Search results for "Ataxia"

showing 10 items of 150 documents

A bi-allelic loss-of-function SARS1 variant in children with neurodevelopmental delay, deafness, cardiomyopathy, and decompensation during fever

2021

Aminoacyl-tRNA synthetases (aaRS) are ubiquitously expressed enzymes responsible for ligating amino acids to their cognate tRNA molecules through an aminoacylation reaction. The resulting aminoacyl-tRNA is delivered to ribosome elongation factors to participate in protein synthesis. Seryl-tRNA synthetase (SARS1) is one of the cytosolic aaRSs and catalyzes serine attachment to tRNASer . SARS1 deficiency has already been associated with moderate intellectual disability, ataxia, muscle weakness, and seizure in one family. We describe here a new clinical presentation including developmental delay, central deafness, cardiomyopathy, and metabolic decompensation during fever leading to death, in a…

AtaxiabrainCardiomyopathySARS1Loss of HeterozygosityBiologyAmino Acyl-tRNA Synthetaseschemistry.chemical_compounddeafnessdeathGeneticsmedicineProtein biosynthesisMissense mutationHumansDecompensationaminoacyl-tRNA synthetaseChildtRNAGenetics (clinical)GeneticsaminoacylationAminoacyl tRNA synthetasemedicine.diseaseElongation factorchemistryintellectual disabilityTransfer RNAmedicine.symptomCardiomyopathiesHuman mutation
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Senataxin defective in ataxia oculomotor apraxia type 2 is involved in the defence against oxidative DNA damage

2007

Adefective response to DNA damage is observed in several human autosomal recessive ataxias with oculomotor apraxia, including ataxia-telangiectasia. We report that senataxin, defective in ataxia oculomotor apraxia (AOA) type 2, is a nuclear protein involved in the DNA damage response. AOA2 cells are sensitive to H2O2, camptothecin, and mitomycin C, but not to ionizing radiation, and sensitivity was rescued with full-length SETX cDNA. AOA2 cells exhibited constitutive oxidative DNA damage and enhanced chromosomal instability in response to H2O2. Rejoining of H2O2-induced DNA double-strand breaks (DSBs) was significantly reduced in AOA2 cells compared to controls, and there was no evidence fo…

Ataxiagenetic structuresDNA RepairDNA damageApraxiasBiologyArticlechemistry.chemical_compoundComplementary DNAChromosome instabilitymedicineHumansDNA Breaks Double-StrandedOculomotor apraxiaCells CulturedResearch ArticlesNeurodegenerationMitomycin CDNA HelicasesCell BiologyHydrogen Peroxidemedicine.diseaseMolecular biologyMultifunctional EnzymesOxidative StresschemistryAtaxiamedicine.symptomDNARNA HelicasesDNA Damage
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Sudden sensorineural hearing loss as prodromal symptom of anterior inferior cerebellar artery infarction.

2011

Sudden sensorineural hearing loss is a clinical condition characterized by a sudden onset of unilateral or bilateral hearing loss. In recent years sudden deafness has been frequently described in association with anterior inferior cerebellar artery (AICA) infarction generally presenting along with other brainstem and cerebellar signs such as ataxia, dysmetria and peripheral facial palsy. The authors report a rare clinical case of a 53-year-old man who suddenly developed hearing loss and tinnitus without any brainstem or cerebellar signs. Computed tomography of his brain was normal, and the audiological results localized the lesion causing deafness to the inner ear. Surprisingly, magnetic re…

Brain InfarctionMalemedicine.medical_specialtyAnterior inferior cerebellar artery infarctionAtaxiaHearing lossHearing Loss SensorineuralInfarctionLesionSudden deafneCerebellar DiseasesInternal auditory arteryDysmetriamedicine.arteryInternal medicineCerebellumInner earotorhinolaryngologic diseasesmedicineVertebrobasilar InsufficiencyHumansbusiness.industrySettore MED/37 - NeuroradiologiaMiddle Agedmedicine.diseaseMagnetic Resonance ImagingSudden deafness; Anterior inferior cerebellar artery infarction; Internal auditory artery; Inner earSettore MED/32 - AudiologiaSurgeryAnterior inferior cerebellar arterymedicine.anatomical_structureOtorhinolaryngologyEar InnerAcute DiseaseCardiologySettore MED/26 - Neurologiamedicine.symptomCerebellar arterybusinessTinnitusORL; journal for oto-rhino-laryngology and its related specialties
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MYCN sensitizes human neuroblastoma to apoptosis by HIPK2 activation through a DNA damage response.

