Search results for "Arrhythmia"

showing 10 items of 210 documents

A G613A missense in the Hutchinson's progeria lamin A/C gene causes a lone, autosomal dominant atrioventricular block.

2014

Background LMNA/C mutations have been linked to the premature aging syndrome Hutchinson’s progeria, dilated cardiomyopathy 1A, skeletal myopathies (such as the autosomal dominant variant of Emery-Dreifuss muscular dystrophy and limb-girdle muscular dystrophy), Charcot-Marie-Tooth disorder type 2B1, mandibuloacral dysplasia, autosomal dominant partial lipodystrophy, and axonal neuropathy. Atrioventricular block (AVB) can be associated with several cardiac disorders and it can also be a highly heritable, primitive disease. One of the most common pathologies associated with AVB is dilated cardiomyopathy (DCM), which is characterized by cardiac dilatation and reduced systolic function. In this …

ProbandGeneticsExome sequencingAgingProgeriaLamin A/Cbusiness.industryResearchImmunologyDilated cardiomyopathymedicine.diseaseSudden deathLMNAMandibuloacral dysplasiaAgeingMedicineMissense mutationMuscular dystrophybusinessExome sequencingArrhythmiaAtrioventricular blockImmunityageing : IA
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Genotype–phenotype correlation in a new Fabry-disease-causing mutation

2019

Background: Fabry disease (FD) is a rare X-linked inherited lysosomal storage disorder caused by α-galactosidase A deficiency leading to intracellular glycosphingolipid accumulation. FD manifestation is multisystem, and can differ depending on disease-related genetic variants. Currently, more than 700 different FD-causing mutations have been identified in the human GLA gene. We identified a novel mutation in a Lithuanian family with classical manifestations of Fabry disease, revealing severe effects to the cardiovascular systems of heterozygous women. Case presentation: A 49-year-old woman underwent echocardiography due to progressive dyspnea that lasted seven years, reduced physical a…

Probandmedicine.medical_specialtyAbdominal painMedicine (General)α-galactosidase ACase ReportLeft ventricular hypertrophyGastroenterologyclassical manifestationR5-920Internal medicinemedicineGLA geneFabry diseasemedicine.diagnostic_testbusiness.industryCardiac arrhythmiaGeneral MedicineFabry disease ; α-galactosidase A ; GLA gene ; novel mutation ; classical manifestationmedicine.diseaseFabry diseaseHyperintensityMutation (genetic algorithm)<i>GLA</i> geneRenal biopsymedicine.symptomnovel mutationbusiness
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QT interval heterogeneities induced through local epicardial warming/cooling. An experimental study

2014

[EN] ntroduction and objectives Abnormal QT interval durations and dispersions have been associated with increased risk of ventricular arrhythmias. The present study examines the possible arrhythmogenic effect of inducing QT interval variations through local epicardial cooling and warming. Methods In 10 isolated rabbit hearts, the temperatures of epicardial regions of the left ventricle were modified in a stepwise manner (from 22 °C to 42 °C) with simultaneous electrogram recording in these regions and in others of the same ventricle. QT and activation-recovery intervals were determined during sinus rhythm, whereas conduction velocity and ventricular arrhythmia induction were determined dur…

Programmed stimulationmedicine.medical_specialtyHot TemperatureArritmiaQT intervalNerve conduction velocityElectrofisiologíaTECNOLOGIA ELECTRONICAElectrocardiographyEstimulación eléctricaHeart Conduction SystemHeart RateInternal medicinemedicineAnimalsVentricular FunctionSinus rhythmVentrículo izquierdobusiness.industryArrhythmias CardiacGeneral MedicineHypothermiaLeft ventricleElectric StimulationCold TemperatureElectrophysiologyElectrophysiologyIncreased riskmedicine.anatomical_structureVentricleAnesthesiaElectrical stimulationCardiologyRabbitsmedicine.symptombusinessPericardiumArrhythmia
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Development and Long-Term Follow-Up of an Experimental Model of Myocardial Infarction in Rabbits

2020

Simple Summary Ischemic heart disease is one of the leading causes of death. A series of processes occur during acute myocardial infarction that contribute to the development of ventricular dysfunction, with subsequent heart failure and ventricular arrhythmias, which account for most episodes of sudden cardiac death in these patients. These complications are associated with the adverse cardiac remodeling that occurs during the healing process following an acute episode. The remodeling causes the appearance of a substrate that can trigger life-threatening arrhythmias, such as tachycardia and/or ventricular fibrillation. The development of experimental models for analyzing the basic mechanism…

