Search results for "Antineoplastic agent"

showing 10 items of 1538 documents

Aminobisphosphonate-activated γδ T cells in immunotherapy of cancer: doubts no more

2008

BACKGROUND: Activated V gamma 9 V delta 2 T cells are able to kill most tumour cells because of recognition by T cell receptor and natural killer receptors. OBJECTIVE: We discuss the possibility that the intentional activation of gammadelta T cells in vivo by aminobisphosphonates may represent a promising target for the design of novel and highly innovative immunotherapy in cancer patients. METHODS: The antitumoral effects of gammadelta T cells both in vitro and in vivo have been demonstrated suggesting a new therapeutic approach for translation into the clinical setting. RESULTS/CONCLUSION: V gamma 9 V delta 2 T lymphocytes represent a particularly interesting target for immunotherapeutic …

Aminobisphosphonate Gamma delta T cells cancermedicine.medical_treatmentT cellClinical BiochemistryReceptors Antigen T-CellAntineoplastic AgentsModels BiologicalInterleukin 21Immune systemAntigenT-Lymphocyte SubsetsIn vivoNeoplasmsDrug DiscoveryAnimalsHumansCytotoxic T cellMedicinePharmacologyClinical Trials as TopicDiphosphonatesbusiness.industryT-cell receptorReceptors Antigen T-Cell gamma-deltaImmunotherapyKiller Cells Naturalmedicine.anatomical_structureImmune SystemImmunologyInterleukin-2ImmunotherapybusinessImmunologic MemoryExpert Opinion on Biological Therapy
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Effects of amsacrine (m-AMSA), a new aminoacridine antitumor drug, on the rabbit heart.

1983

There is emerging clinical evidence that amsacrine (m-AMSA) administration may be associated with cardiotoxic effects such as severe, even fatal, ventricular arrhythmias and impairment of the inotropic performance of the heart. Information on the cardiac effects of m-AMSA in animals is scanty. Studies on mice, dogs, and monkeys have not evidenced the cardiotoxicity of the compound. The data presented in this paper show that m-AMSA causes acute ECG alterations in normal rabbits and a dose-related negative inotropic effect on the isolated rabbit heart, suggesting that this species may be a useful model for the study of the cardiac actions of this antiblastic.

AmsacrineDose-Response Relationship DrugAminoacridinesHeart VentriclesAntineoplastic AgentsArrhythmias CardiacHeartModels BiologicalMyocardial ContractionCardiotoxicityElectrocardiographym-Amsaantitumor drugDepression ChemicalHeart Function Testscancer.AnimalsRabbitsCancer treatment reports
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Identification of conjugation and cleavage products in the thiolytic metabolism of the anticancer drug 4'-(9-acridinylamino)methanesulfon-m-anisidide.

1981

Conjugation and cleavage products in the thiolytic metabolism of the anticancer drug 4′ -(9-acridinyl amino)methanesulfon-m-anisidide were identified primarily by high-pressure liquid chromatography in combination with field desorption mass spectrometry. The spontaneous metabolic pathway of the drug, as related to its susceptibility to nucleophilic attack by endogenous thiols at the 9-carbon atom of the acridine moiety, has been studied. Among the metabolite fraction of 4′-(9-acridinylamino)methanesulfon-m-anisidide excreted in rat bile after administration of a therapeutic dose, a conjugate was identified as the 9-acridinyl thioether of glutathione. This conjugation product and the corresp…

AmsacrineMaleStereochemistryMetaboliteAntineoplastic AgentsBiochemistryMass Spectrometrychemistry.chemical_compoundThioetherAnimalsBileSpectroscopyChromatography High Pressure LiquidAminoacridinesRats Inbred StrainsGlutathioneMetabolismGlutathioneRatsMetabolic pathwaychemistryAcridineMolecular MedicineChromatography Thin LayerCysteineConjugateBiomedical mass spectrometry
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Apoptotic effects of different drugs on cultured retinoblastoma Y79 cells

1998

This paper deals with the apoptotic effect exerted in human retinoblastoma Y79 cells by a number of compounds. A remarkable effect was observed after treatment with DNA-damaging agents, such as camptothecin, etoposide, cisplatin and carboplatin; camptothecin was found to be the most efficacious. Treatment with these compounds induced the appearance of morphological features of apoptosis in the cells together with the distinct fragmentation of DNA, as shown by agarose gel electrophoresis. These effects were also accompanied by a remarkable increase in the level of p53. Many other compounds, which are not DNA-damaging agents, induced the morphological features of apoptosis but none of them we…

AmsacrinePaclitaxelDNA damageAntineoplastic AgentsApoptosisSuraminDNA ladderingBiologyretinoblastomaCarboplatinchemistry.chemical_compoundTumor Cells CulturedmedicineHumansFragmentation (cell biology)EtoposideCisplatinSodium butyrateGeneral MedicineAntineoplastic Agents PhytogenicMolecular biologyButyrateschemistryApoptosisAgarose gel electrophoresisImmunologyButyric AcidCamptothecinCisplatinDrug Screening Assays AntitumorCamptothecinDNA Damagemedicine.drug
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A recommended practical approach to the management of target therapy and angiogenesis inhibitors cardiotoxicity: an opinion paper of the working grou…

