Search results for "Amy"

showing 10 items of 1486 documents

Increased blood mercury levels in patients with Alzheimer's disease.

1998

Alzheimer's disease (AD) is a common neurodegenerative disorder that leads to dementia and death. In addition to several genetic parameters, various environmental factors may influence the risk of getting AD. In order to test whether blood levels of the heavy metal mercury are increased in AD, we measured blood mercury concentrations in AD patients (n = 33), and compared them to age-matched control patients with major depression (MD) (n = 45), as well as to an additional control group of patients with various non-psychiatric disorders (n = 65). Blood mercury levels were more than two-fold higher in AD patients as compared to both control groups (p = 0.0005, and p = 0.0000, respectively). In…

Malemedicine.medical_specialtyPathologyNeurologychemistry.chemical_elementtau ProteinsCentral nervous system diseaseDegenerative diseaseAlzheimer DiseaseInternal medicineBlood plasmamedicineDementiaHumansBiological PsychiatryAgedAged 80 and overDepressive DisorderAmyloid beta-PeptidesNeurodegenerationMercuryMiddle Agedmedicine.diseaseMercury (element)Psychiatry and Mental healthEndocrinologyNeurologychemistryLinear ModelsFemaleNeurology (clinical)Alzheimer's diseasePsychologyBiomarkersJournal of neural transmission (Vienna, Austria : 1996)
researchProduct

Prevalence and Cognitive Impact of Medial Temporal Atrophy in a Hospital Stroke Service: Retrospective Cohort Study

2015

Background Cerebrovascular disease and neurodegeneration cause cognitive impairment and frequently coexist. Aims Our objectives were to investigate the prevalence and cognitive impact of medial temporal lobe atrophy – a radiological marker often associated with Alzheimer's disease – in a hospital stroke service. Methods Retrospective cohort study of patients from a hospital stroke service. Patients assessed for suspected ischemic stroke or transient ischemic attack, irrespective of final diagnosis, underwent neuropsychological testing and magnetic resonance imaging. medial temporal lobe atrophy, white matter hyperintensities, lacunes, and cerebral microbleeds were rated using established cr…

Malemedicine.medical_specialtyPathologyPopulationNeuropsychological TestsSeverity of Illness IndexBrain IschemiaInternal medicinePrevalencemedicineHumansCerebral amyloid angiopathyeducationVascular dementiaStrokeRetrospective Studieseducation.field_of_studymedicine.diagnostic_testbusiness.industryMagnetic resonance imagingRetrospective cohort studyOdds ratioMiddle Agedmedicine.diseaseMagnetic Resonance ImagingTemporal LobeHyperintensityHospitalizationStrokeLogistic ModelsNeurologyMultivariate AnalysisCardiologyFemaleAtrophyVerbal memoryCognition DisordersbusinessMedial temporal lobe atrophyInternational Journal of Stroke
researchProduct

CSF APPsα and Phosphorylated Tau Protein Levels in Mild Cognitive Impairment and Dementia of Alzheimer's Type

2008

We exploratively measured APPs alpha, a secreted fragment of the non-amyloidogenic cleavage of amyloid precursor protein via a-secretase, and tau protein phosphorylated at threonine 181 (p tau) in the cerebrospinal fluid of 10 patients with mild cognitive impairment, 20 patients with dementia of Alzheimer's type, and 10 controls. Cerebrospinal fluid APPs alpha and p tau levels were correlated with cognitive performance. P tau levels were significantly elevated in mild cognitive impairment and in patients with dementia of Alzheimer's type, APPs alpha levels were significantly reduced in patients with dementia of Alzheimer's type compared to the controls. APPs alpha levels were associated wit…

