Search results for "Amides"

showing 10 items of 552 documents

Amide-to-triazole switch vs. in vivo NEP-inhibition approaches to promote radiopeptide targeting of GRPR-positive tumors

2017

Abstract Introduction Radiolabeled bombesin (BBN)-analogs have been proposed for diagnosis and therapy of gastrin-releasing peptide receptor (GRPR)-expressing tumors, such as prostate, breast and lung cancer. Metabolic stability represents a crucial factor for the success of this approach by ensuring sufficient delivery of circulating radioligand to tumor sites. The amide-to-triazole switch on the backbone of DOTA-PEG 4 -[Nle 14 ]BBN(7–14) ( 1 ) was reported to improve the in vitro stability of resulting 177 Lu-radioligands. On the other hand, in-situ inhibition of neutral endopeptidase (NEP) by coinjection of phosphoramidon (PA) was shown to significantly improve the in vivo stability and …

MaleCancer ResearchBiodistributionStereochemistryPharmacology[ CHIM ] Chemical Sciences030218 nuclear medicine & medical imagingPolyethylene Glycols03 medical and health scienceschemistry.chemical_compoundHeterocyclic Compounds 1-RingMice0302 clinical medicineIn vivoCell Line TumorRadioligandAnimalsHumans[CHIM]Chemical SciencesRadiology Nuclear Medicine and imagingTissue DistributionNeprilysinTumor targeting GRPR-radioligand 177Lu-bombesin Triazolyl-bombesin NEP-inhibitionPhosphoramidonGlycopeptidesBombesinTriazolesAmidesIn vitro3. Good healthBioavailabilityReceptors Bombesinchemistry030220 oncology & carcinogenesisMolecular MedicineBombesinNeprilysin
researchProduct

Maternal medication use and the risk of brain tumors in the offspring: The SEARCH international case-control study

2006

International audience; N-nitroso compounds (NOC) have been associated with carcinogenesis in a wide range of species, including humans. There is strong experimental data showing that nitrosamides (R(1)NNO.COR(2)), a type of NOC, are potent neuro-carcinogens when administered transplacentally. Some medications are a concentrated source of amides or amines, which in the presence of nitrites under normal acidic conditions of the stomach can form NOC. Therefore, these compounds, when ingested by women during pregnancy, may be important risk factors for tumors of the central nervous system in the offspring. The aim of the present study was to test the association between maternal use of medicat…

MaleCancer ResearchMESH: Maternal-Fetal ExchangeMESH: Pregnancy0302 clinical medicinePregnancyRisk FactorsMESH: Risk FactorsMESH: ChildRecall biasEpidemiologyMedicine030212 general & internal medicineAminesChildMaternal-Fetal Exchangeeducation.field_of_studyBrain NeoplasmsN-nitroso compoundsMESH: AminesMESH: InfantMESH: AmidesMESH: Case-Control StudiesMESH: Mothers3. Good healthOncologyChild Preschool030220 oncology & carcinogenesisMESH: Brain NeoplasmsFemaleDisease SusceptibilityAdultmedicine.medical_specialtyAdolescentOffspringcase-control studyPopulationMESH: Disease SusceptibilityMothers[SDV.CAN]Life Sciences [q-bio]/Cancerchildhood brain tumors03 medical and health sciencesInternal medicineGliomamaternal medicationHumansRisk factoreducationMESH: AdolescentPregnancyMESH: Humansbusiness.industryMESH: Child PreschoolCase-control studyInfantMESH: Adultmedicine.diseaseAmidesMESH: MaleCase-Control StudiesbusinessMESH: FemaleInternational Journal of Cancer
researchProduct

Development of a [177Lu]BPAMD Labeling Kit and an Automated Synthesis Module for Routine Bone Targeted Endoradiotherapy

2015

Painful bone lesions, both benign and metastatic, are often managed using conventional analgesics. However, the treatment response is not immediate and is often associated with side-effects. Radionuclide therapy is used for pain palliation in bone metastases as well as some benign neoplasms. Endoradiotherapy has direct impact on the pain-producing bone elements, and hence, response is significant, with minimal or no side-effects. A new potential compound for endoradiotherapy is [(177)Lu]BPAMD. It combines a highly affine bisphosphonate, covalently bridged with DOTA through an amide bond, with the low-energy β(-) emitting therapeutic radiolanthanide (177)Lu. For routine chemical application,…

