Search results for "Allele frequency"

showing 10 items of 224 documents

Mutational screening of the USH2A gene in Spanish USH patients reveals 23 novel pathogenic mutations

2011

Abstract Background Usher Syndrome type II (USH2) is an autosomal recessive disorder, characterized by moderate to severe hearing impairment and retinitis pigmentosa (RP). Among the three genes implicated, mutations in the USH2A gene account for 74-90% of the USH2 cases. Methods To identify the genetic cause of the disease and determine the frequency of USH2A mutations in a cohort of 88 unrelated USH Spanish patients, we carried out a mutation screening of the 72 coding exons of this gene by direct sequencing. Moreover, we performed functional minigene studies for those changes that were predicted to affect splicing. Results As a result, a total of 144 DNA sequence variants were identified.…

AdultMaleSequence VariantsAdolescentGenotypegenetic structuresUsher syndromeDNA Mutational AnalysisMutation Missenselcsh:MedicineBiologymedicine.disease_causeExonYoung AdultUSH2ARetinitis pigmentosaGenotypemedicineotorhinolaryngologic diseasesHumansGenetics(clinical)Pharmacology (medical)<it>USH2A</it>GeneAllele frequencyGenetics (clinical)GeneticsMedicine(all)MutationExtracellular Matrix ProteinsResearchlcsh:RGeneral MedicineExonsMiddle Agedmedicine.diseaseeye diseasesPhenotypeSpainMutationFemalesense organsUsher SyndromeUsher SyndromesMutationsMinigeneOrphanet Journal of Rare Diseases
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HLA and killer cell immunoglobulin-like receptors influence the natural course of CMV infection.

2014

Background. Natural killer (NK) cells provide a major defense against cytomegalovirus (CMV) infection through the interaction of their surface receptors, including the activating and inhibitory killer immunoglobulinlike receptors (KIRs), and human leukocyte antigens (HLA) class I molecules. This study assessed whether the KIR and HLA repertoire may influence the risk of developing symptomatic or asymptomatic disease after primary CMV infection in the immunocompetent host. Methods. Sixty immunocompetent patients with primary symptomatic CMV infection were genotyped for KIR and their HLA ligands, along with 60 subjects with a previous asymptomatic infection as controls. Results. The frequency…

AdultMaleSettore MED/07 - Microbiologia E Microbiologia ClinicacytomegaloviruSettore MED/17 - Malattie InfettiveAdolescentGenotypeCytomegalovirusHuman leukocyte antigenAsymptomaticYoung AdultGene FrequencyReceptors KIRmedicineSettore MED/05 - Patologia ClinicaImmunology and AllergyHumansGenetic Predisposition to DiseaseAlleleReceptorAllele frequencyAgedSettore MED/04 - Patologia GeneralebiologyHaplotypeHistocompatibility Antigens Class IMiddle AgedVirologyKIRHLAInfectious DiseasesImmunologyCytomegalovirus Infectionsbiology.proteinFemaleAntibodymedicine.symptomKIR2DS4The Journal of infectious diseases
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Polymorphisms of cyclo-oxygenases and 5-lipo-oxygenase-activating protein are associated with chronic spontaneous urticaria and urinary leukotriene E4

2011

The mechanisms of chronic spontaneous urticaria (CSU) continue to be unknown. Our working hypothesis is that polymorphisms of cyclo-oxygenases and 5-lipo-oxygenase-activating protein may be involved in the pathways leading to CSU. We examined five candidate polymorphisms of cyclo-oxygenases 1 and 2 and of 5-lipo-oxygenase-activating protein in 109 controls and in 94 CSU patients from Northern Italy. We also examined the levels of urinary leukotriene E4 (LTE4) before and after challenge with ASA. A multiple regression model was found to show that COX-2 5'UTR T/G, COX-2 Exon 10 T/C, and FLAP -336 G/A polymorphisms were significantly associated with CSU, with the minor allele more represented …

