Search results for "Association."

showing 10 items of 1723 documents

Life-course leisure-time physical activity trajectories in relation to health-related behaviors in adulthood: the Cardiovascular Risk in Young Finns …

2021

Background Evidence on whether leisure-time physical activity (LTPA) facilitates individuals’ adoption of multiple healthy behaviors remains scarce. This study investigated the associations of diverse longitudinal LTPA trajectories from childhood to adulthood with diet, screen time, smoking, binge drinking, sleep difficulties, and sleep duration in adulthood. Methods Data were drawn from the Cardiovascular Risk in Young Finns Study. Participants were aged 9–18 years (N = 3553; 51% females) in 1980 and 33–49 years at the latest follow-up in 2011. The LTPA trajectories were identified using a latent profile analysis. Differences in self-reported health-related behaviors across the LTPA trajec…

MaleGATEWAY BEHAVIORHealth BehaviorCHILDHOODTrajectoryphysical activityBinge drinkingruokavaliotRisk FactorsChildFinlandALL-CAUSE MORTALITYASSOCIATIONSalcohollcsh:Public aspects of medicineSmoking3142 Public health care science environmental and occupational healthCardiovascular DiseasesADOLESCENCEscreen timetrajectorySLEEP DURATIONFemaleAlcoholfyysinen aktiivisuusResearch ArticleAdultelintavatAdolescentlongitudinallife-courseruutuaikapitkittäistutkimuselämänkaarismokinguni (lepotila)Young AdultLeisure ActivitiesLife-coursetupakointiHumanssleepExercisealkoholi (päihteet)FOOD FREQUENCY QUESTIONNAIREGENDER-DIFFERENCESPhysical activitylcsh:RA1-1270Screen timebinge drinkingDiethumalahakuisuusHeart Disease Risk FactorsterveyskäyttäytyminenLongitudinaldietSleepBMC Public Health
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Comprehensive exploration of the effects of miRNA SNPs on monocyte gene expression.

2012

We aimed to assess whether pri-miRNA SNPs (miSNPs) could influence monocyte gene expression, either through marginal association or by interacting with polymorphisms located in 3'UTR regions (3utrSNPs). We then conducted a genome-wide search for marginal miSNPs effects and pairwise miSNPs × 3utrSNPs interactions in a sample of 1,467 individuals for which genome-wide monocyte expression and genotype data were available. Statistical associations that survived multiple testing correction were tested for replication in an independent sample of 758 individuals with both monocyte gene expression and genotype data. In both studies, the hsa-mir-1279 rs1463335 was found to modulate in cis the expres…

MaleGene Expressionlcsh:MedicineGenome-wide association studyCoronary Artery DiseaseLinkage DisequilibriumMonocytes0302 clinical medicineGene expressionGenotypelcsh:Science3' Untranslated RegionsOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesMultidisciplinaryGenomicsMiddle Aged3. Good healthFemaleRNA InterferenceEpigeneticsResearch ArticleAdultmedicine.medical_specialtyImmune CellsImmunologyLocus (genetics)Single-nucleotide polymorphismBiologyPolymorphism Single Nucleotide03 medical and health sciencesMolecular geneticsmedicineGeneticsHumansGeneBiology030304 developmental biologyAgedPopulation BiologyHaplotypelcsh:RComputational BiologyMicroRNAsCase-Control StudiesLeukocytes MononuclearLinear ModelsGenetic Polymorphismlcsh:QTranscriptomeGenome Expression Analysis030217 neurology & neurosurgeryPopulation GeneticsGenome-Wide Association StudyPLoS ONE
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Exploring by whole exome sequencing patients with initial diagnosis of Rubinstein-Taybi syndrome: the interconnections of epigenetic machinery disord…

2019

Rubinstein–Taybi syndrome (RSTS) is an autosomal-dominant neurodevelopmental disease affecting 1:125,000 newborns characterized by intellectual disability, growth retardation, facial dysmorphisms and skeletal abnormalities. RSTS is caused by mutations in genes encoding for writers of the epigenetic machinery: CREBBP (~ 60%) or its homologous EP300 (~ 10%). No causative mutation is identified in up to 30% of patients. We performed whole-exome sequencing (WES) on eight RSTS-like individuals who had normal high-resolution array CGH testing and were CREBBP- and EP300-mutation -negative, to identify the molecular cause. In four cases, we identified putatively causal variants in three genes (ASXL…

