Search results for "Apolipoprotein"

showing 10 items of 354 documents

The Chaperone Activity of Clusterin is Dependent on Glycosylation and Redox Environment

2014

Background/Aims: Clusterin (CLU), also known as Apolipoprotein J (ApoJ) is a highly glycosylated extracellular chaperone. In humans it is expressed from a broad spectrum of tissues and related to a plethora of physiological and pathophysiological processes, such as Alzheimer's disease, atherosclerosis and cancer. In its dominant form it is expressed as a secretory protein (secreted CLU, sCLU). During its maturation, the sCLU-precursor is N-glycosylated and cleaved into an α- and a β-chain, which are connected by five symmetrical disulfide bonds. Recently, it has been demonstrated that besides the predominant sCLU, rare intracellular CLU forms are expressed in stressed cells. Since these for…

DNA ComplementaryGlycosylationGlycosylationPhysiologyMutantCarbohydrateslcsh:Physiologylcsh:Biochemistrychemistry.chemical_compoundChaperonesHumanslcsh:QD415-436Redox biologySecretory pathwaylcsh:QP1-981ClusterinbiologyRetro-translocationProprotein convertaseProteostasis networkOxidative StressClusterinSecretory proteinHeat shockchemistryBiochemistryApolipoprotein JChaperone (protein)Proteolysisbiology.proteinOxidation-ReductionIntracellularMolecular ChaperonesFurin-like proprotein convertasesCellular Physiology and Biochemistry
researchProduct

Dissecting Abdominal Aortic Aneurysm Is Aggravated by Genetic Inactivation of LIGHT (TNFSF14)

2021

Abdominal aortic aneurysm (AAA), is a complex disorder characterized by vascular vessel wall remodeling. LIGHT (TNFSF14) is a proinflammatory cytokine associated with vascular disease. In the present study, the impact of genetic inactivation of Light was investigated in dissecting AAA induced by angiotensin II (AngII) in the Apolipoprotein E-deficient (Apoe−/−) mice. Studies in aortic human (ah) vascular smooth muscle cells (VSMC) to study potential translation to human pathology were also performed. AngII-treated Apoe−/−Light−/− mice displayed increased abdominal aorta maximum diameter and AAA severity compared with Apoe−/− mice. Notably, reduced smooth muscle α-actin+ area and Acta2 and C…

Dissecting Abdominal Aortic Aneurysmmedicine.medical_specialtyVascular smooth musclebiologyApolipoprotein BQH301-705.5ChemistryMedicine (miscellaneous)Angiotensin IIArticleTNFSF14/LIGHTGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineabdominal aortic aneurysmEndocrinologyLymphotoxinInternal medicinecardiovascular systembiology.proteinmedicinevascular smooth muscle cellsGene silencingBiology (General)ACTA2Biomedicines
researchProduct

Epigenomics and metabolomics reveal the mechanism of the APOA2-saturated fat intake interaction affecting obesity

2018

BACKGROUND: The putative functional variant −265T>C (rs5082) within the APOA2 promoter has shown consistent interactions with saturated fatty acid (SFA) intake to influence the risk of obesity. OBJECTIVE: The aim of this study was to implement an integrative approach to characterize the molecular basis of this interaction. DESIGN: We conducted an epigenome-wide scan on 80 participants carrying either the rs5082 CC or TT genotypes and consuming either a low-SFA (C genotype, promoting an APOA2 expression difference between APOA2 genotypes on a high-SFA diet, and modulating BCAA and tryptophan metabolic pathways. These findings identify potential mechanisms by which this highly reproducible ge…

EpigenomicsMale0301 basic medicineGenotypeMedicine (miscellaneous)030204 cardiovascular system & hematologyBiologyBioinformatics03 medical and health sciences0302 clinical medicineFramingham Heart StudyMetabolomicsGenotypemedicineHumansMetabolomicsDrug InteractionsObesityEpigeneticsAgedEpigenomicsNutrition and DieteticsApolipoprotein A-Ifood and beveragesGenetic VariationEpigenomeDNA MethylationMiddle AgedLipid Metabolismmedicine.diseaseDietary FatsObesityOriginal Research Communications030104 developmental biologyGene Expression RegulationSaturated fatty acidCpG IslandsFemaleApolipoprotein A-IIThe American Journal of Clinical Nutrition
researchProduct

Hypercholesterolemic patients have higher eryptosis and erythrocyte adhesion to human endothelium independently of statin therapy

2021

BACKGROUND Phosphatidylserine (PS) externalization out of the membrane facilitates the eryptotic erythrocytes (EE) binding to endothelial cells (EC), potentially leading to atherosclerosis. Thus, the levels of eryptosis and interactions of EE-EC in hypercholesterolemic patients, either non-medicated or medicated, compared with healthy subjects were studied. METHODS A total of 56 subjects clustered into three groups: (control (n = 20), hypercholesterolemic non-treated (HCNT) (n = 15), and statin-treated (HCT) (n = 21)) were enrolled in this cross-sectional study. Biochemical parameters were determined with validated and standard methods. PS exposure was estimated from annexin-V-binding, cell…