MaleCancer ResearchTreatment responsePathologymedicine.medical_specialtymedicine.medical_treatmentPainBone NeoplasmsLutetiumBone and BonesPain palliationchemistry.chemical_compoundmedicineHumansDOTARadiology Nuclear Medicine and imagingBenign neoplasmsAgedRadioisotopesPharmacologyDiphosphonatesStaining and Labelingbusiness.industryGeneral MedicineBisphosphonateAmidesOncologychemistryBone lesionRadionuclide therapyReagent Kits DiagnosticRadiopharmaceuticalsNuclear medicinebusinessCancer Biotherapy and Radiopharmaceuticals
researchProduct

Reduced anxiety-like behaviour induced by genetic and pharmacological inhibition of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (…

2007

Anandamide and 2-arachidonoyl glycerol, referred to as endocannabinoids (eCBs), are the endogenous agonists for the cannabinoid receptor type 1 (CB1). Several pieces of evidence support a role for eCBs in the attenuation of anxiety-related behaviours, although the precise mechanism has remained uncertain. The fatty acid amid hydrolase (FAAH), an enzyme responsible for the degradation of eCBs, has emerged as a promising target for anxiety-related disorders, since FAAH inhibitors are able to increase the levels of anandamide and thereby induce anxiolytic-like effects in rodents. The present study adopted both genetic and pharmacological approaches and tested the hypothesis that FAAH-deficient…

MaleCannabinoid receptorPolyunsaturated Alkamidesmedicine.medical_treatmentArachidonic AcidsAnxietyPharmacologyAmidohydrolasesGlyceridesMiceCellular and Molecular Neurosciencechemistry.chemical_compoundPiperidinesReceptor Cannabinoid CB1RimonabantFatty acid amide hydrolaseCannabinoid receptor type 1medicineAnimalsMaze LearningMice KnockoutPharmacologyAnalysis of VarianceBehavior AnimalAnandamideURB597Endocannabinoid systemMice Inbred C57BLDisease Models Animalnervous systemchemistryBenzamidesPyrazoleslipids (amino acids peptides and proteins)CarbamatesCannabinoidRimonabantpsychological phenomena and processesEndocannabinoidsmedicine.drugNeuropharmacology
researchProduct

Genetic dissection of the role of cannabinoid type-1 receptors in the emotional consequences of repeated social stress in mice.

2012

International audience; The endocannabinoid system (ECS) tightly controls emotional responses to acute aversive stimuli. Repeated stress alters ECS activity but the role played by the ECS in the emotional consequences of repeated stress has not been investigated in detail. This study used social defeat stress, together with pharmacology and genetics to examine the role of cannabinoid type-1 (CB(1)) receptors on repeated stress-induced emotional alterations. Seven daily social defeat sessions increased water (but not food) intake, sucrose preference, anxiety, cued fear expression, and adrenal weight in C57BL/6N mice. The first and the last social stress sessions triggered immediate brain reg…

MaleCannabinoid receptorPolyunsaturated Alkamidesmedicine.medical_treatmentPopulationEmotionsDrinkingArachidonic AcidsMotor ActivitySerotonergicGlyceridesSocial defeat03 medical and health sciencesEatingFood PreferencesMice0302 clinical medicinePiperidinesReceptor Cannabinoid CB1Adrenal GlandsmedicineAnimals[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]education030304 developmental biologyPharmacologySocial stressMice KnockoutNeurons0303 health scienceseducation.field_of_studyBrainImmobility Response TonicExtinction (psychology)Endocannabinoid systemMice Inbred C57BLPsychiatry and Mental healthnervous systemPyrazoles[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]lipids (amino acids peptides and proteins)Original ArticleCannabinoidRimonabantPsychologyNeuroscience030217 neurology & neurosurgeryStress PsychologicalEndocannabinoidsNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
researchProduct