MaleGenetic Association StudieCompound heterozygosityWhole Exome SequencingArticleEpigenesis Genetic03 medical and health scienceswhole exome sequencing Rubinstein–Taybi syndrome epigenetic mutationsExome SequencingGeneticsmedicineHumansEpigeneticsEP300ChildGenetics (clinical)Exome sequencingGenetic Association Studies030304 developmental biologyGeneticsRubinstein-Taybi Syndrome0303 health sciencesComparative Genomic HybridizationbiologyRubinstein–Taybi syndrome030305 genetics & heredityInfant NewbornFaciesInfantmedicine.diseaseFacieCREB-Binding ProteinHuman geneticsRSTSKMT2APhenotypeChild PreschoolMutationbiology.proteinNeurodegenerative disordersFemaleHaploinsufficiencyE1A-Associated p300 ProteinHumanHuman genetics
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Novel loci for childhood body mass index and shared heritability with adult cardiometabolic traits

2020

The genetic background of childhood body mass index (BMI), and the extent to which the well-known associations of childhood BMI with adult diseases are explained by shared genetic factors, are largely unknown. We performed a genome-wide association study meta-analysis of BMI in 61,111 children aged between 2 and 10 years. Twenty-five independent loci reached genome-wide significance in the combined discovery and replication analyses. Two of these, located nearNEDD4LandSLC45A3, have not previously been reported in relation to either childhood or adult BMI. Positive genetic correlations of childhood BMI with birth weight and adult BMI, waist-to-hip ratio, diastolic blood pressure and type 2 d…

MaleGenetic-variationNedd4 Ubiquitin Protein LigasesPooled AnalysisBlood Pressure0302 clinical medicineHuman geneticsMendelian RandomizationYoung adultChildhealth care economics and organizationsBody mass indexAdiposityGenetics & Heredity0303 health sciencesStatistics1184 Genetics developmental biology physiologyGenomicsadulto3. Good healthCardiovascular DiseasesChild PreschoolPhysical SciencesMenarchegenetic-variationpresión sanguíneaMonosaccharide Transport ProteinsGenetic locieducationenfermedades cardiovascularesProstate-specific AntigenGenetic correlation03 medical and health sciencesSDG 3 - Good Health and Well-beingDiabetes MellitusGeneticsHumansprostate-specific antigenStatistical MethodsMolecular BiologyEcology Evolution Behavior and Systematicschildhood0604 Genetics[SDV.GEN]Life Sciences [q-bio]/GeneticsScience & TechnologyEarly Growth Genetics ConsortiumBiology and Life SciencesComputational Biologynutritional and metabolic diseasesSingle nucleotide polymorphismsMendelian Randomization AnalysisBiological TissueDiabetes Mellitus Type 2estudio de asociación genómica completagenetic factorsmendelian randomizationanálisis de la aleatorización mendelianaproteínas de transporte de monosacáridosBody mass index030217 neurology & neurosurgeryMathematicsDemographyDevelopmental BiologyCardiovascular RiskCancer ResearchobesityPhysiologyhumanosadolescenteOverweightQH426-470Genome-wide association studiesWaist–hip ratioMathematical and Statistical TechniquesMedicine and Health Sciencesbody mass index (BMI)Genetics of diseaseGenetics (clinical)2. Zero hungeradiposityMetaanalysisPhysiological ParametersConnective Tissue/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemalemedicine.symptomAnatomypooled analysisLife Sciences & BiomedicineResearch ArticleAdultcardiovascular riskAdolescentBirth weightmenarquiaAdipose tissueBiology3121 Internal medicineResearch and Analysis MethodsmedicineoverweightGenetic Predisposition to DiseaseObesity030304 developmental biologyMenarcheWaist-Hip Ratioíndice de masa corporalBody WeightCardiometabolic Risk Factorspredisposición genética a la enfermedadHeritabilityOverweightGenome Analysisyoung-adultsGenome-wide Associationíndice cintura-caderaYoung-adultsgenome-wide associationGenome-Wide Association StudyPLoS genetics
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A high-density SNP linkage scan with 142 combined subtype ADHD sib pairs identifies linkage regions on chromosomes 9 and 16

