Search results for "Autoimmune"

showing 10 items of 648 documents

Family studies in scleroderma (systemic sclerosis) demonstrating an HLA-linked increased chromosomal breakage rate in cultured lymphocytes

1988

An increased chromosomal breakage rate (ICBR) was found in 27 of 28 patients with scleroderma (systemic sclerosis, SS) - 5 with the syndrome including calcinosis cutis, Raynaud phenomenon, esophagus hypomotility, sclerodactyly and telangiectasia (CREST), 4 incomplete CREST, 1 overlapping syndrome, 18 progressive systemic sclerosis (PSS). Not only the patients, but also about half of their first-degree relatives showed an increased chromosomal breakage rate (more than 5 breaks per 100 metaphases). This character segregated as a dominant marker in nine families of scleroderma patients. In the six informative of the nine families, the ICBR trait showed close linkage with the HLA region on chro…

Genetic MarkersMaleSystemic diseaseGenetic LinkageHuman leukocyte antigenBiologySclerodermaCalcinosis cutisHLA AntigensGeneticsmedicineHumansLymphocytesCells CulturedGenetics (clinical)Chromosome AberrationsAutoimmune diseaseScleroderma SystemicSclerodactylyChromosome Fragilitymedicine.diseaseConnective tissue diseasePedigreeHaplotypesImmunologyFemalemedicine.symptomHuman Genetics
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Impairment of TGF-β signaling in T cells increases susceptibility to experimental autoimmune hepatitis in mice

2002

In autoimmune hepatitis, strong TGF-beta1 expression is found in the inflamed liver. TGF-beta overexpression may be part of a regulatory immune response attempting to suppress autoreactive T cells. To test this hypothesis, we determined whether impairment of TGF-beta signaling in T cells leads to increased susceptibility to experimental autoimmune hepatitis (EAH). Transgenic mice of strain FVB/N were generated expressing a dominant-negative TGF-beta type II receptor in T cells under the control of the human CD2 promoter/locus control region. On induction of EAH, transgenic mice showed markedly increased portal and periportal leukocytic infiltrations with hepatocellular necroses compared wit…

Genetically modified mouseCellular immunityPhysiologyT-LymphocytesEnzyme-Linked Immunosorbent AssayMice TransgenicAutoimmune hepatitisBiologyMiceImmune systemTransforming Growth Factor betaPhysiology (medical)medicineAnimalsHomeostasisCells CulturedAutoimmune diseaseHepatitisB-LymphocytesHepatologyGastroenterologyT lymphocyteBlotting Northernmedicine.diseaseImmunohistochemistryHepatitis AutoimmunePhenotypeNeutrophil InfiltrationImmunologySignal transductionSignal TransductionAmerican Journal of Physiology-Gastrointestinal and Liver Physiology
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A framework for remission in SLE

2017

ObjectivesTreat-to-target recommendations have identified ‘remission’ as a target in systemic lupus erythematosus (SLE), but recognise that there is no universally accepted definition for this. Therefore, we initiated a process to achieve consensus on potential definitions for remission in SLE.MethodsAn international task force of 60 specialists and patient representatives participated in preparatory exercises, a face-to-face meeting and follow-up electronic voting. The level for agreement was set at 90%.ResultsThe task force agreed on eight key statements regarding remission in SLE and three principles to guide the further development of remission definitions:1. Definitions of remission wi…

Genetics and Molecular Biology (all)PediatricsAutoimmune diseasesNEPHRITIS PATIENTSDISEASE-ACTIVITYSeverity of Illness IndexBiochemistryRETROSPECTIVE ANALYSIS0302 clinical medicineQuality of lifePrednisoneAdrenal Cortex HormonesLupus Erythematosus SystemicImmunology and AllergyCHINESE PATIENTS030212 general & internal medicineSYSTEMIC-LUPUS-ERYTHEMATOSUSskin and connective tissue diseasesPREDICTORSOUTCOMESSystemic lupus erythematosusMalalties autoimmunitàriesRemission InductionSYSTEMIC-LUPUS-ERYTHEMATOSUS; DISEASE-ACTIVITY; RETROSPECTIVE ANALYSIS; INITIAL VALIDATION; NEPHRITIS PATIENTS; AMERICAN-COLLEGE; CHINESE PATIENTS; RENAL FLARES; PREDICTORS; OUTCOMESSymptom Flare UpConnective tissue diseaseManchester Institute for Collaborative Research on AgeingEstudi de casosOutcomes researchAntibodies AntinuclearDNA/immunologyImmunosuppressive Agentsmedicine.drugmedicine.medical_specialtyFarmacologiaResearchInstitutes_Networks_Beacons/MICRAConsensusImmunologyAdrenal Cortex Hormones/therapeutic useAMERICAN-COLLEGELupus Erythematosus Systemic/bloodSystemic Lupus ErythematosusGeneral Biochemistry Genetics and Molecular BiologyMaintenance Chemotherapy03 medical and health sciencesAntimalarialsRheumatologySeverity of illnessmedicineDisease Activity; Outcomes research; Systemic Lupus Erythematosus; Immunology and Allergy; Rheumatology; Immunology; Biochemistry Genetics and Molecular Biology (all)HumansDisease Activity030203 arthritis & rheumatologyPharmacologyAntibodies Antinuclear/bloodLupus erythematosusbusiness.industryTask forceConstruct validityRENAL FLARESComplement System ProteinsDNAINITIAL VALIDATIONDisease Activity; Outcomes research; Systemic Lupus Erythematosusmedicine.diseaseLupus eritematósAntimalarials/therapeutic usePhysical therapyImmunosuppressive Agents/therapeutic useComplement System Proteins/metabolismCase studiesOutcomes researchbusinessAnnals of the Rheumatic Diseases
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Genetics of Polyglandular Failure

