Search results for "Axia"

showing 10 items of 638 documents

Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

1996

International audience; Friedreich's ataxia (FRDA) is an autosomal recessive, degenerative disease that involves the central and peripheral nervous systems and the heart. A gene, X25, was identified in the critical region for the FRDA locus on chromosome 9q13. This gene encodes a 210-amino acid protein, frataxin, that has homologs in distant species such as Caenorhabditis elegans and yeast. A few FRDA patients were found to have point mutations in X25, but the majority were homozygous for an unstable GAA trinucleotide expansion in the first X25 intron.

MaleIron-sulfur cluster assemblyPolymerase Chain Reaction0302 clinical medicineTrinucleotide RepeatsIron-Binding ProteinsGenetics0303 health sciencesMultidisciplinaryAutosomal recessive cerebellar ataxiaPedigree3. Good healthFemalemedicine.symptomChromosomes Human Pair 9HumanPair 9Heterozygotecongenital hereditary and neonatal diseases and abnormalitiesAtaxiaMolecular Sequence DataGenes RecessiveLocus (genetics)BiologyChromosomes03 medical and health sciencesGene mappingAlleles; Amino Acid Sequence; Base Sequence; Chromosomes Human Pair 9; DNA Primers; Female; Friedreich Ataxia; Genes Recessive; Heterozygote; Humans; Male; Molecular Sequence Data; Pedigree; Point Mutation; Polymerase Chain Reaction; Proteins; Sequence Alignment; Introns; Iron-Binding Proteins; Trinucleotide RepeatsmedicineRecessiveHumansPoint MutationAmino Acid SequenceAlleleAllelesDNA Primers030304 developmental biologyBase SequencePoint mutationProteins[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologymedicine.diseaseMolecular biologyIntronsGenes[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsFriedreich AtaxiaFrataxinbiology.proteinSequence Alignment030217 neurology & neurosurgeryScience
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Comparison of three ultrasonic axial transmission methods for bone assessment.

2005

Abstract This study compared three approaches to bone assessment using ultrasonic axial transmission. In 41 fresh human radii, velocity of the first arriving signal was measured with a commercial device (Sunlight Omnisense ™ ) operating at 1.25 MHz, a prototype based on 1-MHz bidirectional axial transmission and a low-frequency (200 kHz) prototype, also measuring the velocity of a slower wave. Cortical and trabecular bone mineral density, cortical thickness and cross-sectional area were determined by peripheral quantitative computed tomography. Significant but modest correlation between velocities reflects differences in the nature of the propagating waves and methodological differences. Of…

MaleMaterials scienceAcoustics and UltrasonicsBiophysicsSignalBone and BonesBone DensitymedicineCadaverHumansRadiology Nuclear Medicine and imagingUltrasonicsQuantitative computed tomographyAxial transmissionAgedUltrasonographyAged 80 and overRadiological and Ultrasound Technologymedicine.diagnostic_testAnatomyMiddle AgedTrabecular boneRadiusmedicine.anatomical_structureMineral densityCortical boneUltrasonic sensorFemaleBiomedical engineeringUltrasound in medicinebiology
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Bone morphogenetic protein-4 is required for mesoderm formation and patterning in the mouse.

1995

Bone morphogenetic protein-4 (BMP-4) is a member of the TGF-beta superfamily of polypeptide signaling molecules, closely related to BMP-2 and to Drosophila decapentaplegic (DPP). To elucidate the role of BMP-4 in mouse development the gene has been inactivated by homologous recombination in ES cells. Homozygous mutant Bmp-4tm1blh embryos die between 6.5 and 9.5 days p.c., with a variable phenotype. Most Bmp-4tm1blh embryos do not proceed beyond the egg cylinder stage, do not express the mesodermal marker T(Brachyury), and show little or no mesodermal differentiation. Some homozygous mutants develop to the head fold or beating heart/early somite stage or beyond. However, they are development…

