Search results for "Allergy"

showing 10 items of 3181 documents

IL-2 Expression in Activated Human Memory FOXP3(+) Cells Critically Depends on the Cellular Levels of FOXP3 as Well as of Four Transcription Factors …

2012

The human CD4(+)FOXP3(+) T cell population is heterogeneous and consists of various subpopulations which remain poorly defined. Anergy and suppression are two main functional characteristics of FOXP3(+)Treg cells. We used the anergic behavior of FOXP3(+)Treg cells for a better discrimination and characterization of such subpopulations. We compared IL-2-expressing with IL-2-non-expressing cells within the memory FOXP3(+) T cell population. In contrast to IL-2-non-expressing FOXP3(+) cells, IL-2-expressing FOXP3(+) cells exhibit intermediate characteristics of Treg and Th cells concerning the Treg cell markers CD25, GITR, and Helios. Besides lower levels of FOXP3, they also have higher levels…

lcsh:Immunologic diseases. AllergyT cellLymphocytePopulationImmunologychemical and pharmacologic phenomenaBiologylymphocyteFlow cytometrytranscription factorsmedicineImmunology and Allergycytokine expressionIL-2 receptorddc:610educationTranscription factorOriginal Researcheducation.field_of_studyIL-2 expressionmedicine.diagnostic_testT cell activationflow cytometryhuman Treg cellsFOXP3T cellhemic and immune systemsmemory Th cellsPhenotypeCell biologymedicine.anatomical_structureImmunologylcsh:RC581-607610 Medizin und GesundheitFrontiers in immunology
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Quantitative Prediction of the Landscape of T Cell Epitope Immunogenicity in Sequence Space

2019

Immunodominant T cell epitopes preferentially targeted in multiple individuals are the critical element of successful vaccines and targeted immunotherapies. However, the underlying principles of this "convergence" of adaptive immunity among different individuals remain poorly understood. To quantitatively describe epitope immunogenicity, here we propose a supervised machine learning framework generating probabilistic estimates of immunogenicity, termed "immunogenicity scores," based on the numerical features computed through sequence-based simulation approximating the molecular scanning process of peptides presented onto major histocompatibility complex (MHC) by the human T cell receptor (T…

lcsh:Immunologic diseases. AllergyT cellT-LymphocytesImmunologyReceptors Antigen T-CellDatasets as TopicEpitopes T-Lymphocytechemical and pharmacologic phenomenaComputational biologyBiologyAdaptive ImmunityimmunogenicityMajor histocompatibility complexEpitopeMajor Histocompatibility ComplexmedicineImmunology and AllergyHumansComputer SimulationAntigen PresentationImmunodominant EpitopesRepertoireImmunogenicityT-cell receptorComputational BiologyAcquired immune systemmedicine.anatomical_structuremachine learningescape mutationbiology.proteinThermodynamicsT cell receptor repertoireSequence space (evolution)lcsh:RC581-607T cell epitopeFrontiers in Immunology
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γδ T Cells Cross-Link Innate and Adaptive Immunity in Mycobacterium tuberculosis Infection

2011

Protective immunity against mycobacterial infections such asMycobacterium tuberculosisis mediated by interactions between specific T cells and activated antigen presenting cells. To date, many aspects of mycobacterial immunity have shown that innate cells could be the key elements that substantially may influence the subsequent adaptive host response. During the early phases of infection, innate lymphocyte subsets play a pivotal role in this context. Here we summarize the findings of recent investigations onγδT lymphocytes and their role in tuberculosis immunity.

lcsh:Immunologic diseases. AllergyT-LymphocytesT cellImmunologyReview ArticleAdaptive ImmunityLymphocyte ActivationMycobacterium tuberculosisImmune systemAntigenImmunitymedicineAnimalsHumansTuberculosisImmunology and AllergyIL-2 receptorAntigen-presenting cellbiologyReceptors Antigen T-Cell gamma-deltaMycobacterium tuberculosisGeneral MedicineAcquired immune systembiology.organism_classificationVirologyImmunity Innategamma delta T cells Mycobacterium tuberculosismedicine.anatomical_structureImmunologylcsh:RC581-607Immunologic Memory
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Are zinc-bound metallothionein isoforms (I+II and III) involved in impaired thymulin production and thymic involution during ageing?

