Search results for "Anticarcinogen"

showing 10 items of 30 documents

Comparison of the chemopreventive efficacies of garlic powders with different alliin contents against aflatoxin B1 carcinogenicity in rats

2004

Garlic (Allium sativum) is well known for its beneficial effects on health and particularly for its chemopreventive potential against cancer. The present study was designed to compare the chemopreventive efficacies of several garlic powders with various levels of alliin, a precursor of active sulfur compounds. For this purpose we used the medium-term hepatocarcinogenesis protocol (resistant hepatocyte model), which allows the detection of preneoplasic foci expressing the placental form of glutathione S-transferase (GST-P) as an end-point. Rats were fed diets containing three garlic powders (5% of the diet) with various alliin contents for 3 weeks. Garlic powders were obtained from bulbs gro…

GARLIC POWDERMale[SDE] Environmental SciencesCancer ResearchAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]Antineoplastic AgentsAlliinChemoprevention03 medical and health scienceschemistry.chemical_compound0302 clinical medicinefoodLiver Neoplasms ExperimentalCytochrome P-450 CYP1A1IngestionAnimalsFood scienceCysteineGlucuronosyltransferaseRats WistarGarlicAnticarcinogenCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologyGlutathione Transferase2. Zero hunger0303 health sciencesfood and beveragesGeneral MedicineGlutathioneAllium sativumCANCERfood.food3. Good healthDietRats[SDV] Life Sciences [q-bio]chemistryBiochemistry030220 oncology & carcinogenesis[SDE]Environmental SciencesRATPowdersCARCINOGENESEPrecancerous Conditions
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n-3 polyunsaturated fatty acids and HER2-positive breast cancer: interest of the fat-1 transgenic mouse model over conventional dietary supplementati…

2013

Overexpression of the tyrosine kinase receptor ErbB2/HER2/Neu, occurs in 25%-30% of invasive breast cancer (BC) with poor patient prognosis. Even if numerous studies have shown prevention of breast cancer by n-3 fatty acid intake, the experimental conditions under which n-3 fatty acids exert their protective effect have been variable from study to study, preventing unifying conclusions. Due to confounding factors, inconsistencies still remain regarding protective effects of n-3 polyunsaturated fatty acids (PUFA) on BC. When animals are fed with dietary supplementation in n-3 fatty acids (the traditional approach to modify tissue content and decrease the n-6/n-3 ratio) complex dietary intera…

Genetically modified mouseFatty Acid Desaturasesmedicine.medical_specialtyReceptor ErbB-2Breast NeoplasmsMice TransgenicBiologyBiochemistryReceptor tyrosine kinaseMiceBreast cancerInternal medicineFatty Acids Omega-3medicineAnimalsAnticarcinogenic AgentsHumansDietary supplementationCaenorhabditis elegans Proteinschemistry.chemical_classificationConfoundingFatty acidGeneral Medicinemedicine.disease3. Good healthDisease Models AnimalEndocrinologychemistryDietary Supplementsbiology.proteinFemaleSignal transductionPolyunsaturated fatty acidBiochimie
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Effect of some indole derivatives on xenobiotic metabolism and xenobiotic-induced toxicity in cultured rat liver slices.

1999

In this study the effect of some indole derivatives on xenobiotic metabolizing enzymes and xenobiotic-induced toxicity has been examined in cultured precision-cut liver slices from male Sprague-Dawley rats. While treatment of rat liver slices for 72 hours with 2-200 microM of either indole-3-carbinol (I3C) or indole-3-acetonitrile (3-ICN) had little effect on cytochrome P-450 (CYP)-dependent enzyme activities, enzyme induction was observed after in vivo administration of I3C. The treatment of rat liver slices with 50 microM 3,3'-diindolylmethane (DIM; a dimer derived from I3C under acidic conditions) for 72 hours resulted in a marked induction of CYP-dependent enzyme activities. DIM appears…

Male33'-DiindolylmethaneAflatoxin B1IndolesCarcinogenicity TestsDiindolylmethaneIn Vitro TechniquesToxicologyXenobioticsRats Sprague-Dawleychemistry.chemical_compoundCytochrome P-450 Enzyme SystemAnimalsAnticarcinogenic AgentsDrug InteractionsEnzyme inducerMonocrotalinebiologyCytochrome P450General MedicineGlutathioneRatschemistryBiochemistryLiverToxicitybiology.proteinCarcinogensXenobioticDrug metabolismFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Mechanisms involved in the chemoprevention of flavonoids

2001

Flavonoids, widespread in edible plants, have been studied extensively for their anticarcinogenic properties. However, only few studies have been done with these constituents being administered by the dietary route. In our research, the effects of feeding rats with flavone, flavanone, tangeretin, and quercetin were investigated on two steps of aflatoxin B1 (AFB1)-induced hepatocarcinogenesis (initiation and promotion). Nonpolar flavonoids such as flavone, flavanone and tangeretin administered through the initiation period, decreased the number of -gamma-glutamyl transpeptidase-preneoplastic foci. In the same conditions of administration, quercetin, a polyhydroxylated flavonoid, showed no pr…

MaleAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]Clinical BiochemistryFlavonoidChemopreventionBiochemistryFlavones03 medical and health scienceschemistry.chemical_compoundTangeretinCytosolLiver Neoplasms Experimental0302 clinical medicineAnimalsAnticarcinogenic Agentsheterocyclic compoundsRats WistarComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidschemistry.chemical_classification0303 health sciencesDNAGeneral MedicineGlutathioneFlavonesGlutathioneCANCERDietRats3. Good health[SDV] Life Sciences [q-bio]LiverchemistryBiochemistryPhenobarbital030220 oncology & carcinogenesisFlavanonesCarcinogensChemoprotectiveMolecular MedicineQuercetinQuercetinFlavanoneBioFactors
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Protective Effects of Fruits and Vegetables against In Vivo Clastogenicity of Cyclophosphamide or Benzo[a]pyrene in Mice

1998

Seven fruits and 10 vegetables commonly consumed in Germany were investigated for their anticlastogenic potencies against cyclophosphamide (CP) and benzo[a]pyrene (BaP) in the in vivo mouse bone marrow micronucleus assay. We detected protective effects in 76.5% and 70.6% of the samples, respectively, and more or less distinct quantitative differences between the various plant materials and the two clastogens investigated. With respect to CP, moderate activities were exerted by sweet cherries, strawberries, cucumber, radish and tomatoes, average activities by bananas, oranges, peaches, asparagus and red beets and strong activities by yellow red peppers and especially spinach. Apples (cultiva…

MaleCitrusRed peppersBone Marrow CellsToxicologytheaterMiceClastogenchemistry.chemical_compoundVegetablesBotanyBenzo(a)pyreneAnimalsAnticarcinogenic AgentsAsparagusCultivarFood scienceAntineoplastic Agents AlkylatingCyclophosphamideLegumeMicronucleus TestsbiologyPlant ExtractsChemistryfungifood and beveragesGeneral Medicinebiology.organism_classificationDietBenzo(a)pyreneFruitCarcinogensSpinachtheater.playAntimutagenMutagensFood ScienceFood and Chemical Toxicology
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Coffee and its chemopreventive components Kahweol and Cafestol increase the activity of O6-methylguanine-DNA methyltransferase in rat liver—compariso…

2003

Abstract A lower rate of colon cancer was observed in consumers of coffee with a high content of the diterpenes Kahweol and Cafestol (K/C). In animal models, K/C have been found to protect against the mutagenic/carcinogenic effects of compounds such as 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP), aflatoxin B1, and 7,12-dimethylbenz[a]anthracene. Thus far, such chemoprotection by K/C has been attributed to modifications of xenobiotic metabolism, e.g. enhanced detoxification by UDP-glucuronosyltransferase (UDPGT) and/or glutathione transferase (GST). In the present study, we investigated the potential of several coffee-related treatments (K/C [1:1], Cafestol-alone, Turkish coffee) …

MaleDNA damageHealth Toxicology and MutagenesisCafestolBiologyCoffeeXenobioticschemistry.chemical_compoundGeneticsmedicineAnimalsDNA Modification MethylasesMolecular BiologyAnticarcinogenCarcinogenKahweolGlutathioneMolecular biologyRatsLiverchemistryBiochemistryDiterpenesAntimutagenDrug metabolismmedicine.drugMutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
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Modification of hepatic drug-metabolizing enzymes in rat fed naturally occurring allyl sulphides

1994

1. The effects of feeding allyl sulphides to rat (2000 ppm of the diet for 15 days) were investigated on various microsomal hepatic drug-metabolizing enzymes by their immunochemical detection and catalytic activity. 2. Allyl sulphides provoked a temporary dietary restriction, which enhanced the microsomal level of P450 and the activities of NADH-cytochrome c reductase and p-hydroxybiphenyl UDP-glucuronyltransferase (UDPGT 2), and lowered the activities of p-nitrophenol hydroxylase (PNPH), N-nitrosodimethylamine demethylase (NDMAD), laurate omega-hydroxylase (LAH) and glutathione S-transferase (GST). Therefore, pair-fed animals were used as a more relevant control for the dietary effects of …

MaleHealth Toxicology and MutagenesisImmunoblottingAllyl compoundAntineoplastic Agents[SDV.BID]Life Sciences [q-bio]/BiodiversitySulfidesReductaseToxicologyBiochemistryEating03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIMMUNOCHIMIECytochrome P-450 Enzyme SystemAnimalsDisulfidesGlucuronosyltransferaseRats WistarEpoxide hydrolaseAnticarcinogenGlutathione Transferase030304 developmental biologyEpoxide HydrolasesPharmacologychemistry.chemical_classification0303 health sciencesDose-Response Relationship DrugbiologyChemistryBody WeightCytochrome P450Organ SizeGeneral MedicineGlutathioneDietRatsAllyl CompoundsEnzymeLiverBiochemistryTOXICOLOGIE030220 oncology & carcinogenesisMicrosomes Liverbiology.proteinMicrosomeRAT[SDV.BID] Life Sciences [q-bio]/Biodiversity
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In vivo antigenotoxic effects of dietary allyl sulfides in the rat