2010

Abstract MYCN amplification occurs in approximately 20% of human neuroblastomas and is associated with early tumor progression and poor outcome, despite intensive multimodal treatment. However, MYCN overexpression also sensitizes neuroblastoma cells to apoptosis. Thus, uncovering the molecular mechanisms linking MYCN to apoptosis might contribute to designing more efficient therapies for MYCN-amplified tumors. Here we show that MYCN-dependent sensitization to apoptosis requires activation of p53 and its phosphorylation at serine 46. The p53S46 kinase HIPK2 accumulates on MYCN expression, and its depletion by RNA interference impairs p53S46 phosphorylation and apoptosis. Remarkably, MYCN ind…

Cancer ResearchApoptosisCell Cycle ProteinsAtaxia Telangiectasia Mutated ProteinsProtein-Serine-Threonine KinaseAtaxia Telangiectasia Mutated ProteinNeuroblastomaCell Cycle ProteinSerinePhosphorylationNuclear ProteinOncogene Proteinseducation.field_of_studyN-Myc Proto-Oncogene ProteinAntibiotics AntineoplasticKinaseOncogene ProteinNuclear ProteinsDNA-Binding ProteinsOncologyPhosphorylationRNA InterferenceHumanDNA damageDNA-Binding ProteinPopulationBlotting WesternBiologyProtein Serine-Threonine KinasesN-Myc Proto-Oncogene ProteinBleomycinNeuroblastomaCell Line TumormedicineHumanseducationneoplasmsMolecular BiologyTumor Suppressor ProteinTumor Suppressor ProteinsApoptosimedicine.diseaseTumor progressionApoptosisMutationCancer researchTumor Suppressor Protein p53Carrier ProteinCarrier ProteinsDNA DamageMolecular cancer research : MCR
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Artesunate induces oxidative DNA damage, sustained DNA double-strand breaks, and the ATM/ATR damage response in cancer cells.

2011

Abstract Artesunate, the active agent from Artemisia annua L. used in the traditional Chinese medicine, is being applied as a first-line drug for malaria treatment, and trials are ongoing that include this drug in cancer therapy. Despite increasing interest in its therapeutic application, the mode of cell killing provoked by artesunate in human cells is unknown. Here, we show that artesunate is a powerful inducer of oxidative DNA damage, giving rise to formamidopyrimidine DNA glycosylase–sensitive sites and the formation of 8-oxoguanine and 1,N6-ethenoadenine. Oxidative DNA damage was induced in LN-229 human glioblastoma cells dose dependently and was paralleled by cell death executed by ap…

Cancer ResearchProgrammed cell deathDNA RepairRAD51Drug Evaluation PreclinicalArtesunateApoptosisCell Cycle ProteinsAtaxia Telangiectasia Mutated ProteinsBiologyProtein Serine-Threonine KinasesModels Biologicalchemistry.chemical_compoundNeoplasmsTumor Cells CulturedHumansDNA Breaks Double-StrandedTumor Suppressor ProteinsMolecular biologyAntineoplastic Agents PhytogenicArtemisininsUp-RegulationNon-homologous end joiningDNA-Binding ProteinsOxidative StressCell killingOncologychemistryArtesunateApoptosisCancer cellHomologous recombinationDNA DamageMolecular cancer therapeutics
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Genomic portrait of a sporadic amyotrophic lateral sclerosis case in a large spinocerebellar ataxia type 1 family

2020

Background: Repeat expansions in the spinocerebellar ataxia type 1 (SCA1) gene ATXN1 increases the risk for amyotrophic lateral sclerosis (ALS), supporting a relationship between these disorders. We recently reported the co-existence, in a large SCA1 family, of a clinically definite ALS individual bearing an intermediate ATXN1 expansion and SCA1 patients with a full expansion, some of which manifested signs of lower motor neuron involvement. Methods: In this study, we employed a systems biology approach that integrated multiple genomic analyses of the ALS patient and some SCA1 family members. Results: Our analysis identified common and distinctive candidate genes/variants and related biolog…

Candidate geneSpinocerebellar Ataxia Type 1Medicine (miscellaneous)lcsh:MedicineNetworkBiologyArticle03 medical and health sciences0302 clinical medicinemulti-omics; networkC9orf72medicineCustomized aCGHAmyotrophic lateral sclerosisGene030304 developmental biologyTAF15Genetics0303 health sciencesMulti-omicslcsh:Rmedicine.diseaseAmyotrophic lateral sclerosisPhenotypeSCA1-MNNGSSpinocerebellar ataxiaSpinocerebellar ataxia030217 neurology & neurosurgeryPathway
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Rare cardiomyopathies: Diagnostic features

2013

Cardiomyopathies (CM) are an important and heterogeneous group of diseases affecting the myocardium. They can induce mechanical and/or electrical disorders and are due to a variety of causes, they frequently are genetic. However, since their high number and their clinical complexity, the identification is still a challenge. Echocardiography is a very useful tool in the assessment of CM. In this review we aim to define the typical clinical features and to discuss the main diagnostic tool, above all echocardiography that can help physicians in the correct assessment of CM.

