Search results for "Arachidonate 5-lipoxygenase"

showing 10 items of 43 documents

Avarol restores the altered prostaglandin and leukotriene metabolism in monocytes infected with human immunodeficiency virus type 1

1991

Abstract Infection of monocytes with human immunodeficiency virus type 1 (HIV-1) (strain Ada-M) caused increased levels of leukotriene B 4 (LTB 4 ) and prostaglandin E 2 (PGE 2 ) in vitro. These two products result from the activities of the two enzymes cyclooxygenase and 5-lipoxygenase. The addition of the sesquiterpenoid hydroquinone Avarol, an HIV inhibitor, strongly reduced the levels of LTB 4 and PGE 2 via inhibition of both cyclooxygenase and lipoxygenase in monocytes. The 50% inhibition concentrations (IC 50 ) for the enzymes were determined to be 2.26 μM (cyclooxygenase) and 1.97 μM (lipoxygenase). A 50% reduction of the extent of PGE 2 and LTB 4 production in HIV-infected monocytes…

Cancer ResearchLeukotriene B4medicine.medical_treatmentProstaglandinBiologyAntiviral AgentsLeukotriene B4DinoprostoneMonocytesLipoxygenasechemistry.chemical_compoundVirologymedicineCyclooxygenase InhibitorsLipoxygenase InhibitorsProstaglandin E2Arachidonate 5-LipoxygenaseMonocyteMolecular biologyInfectious Diseasesmedicine.anatomical_structurechemistryBiochemistryProstaglandin-Endoperoxide SynthasesArachidonate 5-lipoxygenaseHIV-1biology.proteinCyclooxygenaseSesquiterpenesProstaglandin Emedicine.drugVirus Research
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Altered expression of inflammation-related genes in human carotid atherosclerotic plaques.

2011

Abstract Objective Inflammation is a pivotal process in atherosclerosis development and progression, but the underlying molecular mechanisms remain largely obscure. We have conducted an extensive expression study of atherosclerotic plaques to identify the inflammatory pathways involved in atherosclerosis. Methods We studied 11 human carotid plaques, their respective adjacent regions and 7 control arteries from different subjects. Expression of 92 genes was studied by TaqMan low-density array human inflammation panel. Human aortic endothelial and smooth muscle cells were used for in vitro experiments. Results The mRNA levels of 44/92 genes (48%) differed significantly between the tissues exa…

Carotid Artery DiseasesMalemedicine.medical_specialtyMyocytes Smooth MuscleReceptors ProstaglandinPTGS1InflammationReceptors EpoprostenolSettore MED/22 - Chirurgia VascolareMuscle Smooth VascularCytochrome P-450 Enzyme SystemInternal medicineGene expressionmedicineHumansRNA MessengerReceptors CytokineCells CulturedAgedRegulation of gene expressionInflammationbiologyTumor Necrosis Factor-alphaGene Expression ProfilingMacrophagesEndothelial CellsMiddle AgedCoculture TechniquesPlaque AtheroscleroticGene expression profilingLipoproteins LDLEndocrinologyEicosanoidEicosanoid pathwayGene Expression RegulationItalyAtherosclerosiCase-Control StudiesArachidonate 5-lipoxygenasebiology.proteinCancer researchOxidative streTumor necrosis factor alphaFemaleGene expressionmedicine.symptomInflammation MediatorsCardiology and Cardiovascular MedicineCell Adhesion MoleculesAtherosclerosis
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Phenylsulphonyl urenyl chalcone derivatives as dual inhibitors of cyclo-oxygenase-2 and 5-lipoxygenase

2005

Two series of phenylsulphonyl urenyl chalcone derivatives (UCH) with various patterns of substitution were tested for their effects on nitric oxide (NO) and prostaglandin E2 (PGE2) overproduction in RAW 264.7 macrophages. None of the tested compounds reduced NO production more than 50% at 10 microM but most of them inhibited the generation of PGE2 with IC50 values under the micromolar range. Me-UCH 1, Me-UCH 5, Me-UCH 9, Cl-UCH 1, and Cl-UCH 9 were selected to evaluate their influence on human leukocyte functions and eicosanoids generation. These derivatives selectively inhibited cyclo-oxygenase-2 (COX-2) activity in human monocytes being Me-UCH 5 the most potent (IC50 0.06 microM). Selecte…