1997

The effects of dietary administration of diallyl sulfide (DAS), diallyl disulfide (DADS) and allyl mercaptan (AM) on the genotoxicity of different chemicals were studied in two experimental systems: (i) measurement of hepatic DNA single-strand breaks induced in rats by aflatoxin B1 (AFB1), N-nitrosodimethylamine (NDMA) or methylnitrosourea (MNU); (ii) mutagenicity of AFB1 or NDMA on Salmonella typhimurium TA100 using hepatic S9 from rats fed allyl sulfides as the activation system. All compounds strongly reduced hepatic DNA breaks induced by AFB1 and NDMA but did not modify the genotoxicity of MNU. In the Ames test, the mutagenicity of NDMA was strongly inhibited by hepatic S9 from rats fed…

MaleSalmonella typhimuriumCancer ResearchAflatoxin B1[SDV]Life Sciences [q-bio]Allyl compoundMutagenSulfidesmedicine.disease_cause030226 pharmacology & pharmacyDimethylnitrosamineAmes test03 medical and health scienceschemistry.chemical_compound0302 clinical medicineN-NitrosodimethylaminemedicineAnimalsAnticarcinogenic AgentsDisulfidesComputingMilieux_MISCELLANEOUSMutagenicity TestsDiallyl disulfidefood and beveragesAntimutagenic AgentsMethylnitrosoureaRats3. Good healthAllyl Compounds[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistry030220 oncology & carcinogenesisRATAllyl MercaptanCARCINOGENESEAllyl SulfideGenotoxicityDNA DamageMutagensCancer Letters
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Modulation of aflatoxin B1 carcinogenicity, genotoxicity and metabolism in rat liver by dietary carotenoids: evidence for a protective effect of CYP1…

1997

The effects of several carotenoids of vitamin A and of 3-methylcholanthrene have been tested on the initiation of hepatocarcinogenesis by aflatoxin B1, using the sequential protocol of Solt and Farber. AFB1-induced DNA single-strand breaks and AFB1-metabolism were also assessed. The P4501A inducer carotenoids (canthaxanthin, astaxanthin, beta-apo-8'-carotenal) and 3-methylcholanthrene reduce the carcinogenicity of AFB1, divert AFB1-metabolism into the less genotoxic aflatoxin M1 and reduce AFB1-induced DNA single-strand breaks: we conclude that these carotenoids exert their protective effect through the deviation of AFB1 metabolism towards detoxification pathways. beta-Carotene decreased AF…

MaleVitaminCancer ResearchAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]MutagenBiologymedicine.disease_causeAntioxidants03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyAstaxanthinmedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACarotenoidCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesINDUCTIONfood and beverages04 agricultural and veterinary sciencesCarotenoids040401 food scienceDietRats3. Good health[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistryCarcinogensRATCARCINOGENESEGenotoxicityDNA DamageMethylcholanthrene
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Dietary carotenoids inhibit aflatoxin B1-induced liver preneoplastic foci and DNA damage in the rat: Role of the modulation of aflatoxin B1 metabolism

1998

To study the effects of carotenoids on the initiation of liver carcinogenesis by aflatoxin B1 (AFB1), male weanling rats were fed beta-carotene, beta-apo-8'-carotenal, canthaxanthin, astaxanthin or lycopene (300 mg/kg diet), or an excess of vitamin A (21000 RE/kg diet), or were injected i.p. with 3-methylcholanthrene (3-MC) (6 x 20 mg/kg body wt) before and during i.p. treatment with AFB1 (2 x 1 mg/kg body wt). The rats were later submitted to 2-acetylaminofluorene treatment and partial hepatectomy, and placental glutathione S-transferase-positive liver foci were detected and quantified. The in vivo effects of carotenoids or of 3-MC on AFB1-induced liver DNA damage were evaluated using diff…

MaleVitaminCancer Researchmedicine.medical_specialtyAflatoxinAflatoxin B1DNA damage[SDV]Life Sciences [q-bio]Biology03 medical and health scienceschemistry.chemical_compoundLiver Neoplasms Experimental0302 clinical medicineAstaxanthinInternal medicinemedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACYTOCHROME P 450CarotenoidSerum Albumin030304 developmental biologychemistry.chemical_classification0303 health sciencesRetinolGeneral MedicineCarotenoidsDietRats3. Good health[SDV] Life Sciences [q-bio]Endocrinologychemistry030220 oncology & carcinogenesisCarcinogensMicrosomes LiverRAT2-AcetylaminofluoreneCARCINOGENESEPrecancerous ConditionsDNA DamageMethylcholanthrene
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