Search results for "Alcohol"

showing 10 items of 1798 documents

Incorporating Functional Genomic Information to Enhance Polygenic Signal and Identify Variants Involved in Gene-by-Environment Interaction for Young …

2018

BACKGROUND: Characterizing aggregate genetic risk for alcohol misuse and identifying variants involved in gene-by-environment (G × E) interaction effects has so far been a major challenge. We hypothesized that functional genomic information could be used to enhance detection of polygenic signal underlying alcohol misuse and to prioritize identification of single nucleotide polymorphisms (SNPs) most likely to exhibit G × E effects.METHODS: We examined these questions in the young adult FinnTwin12 sample (n = 1,170). We used genomewide association estimates from an independent sample to derive 2 types of polygenic scores for alcohol problems in FinnTwin12. Genomewide polygenic scores included…

Male0301 basic medicineMultifactorial InheritanceTwinsMedicine (miscellaneous)AlcoholToxicologyBioinformaticschemistry.chemical_compound0302 clinical medicineGene–environment interactionYoung adultta515FinlandGeneticsalcoholta3142GenomicsgenomiikkaPhenotypeAlcoholismPsychiatry and Mental health/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemaleGenomic informationgeneettiset tekijätfunctional genomicsFunctional genomicsAdultympäristötekijätSingle-nucleotide polymorphismBiologyta3111Polymorphism Single NucleotideArticleYoung Adult03 medical and health sciencesSDG 3 - Good Health and Well-beingHumansGenetic Predisposition to Diseasenuoret aikuisetGenetic Variationalkoholiongelmatpolygenic scores030104 developmental biologychemistryGenomewide associationgene-environment interplayGene-Environment Interaction030217 neurology & neurosurgeryGenome-Wide Association StudyAlcoholism, Clinical and Experimental Research
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Comprehensive Genomic and Transcriptomic Analysis of Three Synchronous Primary Tumours and a Recurrence from a Head and Neck Cancer Patient

2021

Synchronous primary malignancies occur in a small proportion of head and neck squamous cell carcinoma (HNSCC) patients. Here, we analysed three synchronous primaries and a recurrence from one patient by comparing the genomic and transcriptomic profiles among the tumour samples and determining the recurrence origin. We found remarkable levels of heterogeneity among the primary tumours, and through the patterns of shared mutations, we traced the origin of the recurrence. Interestingly, the patient carried germline variants that might have predisposed him to carcinogenesis, together with a history of alcohol and tobacco consumption. The mutational signature analysis confirmed the impact of alc…

Male0301 basic medicineOncologyAlcohol exposuremedicine.disease_causeGermlineNeoplasms Multiple PrimaryTranscriptomeFatal Outcome0302 clinical medicineMedicineBiology (General)Immune cell infiltrationSpectroscopysynchronous multiple primary malignanciesSmokersimmune cell infiltrationGenomicsGeneral MedicineComputer Science Applicationswhole exome sequencing (WES)Chemistrysomatic single nucleotide variantsHead and Neck Neoplasms030220 oncology & carcinogenesismedicine.medical_specialtyrecurrent head and neck squamous cell carcinoma (HNSCC)Alcohol DrinkingQH301-705.5ArticleCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineHumansPhysical and Theoretical ChemistryQD1-999Molecular BiologyAgedNeoplasm Stagingbusiness.industryGene Expression ProfilingOrganic ChemistryHead and neck cancerRNA sequencing (RNA-seq)medicine.diseaseHead and neck squamous-cell carcinoma030104 developmental biologygermline variantsMutationClinical valueNeoplasm Recurrence LocalbusinessCarcinogenesisInternational Journal of Molecular Sciences
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Shared genetic etiology between alcohol dependence and major depressive disorder

2018

Supplemental Digital Content is available in the text.