2008

As part of the International Multi-centre ADHD Gene (IMAGE) project we have completed an affected sibling pair study of 142 narrowly defined DSM-IV combined type ADHD proband-sibling pairs. We found suggestive linkage on chromosomes 9 and 16 with non-parametric multipoint peak LOD scores of 2.13 and 3.1 respectively. There have been several previous ADHD linkage scans. The UCLA study (Fisher et al. 2002; Ogdie et al. 2004; Ogdie et al. 2003), the Dutch study (Bakker et al. 2003), the German study (Hebebrand et al. 2006) and the MGH Study (Faraone et al., submitted) applied the affected sib pair (ASP) strategy; the Columbian study used extended pedigrees ascertained from a population isolate…

MaleGenetics and epigenetic pathways of disease [NCMLS 6]GENOMEWIDE SCANMedizin2804 Cellular and Molecular NeuroscienceCHILDRENComorbidityNeuroinformatics [DCN 3]Severity of Illness IndexDevelopmental psychology2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]HETEROGENEITYIsraelChildGeneticsObserver Variation0303 health sciencesATTENTION-DEFICIT/HYPERACTIVITY DISORDERPSYCHIATRIC-DISORDERSDOPAMINE TRANSPORTER GENEASSOCIATION10058 Department of Child and Adolescent PsychiatryEuropePsychiatry and Mental healthFemalePsychologyChromosomes Human Pair 9linkageFunctional Neurogenomics [DCN 2]GenotypeDEFICIT HYPERACTIVITY DISORDER610 Medicine & healthPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismWhite PeopleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]Genetic linkage1312 Molecular BiologymedicineSNPAttention deficit hyperactivity disorderHumansADHDSiblingMolecular Biology030304 developmental biologyLinkage (software)SiblingsChromosomemedicine.diseaseSib pairsUnited Statesaffected sib pairsGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with HyperactivityCONDUCT DISORDERLod ScoreDISEQUILIBRIUM030217 neurology & neurosurgeryChromosomes Human Pair 16
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Sex-specific association of the ST8SIAII gene with schizophrenia in a Spanish population

2013

We investigated the association between ST8SIAII and schizophrenia in a sample of Spanish origin. We found that the G allele (P=0.044) and the AG genotype (P=0.040) of rs3759916 were associated in females. The ACAG haplotype (rs3759914, rs3759915, rs3759916 and rs2305561) was associated in males (P=0.028).

MaleGenotypeBiologyPolymorphism Single NucleotideWhite PeopleSex FactorsGenotypemedicineHumansGenetic Predisposition to DiseaseAllelePromoter Regions GeneticAssociation (psychology)GeneAllelesBiological PsychiatryGeneticsHaplotypemedicine.diseaseSex specificSialyltransferasesSpanish populationPsychiatry and Mental healthHaplotypesSpainSchizophreniaSchizophreniaFemalePsychiatry Research
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Interstitial deletion of chromosome 2p15-16.1: Report of two patients and critical review of current genotype–phenotype correlation

2011

Abstract We report two individuals with developmental delay and dysmorphic features, in whom array-based comparative genomic hybridization (array CGH) led to the identification of a 2p15p16.1 de novo deletion. In the first patient (Patient 1) a familial deletion of 6q12, inherited from her father, was also detected. In the second patient (Patient 2) in addition to the 2p15p16.1 microdeletion a de novo deletion in Xq28 was detected. Both individuals shared dysmorphic features and developmental delay with the six reported patients with a 2p15p16.1 microdeletion described in medical literature. Conclusion: in the first patient a 642 kb 2p16.1 deletion (from 60.604 to 61.246 Mb), and a 930 kb 6…