CB1 Cannabinoid Receptors and On-Demand Defense Against Excitotoxicity

2003

Abnormally high spiking activity can damage neurons. Signaling systems to protect neurons from the consequences of abnormal discharge activity have been postulated. We generated conditional mutant mice that lack expression of the cannabinoid receptor type 1 in principal forebrain neurons but not in adjacent inhibitory interneurons. In mutant mice,the excitotoxin kainic acid (KA) induced excessive seizures in vivo. The threshold to KA-induced neuronal excitation in vitro was severely reduced in hippocampal pyramidal neurons of mutants. KA administration rapidly raised hippocampal levels of anandamide and induced protective mechanisms in wild-type principal hippocampal neurons. These protecti…

MaleCannabinoid receptorReceptors Drugmedicine.medical_treatment2-ArachidonoylglycerolExcitotoxicityHippocampal formationmedicine.disease_causeHippocampusMicechemistry.chemical_compoundPiperidinesCannabinoid receptor type 1Excitatory Amino Acid AgonistsReceptors Cannabinoidgamma-Aminobutyric AcidMice KnockoutNeuronsKainic AcidMultidisciplinaryBrainEndocannabinoid systemNeuroprotective AgentsMitogen-Activated Protein KinasesRimonabantSignal Transductionmedicine.medical_specialtyKainic acidPolyunsaturated AlkamidesGlutamic AcidMice TransgenicArachidonic AcidsIn Vitro TechniquesBiologyGlyceridesProsencephalonInternal medicinemedicineAnimalsFuransGenes Immediate-EarlyEpilepsyCannabinoidsBrain-Derived Neurotrophic FactorExcitatory Postsynaptic PotentialsMice Inbred C57BLEndocrinologyGene Expression Regulationnervous systemchemistryMutationPyrazolesCannabinoidNeuroscienceEndocannabinoidsScience
researchProduct

Inhibition by Anandamide and Synthetic Cannabimimetics of the Release of [3H]d-Aspartate and [3H]GABA from Synaptosomes Isolated from the Rat Hippoca…

2004

Cannabinoids (CB) can act as retrograde synaptic mediators of depolarization-induced suppression of inhibition or excitation in hippocampus. This mechanism may underlie the impairment of some cognitive processes produced by these compounds, including short-term memory formation in the hippocampus. In this study, we investigated several compounds known to interact with CB receptors, evaluating their effects on K +-evoked release of [ 3H]d-aspartate ([ 3H]d-ASP) and [ 3H]GABA from superfused synaptosomes isolated from the rat hippocampus. [ 3H]d-ASP and [ 3H]GABA release were inhibited to different degrees by the synthetic cannabinoids WIN 55,212-2; CP 55,940, and arachidonyl-2′- chloroethyla…

MaleCannabinoid receptorSettore BIO/14 - FARMACOLOGIAPolyunsaturated Alkamidesmedicine.medical_treatmentHippocampusArachidonic AcidsPharmacologyHippocampal formationDepolarization-induced suppression of inhibitionHippocampusBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundglutamate releasemedicineAnimalsRats WistarCannabinoidgamma-Aminobutyric AcidCannabinoid Receptor AgonistsAspartic AcidCannabinoidsChemistryGeneral MedicineAnandamideCyclohexanolsgaba releaseEndocannabinoid systemRatsKineticsnervous systemBiochemistryAnimals Arachidonic Acids Aspartic Acid Calcium Cannabinoids Capsaicin Cyclohexanols gamma-Aminobutyric Acid Hippocampus Kinetics Polyunsaturated Alkamides Potassium Rats Receptors Cannabinoid SynaptosomesPotassiumCalciumlipids (amino acids peptides and proteins)CannabinoidCapsaicinCapsazepineEndocannabinoidsSynaptosomesNeurochemical Research
researchProduct