Male0301 basic medicineOncologyAlcoholism/geneticsMultifactorial Inheritancealcohol dependenceMedizinGenome-wide association study0302 clinical medicineRisk FactorsGermanyMedicineGenetics (clinical)Brief ReportRegression analysisAlcoholismPsychiatry and Mental healthpolygenic risk scoresMeta-analysisComputingMethodologies_DOCUMENTANDTEXTPROCESSINGRegression AnalysisMajor depressive disorderdisease comorbidityDepressive Disorder Major/geneticsmedicine.medical_specialtyBFPolymorphism Single Nucleotide03 medical and health sciencespsychiatric genomics consortiumInternal medicineGeneticsHumansGenetic Predisposition to Diseaseddc:610Biological PsychiatryDepressive Disorder Majormajor depressive disorderbusiness.industryAlcohol dependenceCase-control studymedicine.diseaseComorbidity030104 developmental biologySample size determinationCase-Control Studiesgenome-wide association studiesRC0321business030217 neurology & neurosurgeryGenome-Wide Association Study
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Podoplanin discriminates distinct stromal cell populations and a novel progenitor subset in the liver

2015

Podoplanin/gp38+ stromal cells present in lymphoid organs play a central role in the formation and reorganization of the extracellular matrix and in the functional regulation of immune responses. Gp38+ cells are present during embryogenesis and in human livers of primary biliary cirrhosis. Since little is known about their function, we studied gp38+ cells during chronic liver inflammation in models of biliary and parenchymal liver fibrosis and steatohepatitis. Gp38+ cells were analyzed using flow cytometry and confocal microscopy, and the expression of their steady state and inflammation-associated genes was evaluated from healthy and inflamed livers. Gp38+ cells significantly expanded in …

Male0301 basic medicinePathologymedicine.medical_specialtyATP Binding Cassette Transporter Subfamily BStromal cellPhysiologyLiver and Biliary Tract Physiology/PathophysiologyPopulationCell SeparationBiologyLiver Cirrhosis Experimental03 medical and health sciencesPrimary biliary cirrhosisAntigens CDNon-alcoholic Fatty Liver DiseasePhysiology (medical)medicineAnimalsAC133 AntigenProgenitor celleducationCells CulturedGlycoproteinsMice KnockoutLiver injuryeducation.field_of_studyMembrane GlycoproteinsMicroscopy ConfocalHepatologyLiver Cirrhosis BiliaryStem CellsLiver cellGastroenterologyFlow Cytometrymedicine.diseaseMolecular biologyMice Inbred C57BLPhenotype030104 developmental biologyGene Expression RegulationLiverChemical and Drug Induced Liver InjuryInflammation MediatorsStromal CellsStem cellSteatohepatitisPeptidesBiomarkersAmerican Journal of Physiology-Gastrointestinal and Liver Physiology
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Increased ethanol consumption after interruption of fat bingeing

2018

There is a marked comorbidity between alcohol abuse and eating disorders, especially in the young population. We have previously reported that bingeing on fat during adolescence increases the rewarding effects of ethanol (EtOH). The aim of the present work was to study if vulnerability to EtOH persists after cessation of binge eating. OF1 mice binged on fat (HFB: high-fat binge) during adolescence (PND 25-43) and were tested for 15 days after the last access to HFB (on PND 59) using the self-administration paradigm, the conditioned place preference (CPP) and locomotor sensitization to ethanol. Our results showed that after 15 days of cessation of fat ingestion, mice increased their consumpt…

Male0301 basic medicinePhysiologySocial Scienceslcsh:MedicineAlcohol abuseDrug AddictionBiochemistryFatsMicechemistry.chemical_compound0302 clinical medicineCocaineMedicine and Health SciencesPsychologyIngestionPublic and Occupational HealthBulimiaOvereatinglcsh:ScienceMultidisciplinaryOrganic CompoundsLipidsBody FluidsChemistryEating disordersBloodBehavioral PharmacologyPhysical SciencesAnatomymedicine.symptomResearch Articlemedicine.medical_specialtyAdolescentAlcohol DrinkingSubstance-Related DisordersAddiction03 medical and health sciencesAlkaloidsRecreational Drug UseInternal medicineMental Health and Psychiatrymental disordersmedicineAnimalsHumansNutritionPharmacologyEthanolEthanolBinge eatingBiological Locomotionbusiness.industryOrganic Chemistrylcsh:RChemical CompoundsBiology and Life Sciencesmedicine.diseaseDietary FatsConditioned place preferenceDietLocomotor sensitization030104 developmental biologyEndocrinologychemistryFoodAlcoholslcsh:Qbusiness030217 neurology & neurosurgeryPLOS ONE
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Lean NAFLD: A Distinct Entity Shaped by Differential Metabolic Adaptation