AdultMaleSettore MED/09 - Medicina InternaAdolescentGenotypeUrticariaUrinary system5-Lipoxygenase-Activating ProteinsSingle-nucleotide polymorphismDermatologyYoung Adultchemistry.chemical_compoundExonchronic spontaneous urticaria hypersensivity to aspirin cyclo-oxygenases 5-lipo-oxygenase-activating protein urinary leukotriene E4GenotypeHumansMedicineAllele5-lipoxygenase-activating proteinAgedLeukotriene E4Settore MED/04 - Patologia GeneraleLeukotriene E4Polymorphism Geneticbiologybusiness.industryMiddle AgedMinor allele frequencychemistryProstaglandin-Endoperoxide SynthasesChronic DiseaseImmunologybiology.proteinFemalebusiness
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Relationship Between Human Leucocyte Antigen Class I and Class II and Chronic Idiopathic Urticaria Associated With Aspirin and/or NSAIDs Hypersensiti…

2006

Background. HLA genes play a role in the predisposition of several diseases. The aim was to analyze the prevalence of HLA class I phenotypes and HLA-DRB1*genotype in patients with CIU associated with ASA and NSAIDs hypersensitivity (AICU).Methods. 69 patients with AICU, and 200 healthy subjects.Results. Subjects with HLA-B44 and HLA-Cw5 antigens were more represented in patients with AICU than in control group. Subjects with HLA-A11, HLA-B13, HLACw4, and HLA-Cw7 antigen were more represented in control group than in patients with AICU. Multiple logistic regression demonstrated an association of HLA-Cw4 and HLA-Cw7 with a lower risk of AICU, whereas carriers of HLA-B44 phenotype had a higher…

AdultMaleSettore MED/09 - Medicina InternaChronic Idiopathic UrticariaGenotypeUrticariahuman leucocyte antigen class IImmunologyGenes MHC Class IIAnti-Inflammatory AgentsHuman leukocyte antigenLower riskDrug HypersensitivityResearch CommunicationAntigenGene FrequencyRisk FactorsGenotypelcsh:PathologyMedicineHumansAlleleAllele frequencyAllelesAspirinAspirinbusiness.industryAnti-Inflammatory Agents Non-SteroidalHistocompatibility Antigens Class ICase-control studyCell BiologyHLA-DR AntigensMiddle AgedNSAIDhuman leucocyte antigen class I; human leucocyte antigen class II; chronic idiopathic urticaria; aspirin; NSAIDs; hypersensitivityhuman leucocyte antigen class IIMHC Class IIPhenotypeGenesCase-Control StudiesImmunologyFemalehypersensitivityNon-Steroidalbusinesslcsh:RB1-214medicine.drugHLA-DRB1 ChainsMediators of Inflammation
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Role of TLR4 Receptor Polymorphisms in Boutonneuse Fever

2005

The genetics of the interaction between host and microbes plays an essential role in the survival of the individual and attainment of longevity. The activation of toll-like receptor (TLR)4 plays a key role in natural and clonotypic immune responses. We evaluated whether TLR4 genotype is a component of genetic background protective versus rickettsiosis and whether this background influences longevity. We genotyped for +896A/G TLR4 polymorphism 78 patients affected by Boutonneuse fever, 78 age-matched controls and 78 advanced age individuals from Sicily. The +869G allele, that attenuates receptor signalling, was significantly overrepresented in patients in comparison with age-matched control…

AdultMaleSettore MED/09 - Medicina InternaGenotypemedia_common.quotation_subjectImmunologyBoutonneuse Feverpolymorphism03 medical and health sciences0302 clinical medicineGene FrequencyPolymorphism (computer science)GenotypemedicineHumansImmunology and AllergyTLR4 receptorAlleleTLR4 receptor; polymorphisms; Boutonneuse feverSicilyAllele frequencyAllelesAgedmedia_commonAged 80 and overPharmacologyPolymorphism Geneticbusiness.industryLongevityDNAMiddle Agedmedicine.diseaseToll-Like Receptor 4Boutonneuse feverRickettsiosis030220 oncology & carcinogenesisImmunologyTLR4Femalebusiness030215 immunologyInternational Journal of Immunopathology and Pharmacology
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Clinical features and molecular genetic analysis in a Turkish family with oral white sponge nevus

