CellsNADPH OxidasesHematologyOxidative StressC-Reactive Proteinmedicine.anatomical_structureEndocrinologyNAD(P)H oxidaseImmunologybiology.proteinCattleP22phoxNAD+ kinaseOxidative stressCytokine
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Effects of Long-Term Nitroglycerin Treatment on Endothelial Nitric Oxide Synthase (NOS III) Gene Expression, NOS III–Mediated Superoxide Production, …

2000

Abstract —Long-term nitroglycerin (NTG) treatment has been shown to be associated with cross-tolerance to endothelium-dependent vasodilators. It may involve increased production of reactive oxygen species (such as superoxide, O 2 ·− ) that rapidly inactivate the nitric oxide (NO) released from the endothelial cells. It remains to be elucidated, however, whether long-term treatment with NTG alters the activity and expression of the endothelial NO synthase (NOS III) and whether this enzyme can contribute to O 2 ·− formation. We studied the influence of long-term NTG treatment on the expression of NOS III as assessed by RNase protection assay and Western blot. Tolerance was measured ex vivo i…

medicine.medical_specialtyIndolesNitric Oxide Synthase Type IIIPhysiologyCarbazolesBiological AvailabilityVasodilationArginineNitric OxideGene Expression Regulation EnzymologicTimeNitric oxideNitroglycerinchemistry.chemical_compoundAlkaloidsSuperoxidesInternal medicinemedicineAnimalsRNA MessengerLucigeninCloning MolecularEnzyme InhibitorsRats WistarCalcimycinProtein Kinase CProtein kinase CBenzophenanthridineschemistry.chemical_classificationReactive oxygen speciesSuperoxideAcetylcholinePhenanthridinesRatsVasodilationEndocrinologychemistryBiochemistryEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicineEx vivoAcetylcholinemedicine.drugCirculation Research
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Lysine triggers apoptosis through a NADPH oxidase-dependent mechanism in human renal tubular cells

2012

Progressive chronic kidney disease (CKD) is common in lysinuric protein intolerance (LPI), a primary inherited aminoaciduria characterized by massive Lysine excretion in urine. However, by which mechanisms Lysine may cause kidney damage to tubule cells is still not understood. This study determined whether Lysine overloading of human proximal tubular cells (HK-2) in culture enhances apoptotic cell loss and its associated mechanisms. Overloading HK-2 with Lysine levels reproducing those observed in urine of patients affected by LPI (10 mM) increased apoptosis (+30%; p < 0.01 vs.C), as well as Bax and Apaf-1 expressions (+30-50% p < 0.05), while downregulated Bcl-2 (-40% p < 0.05). Apoptosis …

medicine.medical_specialtyLysineGene ExpressionApoptosisNADPH Oxidasecomplex mixturesAntioxidantsCell LineExcretionKidney Tubules ProximalInternal medicineGeneticsmedicineHumansRenal Insufficiency ChronicAmino Acid Metabolism Inborn ErrorsProtein SubunitGenetics (clinical)Membrane Potential MitochondrialKidneyNADPH oxidasebiologyLysineAmino Acid Metabolism Inborn ErrorNADPH OxidasesApoptosimedicine.diseaseCaspase InhibitorsLysinuric protein intoleranceIn vitroProtein SubunitsEndocrinologymedicine.anatomical_structureCell cultureApoptosisbiology.proteinCaspase InhibitorDisease ProgressionAntioxidantReactive Oxygen SpeciesReactive Oxygen SpecieHuman
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Mechanisms of C-reactive protein-induced blood-brain barrier disruption.

2009

Background and Purpose— Increased mortality after stroke is associated with brain edema formation and high plasma levels of the acute phase reactant C-reactive protein (CRP). The aim of this study was to examine whether CRP directly affects blood–brain barrier stability and to analyze the underlying signaling pathways. Methods— We used a cell coculture model of the blood–brain barrier and the guinea pig isolated whole brain preparation. Results— We could show that CRP at clinically relevant concentrations (10 to 20 μg/mL) causes a disruption of the blood–brain barrier in both approaches. The results of our study further demonstrate CRP-induced activation of surface Fcγ receptors CD16/32 fo…

medicine.medical_specialtyMyosin light-chain kinaseMyosin Light ChainsGuinea PigsBrain Edemamedicine.disease_causeBlood–brain barrierp38 Mitogen-Activated Protein KinasesMyosin light chain kinase activityTight JunctionsInternal medicineMyosinmedicineAnimalsPhosphorylationReceptorCells CulturedAdvanced and Specialized Nursingbusiness.industryReceptors IgGCoculture TechniquesCell biologyRatsStrokeEndocrinologymedicine.anatomical_structureC-Reactive ProteinBlood-Brain BarrierPhosphorylationNeurology (clinical)Endothelium VascularSignal transductionCardiology and Cardiovascular MedicinebusinessReactive Oxygen SpeciesOxidative stressSignal TransductionStroke
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Comparative cytoprotective effects of carbocysteine and fluticasone propionate in cigarette smoke extract-stimulated bronchial epithelial cells