2020

Background and Aims: Nonalcoholic fatty liver disease (NAFLD) affects a quarter of the adult population. A significant subset of patients are lean, but their underlying pathophysiology is not well understood. Approach and Results: We investigated the role of bile acids (BAs) and the gut microbiome in the pathogenesis of lean NAFLD. BA and fibroblast growth factor (FGF) 19 levels (a surrogate for intestinal farnesoid X receptor [FXR] activity), patatin-like phospholipase domain containing 3 (PNPLA3), and transmembrane 6 superfamily member 2 (TM6SF2) variants, and gut microbiota profiles in lean and nonlean NAFLD were investigated in a cohort of Caucasian patients with biopsy-proven NAFLD (n …

Male0301 basic medicineReceptors Cytoplasmic and NuclearGut floraMice0302 clinical medicineNon-alcoholic Fatty Liver DiseaseFibrosisNonalcoholic fatty liver diseasebiologyMiddle AgedNAFLD; bile acids; fibrosis; gut microbiota; leanPhospholipases A2 Calcium-IndependentFemale030211 gastroenterology & hepatologyfibrosiAdultmedicine.medical_specialtydigestive systemBile Acids and SaltsCyclic N-Oxides03 medical and health sciencesThinnessInternal medicineNAFLDmedicinebile acidAnimalsHumansbile acidsHepatologygut microbiotabusiness.industryFGF15fibrosisnutritional and metabolic diseasesFGF19leanmedicine.diseasebiology.organism_classificationNAFLD fibrosis lean bile acids gut microbiotadigestive system diseasesGastrointestinal MicrobiomeFibroblast Growth FactorsMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologyFarnesoid X receptorSteatohepatitisbusinessTropanesTM6SF2
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Sample size planning of two-arm superiority and noninferiority survival studies with discrete follow-up

2016

In clinical trials using lifetime as primary outcome variable, it is more the rule than the exception that even for patients who are failing in the course of the study, survival time does not become known exactly since follow-up takes place according to a restricted schedule with fixed, possibly long intervals between successive visits. In practice, the discreteness of the data obtained under such circumstances is plainly ignored both in data analysis and in sample size planning of survival time studies. As a framework for analyzing the impact of making no difference between continuous and discrete recording of failure times, we use a scenario in which the partially observed times are assig…

Male0301 basic medicineStatistics and ProbabilityScheduleTime FactorsEpidemiologyBiostatistics01 natural sciences010104 statistics & probability03 medical and health sciencesStatisticsEconometricsHumans0101 mathematicsRepresentation (mathematics)Proportional Hazards ModelsMathematicsClinical Trials as TopicLikelihood FunctionsModels StatisticalProstatic NeoplasmsEstimatorGridSurvival AnalysisConfidence intervalAlcoholismVariable (computer science)030104 developmental biologySample size determinationSample SizeParametric modelFollow-Up StudiesStatistics in Medicine
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Ketogenic Diet Decreases Alcohol Intake in Adult Male Mice

2021

The classic ketogenic diet is a diet high in fat, low in carbohydrates, and well-adjusted proteins. The reduction in glucose levels induces changes in the body’s metabolism, since the main energy source happens to be ketone bodies. Recent studies have suggested that nutritional interventions may modulate drug addiction. The present work aimed to study the potential effects of a classic ketogenic diet in modulating alcohol consumption and its rewarding effects. Two groups of adult male mice were employed in this study, one exposed to a standard diet (SD, n = 15) and the other to a ketogenic diet (KD, n = 16). When a ketotic state was stable for 7 days, animals were exposed to the oral self-a…

