Search results for "Amplification"

showing 10 items of 258 documents

Phenotype Correlation and Intergenerational Dynamics of the Friedreich Ataxia GAA Trinucleotide Repeat

1997

Summary The Friedreich ataxia (FA) mutation has recently been identified as an unstable trinucleotide GAA repeat present 7–22 times in the normal population but amplified as many as > 1, 000 times in FA. Since it is an autosomal recessive disease, FA does not show typical features observed in other dynamic mutation disorders, such as genetic anticipation. We have analyzed the GAA repeat in 104 FA patients and 163 carrier relatives previously defined by linkage analysis. The GAA expansion was detected in all patients, most (94%) of them being ho-mozygous for the mutation. We have demonstrated that clinical variability in FA is related to the size of the expanded alleles: milder forms of the …

AtaxiaAdolescentGenetic LinkagePopulationBiologyTrinucleotide RepeatsMeiosisGenetic linkageGene duplicationGeneticsmedicineHumansGenetics(clinical)AlleleChildeducationGenetics (clinical)Geneticseducation.field_of_studyGene AmplificationPhenotypeFriedreich AtaxiaMutationMutation (genetic algorithm)Dynamic mutationmedicine.symptomResearch ArticleThe American Journal of Human Genetics
researchProduct

The Arctic Cloud Puzzle: Using ACLOUD/PASCAL Multiplatform Observations to Unravel the Role of Clouds and Aerosol Particles in Arctic Amplification

2019

A consortium of polar scientists combined observational forces in a field campaign of unprecedented complexity to uncover the secrets of clouds and their role in Arctic amplification. Two research aircraft, an icebreaker research vessel, an ice-floe camp including an instrumented tethered balloon, and a permanent ground-based measurement station were employed in this endeavour. Clouds play an important role in Arctic amplification. This term represents the recently observed enhanced warming of the Arctic relative to the global increase of near-surface air temperature. However, there are still important knowledge gaps regarding the interplay between Arctic clouds and aerosol particles, surfa…

Atmospheric Science010504 meteorology & atmospheric sciencesbusiness.industryCloud computingPascal (programming language)010502 geochemistry & geophysics01 natural sciencesAerosolThe arcticEarth sciencesClimatologyddc:550Polar amplificationEnvironmental sciencebusinesscomputer0105 earth and related environmental sciencescomputer.programming_languageBulletin of the American Meteorological Society
researchProduct

Usefulness of omp1 restriction mapping for avian Chlamydia psittaci isolate differentiation

1995

Abstract Sixty-five avian Chlamydia psittaci isolates collected worldwide, including 27 previously characterized reference strains, were analysed by restriction mapping of the major outer membrane protein gene ( omp1 ) obtained after DMA amplification by PCR. They were compared to 2 ruminant isolates, a feline pneumonitis and a guinea pig inclusion conjunctivitis (GPIC) isolate. According to their omp1 restriction patterns, avian strains were heterogeneous in that they exhibited 6 and 4 distinct patterns using Alu I and Mbo II restriction enzymes, respectively, thus defining 7 groups. However, 84% of the studied strains belonged to groups 1 to 4, which share a specific fragment triplet of 4…

Base pairGuinea PigsRestriction MappingAMPLIFICATION CHAINE POLYMERASEIn Vitro TechniquesMicrobiologyBirdsRestriction mapAnimalsMolecular BiologyGene[SDV.MP] Life Sciences [q-bio]/Microbiology and ParasitologyElectrophoresis Agar GelChlamydia psittaciGeneticsbiologyDNA–DNA hybridizationGene AmplificationGeneral Medicinebiology.organism_classificationRestriction enzyme[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyChlamydophila psittaciGenes BacterialGenetic markerCatsCattleRestriction fragment length polymorphismBacterial Outer Membrane Proteins
researchProduct

Growth of immobilized DNA by polymerase: bridging nanoelectrodes with individual dsDNA molecules.

2011

We present a method for controlled connection of gold electrodes with dsDNA molecules (locally on a chip) by utilizing polymerase to elongate single-stranded DNA primers attached to the electrodes. Thiol-modified oligonucleotides are directed and immobilized to nanoscale electrodes by means of dielectrophoretic trapping, and extended in a procedure mimicking PCR, finally forming a complete dsDNA molecule bridging the gap between the electrodes. The technique opens up opportunities for building from the bottom-up, for detection and sensing applications, and also for molecular electronics.