MaleGenotypeDevelopmental delayDevelopmental DisabilitiesBioinformaticsContiguous gene syndromeGenotype phenotypeCorrelationGeneticsHumansChromosomal delectionMedicineAbnormalities MultipleClinical phenotypeGenetic Association StudiesIn Situ Hybridization FluorescenceSex Chromosome AberrationsGenetics (clinical)Sequence DeletionGeneticsChromosomes Human XComparative Genomic Hybridizationbusiness.industryInfantChromosomeSyndromeGeneral MedicineMicrodeletion syndromemedicine.diseaseXq28PhenotypeChild PreschoolChromosomes Human Pair 2FemaleChromosome DeletionbusinessComparative genomic hybridizationEuropean Journal of Medical Genetics
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Genotyping of sex hormone-related pathways in benign and malignant human prostate tissues: data of a preliminary study.

2011

Prostate cancer (PCa) is a major health issue in Westernized countries, representing a common cause of morbidity and mortality in the elderly male population. Endogenous sex steroids, along with environmental factors (notably diet) and host immune and inflammatory responses, are likely to cooperate in the pathogenesis of the disease. Based on the assumption that a complex endocrine–inflammatory-immune interaction is primarily implicated in human PCa, we have investigated the interplay between sex steroids and inflammation in development and growth of human PCa. To this end, we have assessed nine functional single nucleotide polymorphisms (SNP)s of five genes involved in sex hormone-related …

MaleGenotypeProstatic HyperplasiaSingle-nucleotide polymorphismDiseaseBioinformaticsBiochemistryPolymorphism Single NucleotideCohort StudiesProstate cancerSex hormone-binding globulinAromataseprostate cancer sex hormone related pathways3-Oxo-5-alpha-Steroid 4-DehydrogenaseGene FrequencyGeneticsmedicineSNPHumansMolecular BiologyAllele frequencyGenotypingSicilyGenetic Association StudiesAgedSettore MED/04 - Patologia GeneraleAged 80 and overbiologyCase-control studyMembrane ProteinsProstatic NeoplasmsSequence Analysis DNAMiddle Agedmedicine.diseaseReceptors EstrogenReceptors AndrogenCase-Control StudiesImmunologybiology.proteinMolecular MedicineBiotechnologyOmics : a journal of integrative biology
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No Major Host Genetic Risk Factor Contributed to A(H1N1)2009 Influenza Severity

2015

Garcia-Etxebarria, Koldo et al.

MaleGenotypeRisk factors in diseaseslcsh:MedicineGenome-wide association studySingle-nucleotide polymorphismDiseaseBiologyBioinformaticsmedicine.disease_causePolymorphism Single NucleotideSeverity of Illness IndexGripInfluenza A Virus H1N1 SubtypeGene FrequencyRisk FactorsGenotypeSeverity of illnessInfluenza HumanInfluenza A virusmedicineSNPHumanslcsh:ScienceAllele frequencyMultidisciplinaryFactors de risc en les malaltieslcsh:RGenomicsInfluenzaGenòmicaEstudi de casosRNA ViralRNAFemalelcsh:QCase studiesResearch Article
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Large-scale replication and heterogeneity in Parkinson disease genetic loci

2012

Objective: Eleven genetic loci have reached genome-wide significance in a recent meta-analysis of genome-wide association studies in Parkinson disease (PD) based on populations of Caucasian descent. The extent to which these genetic effects are consistent across different populations is unknown. Methods: Investigators from the Genetic Epidemiology of Parkinson9s Disease Consortium were invited to participate in the study. A total of 11 SNPs were genotyped in 8,750 cases and 8,955 controls. Fixed as well as random effects models were used to provide the summary risk estimates for these variants. We evaluated between-study heterogeneity and heterogeneity between populations of different ances…

MaleGenotypeSingle-nucleotide polymorphismGenome-wide association studyCase-control studiesBiologyPolymorphism Single NucleotideGene Frequencygenetics [Parkinson Disease]HumansGenetic Predisposition to Diseaseddc:610AlleleParkinson Disease/geneticsAllele frequencyAllelesGenetic associationAgedGeneticsMedicine(all)Case-control studyParkinson DiseaseOdds ratioMiddle Agedddc:616.8Genetic epidemiologyGenetic LociCase-Control StudiesFemaleNeurology (clinical)Human medicineGenome-Wide Association Study
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