Programmed stimulationmedicine.medical_specialtyLong term follow upmedicine.medical_treatment030204 cardiovascular system & hematologyischemia/reperfusion injuryventricular remodelingArticle03 medical and health sciencesexperimental surgery0302 clinical medicineRisk areaInternal medicinelcsh:ZoologymedicineMyocardial infarctionlcsh:QL1-991cardiovascular diseasesVentricular remodelingLigature030304 developmental biology0303 health scienceslcsh:Veterinary medicineGeneral Veterinarybusiness.industryExperimental modelventricular arrhythmiasInfarct sizemedicine.diseasemyocardial infarctionCardiologycardiovascular systemlcsh:SF600-1100Animal Science and Zoologycardiac mappingbusinessAnimals : an Open Access Journal from MDPI
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Cardiovascular Events in Moderately to Severely Obese Obstructive Sleep Apnea Patients on Positive Airway Pressure Therapy.

2016

&lt;b&gt;&lt;i&gt;Background:&lt;/i&gt;&lt;/b&gt; In moderately to severely obese patients with obstructive sleep apnea (OSA), the effects of long-term positive airway pressure (PAP) treatment on cardiovascular risk are poorly defined. &lt;b&gt;&lt;i&gt;Purpose:&lt;/i&gt;&lt;/b&gt; To assess the effect of continuous positive airway pressure (CPAP) or noninvasive ventilation (NIV) on the occurrence of cardiovascular events in obese OSA patients. &lt;b&gt;&lt;i&gt;Methods:&lt;/i&gt;&lt;/b&gt; We performed a noninterventional observational study in obese OSA patients recruited between 2007 and 2010 at the Sleep Center, University of Grenoble, treated with CPAP or NIV, and followed for 5.6 year…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_treatmentMyocardial Infarctionmacromolecular substancesComorbiditySettore MED/10 - Malattie Dell'Apparato Respiratorio030204 cardiovascular system & hematologySeverity of Illness Index03 medical and health sciences0302 clinical medicineSleep and breathingRisk FactorsSeverity of illnessPositive airway pressuremedicineMyocardial RevascularizationHumansContinuous positive airway pressureMyocardial infarctionAngina UnstableObesityAcute Coronary SyndromeStrokeAgedSleep-disordered breathing · Longitudinal studies · Continuous positive airway pressure · Noninvasive ventilation · PrognosisSleep Apnea ObstructiveContinuous Positive Airway Pressurebusiness.industryArrhythmias CardiacMiddle Agedmedicine.diseaseObesityrespiratory tract diseasesObstructive sleep apneaStroke030228 respiratory systemCardiovascular DiseasesAnesthesiaFemalebusinessRespiration; international review of thoracic diseases
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Drug-induced and iatrogenic infiltrative lung disease.

2004

At present more than 350 drugs are known to cause injury of the lung parenchyma,upper and lower airways, pulmonary circulation, pleura, mediastinum, lymph nodes,and neuromuscular system. Infiltrative lung disease (ILD) is the most common pattern of drug-induced injury. This article, which is clinically oriented rather than drug oriented, reviews the patterns of ILD produced by therapeutic drugs and radiation therapy.

Pulmonary and Respiratory MedicineDrugLung Diseasesmedicine.medical_specialtyDrug-Related Side Effects and Adverse Reactionsmedia_common.quotation_subjectIatrogenic DiseaseMEDLINEAmiodaronePulmonary EdemaSarcoidosis PulmonaryIatrogenic diseaseMedicineHumansIntensive care medicinemedia_commonbusiness.industryPneumoniamedicine.diseaseBreast radiationDermatologyPulmonary AlveoliMethotrexateLung diseaseSarcoidosisbusinessAnti-Arrhythmia AgentsClinics in chest medicine
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Amiodarone pulmonary toxicity

2004

The amount of literature on amiodarone pulmonary toxicity (APT) peaked in 1983-1984 with several hundred cases reported cumulatively, and declined thereafter. Since the mid-1990s, publications have increased, which suggests that APT remains a current problem in clinical practice. Amiodarone remains difficult to diagnose noninvasively, and although the outcome is good in the majority of patients, not all cases of APT can be controlled satisfactorily.