Cardiomyopathy RestrictiveCardiomyopathyRestrictiveReproducibility of ResultsSettore MED/11 - Malattie Dell'Apparato CardiovascolareSensitivity and Specificitycardiomyopahies echocardiographyDiagnosis DifferentialArrhythmogenic Right Ventricular Dysplasia; Cardiomyopathies; Cardiomyopathy Restrictive; Diagnosis Differential; Fabry Disease; Friedreich Ataxia; Humans; Predictive Value of Tests; Reproducibility of Results; Risk Factors; Sensitivity and Specificity; Takotsubo Cardiomyopathy; EchocardiographyFriedreich AtaxiaPredictive Value of TestsRisk FactorsTakotsubo CardiomyopathyEchocardiographyDiagnosisDifferentialFabry DiseaseHumansCardiomyopathiesArrhythmogenic right ventricular dysplasia; Cardiomyopathy restrictive; Fabry diseaseArrhythmogenic Right Ventricular Dysplasia
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Glutamate Excitotoxicity in the Cerebellum Mediated by IL-1β

2013

Multiple sclerosis (MS) is the prototypic inflammatory demyelinating disorder of the CNS. Symptoms of cerebellar dysfunction, such as tremors and ataxia, are relatively common in MS, but available treatment options are generally of limited value. Although many clinical manifestations of MS are

CerebellumAtaxiabusiness.industryGeneral NeuroscienceMultiple sclerosisEncephalomyelitisExcitotoxicityGlutamate receptorTreatment optionsmedicine.disease_causemedicine.diseasemedicine.anatomical_structureImmunologymedicinemedicine.symptomDemyelinating DisorderbusinessThe Journal of Neuroscience
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Effects of Neuromodulation on Gait

2018

In the last decades, non-invasive brain stimulation (NIBS) has evolved to become a valuable tool in both basic and clinical neuroscience. Various methods of transcranial magnetic stimulation (TMS) and transcranial electrical stimulation (TES) have been widely used for diagnostic, prognostic and even therapeutic applications in a broad range of neurological and psychiatric disorders. The rationale for using NIBS techniques lies in the possibility to modulate, in a targeted manner, the activity of different cerebral and cerebellar cortical regions, as well as the functional connections between these areas and distant brain regions also including subcortical structures. The neural circuitry in…

CerebellumCerebellar ataxiaClinical neurosciencebusiness.industrymedicine.medical_treatmentmedicine.diseaseNeuromodulation (medicine)Transcranial magnetic stimulationGait (human)medicine.anatomical_structureNIBS TMS rTMS tES balance gaitBrain stimulationmedicineSettore MED/26 - Neurologiamedicine.symptombusinessSpinal cord injuryNeuroscience
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Degeneration of the Cerebellum in Huntington's Disease (HD): Possible Relevance for the Clinical Picture and Potential Gateway to Pathological Mechan…

2012

Huntington's disease (HD) is a polyglutamine disease and characterized neuropathologically by degeneration of the striatum and select layers of the neo- and allocortex. In the present study, we performed a systematic investigation of the cerebellum in eight clinically diagnosed and genetically confirmed HD patients. The cerebellum of all HD patients showed a considerable atrophy, as well as a consistent loss of Purkinje cells and nerve cells of the fastigial, globose, emboliform and dentate nuclei. This pathology was obvious already in HD brains assigned Vonsattel grade 2 striatal atrophy and did not correlate with the extent and distribution of striatal atrophy. Therefore, our findings sug…

CerebellumPathologymedicine.medical_specialtyAtaxiaGeneral NeuroscienceNeurodegenerationmedicine.diseasePathology and Forensic MedicinePathogenesisDysarthriamedicine.anatomical_structureAtrophynervous systemHuntington's diseasemedicineCorticobasal degenerationNeurology (clinical)medicine.symptomPsychologyNeuroscienceBrain Pathology
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