Bridging (networking)Sensing applicationsFOS: Physical sciencesNanotechnology02 engineering and technologyDNA-Directed DNA PolymeraseCondensed Matter - Soft Condensed Matter03 medical and health sciencesMoleculeNanotechnologyGeneral Materials SciencePhysics - Biological PhysicsElectrodesPolymerase030304 developmental biologyDNA PrimersFluorescent Dyes0303 health sciencesbiologyImmobilized DNAta114OligonucleotideChemistryta1182Molecular electronicsDNA021001 nanoscience & nanotechnologyCondensed Matter - Other Condensed MatterBiological Physics (physics.bio-ph)Electrodebiology.proteinSoft Condensed Matter (cond-mat.soft)Gold0210 nano-technologyNucleic Acid Amplification TechniquesOther Condensed Matter (cond-mat.other)Nanoscale
researchProduct

MYCN gain and MYCN amplification in a stage 4S neuroblastoma.

2003

Abstract Stage 4S neuroblastoma is a disease associated with spontaneous regression and good survival. We present a patient whose evolution has shown the variety and complexity of this disease in infants. Biologic factors, such as ploidy, MYCN copy number, loss of 1p36, and other chromosomal gains and losses were determined. A complex pattern of genetic abnormalities, such as near-diploidy, MYCN gain (2–4 copies per haploid genome) and imbalance/deletion of 1p36 was seen in the diagnostic sample. An extensive disseminated disease after a latent period of 26 months was associated with a special genetic evolution, such as tetraploidy, MYCN amplification (2:100–500 copies), 1p36 deletion, and …

Cancer ResearchAdrenal Gland NeoplasmsGenes mycDiseaseBiologymedicine.disease_causeNeuroblastomaFatal OutcomeNeuroblastomaGene duplicationGeneticsmedicineHumansneoplasmsMolecular BiologyNeoplasm StagingGeneticsMutationTransition (genetics)Gene AmplificationInfantmedicine.diseaseAneuploidyPrimary tumorChromosomes Human Pair 1Stage 4S NeuroblastomaCancer researchDisease ProgressionFemalePloidyChromosome DeletionChromosomes Human Pair 17Cancer genetics and cytogenetics
researchProduct

Centrosome amplification induced by hydroxyurea leads to aneuploidy in pRB deficient human and mouse fibroblasts.

2006

Alterations in the number and/or morphology of centrosomes are frequently observed in human tumours. However, it is still debated if a direct link between supernumerary centrosomes and tumorigenesis exists and if centrosome amplification could directly cause aneuploidy. Here, we report that hydroxyurea treatment induced centrosome amplification in both human fibroblasts expressing the HPV16 -E6-E7 oncoproteins, which act principally by targeting p53 and pRB, respectively, and in conditional pRB deficient mouse fibroblasts. Following hydroxyurea removal both normal and p53 deficient human fibroblasts arrested. On the contrary pRB deficient fibroblasts entered the cell cycle generating aneupl…

Cancer ResearchAneuploidyCentrosome amplificationBiologymedicine.disease_causeRetinoblastoma ProteinCell LineMicepRBChromosomal InstabilitymedicineDeficient mouseAnimalsHumansHydroxyureaCINCells CulturedCentrosomeDNA synthesisCell cycleFibroblastsmedicine.diseaseAneuploidyCell biologySettore BIO/18 - GeneticaOncologyCentrosomeAneuploid CellsCarcinogenesisCancer letters
researchProduct

Expression of type I interferon receptor and its relation with other prognostic factors in human neuroblastoma.

1998

Expression of type I interferon receptor (IFN-R) has been found in several normal tissues and in malignant neoplasms, mainly those with epithelial differentiation. In order to analyze the immunohistochemical expression of type I IFN-R we studied 79 cases of neuroblastoma. Results of expression of type I IFN-R were statistically correlated with histopathology, stage, bcl-2 and PCNA expression, N-myc amplification and apoptosis. We found expression of type I IFN-R in 54/79 cases showing statistical correlation with bcl-2 expression (P=0.017) and favourable histopathology (P=0.015). The overexpression found in ganglion cells suggests that IFN-R could be involved in the pathway of neuroblastoma…