2018

Background Oral white sponge nevus (WSN) is a rare autosomal dominant benign condition, characterized by asymptomatic spongy white plaques. Mutations in Keratin 4 (KRT4) and 13 (KRT13) have been shown to cause WSN. Familial cases are uncommon due to irregular penetrance. Thus, the aim of the study was: a) to demonstrate the clinical and histopathological features of a three-generation Turkish family with oral WSN b) to determine whether KRT4 or KRT13 gene mutation was the molecular basis of WSN. Material and Methods Out of twenty members of the family ten were available for assessment. Venous blood samples from six affected and five unaffected members and 48 healthy controls were obtained f…

AdultMaleTurkish populationAdolescentTurkeyLeukokeratosis Hereditary MucosalGene mutationBiology030207 dermatology & venereal diseases03 medical and health sciencesExonYoung Adult0302 clinical medicineWhite sponge nevusmedicineHumansChildGeneral DentistryGeneAllele frequencyGeneticsOral Medicine and PathologyResearchKeratin-13030206 dentistryMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseasePenetrancePedigreeOtorhinolaryngologyKeratin 4Case-Control StudiesUNESCO::CIENCIAS MÉDICASCytogenetic AnalysisMutationbiology.proteinSurgeryKeratin-4Medicina Oral, Patología Oral y Cirugía Bucal
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The Atrial Natriuretic Peptide Genetic Variant rs5068 Is Associated With a Favorable Cardiometabolic Phenotype in a Mediterranean Population

2013

OBJECTIVE We hypothesized that the minor allele of the atrial natriuretic peptide (ANP) genetic variant rs5068 is associated with a favorable cardiometabolic phenotype in a general Mediterranean population. RESEARCH DESIGN AND METHODS We genotyped a random sample of the residents of Ventimiglia di Sicilia, Sicily, for rs5068. RESULTS Genotype frequencies of rs5068 are AA, 93.5%; AG, 6.4%; and GG, 0.1%. All subsequent analyses are AA versus AG+GG. After adjusting for age and sex, the minor G allele is associated with lower BMI (estimate [SE]: −1.7 kg/m2 [0.8], P = 0.04). In the AG+GG group, males with HDL cholesterol levels &amp;lt;40 mg/dL are less frequent (P = 0.05) and obesity tends to …

AdultMalegenetic variantCardiovascular and Metabolic Riskmedicine.medical_specialtySettore MED/09 - Medicina InternaGenotypeEndocrinology Diabetes and MetabolismPopulationBlood PressureGene Frequencycardiometabolic phenotypeAtrial natriuretic peptideInternal medicineNatriuretic Peptide Brainatrial natriuretic peptideInternal MedicinemedicineHumansAlleleeducationAllele frequencyAgedOriginal ResearchAdvanced and Specialized Nursingeducation.field_of_studybusiness.industryOdds ratioMiddle Agedmedicine.diseaseGenotype frequencyMinor allele frequencyPhenotypeEndocrinologyFemaleMetabolic syndromebusinessAtrial Natriuretic FactorDiabetes Care
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Association between the MHC class I gene HFE polymorphisms and longevity: a study in Sicilian population.

2001

Classes I and II human leukocyte antigens (HLA) genes encode highly polymorphic heterodimeric glycoproteins involved in the control of immune responses. The HLA class I gene HFE seemingly no longer participates in immunity because it has lost its ability to bind peptides and it has acquired the ability to form complex with the receptor for iron-binding transferrin by regulating iron uptake by intestinal cells. Thus, it indirectly regulates immune responses too, because iron availability plays a role in specific and non-specific immune responses. The distribution of HFE polymorphisms in Sicilian centenarians and nonagenarians was studied to evaluate if HFE alleles might be represented differ…