ChalconeNeutrophilsNitric OxideLeukotriene B4DinoprostoneGeneral Biochemistry Genetics and Molecular BiologyCell LineNitric oxideMiceStructure-Activity Relationshipchemistry.chemical_compoundChalconesmedicineAnimalsHumansCyclooxygenase InhibitorsLipoxygenase InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsProstaglandin E2IC50Molecular StructurebiologySuperoxideMacrophagesElastaseGeneral MedicinechemistryBiochemistryCyclooxygenase 2MyeloperoxidaseArachidonate 5-lipoxygenasebiology.proteinmedicine.drugLife Sciences
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Synthesis and anti-inflammatory activity of chalcone derivatives

1998

Chalcones and their derivatives were synthesized and evaluated for their anti-inflammatory activity. In vitro, chalcones 2, 4, 8, 10 and 13 inhibited degranulation and 5-lipoxygenase in human neutrophils, whereas 11 behaved as scavenger of superoxide. Only four compounds (4-7) inhibited cyclo-oxygenase-2 activity. The majority of these samples showed anti-inflammatory effects in the mouse air pouch model.

ChalconeNeutrophilsmedicine.drug_classLeukotriene B4Clinical BiochemistryPharmaceutical ScienceLeukotriene B4BiochemistryChemical synthesisAnti-inflammatoryMiceStructure-Activity Relationshipchemistry.chemical_compoundChalconeIn vivoDrug DiscoverymedicineAnimalsHumansCyclooxygenase InhibitorsLipoxygenase InhibitorsMolecular BiologyPropiophenonesArachidonate 5-LipoxygenaseCyclooxygenase 2 InhibitorsMolecular StructureChemistrySuperoxideAnti-Inflammatory Agents Non-SteroidalOrganic ChemistryDegranulationMembrane ProteinsIn vitroIsoenzymesBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesDrug DesignMolecular MedicineBioorganic & Medicinal Chemistry Letters
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SAR-studies of γ-secretase modulators with PPARγ-agonistic and 5-lipoxygenase-inhibitory activity for Alzheimer’s disease

2014

Abstract We present the design, synthesis and biological evaluation of compounds containing a 2-(benzylidene)hexanoic acid scaffold as multi-target directed γ-secretase-modulators. Broad structural variations were undertaken to elucidate the structure–activity-relationships at the 5-position of the aromatic core. Compound 13 showed the most potent activity profile with IC50 values of 0.79 μM (Aβ42), 0.3 μM (5-lipoxygenase) and an EC50 value of 4.64 μM for PPARγ-activation. This derivative is the first compound exhibiting low micromolar to nanomolar activities for these three targets. Combining γ-secretase-modulation, PPARγ-agonism and inhibition of 5-lipoxygenase in one compound could be a …

Clinical BiochemistryPharmaceutical SciencePeroxisome proliferator-activated receptorInflammationDiseasePharmacologyInhibitory postsynaptic potentialBiochemistryStructure-Activity Relationshipchemistry.chemical_compoundAlzheimer DiseaseDrug DiscoverymedicineHumansLipoxygenase Inhibitorsγ secretaseCaproatesMolecular BiologyHexanoic acidchemistry.chemical_classificationArachidonate 5-LipoxygenasebiologyOrganic ChemistryPPAR gammachemistryBiochemistryArachidonate 5-lipoxygenasebiology.proteinMolecular MedicineAmyloid Precursor Protein Secretasesmedicine.symptomDerivative (chemistry)Bioorganic & Medicinal Chemistry Letters
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Licofelone, a novel 5-LOX/COX-inhibitor, attenuates leukocyte rolling and adhesion on endothelium under flow