Malemedicine.medical_specialtyPathologyTau proteintau ProteinsNeuropsychological TestsSeverity of Illness IndexCerebrospinal fluidDegenerative diseaseAlzheimer DiseaseInternal medicineTask Performance and AnalysismedicineAmyloid precursor proteinHumansDementiaSex DistributionThreonineAgedAnalysis of VariancebiologyChemistrymedicine.diseasePsychiatry and Mental healthMemory Short-TermEndocrinologyMental Recallbiology.proteinPhosphorylationFemaleNeurology (clinical)Amyloid Precursor Protein SecretasesGeriatrics and GerontologyAlzheimer's diseaseCognition DisordersBiomarkersJournal of Geriatric Psychiatry and Neurology
researchProduct

Comparison of enzyme phenotypes in human bladder tumours and experimentally induced hyperplastic and neoplastic lesions of the rat urinary bladder. A…

1988

The expression of a number of enzymes involved in drug metabolism, membrane function etc. was compared in hyperplastic and neoplastic lesions of the rat bladder and in human bladder tumours. Transitional cell carcinomas (TCC) in both rat and Man were characterized by decreased alkaline phosphatase (ALP) and increased gamma-glutamyl transpeptidase (GGT), beta-glucuronidase (beta-G1), succinate dehydrogenase (SD) and glucose-6-phosphate dehydrogenase (G6PD) activities. In addition, binding for antibodies specific for different cytochrome P-450 species (UT50, PB3a, MC1, MC2) and microsomal epoxide hydrolase (mEHb) was elevated in both murine and human tumours. Comparison of the enzyme phenotyp…

Malemedicine.medical_specialtyPathologyUrinary BladderGlucosephosphate DehydrogenaseInternal medicinemedicineAnimalsHumansNeoplastic transformationEnzyme inducerGlucuronidaseCarcinoma Transitional CellUrinary bladderHyperplasiabiologyHistocytochemistrySuccinate dehydrogenasegamma-GlutamyltransferaseHyperplasiamedicine.diseaseAlkaline PhosphataseImmunohistochemistryRats Inbred F344RatsSuccinate DehydrogenaseEndocrinologymedicine.anatomical_structurePhenotypeUrinary Bladder NeoplasmsMicrosomal epoxide hydrolasebiology.proteinImmunohistochemistryDrug metabolismVirchows Archiv. B, Cell pathology including molecular pathology
researchProduct

FACTORS AFFECTING THE DIAGNOSTIC DELAY IN AMYOTROPHIC LATERAL SCLEROSIS

2012

Abstract Background Although amyotrophic lateral sclerosis (ALS) is a relentlessly progressive disorder, early diagnosis allows a prompt start with the specific drug riluzole and an accurate palliative care planning. ALS at onset may however mimic several disorders, some of them treatable ( e.g. , multifocal motor neuropathy) or epidemiologically more frequent ( e.g. , cervical myelopathy). Objective To study the delay from onset to diagnosis in a cohort of ALS patients and to the variables that may affect it. Methods We performed a retrospective analysis of the diagnostic delays in a cohort of 260 patients affected by ALS (M/F = 1.32) followed at our tertiary referral ALS Center between 20…

Malemedicine.medical_specialtyPediatricsDelayed DiagnosisPalliative careALS diagnostic delay cognitive errorsCohort StudiesHumansMedicineAge of OnsetDiagnostic ErrorsAmyotrophic lateral sclerosisAgedRetrospective Studiesbusiness.industryAmyotrophic Lateral SclerosisRetrospective cohort studyGeneral MedicineMiddle Agedmedicine.diseaseMultivariate AnalysisCohortPhysical therapyFemaleSurgerySettore MED/26 - NeurologiaNeurology (clinical)Age of onsetDifferential diagnosisbusinessCohort studyMultifocal motor neuropathy
researchProduct

Parenteral nutrition improves nutritional status, autonomic symptoms and quality of life in transthyretin amyloid polyneuropathy

2016

Transthyretin familial amyloid polyneuropathy (TTR-FAP) is an inherited amyloidosis, leading to death in about ten years in most cases due to cardiac failure or wasting syndrome. Previous studies showed that modified body mass index was related to time before death, duration of gastrointestinal disturbances, malabsorption and functional capacity. We report two patients in whom nutritional status worsened despite diet modification, hypercaloric supplement and two relevant therapeutic approaches such as liver transplant and tafamidis meglumine, respectively. The first patient, a 52-year-old lady carrying Thr49Ala mutation, had a disease duration of twelve years and had lost weight up to 35 kg…