Male0301 basic medicinemedicine.medical_specialtyAdenosineketosisAlcohol DrinkingDopaminemedicine.medical_treatmentmedia_common.quotation_subjectGene ExpressionArticleEatingMice03 medical and health sciences0302 clinical medicineDopamineInternal medicinemedicineAnimalsTX341-641media_commonMotivationNutrition and DieteticsEthanolNutrition. Foods and food supplyCannabinoidsalcoholbusiness.industryAddictionketonemedicine.diseaseAdenosineketogenicAlcoholismDisease Models Animal030104 developmental biologyEndocrinologyadenosineKetone bodiesCannabinoiddopamineKetosisDiet KetogenicEnergy sourcebusiness030217 neurology & neurosurgeryFood Sciencemedicine.drugKetogenic dietNutrients
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Influence of lifestyle factors and staple foods from the Mediterranean diet on non-alcoholic fatty liver disease among older individuals with metabol…

2020

Objective: Non-alcoholic fatty liver disease (NAFLD) is the most common cause of chronic liver morbidity. This condition often is accompanied by obesity, diabetes, and metabolic syndrome (MetS). The aim of this study was to evaluate the connection between lifestyle factors and NAFLD in individuals with MetS. Methods: A cross-sectional study with 328 participants (55-75 y of age) diagnosed with MetS participating in the PREDIMED-Plus trial was conducted. NAFLD status was evaluated using the non-invasive hepatic steatosis index (HSI). Sociodemographic, clinical, and dietary data were collected. Adherence to the Mediterranean diet (mainly assessed by the consumption of olive oil, nuts, legumes…

Male0301 basic medicinemedicine.medical_specialtyMediterranean dietEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismDiet MediterraneanDiet SurveysSeverity of Illness Index03 medical and health sciences0302 clinical medicineNon-alcoholic Fatty Liver DiseaseRisk FactorsSurveys and QuestionnairesNAFLDInternal medicineDiabetes mellitusmedicineHumansObesityExerciseLife StyleAgedRandomized Controlled Trials as TopicNutritionMetabolic SyndromeInflammation2. Zero hunger030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryFatty liverMiddle Agedmedicine.diseaseObesityConfidence interval3. Good healthCross-Sectional StudiesSpainRelative riskLinear ModelsPatient ComplianceFemaleSteatosisMetabolic syndromebusinessNutrition
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Insulin resistance alters hepatic ethanol metabolism: studies in mice and children with non-alcoholic fatty liver disease

2015

Objective Increased fasting blood ethanol levels, suggested to stem from an increased endogenous ethanol synthesis in the GI tract, are discussed to be critical in the development of non-alcoholic fatty liver disease (NAFLD). The aim of the present study was to further delineate the mechanisms involved in the elevated blood ethanol levels found in patients with NAFLD. Design In 20 nutritionally and metabolically screened children displaying early signs of NAFLD and 29 controls (aged 5–8 years), ethanol plasma levels were assessed. Ethanol levels along the GI tract, in vena cava and portal vein, intestinal and faecal microbiota, and activity of alcohol dehydrogenase (ADH) and cytochrome P450…

Male0301 basic medicinemedicine.medical_specialtyStatistics as TopicEndogenyBody Mass IndexMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceNon-alcoholic Fatty Liver DiseaseInternal medicinemedicineAnimalsHumansNutritional Physiological PhenomenaEthanol metabolismChildAlcohol dehydrogenaseEthanolEthanolbiologyFatty liverAlcohol DehydrogenaseGastroenterologyCytochrome P-450 CYP2E1CYP2E1medicine.disease030104 developmental biologyEndocrinologyLiverchemistryChild Preschoolbiology.proteinFemale030211 gastroenterology & hepatologyInsulin ResistanceAntibodyGut
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