2004

Abstract Background With advancing age, thymic efficiency shows progressive decline due to thymic involution allowing impaired cell-mediated immunity and the appearance of age-related diseases. The intrinsic cause of thymic involution is still undefined. Chronic inflammation and high glucocorticoids (GCs) may be involved. However, transgenic mice, with increased GC sensitivity and over expression of GC receptors, display delayed age-associated thymic involution. This fact suggests that other substances may affect thymic involution. Among them, both isoforms of metallothioneins (MTs) I+II and III are the major candidates because their increments leads to organ atrophy in constant stress and …

lcsh:Immunologic diseases. AllergyThymic involutionIL-6glucocorticoidsResearchzinclcsh:Geriatricslcsh:RC952-954.6TECslongevityinflammationageinglcsh:RC581-607MetallothioneinsImmunityageing : IA
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Functional Signatures of Human CD4 and CD8 T Cell Responses to Mycobacterium tuberculosis

2014

With 1.4 million deaths and 8.7 million new cases in 2011, tuberculosis (TB) remains a global health care problem and together with HIV and Malaria represents one of the three infectious diseases world-wide. Control of the global TB epidemic has been impaired by the lack of an effective vaccine, by the emergence of drug-resistant forms of Mycobacterium tuberculosis (Mtb) and by the lack of sensitive and rapid diagnostics. It is estimated, by epidemiological reports, that one third of the world's population is latently infected with Mtb, but the majority of infected individuals develop long-lived protective immunity, which controls and contains Mtb in a T cell-dependent manner. Development o…

lcsh:Immunologic diseases. AllergyTuberculosisPopulationImmunologyReview ArticleDiseaseMycobacterium tuberculosis03 medical and health sciences0302 clinical medicineImmunitymedicineM. tuberculosiscytokineinfection.Immunology and AllergyCytotoxic T celleducationImmunodeficiency030304 developmental biologySettore MED/04 - Patologia Generale0303 health scienceseducation.field_of_studydiseasebiologybusiness.industrymedicine.diseasebiology.organism_classificationVirologyinfection3. Good healthhuman memory T cellsImmunologyCytokinesbusinesslcsh:RC581-607human memory T cellMalaria030215 immunologyM. tuberculosi
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Immunological Markers for PML Prediction in MS Patients Treated with Natalizumab

2015

International audience; Natalizumab (NTZ), a monoclonal antibody recognizing the alpha4 integrin chain, has been approved for the treatment of active multiple sclerosis, but expose to the onset of a rare side effect, progressive multifocal leukoencephalopathy (PML). Estimating the individual risk of PML in NTZ-treated patients is a major challenge, and therapeutic strategies are mainly guided by the overall PML risk assessed by identified risk factors: JC virus (JCV) seropositivity, treatment duration (with peak incidence after 24 months), and the previous use of immunosuppressive therapies. Given that this stratification does not yet allow a precise individual prediction of PML, other pred…

lcsh:Immunologic diseases. Allergy[SDV.IMM] Life Sciences [q-bio]/ImmunologySide effectmedicine.drug_classvirusesImmunologyJC virusReview Articlerisk stratificationCD11aJC virusmultiple sclerosismedicine.disease_causeMonoclonal antibodyCD49dprogressive multifocal leukoencephalopathyNatalizumabeffector memory T-cellst effector memory cellsImmunology and AllergyMedicineselectinPMLbusiness.industryMultiple sclerosisProgressive multifocal leukoencephalopathyvirus diseasesmedicine.disease3. Good healthJCVImmunologySelectins[SDV.IMM]Life Sciences [q-bio]/Immunologylcsh:RC581-607businessmedicine.drugFrontiers in Immunology
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Memory CD8+ T Cell Protection From Viral Reinfection Depends on Interleukin-33 Alarmin Signals