Pulmonary and Respiratory Medicinemedicine.diagnostic_testbusiness.industryPulmonary toxicityIatrogenic DiseaseAmiodaronePneumoniaAmiodaroneBronchoalveolar LavageRespiratory Function TestsClinical PracticeBronchoalveolar lavageAnesthesiaIatrogenic diseaseHumansPleuraMedicinebusinessAnti-Arrhythmia AgentsLungmedicine.drugClinics in Chest Medicine
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The mutual relationship between heart failure and atrial fibrillation

2020

Atrial fibrillation (AF) and heart failure (HF) are evolving epidemies, together responsible for substantial human suffering and health-care expenditure. The simultaneous co-hexistence of the two conditions is associated with mortality rates higher than those observed in individuals with only one or none of them. Patients with concomitant HF and AF suffer from even worse symptoms and poorer prognosis, yet evidence-based evaluation and management of this group of patients is lacking. In this review, we evaluate the common mechanisms for the development of AF in HF patients and vice versa, focusing on the evidence for potential treatment strategies. Recent data have suggested that these patie…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyAdverse outcomesbusiness.industryMortality ratelcsh:Rheart failurelcsh:MedicineAtrial fibrillationArrhythmia-induced cardiomyopathymedicine.diseaseAtrial fibrillationHeart failure.Risk FactorsHeart failureConcomitantmedicineHumansTreatment strategyBest evidenceCardiology and Cardiovascular MedicineIntensive care medicinebusinessHealthcare system
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Association between Tpeak-Tend/QT and major adverse cardiovascular events in patients with Takotsubo syndrome

2020

Background:Conflicting results have been described in the scientific literature regarding the relationship between electrocardiographic parameters and complications in patients with Takotsubo syndrome (TTS). Aim of the present study was to investigate whether there is an association between markers of ventricular repolarization and major adverse cardiovascular events (MACE) during hospitalisation. Methods:A retrospective chart review was conducted on a sample of patients with diagnosis of TTS, based on the fulfilment of the revised Mayo Clinic criteria. MACE included acute heart failure, cardiogenic shock, sustained ventricular tachycardia, ventricular fibrillation, and death. The following…

QT intervalmedicine.medical_specialtyelectrocardiogram030204 cardiovascular system & hematologyQT intervalTpeak–Tend intervalElectrocardiography03 medical and health sciences0302 clinical medicineTakotsubo CardiomyopathyInternal medicineHumansMedicineRepolarizationIn patientcardiovascular diseases030212 general & internal medicineAssociation (psychology)TakotsuboRetrospective StudiesrepolarizationTakotsubo syndromebusiness.industrymajor cardiovascular eventsArrhythmias CardiacGeneral MedicineVentricular FibrillationCardiologyTpeak tendCardiology and Cardiovascular MedicinebusinessActa Cardiologica
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The effects of the tricyclic antidepressants desipramine, doxepin and iprindole on the isolated perfused rabbit heart.

1974

1. The right sympathetic nerves of isolated perfused rabbit hearts were stimulated electrically (1 msec, supramaximal strength, 15 sec) with increasing frequencies (0.25–20 Hz) at 3 min intervals before and 20 min after starting perfusion with desipramine, doxepin or iprindole. Ventricular rate, right atrial and right ventricular tensions were recorded using the transverse method. 2. Sympathic nerve stimulation caused ventricular arrhythmias in the presence of desipramine (3.3 and 5.0 · 10−6 M) and doxepin (1.6−4.7×10−6 M) but failed to produce arrhythmias in hearts not exposed to drugs, or after iprindole, cocaine and atropine. 3. When desipramine or doxepin was added to Tyrode solution co…

QuinidineAtropineMaleSympathetic Nervous SystemTime FactorsStimulationPropranololPharmacologyCocaineHeart RateDesipraminemedicineAnimalscardiovascular diseasesPharmacologyIprindolebusiness.industryDesipramineArrhythmias CardiacHeartGeneral MedicineDoxepinPropranololQuinidineAntidepressive AgentsElectric StimulationPerfusionAtropinecardiovascular systemIprindoleAutonomic Fibers PostganglionicFemaleDoxepinRabbitsbusinessPerfusionDrug Antagonismmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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