2005

The main mechanism of action of non-steroidal anti-inflammatory drugs (NSAIDs) is the inhibition of cycloxygenases COX-1 and COX-2. During recent years, combined 5-LOX/COX-inhibition, interfering with the biosynthesis of both prostaglandins and leukotrienes (LTs), has emerged as a possibility to avoid side effects related to COX-inhibition. The aim of the present study was to investigate if there is a contribution of mechanisms other than the reduction of inflammatory prostaglandins and leukotrienes to the anti-inflammatory effect of the LOX/COX inhibitor licofelone. In a flow chamber assay, licofelone (10-30 microM) dose-dependently decreased both the rolling and adhesion of leukocytes on …

EndotheliumAcetatesPharmacologyBiochemistrychemistry.chemical_compoundCell MovementIn vivoCell AdhesionLeukocytesmedicineHumansCyclooxygenase InhibitorsPyrrolesLipoxygenase InhibitorsRNA MessengerCells CulturedPharmacologybiologyChemistryEndothelial Cellsmedicine.anatomical_structureMechanism of actionImmunologyArachidonate 5-lipoxygenaseCelecoxibbiology.proteinCyclooxygenasemedicine.symptomLicofeloneCell Adhesion MoleculesSelectinmedicine.drugBiochemical Pharmacology
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Effects of naturally occurring dihydroflavonols from Inula viscosa on inflammation and enzymes involved in the arachidonic acid metabolism

2007

Abstract The anti-inflammatory properties of three flavanones isolated from Inula viscosa , sakuranetin, 7- O -methylaromadendrin, and 3-acetyl-7- O -methylaromadendrin, have been tested both in vitro and in vivo. Acute inflammation in vivo was induced by means of topical application of 12- O -tetradecanoylphorbol 13-acetate (TPA) to mouse ears or by subcutaneous injection of phospholipase A 2 (PLA 2 ) into mouse paws. The test compounds were evaluated in vitro for their effect on both the metabolism of arachidonic acid and on the release and/or activity of enzymes involved in the inflammatory response such as elastase, myeloperoxidase (MPO), and protein kinase C (PKC). The most active comp…

FlavonolsCell SurvivalNeutrophilsIn Vitro TechniquesPharmacologyHistamine ReleaseLeukotriene B4DinoprostonePhospholipases AGeneral Biochemistry Genetics and Molecular BiologySakuranetinMicechemistry.chemical_compoundIn vivoAnimalsEdemaHumansGeneral Pharmacology Toxicology and PharmaceuticsProtein Kinase CProtein kinase CPeroxidaseInflammationLeukotrieneArachidonate 5-LipoxygenaseArachidonic AcidbiologyAnti-Inflammatory Agents Non-SteroidalElastaseGeneral MedicineRatschemistryBiochemistryMyeloperoxidasebiology.proteinTetradecanoylphorbol AcetateFemaleArachidonic acidInulaLeukocyte ElastaseHistamineLife Sciences
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Monohydroxylated fatty acid substrate specificity of human leukocyte 5-lipoxygenase and ω-hydroxylase

1988

Various monohydroxylated fatty acids were synthesized from eicosapolyenoic acids, namely arachidonic (20:4 omega-6), timnodonic (20:5 omega-3), dihomogammalinolenic (20:3 omega-6) and mead (20:3 omega-9) acids. 12-Hydroxy derivatives, as well as 12-hydroxy-5,8,10-heptadecatrienoic acid (HHT), were produced with platelets as the enzyme source, and 15-hydroxy derivatives were produced by soya bean lipoxygenase treatment. Each monohydroxylated fatty acid was incubated with human leukocytes in the presence or absence of the calcium ionophore A23187, and dihydroxylated products were analysed by h.p.l.c. 12-Hydroxy derivatives of 20:4 omega-6, 20:5 omega-3 and 20:3 omega-9 were similarly oxygenat…