ErythrocytesApolipoprotein BEndotheliumEryptosisPharmacologymedicine.disease_causeMicrocirculationFlow cytometrychemistry.chemical_compoundmedicineHumansEndotheliumbiologymedicine.diagnostic_testbusiness.industryEndothelial CellsGeneral MedicineGlutathionePhosphatidylserineAdhesionCross-Sectional Studiesmedicine.anatomical_structurechemistrybiology.proteinCalciumHydroxymethylglutaryl-CoA Reductase InhibitorsbusinessOxidative stressInternational Journal of Clinical Practice
researchProduct

Cell-mediated lipoprotein transport: A novel anti-atherogenic concept

2010

Lipoprotein transport is thought to occur in the plasma compartment of the blood, where lipoproteins are modulated by various enzymatic reactions. Subsequently, lipoproteins can migrate through the endothelial barrier to the subendothelial space or are taken up by the liver. The interaction between pro-atherogenic (apoB-containing) lipoproteins and blood cells (especially monocytes and macrophages) in the subendothelial space is well known. This lipoprotein-inflammatory cell interplay is central in the development of the atherosclerotic plaque. In this review, a novel interaction is described between lipoproteins and both leukocytes and erythrocytes in the blood compartment. This lipoprotei…

ErythrocytesApolipoprotein BLipoproteinsComplement receptor 1Blood lipidsLeukocytesInternal MedicinemedicineAnimalsHumansEndothelial dysfunctionComplement ActivationApolipoproteins BbiologyBiological TransportGeneral MedicineLipoprotein(a)Atherosclerosismedicine.diseaseComplement systemCell biologyLiverBiochemistryLipoprotein transportbiology.proteinlipids (amino acids peptides and proteins)Inflammation Mediatorsbiology.geneCardiology and Cardiovascular MedicineLipoproteinAtherosclerosis Supplements
researchProduct

Exome sequencing of three cases of familial exceptional longevity

2014

Exceptional longevity (EL) is a rare phenotype that can cluster in families, and co-segregation of genetic variation in these families may point to candidate genes that could contribute to extended lifespan. In this study, for the first time, we have sequenced a total of seven exomes from exceptionally long-lived siblings (probands ≥ 103 years and at least one sibling ≥ 97 years) that come from three separate families. We have focused on rare functional variants (RFVs) which have ≤ 1% minor allele frequency according to databases and that are likely to alter gene product function. Based on this, we have identified one candidate longevity gene carrying RFVs in all three families, APOB. Inter…

Exome sequencingCienciaMaleAgingCandidate genemedia_common.quotation_subjectLongevityEnvejecimientoBiologyGene FrequencyCentenariansGenetic variationapolipoprotein BHumansExomeAllele frequencyGeneExomeExome sequencingmedia_commonGeneticsShort TakesAged 80 and overFamily HealthLongevityrare variantsGenetic VariationRare variantsCell BiologyGenéticaMinor allele frequencyApolipoprotein B-100FemalecentenariansApolipoprotein Bexome sequencingAging Cell
researchProduct

Atherogenic Ratios in Patients with Recurrent Acute Coronary Syndrome and Receiving Statin Therapy: Clinical Usefullness as Cardiovascular Predictors

2015

Patients who have already suffered a vascular event require more and better control of cardiovascular risk factors. Different atherogenic indexes such as TC/HDLc, LDLc/HDLc, apoB/apoA-I, LDLc/apoB and non-HDLc/HDLc have been used to follow-up the patients because of their predictive capacity of the lipid profile. The aim of this study was to evaluate atherogenic ratios as a marker of the lipid residual risk in high-risk patients receiving statin therapy and to know the changes produced by previous lipid-lowering drugs treatment for a previous coronary event. The study including patients admitted to coronary care units of six Spanish tertiary hospitals for Acute Coronary Syndrome (ACS). A to…

First episodemedicine.medical_specialtyAcute coronary syndromeStatinApolipoprotein Bbiologymedicine.diagnostic_testmedicine.drug_classbusiness.industrymedicine.diseaseSurgeryCor MalaltiesResidual riskAnginaInternal medicinebiology.proteinmedicineCardiologylipids (amino acids peptides and proteins)Myocardial infarctionCardiology and Cardiovascular MedicineLipid profilebusiness
researchProduct

Anti-angiogenic drug loaded liposomes: Nanotherapy for early atherosclerotic lesions in mice.