Malemedicine.medical_specialtyPediatricsMalabsorptionNausea030204 cardiovascular system & hematologyPediatrics03 medical and health sciences0302 clinical medicineQuality of lifeNutritional statusMedicineHumansWasting SyndromeTTR-FAPGenetics (clinical)Amyloid Neuropathies FamilialFamilial amyloid polyneuropathy; Modified body mass index (mBMI); Nutritional status; Parenteral nutrition; TTR-FAP; Neurology (clinical); Pediatrics Perinatology and Child Health; Genetics (clinical); Neurologybiologybusiness.industryAmyloidosisMiddle AgedPerinatology and Child Healthmedicine.diseaseParenteral nutritionSurgeryTransthyretinParenteral nutritionTreatment OutcomeAutonomic Nervous System DiseasesNeurologyFamilial amyloid polyneuropathyPediatrics Perinatology and Child Healthbiology.proteinQuality of LifeFemaleNeurology (clinical)medicine.symptombusinessBody mass index030217 neurology & neurosurgeryModified body mass index (mBMI)
researchProduct

Cognitive correlates in amyotrophic lateral sclerosis: a population-based study in Italy.

2014

Background There is less data available regarding the characteristics of cognitive impairment in patients with amyotrophic lateral sclerosis (ALS) in a population-based series. Methodology Patients with ALS incident in Piemonte, Italy, between 2009 and 2011 underwent an extensive neuropsychological battery. Cognitive status was classified as follows: normal cognition, frontotemporal dementia (ALS-FTD), executive cognitive impairment (ALS-ECI), non-executive cognitive impairment (ALS-NECI), behavioural impairment (ALS-Bi), non-classifiable cognitive impairment. We also assessed 127 age-matched and gender-matched controls identified through patients’ general practitioners. Results Out of the …

Malemedicine.medical_specialtyPediatricsNeuromuscular diseasePopulationNeuropsychological TestsSuperoxide Dismutase-1Risk FactorsmedicineDementiaHumansEPIDEMIOLOGYAmyotrophic lateral sclerosisPsychiatryeducationCognitive reserveAgededucation.field_of_studyC9orf72 ProteinSuperoxide DismutaseDEMENTIAAmyotrophic Lateral SclerosisProteinsCognitionmedicine.diseaseSurvival AnalysisALS DEMENTIA EPIDEMIOLOGYDNA-Binding ProteinsPsychiatry and Mental healthItalyCase-Control StudiesMutationSurgeryFemaleSettore MED/26 - NeurologiaNeurology (clinical)ALSPsychologyCognition DisordersMotor neurone diseaseFrontotemporal dementia
researchProduct

Circadian System Functionality, Hippocampal Oxidative Stress, and Spatial Memory in the APPswe/PS1dE9 Transgenic Model of Alzheimer Disease: Effects …

2012

Alzheimer disease (AD) is a neurodegenerative disorder that primarily causes β-amyloid accumulation in the brain, resulting in cognitive and behavioral deficits. AD patients, however, also suffer from severe circadian rhythm disruptions, and the underlying causes are still not fully known. Patients with AD show reduced systemic melatonin levels. This may contribute to their symptoms, since melatonin is an effective chronobiotic and antioxidant with neuroprotective properties. Here, the authors critically assessed the effects of long-term melatonin treatment on circadian system function, hippocampal oxidative stress, and spatial memory performance in the APPswe/PS1 double transgenic (Tg) mou…

