Search results for "Angiogenesis"

showing 10 items of 552 documents

A Reassessment of the Therapeutic Potential of a Dopamine Receptor 2 Agonist (D2-AG) in Endometriosis by Comparison against a Standardized Antiangiog…

2021

Dopamine receptor 2 agonists (D2-ags) have been shown to reduce the size of tumors by targeting aberrant angiogenesis in pathological tissue. Because of this, the use of a D2-ag was inferred for endometriosis treatment. When assayed in mouse models however, D2-ags have been shown to cause a shift of the immature vessels towards a more mature phenotype but not a significant reduction in the amount of vascularization and size of lesions. These has raised concerns on whether the antiangiogenic effects of these compounds confer a therapeutic value for endometriosis. In the belief that antiangiogenic effects of D2-ags in endometriosis were masked due to non-optimal timing of pharmacological inte…

endometriosis0301 basic medicineAgonistAngiogenesismedicine.drug_classEndometriosisdopamine agonistMedicine (miscellaneous)Dopamine agonistArticleGeneral Biochemistry Genetics and Molecular BiologyLesion03 medical and health sciencesheterologous mouse model0302 clinical medicineIn vivoCabergolinemedicinelcsh:QH301-705.5030219 obstetrics & reproductive medicinebusiness.industryantiangiogenicmedicine.disease030104 developmental biologylcsh:Biology (General)anti-VEGFDopamine receptorCancer researchmedicine.symptombusinessmedicine.drugBiomedicines
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Interplay Between MicroRNAs and Oxidative Stress in Ovarian Conditions with a Focus on Ovarian Cancer and Endometriosis

2019

Ovarian cancer and endometriosis are two distinct gynaecological conditions that share many biological aspects incuding proliferation, invasion of surrounding tissue, inflammation, inhibition of apoptosis, deregulation of angiogenesis and the ability to spread at a distance. miRNAs are small non-coding RNAs (19–22 nt) that act as post-transcriptional modulators of gene expression and are involved in several of the aforementioned processes. In addition, a growing body of evidence supports the contribution of oxidative stress (OS) to these gynaecological diseases: increased peritoneal OS due to the decomposition of retrograde menstruation blood facilitates both endometriotic lesion developmen…

endometriosis0301 basic medicineAngiogenesisEndometriosisInflammationReviewCatalysisendometriosis-associated ovarian cancerMalignant transformationlcsh:ChemistryInorganic Chemistry03 medical and health sciences0302 clinical medicinehigh-grade serous ovarian cancermicroRNAmedicineAnimalsHumansRNA NeoplasmEpithelial–mesenchymal transitionPhysical and Theoretical ChemistryCàncerlcsh:QH301-705.5Molecular BiologySpectroscopyOvarian Neoplasmsbusiness.industryOrganic ChemistrychemoresistanceGeneral Medicinemedicine.diseaseComputer Science ApplicationsMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999Ginecologia030220 oncology & carcinogenesismiRNAsCancer researchFemaleepithelial-to-mesenchymal transitionmedicine.symptombusinessOvarian cancerFallopian tubeInternational Journal of Molecular Sciences
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Evaluation of PAI-1 in endometriosis using a homologous immunocompetent mouse model

2018

To analyze the role of PAI-1 (plasminogen activator inhibitor 1) in endometriotic lesion growth, we studied the effect of PAI-1 inhibition by PAI-039 using a homologous mouse model of endometriosis that allows noninvasive monitoring. Endometrial tissue from donor mice was collected, labeled with mCherry adenovirus, and implanted into a subcutaneous pocket on the ventral abdomen of recipient mice. Seven days after transplantation, mice were randomly allocated in two groups and treated once daily for 2 weeks with either vehicle (control group) or PAI-1 inhibitor (PAI-039 group). Endometriotic lesion size generated in recipient mice was monitored by mCherry signal. Animals were euthanized 21 d…

endometriosisPathologymedicine.medical_specialtyAngiogenesismouse modelnoninvasive monitoringEndometriosisEndometriosisPAI-1FibrinLesionNeovascularization03 medical and health scienceschemistry.chemical_compoundEndometriumMiceangiogenesis0302 clinical medicineIn vivoSerpin E2medicineAnimalsCell Proliferation030219 obstetrics & reproductive medicinebiologyIndoleacetic AcidsNeovascularization PathologicCell BiologyGeneral Medicinemedicine.diseaseTransplantationDisease Models AnimalReproductive Medicinechemistry030220 oncology & carcinogenesisPlasminogen activator inhibitor-1biology.proteinFemalefibrinolysismedicine.symptom
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Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio…