2010

Publisher Summary This chapter discusses the genetic aspects of polyglandular failure syndromes (APS).The polyglandular failure syndromes comprise of a wide spectrum of autoimmune disorders and encompass a rare juvenile type (APS1) and more frequent adult types (APS2 and APS3). The juvenile type APS1 is also known as autoimmune polyendocrinopathy candidiasis ectodermal dystrophy because it consists of three main disorders, namely chronic mucocutaneous candidiasis, autoimmune hypoparathyroidism and autoimmune Addison's disease. APS2 is defined as the association between Addison's disease and either autoimmune thyroid disease or type 1 diabetes mellitus and APS3 is characterized by the presen…

GeneticsType 1 diabetesendocrine system diseasesmedicine.diagnostic_testAutoimmune polyendocrinopathyDiseaseBiologymedicine.diseasePathophysiologyHypoparathyroidismImmunologymedicineChronic mucocutaneous candidiasisChromosome 21Genetic testing
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Differences between the Glycosylation Patterns of Haptoglobin Isolated from Skin Scales and Plasma of Psoriatic Patients

2012

Improved diagnosis of psoriasis, by new biomarkers, is required for evaluating the progression rate of the disease and the response to treatment. Haptoglobin (Hpt), a glycoprotein secreted by hepatocytes and other types of cells including keratinocytes, was found with glycan changes in psoriasis and other diseases. We previously reported that Hpt isolated from plasma of psoriatic patients is more fucosylated than Hpt of healthy subjects. The aim of this study was to compare the glycosylation pattern of Hpt isolated from skin scales or plasma of patients with psoriasis with that of Hpt from cornified epidermal layer or plasma of healthy subjects. High performance liquid chromatography analys…

GlycosylationGlycobiologylcsh:MedicineDermatologic PathologyBiochemistryMass SpectrometryFucosechemistry.chemical_compoundLectinsBlood plasmalcsh:ScienceChromatography High Pressure LiquidFucosylationchemistry.chemical_classificationglycanMultidisciplinarybiologyHaptoglobinGlycopeptideshaptoglobinMedicineResearch ArticleAdultskinProtein Structuremedicine.medical_specialtyGlycanGlycosylationInflammatory DiseasesImmunologyDermatologyAutoimmune DiseasesPolysaccharidesInternal medicinePsoriasismedicinePsoriasisHumansAmino Acid SequenceImmunoassaysBiologyGlycoproteinsHaptoglobinslcsh:RProteinsmedicine.diseasecarbohydrates (lipids)EndocrinologychemistryImmunologyImmunologic Techniquesbiology.proteinglycanslectinClinical Immunologylcsh:QGlycoproteinBiomarkers
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Leflunomide (HWA 486), a novel immunomodulating compound for the treatment of autoimmune disorders and reactions leading to transplantation rejection.

1991

Leflunomide has been shown to be very effective in preventing and curing several autoimmune animal diseases. Further, this agent is as effective as cyclosporin A in preventing the rejection of skin and kidney transplants in rats. Preliminary results from patients suffering from severe cases of rheumatoid arthritis demonstrated that clinical and immunological parameters could be improved with leflunomide therapy. Mode of action studies revealed that this substance antagonizes the proliferation inducing activity of several cytokines and is cytostatic for certain cell types. In this light, we could show that tyrosine phosphorylation of the RR-SRC peptide substrate and the autophosphorylation o…

Graft RejectionImmunologyMolecular Sequence DataGraft vs Host DiseasePharmacologyToxicologyAutoimmune Diseaseschemistry.chemical_compoundEpidermal growth factorCyclosporin amedicineAnimalsHumansPharmacology (medical)Amino Acid SequenceMode of actionLeflunomidePharmacologybusiness.industryAnti-Inflammatory Agents Non-SteroidalTyrosine phosphorylationIsoxazolesmedicine.diseaseTransplantationDisease Models AnimalchemistryRheumatoid arthritisImmunologybusinessTyrosine kinaseImmunosuppressive AgentsLeflunomidemedicine.drugAgents and actions
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Effects of leflunomide on immune responses and models of inflammation.