Malemedicine.medical_specialtyPhysiologyChronobioticRamelteonReceptors MelatoninHippocampusMice TransgenicMotor Activitymedicine.disease_causeHippocampusNeuroprotectionBody TemperatureMelatoninAmyloid beta-Protein PrecursorMiceAlzheimer DiseaseMemoryPhysiology (medical)Internal medicinePresenilin-1medicineAnimalsCircadian rhythmMelatoninmedicine.diseaseCircadian RhythmDisease Models AnimalOxidative StressEndocrinologyIndenesMutant ProteinsAlzheimer's diseasePsychologyNeuroscienceOxidative stressmedicine.drugChronobiology International
researchProduct

Muscle protein synthesis, mTORC1/MAPK/Hippo signaling, and capillary density are altered by blocking of myostatin and activins

2012

Loss of muscle mass and function occurs in various diseases. Myostatin blocking can attenuate muscle loss, but downstream signaling is not well known. Therefore, to elucidate associated signaling pathways, we used the soluble activin receptor IIb (sActRIIB-Fc) to block myostatin and activins in mice. Within 2 wk, the treatment rapidly increased muscle size as expected but decreased capillary density per area. sActRIIB-Fc increased muscle protein synthesis 1–2 days after the treatment correlating with enhanced mTORC1 signaling (phosphorylated rpS6 and S6K1, r = 0.8). Concurrently, increased REDD1 and eIF2Bε protein contents and phosphorylation of 4E-BP1 and AMPK was observed. In contrast, pr…

Malemedicine.medical_specialtyPhysiologyEndocrinology Diabetes and MetabolismMuscle ProteinsCell CountP70-S6 Kinase 1MyostatinMechanistic Target of Rapamycin Complex 1Protein Serine-Threonine KinasesBiologyMice03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicinemedicineAnimalsHippo Signaling PathwayExtracellular Signal-Regulated MAP KinasesMuscle Skeletalta315030304 developmental biology0303 health sciencesHippo signaling pathwayMyogenesisTOR Serine-Threonine KinasesSkeletal muscleActivin receptorMyostatinActivinsCapillariesMice Inbred C57BLmedicine.anatomical_structureEndocrinologyHippo signalingMultiprotein ComplexesProtein Biosynthesisbiology.proteinIntercellular Signaling Peptides and ProteinsPhosphorylation030217 neurology & neurosurgerySignal TransductionAmerican Journal of Physiology-Endocrinology and Metabolism
researchProduct

Up‐regulation of the α‐secretase ADAM10 by retinoic acid receptors and acitretin

2009

Late-onset Alzheimer's disease is often connected with nutritional misbalance, such as enhanced cholesterol intake, deficiency in polyunsaturated fatty acids, or hypovitaminosis. The alpha-secretase ADAM10 has been found to be regulated by retinoic acid, the bioreactive metabolite of vitamin A. Here we show that retinoids induce gene expression of ADAM10 and alpha-secretase activity by nonpermissive retinoid acid receptor/retinoid X receptor (RAR/RXR) heterodimers, whereby alpha- and beta-isotypes of RAR play a major role. However, ligands of other RXR binding partners, such as the vitamin D receptor, do not stimulate alpha-secretase activity. On the basis of these findings, we examined the…

Malemedicine.medical_specialtyReceptors Retinoic AcidReceptors Cytoplasmic and NuclearMice TransgenicTretinoinRetinoic acid receptor betaRetinoid X receptorBiologyBiochemistryCell LineAcitretinADAM10 ProteinAmyloid beta-Protein PrecursorMiceKeratolytic AgentsAlzheimer DiseaseInternal medicineGeneticsmedicineAnimalsHumansPromoter Regions GeneticMolecular BiologyLiver X ReceptorsReceptors Thyroid HormoneMolecular StructureRetinoid X receptor alphaMembrane ProteinsOrphan Nuclear ReceptorsRetinoid X receptor gammaAcitretinUp-RegulationDNA-Binding ProteinsPPAR gammaADAM ProteinsRetinoic acid receptorRetinoid X ReceptorsEndocrinologyGene Expression RegulationRetinoic acid receptor alphaReceptors CalcitriolAmyloid Precursor Protein SecretasesRetinoid X receptor betaBiotechnologymedicine.drugThe FASEB Journal
researchProduct