2018

La endometriosis es una enfermedad con elevada prevalencia en la población de mujeres en edad fértil (Goldstein y cols, 1980; Eskenazi y cols, 1997; Gazvani y cols, 2002), que cursa con dolor pélvico crónico, dismenorrea, sangrados irregulares e infertilidad, principalmente (Goldstein y cols, 1980; Eskenazi y cols, 1997). Esta sintomatología empeora la calidad de vida de la paciente (D’Hooghe T y cols, 2006) y genera un gran gasto en términos económicos y sociales (Simoens y cols, 2007), de lo que se deriva su gran importancia. Ésto, unido a las múltiples cuestiones que se desconocen o están en controversia en relación a la génesis de la enfermedad, patogeniam diagnóstico precoz y tratamien…

endometriosisangiogenesismouse modelUNESCO::CIENCIAS MÉDICASapoptosispICPEI:CIENCIAS MÉDICAS [UNESCO]
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Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for di…

2018

Introduction: Endometriosis is an oestrogen-dependent inflammatory disorder defined by the presence of endometrial-like tissue in ectopic locations, which limits the quality of life of affected women. This pathology affects 10% of reproductive-age women from all ethnic and social groups, although the prevalence in those patients experiencing pain, infertility or both is as high as 35%-50%, being the estimated prevalence of this condition around 176 million worldwide. Endometriosis is associated with an average diagnostic delay of 7 years, what could be partially explained by the lack of non-invasive biomarkers for diagnose, since the gold standard method is laparoscopy followed by histologi…

endometriosisangiogenesisproteolysisDNA methylationperitoneal fluidepigeneticsmiRNAsUNESCO::CIENCIAS DE LA VIDA:CIENCIAS DE LA VIDA [UNESCO]
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Sildenafil inhibits the proliferation of cultured human endothelial cells.

2013

The proliferation of endothelial cells plays a crucial role in the development of intraplaque angiogenesis (IPA). IPA is a major source of intraplaque hemorrhage and therefore contributes to the destabilization of atherosclerotic plaques. Therefore, the aim of the present study was to examine, whether sildenafil inhibits endothelial cell growth. The proliferation of human endothelial cells derived from umbilical cord veins (HUVEC) was examined on DNA level by measurements of (3H)-thymidine incorporation. Cell viability was analyzed using trypan blue staining. The proliferation of cultured human endothelial cells was significantly decreased by 1 μmol/l (-48.4%) and 10 μmol/l (-89.6%) sildena…

endothelial proliferationsildenafilcardiovascular systematherosclerosisbasic fibroblast growth factorArticleplaque angiogenesisrespiratory tract diseasesInternational journal of biomedical science : IJBS
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Magnetofection of acoustically active „magnetic microspheres“ (Magnetobubbles) — Induction of angiogenesis via non-viral transfection of VEGF

2009

Adenoviral transduction of the VEGF gene in an oversized skin flap increases flap survival and perfusion. In this study we investigated the potential of magnetofection of magnetic lipospheres containing VEGF165-cDNA on survival and perfusion of ischemic skin flaps and evaluated the method with respect to the significance of applied magnetic field and ultrasound.

integumentary systembiologybusiness.industryChemistryAngiogenesisVEGF receptorsUltrasoundSkin flapequipment and suppliesNon viral transfectioneye diseasesMicrospherebiology.proteinMagnetofectionCancer researchbusinesshuman activitiesPerfusion
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Osteoclast Immunosuppressive Effects in Multiple Myeloma: Role of Programmed Cell Death Ligand 1

