Search results for "Aerolysin"

showing 3 items of 3 documents

Secretion of haemolysins and proteases by Aeromonas hydrophila EO63: separation and characterization of the serine protease (caseinase) and the metal…

2004

C . E S T E V E A N D T . H . B I R K B E C K . 2004. Aims: To determine the haemolysins and proteases excreted by the virulent strain EO63 of Aeromonas hydrophila grown in complex media and to then fractionate and characterize them, in particular those with elastolytic activity. Methods and Results: The amount of haemolytic and proteolytic activity in EO63 culture supernatants was dependent on the culture media used. In all media, haemolysins appeared during the phase of active growth and haemolytic activity decreased quickly thereafter, as previously described for aerolysin. In contrast, proteases were mainly released during the stationary phase. Serine protease activity in EO63 culture s…

ProteasesAerolysinBiologyApplied Microbiology and BiotechnologyMicrobiologySerineHemolysin ProteinsCaseinaseEndopeptidasesSerine proteaseSerine EndopeptidasesElastaseCaseinsHemolysinGeneral MedicineHydrogen-Ion Concentrationbiology.organism_classificationAeromonas hydrophilaCulture MediaElastinAeromonas hydrophilaBiochemistryMetalloproteasesbiology.proteinElectrophoresis Polyacrylamide GelIsoelectric FocusingBiotechnologyJournal of Applied Microbiology
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Mode of primary binding to target membranes and pore formation induced by Vibrio cholerae cytolysin (hemolysin).

1997

Vibrio cholerae cytolysin (VCC) is produced by many non-choleratoxigenic strains of V. cholerae, and possibly represents a relevant pathogenicity determinant of these bacteria. The protein is secreted as a pro-toxin that is proteolytically cleaved to yield the active toxin with a molecular mass of approximately 63 kDa. We here describe a simple procedure for preparative isolation of mature VCC from bacterial culture supernatants, and present information on its mode of binding and pore formation in biological membranes. At low concentrations, toxin monomers interact with a high-affinity binding site on highly susceptible rabbit erythrocytes. This as yet unidentified binding site is absent on…

Pore-forming toxinBinding SitesToxinCytotoxinsErythrocyte MembraneMolecular Sequence DataAerolysinHemolysinBiologymedicine.disease_causeBiochemistryTransmembrane proteinMolecular WeightBiochemistryVibrio choleraemedicineAnimalsHumansCytolysinAmino Acid SequenceRabbitsBinding siteVibrio choleraeEuropean journal of biochemistry
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Repair of a Bacterial Small β-Barrel Toxin Pore Depends on Channel Width

2017

ABSTRACT Membrane repair emerges as an innate defense protecting target cells against bacterial pore-forming toxins. Here, we report the first paradigm of Ca2+-dependent repair following attack by a small β-pore-forming toxin, namely, plasmid-encoded phobalysin of Photobacterium damselae subsp. damselae. In striking contrast, Vibrio cholerae cytolysin, the closest ortholog of phobalysin, subverted repair. Mutational analysis uncovered a role of channel width in toxicity and repair. Thus, the replacement of serine at phobalysin´s presumed channel narrow point with the bulkier tryptophan, the corresponding residue in Vibrio cholerae cytolysin (W318), modulated Ca2+ influx, lysosomal exocytosi…

0301 basic medicineBacterial ToxinsAerolysinmedicine.disease_causeMicrobiologySerine03 medical and health sciencesNanoporesVirologyExtracellularmedicineHumansVibrio choleraeChemistryToxinPerforinCell MembraneQR1-502Transmembrane proteinCell biology030104 developmental biologyPhotobacterium damselaeVibrio choleraeCalciumCytolysinResearch ArticlemBio
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