2016

The US National Cancer Institute estimates that cardiotoxicity (CTX) from target therapy refers mostly to four groups of drugs: epidermal growth factor receptor 2 inhibitors, angiogenic inhibitors, directed Abelson murine leukemia viral oncogene homolog inhibitors, and proteasome inhibitors. The main cardiotoxic side-effects related to antiepidermal growth factor receptor 2 therapy are left ventricular systolic dysfunction and heart failure. Angiogenesis inhibitors are associated with hypertension, left ventricular dysfunction/heart failure, myocardial ischemia, QT prolongation, and thrombosis. Moreover, other agents may be related to CTX induced by treatment. In this study, we review the g…

AngiogenesisLeftAngiogenesis Inhibitors030204 cardiovascular system & hematologyVentricular Dysfunction Left0302 clinical medicinetyrosine kinase inhibitorNeoplasmstyrosine kinase inhibitorsVentricular DysfunctionMolecular Targeted TherapyEpidermal growth factor receptorSocieties Medicalangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitorABLbiologyDisease ManagementGeneral MedicineItalyCardiovascular DiseasesSupplement Submission030220 oncology & carcinogenesisangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitors; Angiogenesis Inhibitors; Antineoplastic Agents; Cardiology; Cardiomyopathies; Cardiotoxicity; Heart Failure; Humans; Italy; Neoplasms; Practice Guidelines as Topic; Societies Medical; Ventricular Dysfunction Left; Disease ManagementPractice Guidelines as TopicCardiologyCardiology and Cardiovascular MedicineCardiomyopathiesmedicine.medical_specialtyCardiologyAntineoplastic AgentsRisk AssessmentQT interval03 medical and health sciencesGrowth factor receptorInternal medicineMedicalmedicineHumansMonitoring PhysiologicHeart FailureCardiotoxicitybusiness.industryCancerHER2/epidermal growth factor receptor 2medicine.diseaseangiogenesis inhibitors; HER2/epidermal growth factor receptor 2; tyrosine kinase inhibitors; Cardiology and Cardiovascular MedicineCardiotoxicityangiogenesis inhibitorHeart failurebiology.proteinbusinessSocietiesJournal of cardiovascular medicine (Hagerstown, Md.)
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Inhibition of tumor angiogenesis by antibodies, synthetic small molecules and natural products.

2011

Cancer remains one of the major causes of death worldwide. The switch to pathological angiogenesis is a key process in the promotion of cancer and consequently provides several new and promising targets to anticancer therapy. Thus, antagonizing angiogenesis cuts off the tumor's oxygen and nutrition supply. This review focuses on angiogenesis inhibitors as option for cancer treatment. Modes of action, adverse effects, mechanisms of resistance as well as new developments are highlighted. One approach in angiogenesis inhibition is intermitting the further VEGF (vascular endothelial growth factor) signal pathway with monoclonal antibodies. Bevacizumab is a highly specific recombinant humanized …

Angiogenesismedicine.drug_classGenisteinAngiogenesis InhibitorsAntineoplastic AgentsBiologyPharmacologyMonoclonal antibodyBiochemistryReceptor tyrosine kinaseNeovascularizationSmall Molecule Librarieschemistry.chemical_compoundGrowth factor receptorNeoplasmsDrug DiscoverymedicineAnimalsHumansPharmacologyBiological ProductsNeovascularization PathologicVascular Endothelial Growth FactorsOrganic ChemistryCancerAntibodies Monoclonalmedicine.diseaseAntineoplastic Agents PhytogenicVascular endothelial growth factorchemistrybiology.proteinMolecular Medicinemedicine.symptomCurrent medicinal chemistry
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Anthemis wiedemanniana essential oil prevents LPS-induced production of NO in RAW 264.7 macrophages and exerts antiproliferative and antibacterial ac…

2012

Anthemis wiedemanniana is known in folk medicine for the treatment of microbial infections, cancer and also urinary and pulmonary problems. In this study, the chemical composition of the essential oil from A. wiedemanniana was evaluated and its antibacterial activity was tested against 10 bacterial strains. The oil was also tested for its potentiality to inhibit nitric oxide production in RAW 264.7 macrophages and for its cytotoxicity against four human cancer cell lines. A. wiedemanniana oil, rich of oxygenated monoterpenes (25.4%), showed a good antibacterial activity against Gram-positive bacteria and a good activity against the two Gram-negative bacteria, Escherichia coli and Proteus vu…