2018

Immunomodulatory drugs and monoclonal antibody-based immunotherapies have significantly improved the prognosis of the patients with multiple myeloma (MM) in the recent years. These new classes of reagents target malignant plasma cells (PCs) and further modulate the immune microenvironment, which prolongs anti-MM responses and may prevent tumor occurrence. Since MM remains an incurable cancer for most patients, there continues to be a need to identify new tumor target molecules and investigate alternative cellular approaches using gene therapeutic strategies and novel treatment mechanisms. Osteoclasts (OCs), as critical multi-nucleated large cells responsible for bone destruction in >80% …

lcsh:Immunologic diseases. Allergy0301 basic medicineCarcinogenesisAngiogenesismedicine.medical_treatmentOsteoimmunologyT cellPlasma CellsProgrammed Cell Death 1 ReceptorImmunologyOsteoclastsCell CommunicationReviewB7-H1 AntigenImmune tolerance03 medical and health sciencesImmune systemAntigens NeoplasmImmune ToleranceTumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyBone ResorptionImmunologic Surveillancebone marrow microenvironmentTumor microenvironmentbusiness.industryprogrammed cell death ligand 1Immunotherapymultiple myeloma030104 developmental biologymedicine.anatomical_structureprogrammed cell death 1osteoclastosteoblastCancer researchimmunotherapylcsh:RC581-607businessB7-H1 AntigenSignal TransductionFrontiers in Immunology
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Complement Protein C1q Binds to Hyaluronic Acid in the Malignant Pleural Mesothelioma Microenvironment and Promotes Tumor Growth

2017

C1q is the first recognition subcomponent of the complement classical pathway, which acts towards the clearance of pathogens and apoptotic cells. C1q is also known to modulate a range of functions of immune and non-immune cells, including their involvement in placental development and sensorial synaptic pruning. We have recently shown that C1q can promote tumour by encouraging their adhesion, migration and proliferation in addition to angiogenesis and metastasis. In this study, we have examined the role of C1q in the microenvironment of malignant pleuric mesothelioma (MPM), a rare form of cancer commonly associated with exposure to asbestos. We found that C1q was highly expressed in all MPM…

lcsh:Immunologic diseases. Allergy0301 basic medicineComplement system; Malignant pleural mesothelioma; Hyaluronic acid; Mesothelioma cells; C1q; CancerAngiogenesisMPMp38 mitogen-activated protein kinasesImmunologyHAchemical and pharmacologic phenomenaBiologyMetastasisMesothelioma cell03 medical and health sciencesClassical complement pathwaychemistry.chemical_compound0302 clinical medicineImmune systemhyaluronic acidHyaluronic acidmedicinemalignant pleural mesotheliomacancerImmunology and AllergyCell adhesioncomplement systemC1qcomplement system; MPM; HA; Mesothelioma cells; C1q and cancerOriginal ResearchC1q and cancermedicine.diseaseComplement system030104 developmental biologyC1q; Cancer; Complement system; Hyaluronic acid; Malignant pleural mesothelioma; Mesothelioma cells; Immunology and Allergy; Immunologychemistrymesothelioma cells030220 oncology & carcinogenesisImmunologyCancer researchlcsh:RC581-607Frontiers in Immunology
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Blocking Jak/STAT signalling using tofacitinib inhibits angiogenesis in experimental arthritis

2021

Abstract Objective During rheumatoid arthritis (RA), the angiogenic processes, occurring with pannus-formation, may be a therapeutic target. JAK/STAT-pathway may play a role and the aim of this work was to investigate the inhibiting role of a JAK-inhibitor, tofacitinib, on the angiogenic mechanisms occurring during RA. Methods After ethical approval, JAK-1, JAK-3, STAT-1, STAT-3 and VEGF expression was evaluated on RA-synovial-tissues. In vitro, endothelial cells (ECs), stimulated with 20 ng/ml of VEGF and/or 1 μM of tofacitinib, were assessed for tube formation, migration and proliferation, by Matrigel, Boyden chamber assay and ki67 gene-expression. In vivo, 32 mice received collagen (coll…

medicine.medical_specialtyAngiogenesisArthritisDiseases of the musculoskeletal systemPharmacologyPyrroleMiceRheumatoid arthritis Angiogenesis TofacitinibPiperidinePiperidinesIn vivoInternal medicineMedicineAnimalsHumansPyrrolesRheumatoid arthritisRheumatoid arthritiTube formationMatrigelEndothelial CellTofacitinibbusiness.industryAnimalSynovial MembraneEndothelial Cellsmedicine.diseaseArthritis ExperimentalRheumatologyAngiogenesiPyrimidinesPyrimidineRC925-935TofacitinibRheumatoid arthritisAngiogenesisbusinessHumanResearch Article
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