Cancer ResearchCellular differentiationmedicine.medical_treatmentGenes mycAlpha interferonApoptosisReceptor Interferon alpha-betaBiologyImmunoenzyme TechniquesNeuroblastomaProliferating Cell Nuclear AntigenNeuroblastomaGene expressionBiomarkers TumormedicineHumansChildInterferon alfaNeoplasm StagingReceptors InterferonOncogeneGene AmplificationInfantMembrane ProteinsCell DifferentiationGeneral MedicinePrognosismedicine.diseaseNeoplasm ProteinsCytokineProto-Oncogene Proteins c-bcl-2OncologySpainChild PreschoolCancer researchImmunohistochemistrymedicine.drugOncology Reports
researchProduct

Acquired resistance of melanoma cells to the antineoplastic agent fotemustine is caused by reactivation of the DNA repair gene mgmt

2001

Acquired resistance to antineoplastic agents is a frequent obstacle in tumor therapy. Malignant melanoma cells are particularly well known for their unresponsiveness to chemotherapy; only about 30% of tumors exhibit a transient clinical response to treatment. In our study, we investigated the molecular mechanism of acquired resistance of melanoma cells (MeWo) to the chloroethylating drug fotemustine. Determination of O6-methylguanine-DNA methyltransferase (MGMT) activity showed that MeWo cells that acquired resistance to fotemustine upon repeated treatment with the drug display high MGMT activity, whereas the parental cell line had no detectable MGMT. The resistant cell lines exhibit cross-…

Cancer ResearchGuanineMethyltransferaseDNA RepairDNA repairmedicine.medical_treatmentGene ExpressionAntineoplastic AgentsDrug resistanceBiologyNitrosourea CompoundsO(6)-Methylguanine-DNA MethyltransferaseEnzyme ReactivatorsOrganophosphorus CompoundsTumor Cells CulturedmedicineHumansEnzyme InhibitorsPromoter Regions GeneticMelanomaneoplasmsChemotherapyMelanomaGene AmplificationDNA Methylationmedicine.diseaseVirologydigestive system diseasesEnzyme ActivationBlotting SouthernOncologyDrug Resistance NeoplasmDNA methylationAzacitidineCancer researchFotemustinemedicine.drugAlkyltransferaseInternational Journal of Cancer
researchProduct

Chimeric amplicons containing the c-myc gene in HL60 cells

1998

The major amplicon present in HL60 cells is chimeric in nature being composed of 70 kb of DNA sequence derived from the MYC locus linked to 80 kb of novel DNA sequence derived from a non contiguous region located telomeric to the c-myc gene at 8q24 (Feo et al., 1996). Here we show by fluorescence in situ hybridization (FISH) that these coamplified sequences, MCR (Myc Coamplified Region), are derived from a locus located 3-4 Mb telomeric to the c-myc gene in the q24.2-24.3 region of chromosome 8. Genomic cloning and Southern blot analysis indicate the arrangement of chimeric amplicons are in tandem arrays. Analysis of the DNA sequences at the juncture of the MYC locus and the MCR suggest tha…

Cancer ResearchOncogene Proteins FusionInverted repeatMolecular Sequence DataGenes mycHL-60 CellsLocus (genetics)BiologyMolecular cloningDNA sequencingLeukemia Promyelocytic AcuteGene mappingGeneticsHumansCloning MolecularMolecular BiologyGeneIn Situ Hybridization FluorescenceSouthern blotChromosome AberrationsRecombination GeneticGeneticsBase SequenceChromosome FragilityGene AmplificationSequence Analysis DNAAmpliconMolecular biologyBlotting SouthernChromosomes Human Pair 8Oncogene
researchProduct

Abstract 5379: Recurrent inactivation of PARD3, a polarity-related gene, in squamous cell carcinomas of the lung.

2013

Abstract In spite of the recent advances in cancer genomics, the genetics underlying the development of lung squamous cell carcinomas (SCC) is still poorly understood. Here, we investigated the contribution of the cell polarity-related gene, PARD3, to lung SCC carcinogenesis. First, we tested for PARD3 alterations in lung cancer cell lines from various histopathological types. The analysis confirmed an intragenic deletion at the H157 cells and unveiled biallelic mutations in another cell line. Both cell lines are SCCs, which circumscribed PARD3 alterations to this lung cancer type. Next, we extended the genetic screening, which included the determination of mutations and of intragenic delet…

Cancer ResearchPathologymedicine.medical_specialtyCell growthCellWild typeCancerBiologymedicine.diseasemedicine.disease_causemedicine.anatomical_structureOncologymedicineCancer researchMultiplex ligation-dependent probe amplificationLung cancerCarcinogenesisGeneCancer Research
researchProduct