2019

Memory CD8+ cytotoxic T lymphocytes (CTLs) can protect against viral reinfection. However, the signals driving rapid memory CTL reactivation have remained ill-defined. Viral infections can trigger the release of the alarmin interleukin-33 (IL-33) from non-hematopoietic cells. IL-33 signals through its unique receptor ST2 to promote primary effector expansion and activation of CTLs. Here, we show that the transcription factor STAT4 regulated the expression of ST2 on CTLs in vitro and in vivo in primary infections with lymphocytic choriomeningitis virus (LCMV). In the primary antiviral response, IL-33 enhanced effector differentiation and antiviral cytokine production in a CTL-intrinsic manne…

lcsh:Immunologic diseases. Allergyadaptive memoryalarminsIL-33virus infectionhemic and immune systemschemical and pharmacologic phenomenaST2CD8+ T cellslcsh:RC581-607Frontiers in Immunology
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Chlamydia trachomatis Infection and Anti-Hsp60 Immunity: The Two Sides of the Coin

2009

Chlamydia trachomatis (CT) infection is one of the most common causes of reproductive tract diseases and infertility. CT-Hsp60 is synthesized during infection and is released in the bloodstream. As a consequence, immune cells will produce anti-CT-Hsp60 antibodies. Hsp60, a ubiquitous and evolutionarily conserved chaperonin, is normally sequestered inside the cell, particularly into mitochondria. However, upon cell stress, as well as during carcinogenesis, the chaperonin becomes exposed on the cell surface (sf-Hsp60) and/or is secreted from cells into the extracellular space and circulation. Reports in the literature on circulating Hsp and anti-Hsp antibodies are in many cases short on detai…

lcsh:Immunologic diseases. Allergyanimal structuresImmunologyCardiovascular Disorders/Heart FailurePublic Health and Epidemiology/Infectious DiseasesChlamydia trachomatisPathology/Immunologychemical and pharmacologic phenomenaReviewmedicine.disease_causecomplex mixturesMicrobiologyAutoimmune DiseasesInfectious Diseases/Bacterial InfectionsPathogenesisImmune systemImmunityVirologyGeneticsmedicineAnimalsHumansImmunology/Cellular Microbiology and Pathogenesislcsh:QH301-705.5Molecular BiologyRheumatology/Autoimmunity Autoimmune and Inflammatory DiseasesAntigens BacterialbiologySettore BIO/16 - Anatomia UmanaMultiple sclerosisfungiAutoantibodyChaperonin 60Chlamydia Infectionsmedicine.diseaseHSP60 ChlamydiaMicrobiology/Immunity to Infectionslcsh:Biology (General)Immunologybiology.proteinParasitologyHSP60AntibodyDiabetes and Endocrinology/Type 1 Diabeteslcsh:RC581-607Chlamydia trachomatisPLoS Pathogens
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Corrigendum: Cost-Effective, Safe, and Personalized Cell Therapy for Critical Limb Ischemia in Type 2 Diabetes Mellitus.

2020

[This corrects the article DOI: 10.3389/fimmu.2019.01151.].

lcsh:Immunologic diseases. Allergycritical limb ischemiamedicine.medical_specialtyclinical trialsdiabetesbusiness.industrycost-effectivecell-based therapyImmunologyType 2 Diabetes MellitusCorrectionCritical limb ischemiamedicine.diseasecellular medicamentsCell therapyClinical trialDiabetes mellitusInternal medicinemedicineImmunology and Allergymedicine.symptombusinesslcsh:RC581-607
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Molecular epidemiology of HIV-1 subtype B in the Basque Country (Spain)

2012

The goal of this work was to study the HIV-1 subtype B epidemic in the Basque Country (Spain). For this, we used HIV samples submitted for genotypic testing of anti-retroviral resistance mutations from 2005 until 2008. Consequently, 2115 HIV-1 sequences comprising protease and retrotranscriptase (PR/RT) coding regions were analyzed. HIV transmission groups were identified by phylogenetic analysis. The 10 largest such groups were subsequently subjected to Bayesian phylogenetic and coalescent reconstructions, using a relaxed molecular clock model. The results obtained show that these groups have been long-standing: most of them were originated in the late 70s or early 80s, and none after the …

lcsh:Immunologic diseases. Allergyeducation.field_of_studyMolecular epidemiologyPhylogenetic treeTransmission (medicine)PopulationBiologyResistance mutationVirologyCoalescent theoryInfectious DiseasesVirologyPoster Presentationlcsh:RC581-607Molecular clockeducationCladeDemography
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