2013

Cigarette smoke extracts (CSE) induce oxidative stress, an important feature in chronic obstructive pulmonary disease (COPD), and oxidative stress contributes to the poor clinical efficacy of corticosteroids in COPD patients. Carbocysteine, an antioxidant and mucolytic agent, is effec- tive in reducing the severity and the rate of exacerbations in COPD patients. The effects of carbocysteine on CSE-induced oxidative stress in bronchial epithelial cells as well as the comparison of these antioxidant effects of carbocysteine with those of fluticasone propionate are unknown. The present study was aimed to assess the effects of carbocysteine (10−4 M) in cell survival and intracellular reactive o…

medicine.medical_specialtyNecrosisCell SurvivalNF-E2-Related Factor 2Histone Deacetylase 2ApoptosisSettore MED/10 - Malattie Dell'Apparato Respiratoriomedicine.disease_causeBiochemistryFluticasone propionateAntioxidantsCell Linechemistry.chemical_compoundNecrosisInternal medicineparasitic diseasesTobaccomedicineHumansFluticasonechemistry.chemical_classificationReactive oxygen speciesOriginal PaperPlant ExtractsCarbocysteineCarbocysteineEpithelial CellsCell BiologyGlutathioneCigarette smoke . Airway epithelial cells . Reactive oxygen speciesGlutathioneAndrostadienesOxidative StressEndocrinologychemistryApoptosisFluticasonemedicine.symptomReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1medicine.drug
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Chronic periodontitis impairs polymorphonuclear leucocyte-endothelium cell interactions and oxidative stress in humans.

2018

Aim To evaluate the relationship between oxidative stress parameters in polymorphonuclear leucocytes (PMNs) and PMN-endothelial cell interactions in patients with chronic periodontitis (CP) according to different degrees of severity of the disease. Materials and methods For this cross-sectional study, 182 subjects were divided into four groups according to degree of CP: without CP (n = 37), mild CP (n = 59), moderate CP (n = 51), and severe CP (n = 35). We determined anthropometric and biochemical variables, periodontal parameters, inflammatory markers, oxidative stress parameters (superoxide and mitochondrial membrane potential), and PMN-endothelium cell interactions (rolling flux, velocit…

medicine.medical_specialtyNeutrophilsCell Communication030204 cardiovascular system & hematologymedicine.disease_causeSevere periodontitisProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineHumansEndotheliumEndothelial dysfunctionPeriodontitischemistry.chemical_classificationReactive oxygen speciesChemistrySuperoxide030206 dentistrymedicine.diseaseChronic periodontitisOxidative StressEndocrinologyCross-Sectional StudiesChronic PeriodontitisPeriodonticsOxidative stressJournal of clinical periodontology
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Is Myeloperoxidase a Key Component in the ROS-Induced Vascular Damage Related to Nephropathy in Type 2 Diabetes?

2013

AbstractIt is still unclear whether microvascular complications of type 2 diabetes correlate with leukocyte-endothelium interactions and/or myeloperoxidase (MPO) levels. In the present study, we found that serum levels of glucose, the rate of ROS and MPO concentration were higher in type 2 diabetic patients. Patients with nephropathy (39.6%) presented higher MPO levels that correlate positively with the albumin/creatinine ratio (r=0.59, p<0.05). In addition, nephropatic patients showed increased leukocyte-endothelium interactions due to an undermining of polymorphonuclear leukocytes (PMN) rolling velocity and increased rolling flux and adhesion, which was accompanied by a rise in levels of …

medicine.medical_specialtyPhysiologyClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistryNephropathyProinflammatory cytokinechemistry.chemical_compoundInternal medicinemedicineCell AdhesionHuman Umbilical Vein Endothelial CellsLeukocytesHumansNews & ViewsDiabetic NephropathiesCell adhesionMolecular BiologyCells CulturedGeneral Environmental SciencePeroxidaseCreatininebiologyCell adhesion moleculeCell BiologyMiddle Agedmedicine.diseaseEndocrinologychemistryDiabetes Mellitus Type 2Myeloperoxidasebiology.proteinGeneral Earth and Planetary SciencesCytokinesReactive Oxygen SpeciesCell Adhesion MoleculesOxidative stress
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