2018

Este artículo se encuentra disponible en la página web de la revista en la siguiente URL: https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0190540 También participan en la elaboración de este artículo científico: Aracely Calatayud-Pascual, Alicia López-Castellano, Elena P. Albelda, Enrique García-España, Luis Martí-Bonmatí, Juan C. Frias y M. Teresa Albelda. Fumagillin-loaded liposomes were injected into ApoE-KO mice. The animals were divided into several groups to test the efficacy of this anti-angiogenic drug for early treatment of atherosclerotic lesions. Statistical analysis of the lesions revealed a decrease in the lesion size after 5 weeks of treatment.

Fluorescence-lifetime imaging microscopyPathologylcsh:MedicineAngiogenesis Inhibitors02 engineering and technology030204 cardiovascular system & hematologyVascular MedicineBiochemistryArteriosclerosis - Chemotherapy.Diagnostic RadiologyAteroesclerosis - Farmacoterapia.MiceWhite Blood Cells0302 clinical medicineAnimal CellsArteriosclerosis - Farmacoterapia.Medicine and Health SciencesArteries - Diseases - Treatment.Nanotechnologylcsh:ScienceAortaPhospholipidsmedia_commonMice KnockoutLiposomeDrug CarriersMultidisciplinarymedicine.diagnostic_testRadiology and Imaging021001 nanoscience & nanotechnologyMagnetic Resonance ImagingLipidsFatty Acids UnsaturatedEngineering and Technologymedicine.symptomCellular Structures and OrganellesCellular TypesAnatomy0210 nano-technologySesquiterpenesResearch ArticleDrugmedicine.medical_specialtyImaging Techniquesmedia_common.quotation_subjectImmune CellsImmunologyLiposomes.Research and Analysis MethodsLiposomas.Lesion03 medical and health sciencesText miningApolipoproteins ECyclohexanesDiagnostic Medicinemedicine.arteryFluorescence ImagingmedicineAnimalsArterias - Enfermedades - Tratamiento.VesiclesAortaBlood Cellsbusiness.industryMacrophageslcsh:RAnti angiogenicBiology and Life SciencesMagnetic resonance imagingCell BiologyAtherosclerosisFumagillin - Therapeutic use.Atherosclerosis - Chemotherapy.Disease Models AnimalFumagilina - Uso terapéutico.LiposomesCardiovascular AnatomyNanoparticlesBlood Vesselslcsh:QbusinessPloS one
researchProduct

Detection of mutations in the apolipoprotein CII gene by denaturing gradient gel electrophoresis. Identification of the splice site variant apolipopr…

1998

AbstractFamilial apolipoprotein (apo) CII deficiency is a rare autosomal recessive inborn error of metabolism clinically resembling lipoprotein lipase deficiency. A number of mutations of the apo CII gene are known to date; they are located in the promoter region, the coding exons, or in the splice junctions. We present a simple assay based on PCR and denaturing gradient gel electrophoresis, which allows scanning of the promoter, the entire coding sequence, and the splice junctions of the apo CII gene for sequence variants. All gene fragments are amplified using a common PCR protocol and are examined for mutations on a single gradient gel. Using this method and direct sequencing, we identif…

Gel electrophoresisGeneticsMutationApolipoprotein BbiologyBiochemistry (medical)Clinical BiochemistryIntronmedicine.diseasemedicine.disease_causeMolecular biologyExonLipoprotein lipase deficiencymedicinebiology.proteinCoding regionGeneClinical Chemistry
researchProduct

Differential apolipoprotein(a) isoform expression in heterozygosity is an independent contributor to lipoprotein(a) levels variability

2003

Abstract Background and methods : Lipoprotein(a) [Lp(a)] levels represent an independent risk factor for cardio- and cerebrovascular diseases. Since lipoprotein(a) levels show a wide variability even in subjects with similar apolipoprotein(a) isoforms, we investigated the contribution of apolipoprotein(a) heterozygosity to lipoprotein(a) variance. Lipoprotein(a) levels, apolipoprotein(a) isoforms identification and expression, and the correlation with other lipo-apolipoprotein parameters have been investigated in 628 subjects >18 years of age. Results : In our study, 246 subjects were found heterozygous for apolipoprotein(a) isoforms. Lipoprotein(a) levels were higher in females. About 40% …

Gene isoformAdultMalemedicine.medical_specialtyHeterozygoteApolipoprotein BClinical BiochemistryWestern blotApoprotein(a)BiochemistryProtein expressionLoss of heterozygosityWestern blotInternal medicinemedicineHumansProtein IsoformsAgedApolipoprotein(a) phenotypingmedicine.diagnostic_testbiologyMedical screeningBiochemistry (medical)General MedicineLipoprotein(a)Middle AgedEndocrinologyApolipoproteinsbiology.proteinProtein expressionlipids (amino acids peptides and proteins)FemaleLipoproteinLipoprotein(a)
researchProduct