Determination of globotriaosylceramide in plasma and urine by mass spectrometry

2009

Abstract Background: Fabry disease is an X-chromosomally inherited lysosomal storage disorder leading to accumulation of glycosphingolipids, mainly globotriaosylceramide (ceramide-trihexoside, Gb3). Concentrations of Gb3 in plasma and urine have been used to diagnose Fabry disease and to monitor enzyme replacement therapy with recombinant α-galactosidase. Methods: Gb3 was purified from plasma or urine by combined liquid extraction/protein precipitation and solid-phase extraction, and was detected by flow-injection analysis electrospray mass spectrometry (MS) using multi-reaction-monitoring. Calibration was performed via standard addition using C17-Gb3 as internal standard. The most abundant…

MaleCoefficient of variationClinical BiochemistryGlobotriaosylceramideUrinechemistry.chemical_compoundTandem Mass SpectrometryLiquid chromatography–mass spectrometryBlood plasmamedicineHumansProtein precipitationEnzyme Replacement TherapyChromatographyTrihexosylceramidesSolid Phase ExtractionBiochemistry (medical)General MedicineReference Standardsmedicine.diseaseFabry diseaseLysosomal Storage Diseaseschemistryalpha-GalactosidaseStandard additionCalibrationFabry DiseaseFemaleChromatography Liquidcclm
researchProduct

Effects of K(ATP) channel modulators on acetylcholine release from guinea-pig isolated atria and small intestine.

2002

The effects of K(ATP) channel blockers (glibenclamide, HMR 1883, HMR 1372) and openers (cromakalim, pinacidil, diazoxide) on the electrically-evoked (5 Hz) release of [(3)H]acetylcholine were studied in isolated guinea-pig atria and myenteric plexus-longitudinal muscle preparations which had been preincubated with [(3)H]choline. Atria: Cromakalim (0.3 microM and 1 microM), pinacidil (10 microM) and diazoxide (30 microM) significantly reduced the stimulation-evoked release of [(3)H]acetylcholine. The inhibition produced by cromakalim and pinacidil was prevented by 1 microM of either HMR 1883, HMR 1372 or glibenclamide. The blockers alone significantly increased the release at concentrations …

MaleCromakalimPotassium ChannelsGuinea PigsNeuromuscular JunctionMyenteric PlexusPharmacologyIn Vitro Techniqueschemistry.chemical_compoundGlyburideIntestine SmallmedicineDiazoxidePotassium Channel BlockersAnimalsChannel blockerHeart AtriaPharmacologySulfonamidesPinacidilDiazoxideThioureaPotassium channel blockerMuscle SmoothGeneral Medicinemusculoskeletal systemAtrial FunctionMyocardial ContractionHMR 1883Potassium channelAcetylcholinechemistryAnesthesiaPinacidilcardiovascular systemFemaleCromakalimAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Oxidative burst inhibitory and cytotoxic amides and lignans from the stem bark of Fagara heitzii (Rutaceae)

2009

Two amides, heitziamide A and heitziamide B and two phenylethanoids, heitziethanoid A and heitziethanoid B together with thirteen known compounds were isolated from F. heitzii (Letouzey). The structures of all compounds were established by spectroscopic analysis. Nine compounds were evaluated for oxidative burst inhibitory activity in a chemoluminescence assay and for cytotoxicity against PC-3 prostate cancer cells. All compounds exhibited a clear suppressive effect on phagocytosis response upon activation with serum opsonized zymosan at the range of IC50 = 2.0-6.5 mu M, but no cytotoxic effect was observed (IC50 > 100 mu M). (C) 2009 Elsevier Ltd. All rights reserved.

MaleCytotoxicityChemical structurePhagocytosisPlant ScienceHorticultureHeterocyclic Compounds 2-RingBiochemistryAntioxidantsLignansInhibitory Concentration 50chemistry.chemical_compoundPhagocytosisCell Line TumorHumansImmunologic FactorsCytotoxic T cellFagara heitziiCytotoxicityRutaceaeMolecular BiologyRespiratory BurstLignanPlant StemsPlant ExtractsZymosanZymosanProstatic NeoplasmsBiological activityGeneral MedicineAntineoplastic Agents PhytogenicAmidesOxidative burstinhibitionRespiratory burstchemistryBiochemistryPlant BarkPhenylethanoidsPhytochemistry
researchProduct