AdultMalemedia_common.quotation_subjectImmunologyPopulationLongevityGenes MHC Class IHuman leukocyte antigenBiologyCompound heterozygositymedicine.disease_causeGene FrequencyHLA AntigensGeneticsmedicineHumansAlleleeducationHemochromatosis ProteinAllele frequencySicilyGenetics (clinical)Allelesmedia_commonAgedGeneticsAged 80 and overeducation.field_of_studyMutationPolymorphism GeneticHaplotypeHistocompatibility Antigens Class ILongevityMembrane ProteinsMiddle AgedHaplotypesFemaleGenes and immunity
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Influence of CYP3A5 and ABCB1 gene polymorphisms and other factors on tacrolimus dosing in Caucasian liver and kidney transplant patients

2011

Tacrolimus is a substrate of cytochrome P4503A (CYP3A) enzymes as well as of the drug transporter ABCB1. We have investigated the possible influence of CYP3A5 and ABCB1 single nucleotide polymorphisms (SNPs) and other factors (e.g. albumin, hematocrit and steroids) on tacrolimus blood levels achieved in a population of Caucasian liver (n=51) and kidney (n=50) transplant recipients. At 1, 3 and 6 months after transplantation, tacrolimus doses (mg/kg/day) and trough blood levels (C0) were recorded and the weight-adjusted tacrolimus dosage (mg/kg/day) was calculated. Polymerase chain reaction followed by restriction fragment length polymorphism analysis was used for genotyping CYP3A5*1 and *3 …

AdultMalemedicine.medical_specialtyATP Binding Cassette Transporter Subfamily BGenotypemedicine.medical_treatmentDNA Mutational AnalysisPopulationSingle-nucleotide polymorphismLiver transplantationBiologyKidneyPolymorphism Single NucleotideGastroenterologyBiomarkers PharmacologicalTacrolimusWhite PeopleGene FrequencyInternal medicineGeneticsmedicineCytochrome P-450 CYP3AHumansDrug Dosage CalculationsATP Binding Cassette Transporter Subfamily B Member 1educationAllele frequencyAllelesKidney transplantationAgededucation.field_of_studyKidney metabolismGeneral MedicineMiddle Agedmedicine.diseaseKidney TransplantationTacrolimusLiver TransplantationTransplantationsurgical procedures operativeItalyLiverImmunologySettore BIO/14 - FarmacologiaPharmacogenetics CYP3A5 ABCB1 TacrolimusTransplant patientsFemaleImmunosuppressive AgentsPolymorphism Restriction Fragment LengthInternational Journal of Molecular Medicine
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Association between polymorphisms of endothelial nitric oxide synthase gene (NOS3) and left posterior wall thickness (LPWT) of the heart in Fabry dis…

2008

Fabry disease is an X-chromosomal storage disorder due to loss-of-function mutations of the GLA gene encoding the lysosomal enzyme α-galactosidase A. Accumulating glycosphingolipid deposits disturb the function of various cells, in particular that of myocytes, arterial smooth-muscle cells, and vascular endothelium. Hypertrophic cardiomyopathy, for example measured by left posterior wall thickness (LPWT) of the heart, represents a major component of Fabry disease morbidity in adult patients. Endothelium-derived nitric oxide (eNO), produced by eNO synthase (eNOS), is a key regulator of vessel wall function and cardiovascular homeostasis. We analysed the effect of the polymorphisms c.894G > T …

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIMutation MissenseMinisatellite RepeatsYoung AdultSex FactorsGene FrequencyInternal medicineGermanyGenotypeGeneticsMedicineHumansGenetic Predisposition to DiseaseAlleleChildAllele frequencyGenetics (clinical)AgedUltrasonographyGeneticsAlpha-galactosidasePolymorphism Geneticbiologybusiness.industryMyocardiumHaplotypeHypertrophic cardiomyopathyNitric Oxide Synthase Type IIIExonsCardiomyopathy HypertrophicMiddle Agedmedicine.diseaseFabry diseaseIntronsEndocrinologyPhenotypeHaplotypesCase-Control Studiesalpha-Galactosidasebiology.proteinFabry DiseaseRegression AnalysisFemaleHypertrophy Left VentricularbusinessJournal of inherited metabolic disease
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