1993

Leflunomide is an antiphlogistic and immunomodulating agent that has been shown to be effective in preventing and healing autoimmune disorders and reactions leading to organ graft rejection. From our preliminary clinical data [4], we now have hopes that these effects, observed in experimental animals, can truly be transferred to humans. Although we are far from understanding the mode of action of leflunomide, we are slowly gathering some insight. A good many of the immunosuppressive effects of leflunomide can be attributed to the antagonistic effects it has on responses to many cytokines, most likely through receptor expression and signal transduction (tyrosine kinase inhibition). The inhib…

Graft RejectionReceptor expressionImmunologyDrug Evaluation PreclinicalAutoimmune Diseaseschemistry.chemical_compoundMiceImmune systemMedicineAnimalsHumansLeflunomideInflammationImmunity Cellularbusiness.industryAnti-Inflammatory Agents Non-SteroidalAutoantibodyGeneral MedicineIsoxazolesProtein-Tyrosine KinasesRatsDisease Models AnimalchemistryImmunologyAntibody FormationCytokinesSignal transductionbusinessTyrosine kinaseImmunomodulating AgentHistamineImmunosuppressive AgentsLeflunomidemedicine.drugSpringer seminars in immunopathology
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Europäische Leitlinien (S1) für die Anwendung von hochdosierten intravenösen Immunglobulinen in der Dermatologie.

2017

Hintergrund und Ziele Die Behandlung schwerer dermatologischer Autoimmunerkrankungen und der toxischen epidermalen Nekrolyse (TEN) mit hochdosierten intravenosen Immunglobulinen (IVIg) ist ein bewahrtes therapeutisches Verfahren in der Dermatologie. Da eine IVIg‐Therapie in der Regel nur bei seltenen Erkrankungen oder bei schweren Fallen in Betracht gezogen wird, stutzt sich die Anwendung von Immunglobulinen zumeist nicht auf Daten aus randomisierten kontrollierten Studien, wie sie in der evidenzbasierten Medizin erforderlich sind. Da Indikationen fur die Anwendung von IVIg selten sind, ist es unwahrscheinlich, dass solche Studien in absehbarer Zeit durchgefuhrt werden. Wegen der hohen Kost…

Gynecologymedicine.medical_specialtytherapyexpert consensusbusiness.industryDermatologyautoimmune diseases dosing therapy expert consensus decision treedosing030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisdecision treemedicineautoimmune diseasesbusinessJournal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG
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Immunological alterations in hepatitis C virus infection

2013

A higher prevalence of immunological processes has recently been reported in patients with hepatitis C virus (HCV) infection, focusing the attention of physicians and researchers on the close association between HCV and immune disorders. HCV lymphotropism represents the most important step in the pathogenesis of virus-related immunological diseases and experimental, virologic, and clinical evidence has demonstrated a trigger role for HCV both in systemic autoimmune diseases, such as rheumatoid arthritis, Sjogren syndrome, hemolytic anemia and severe thrombocytopenia, and in organ-specific autoimmune diseases, such as autoimmune hepatitis, thyroid disorders and diabetes. This review will out…

Hemolytic anemiaHepacivirusHepatitis C virusAutoimmunityAutoimmune hepatitisHepacivirusmedicine.disease_causeAutoimmunityAutoimmune DiseasesImmune systemmedicineAnimalsHumansTopic Highlightbiologybusiness.industryGastroenterologyAutoantibodyvirus diseasesGeneral MedicineHepatitis C Chronicmedicine.diseasebiology.organism_classificationVirologyHepatitis Cdigestive system diseasesRheumatoid arthritisImmunologybusiness
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Effectiveness of cyclosporine and mycophenolate mofetil in a child with refractory evans syndrome

2011

Evans Syndrome is a rare autoimmune disease consisting of hemolytic anemia, thrombocytopenia and/or neutropenia. It may be associated with other autoimmune or lymphoproliferative diseases. Its course can be extremely serious and, rarely, even life-threatening

Hemolytic anemiaVincristinePediatricsmedicine.medical_specialtyEvans syndromeoutcome.CyclophosphamideEvans’ syndrome Cyclosporine Mycophenolate mofetil Treatment Outcomelcsh:MedicineCase ReportNeutropeniaPediatricshemic and lymphatic diseasesmedicineevans syndromeOutcomeAutoimmune diseasebusiness.industryMycophenolate mofetillcsh:Rlcsh:RJ1-570lcsh:PediatricsEvans’ syndromemedicine.diseaseDiscontinuationTreatmentImmunologyCyclosporineRituximabbusinessmedicine.drugPediatric Reports
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