Leukotriene B4StereochemistryHydroxylationArachidonate LipoxygenasesBiochemistryMixed Function OxygenasesSubstrate SpecificityHydroxylationchemistry.chemical_compoundLipoxygenaseCytochrome P-450 Enzyme SystemHydroxyeicosatetraenoic AcidsLeukocytesHumansMolecular Biologychemistry.chemical_classificationArachidonate 5-LipoxygenasebiologyFatty AcidsFatty acidSubstrate (chemistry)Cell BiologyEnzymechemistryBiochemistryArachidonate 5-lipoxygenasebiology.proteinCytochrome P-450 CYP4AResearch ArticlePolyunsaturated fatty acidBiochemical Journal
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LTB4 is present in exudative pleural effusions and contributes actively to neutrophil recruitment in the inflamed pleural space

2004

SUMMARY The pleural space is a virtual compartment between the lung and chest wall that becomes filled with fluid and inflammatory cells during a variety of respiratory diseases. Here, we study the potential role of the eicosanoid metabolite leukotriene B4 (LTB4) in disparate diseases leading to acute (pneumonia) or chronic (tuberculosis, cancer) inflammation of the pleural space. LTB4 concentrations were significantly higher in pleural fluid due to pneumonia, tuberculosis and cancer with respect to congestive heart failure and correlated with neutrophil elastase, which is used as an indication of state of activation of neutrophils in the pleural space. Moreover, pleural LTB4 was biological…

LipopolysaccharidesPathologyHot TemperatureNeutrophilsLeukotriene B4Gene ExpressionEpitheliumchemistry.chemical_compoundNeoplasmsClinical StudiesImmunology and AllergyMedicineRespiratory systemPancreatic ElastasebiologyNeutrophilMiddle Agedrespiratory systemChemotaxis Leukocytemedicine.anatomical_structureNeutrophil InfiltrationNeutrophil elastaseLTB4Pleuralipids (amino acids peptides and proteins)medicine.symptomAdultmedicine.medical_specialtyImmunologyInflammationGranulocyteLeukotriene B4HumansRNA MessengerTuberculosis PulmonaryAgedArachidonate 5-LipoxygenaseLungbusiness.industryPneumoniaMacrophage Activationmedicine.diseaserespiratory tract diseasesPleural EffusionPneumoniaEicosanoidchemistryImmunologybiology.proteinbusinessClinical and Experimental Immunology
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Inhibition of leukocyte functions by the alkaloid isaindigotone from Isatis indigotica and some new synthetic derivatives.

2001

The alkaloid isaindigotone (1a) and seven derivatives have been synthesized to study their influence on several leukocyte functions and the generation of inflammatory mediators. Isaindigotone (1a) was found to be a scavenger of superoxide generated either by the hypoxanthine/xanthine oxidase system or stimulated human neutrophils. Isaindigotone (1a) and its acetylated derivative (1b) also inhibited 5-lipoxygenase activity and leukotriene B(4) production in these cells, whereas none of the compounds affected degranulation. In RAW 264.7 macrophages stimulated with lipopolysaccharide, synthetic derivatives exerted higher inhibitory effects on prostaglandin E(2) (PGE(2)) and nitric oxide (NO) g…

LipopolysaccharidesXanthine OxidaseMagnetic Resonance SpectroscopyLeukotriene B4StereochemistryNeutrophilsmedicine.medical_treatmentPharmaceutical ScienceLeukotriene B4DinoprostoneAnalytical ChemistryNitric oxidechemistry.chemical_compoundInhibitory Concentration 50MiceStructure-Activity RelationshipAlkaloidsDrug DiscoverymedicineLeukocytesAnimalsHumansLipoxygenase InhibitorsXanthine oxidaseHypoxanthineCells CulturedPharmacologyInflammationPlants MedicinalbiologyMolecular StructureSuperoxideAlkaloidMacrophagesOrganic ChemistryFree Radical ScavengersComplementary and alternative medicineBiochemistrychemistryArachidonate 5-lipoxygenaseBrassicaceaebiology.proteinQuinazolinesMolecular MedicineChromatography Thin LayerInflammation MediatorsNitric Oxide SynthaseProstaglandin EJournal of natural products
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