Anthemis wiedemanniana essential oil antiproliferative activity antibacterial activityLipopolysaccharidesProteus vulgarisPlant ScienceMicrobial Sensitivity Testsmedicine.disease_causeGram-Positive BacteriaBiochemistryAnalytical ChemistryNitric oxideMicrobiologylaw.inventionCell Linechemistry.chemical_compoundMicelawCell Line TumormedicineEscherichia coliOils VolatileAnimalsHumansAnthemisSettore BIO/15 - Biologia FarmaceuticaCytotoxicityEscherichia coliEssential oilNitritesCell ProliferationbiologyOrganic ChemistrySettore CHIM/06 - Chimica Organicabiology.organism_classificationProteusAntineoplastic Agents PhytogenicAnti-Bacterial AgentschemistryMonoterpenesAnthemisAntibacterial activityBacteriaNatural product research
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Anti-cancer activity of di- and tri-organotin(IV) compounds with D-(+)-Galacturonic acid on human tumor cells

2018

Abstract We have compared the anti-proliferative activity in vitro, of R2SnGala (1-3) [R = Me, n-Bu, Ph] and novel R3SnGala (4, 5) [R = Me, n-Bu] with D-(+)-Galacturonic acid [HGala; Galaq-, q = (2) and (1) for R2SnGala and R3SnGala, respectively] compounds, towards human tumor cell lines of intestinal carcinoma (HCT-116) and breast adenocarcinoma (MCF-7). The new synthesized 4 and 5 compounds were characterized, in solution, by 1H, 13C and 119Sn NMR, that showed that HGala acts as monoanionic moiety and evidenced the dynamic behavior of the compounds, due to inter-conversions involving the anomeric carbon atom of the ligand. Cell viability, apoptosis induction and cell cycle distribution w…

Anti cancerCarbohydrateCell SurvivalHCT-116Antineoplastic AgentsApoptosisBreast NeoplasmsOrganotin(IV)Adenocarcinoma010402 general chemistry01 natural sciencesBiochemistryFlow cytometryInorganic ChemistryOrganotin(IV); D-(+)-Galacturonic acid; NMR; Anti cancer; HCT-116; MCF-7Intestinal NeoplasmsmedicineOrganotin CompoundsCytotoxic T cellHumansViability assayCytotoxicityD-(+)-Galacturonic acidmedicine.diagnostic_testAnti-proliferative010405 organic chemistryCell growthChemistryHexuronic AcidsMCF-7 .Cell cycleHCT116 CellsMolecular biologyNMR0104 chemical sciencesCell cultureApoptosisSettore CHIM/03 - Chimica Generale E InorganicaMCF-7 CellsMCF-7Caco-2 Cells
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Inducers of heme oxygenase-1.

2008

Heme oxygenase-1 (HO-1) is an inducible rate-limiting enzyme which catalyzes group heme into carbon monoxide, iron and bilirubin. In the recent years, HO-1 expression has been reported as an important protective endogenous mechanism against physical, chemical and biological stress. In this regard, induction of this enzyme has shown beneficial effects in several pathologic conditions, such as inflammatory processes, atherosclerosis, carcinogenesis, ischemia-reperfusion systems or degenerative diseases. Complex intracellular signalling cascades mediate the expression of HO-1 in response to external stimuli, Transcription factors, as nuclear factor E2-related factor-2, activator protein-1, and…

Anti-Inflammatory AgentsAntineoplastic AgentsApoptosisAntioxidantsCatalysischemistry.chemical_compoundDrug DiscoverymedicineAnimalsHumansEnzyme inducerHemeTranscription factorPharmacologybiologyActivator (genetics)KinaseUp-RegulationHeme oxygenaseBiochemistryMechanism of actionchemistryEnzyme Inductionbiology.proteinmedicine.symptomSignal transductionHeme Oxygenase-1Signal TransductionTranscription FactorsCurrent pharmaceutical design
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Chemical Constituents and Pharmacological Properties ofPoria cocos

2011

Poria cocos (Polyporaceae) is a saprophytic fungus that grows in diverse species of Pinus. Its sclerotium, called fu-ling or hoelen, is used in traditional Chinese and Japanese medicine for its diuretic, sedative, and tonic effects. Various studies of this fungus have demonstrated its marked anti-inflammatory activity in different experimental models of acute and chronic inflammation. It is widely used as a constituent of many preparations in Asian medicine, but the number of research papers on its clinical properties is insufficient for establishing its efficacy and safety from a scientific point of view. In this review, we have compiled all the published data concerning the chemistry, pha…

Anti-Inflammatory AgentsPharmaceutical ScienceAntineoplastic AgentsInflammationBiologyPharmacognosyAnalytical ChemistryImmune systemJapanPolysaccharidesPsoriasisDrug DiscoverymedicineHumansImmunologic FactorsSecretionMedicine Chinese TraditionalPolyporaceaePharmacologyBiological ProductsTraditional medicineOrganic ChemistryPoriamedicine.diseasebiology.organism_classificationTriterpenesPolyporusComplementary and alternative medicinePhytochemicalMolecular Medicinemedicine.symptomPlanta Medica
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