MaleMesodermBrachyuryHeterozygoteanimal structuresMolecular Sequence DataBiologyCell LineMesodermEmbryonic and Fetal DevelopmentMiceGeneticsmedicineParaxial mesodermAnimalsCrosses GeneticDecapentaplegicBase SequenceChimeraStem CellsHomozygoteProteinsGastrulaCell biologyMice Inbred C57BLmedicine.anatomical_structureBone morphogenetic protein 5PhenotypeBone morphogenetic protein 4GDF6embryonic structuresMesoderm formationBone Morphogenetic ProteinsGene TargetingFemaleDevelopmental BiologyGenesdevelopment
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A novel form of ataxia oculomotor apraxia characterized by oxidative stress and apoptosis resistance

2007

Several different autosomal recessive genetic disorders characterized by ataxia with oculomotor apraxia (AOA) have been identified with the unifying feature of defective DNA damage recognition and/or repair. We describe here the characterization of a novel form of AOA showing increased sensitivity to agents that cause single-strand breaks (SSBs) in DNA but having no gross defect in the repair of these breaks. Evidence for the presence of residual SSBs in DNA was provided by dramatically increased levels of poly (ADP-ribose)polymerase (PARP-1) auto-poly (ADP-ribosyl)ation, the detection of increased levels of reactive oxygen/nitrogen species (ROS/RNS) and oxidative damage to DNA in the patie…

MaleMethylnitronitrosoguanidineProgrammed cell deathAtaxiaDNA RepairApraxiasDNA damageMitomycinBlotting WesternPoly (ADP-Ribose) Polymerase-1Apoptosismedicine.disease_causeAntioxidantschemistry.chemical_compoundRadiation IonizingmedicineHumansDNA Breaks Single-StrandedOculomotor apraxiaMolecular BiologyCells CulturedEtoposideMembrane Potential MitochondrialbiologyCytochrome cHydrogen PeroxideCell BiologyFlow Cytometrymedicine.diseaseAntineoplastic Agents PhytogenicReactive Nitrogen SpeciesMolecular biologyOxidative StresschemistryApoptosisbiology.proteinAtaxiaCamptothecinFemalePoly(ADP-ribose) Polymerasesmedicine.symptomDNAOxidative stressDNA DamageCell Death & Differentiation
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Three-Dimensional Reconstruction of the Bony Nasolacrimal Canal by Automated Segmentation of Computed Tomography Images.

2016

Objective To apply a fully automated method to quantify the 3D structure of the bony nasolacrimal canal (NLC) from CT scans whereby the size and main morphometric characteristics of the canal can be determined. Design Cross-sectional study. Subjects 36 eyes of 18 healthy individuals. Methods Using software designed to detect the boundaries of the NLC on CT images, 36 NLC reconstructions were prepared. These reconstructions were then used to calculate NLC volume. The NLC axis in each case was determined according to a polygonal model and to 2nd, 3rd and 4th degree polynomials. From these models, NLC sectional areas and length were determined. For each variable, descriptive statistics and nor…

MaleModels AnatomicCritical Care and Emergency Medicinelcsh:MedicineComputed tomographyPolynomialsDiagnostic RadiologyNormality test0302 clinical medicineMedicine and Health SciencesSegmentationDegree of a polynomiallcsh:ScienceTomographyMusculoskeletal SystemTrauma MedicineMathematicsMultidisciplinaryNasolacrimal ductmedicine.diagnostic_testRadiology and ImagingAnatomyMiddle Agedmedicine.anatomical_structureSurgery Computer-AssistedPhysical SciencesNasolacrimal canalFemaleAnatomyResearch ArticleAdultComputer and Information SciencesImaging TechniquesTrauma SurgeryAutomated segmentationNeuroimagingSurgical and Invasive Medical ProceduresResearch and Analysis MethodsBone and BonesComputer Software03 medical and health sciencesImaging Three-DimensionalDiagnostic MedicinemedicineHumansSkeletonAgedMorphometrySkulllcsh:RBiology and Life SciencesComputing MethodsComputed Axial TomographyCross-Sectional StudiesAlgebra030221 ophthalmology & optometrylcsh:QTomography X-Ray ComputedNasolacrimal DuctMathematics030217 neurology & neurosurgeryNeuroscienceBiomedical engineeringVolume (compression)PLoS ONE
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Cancer in Children With Fanconi Anemia and Ataxia-Telangiectasia—A Nationwide Register-Based Cohort Study in Germany

2021

PURPOSE Fanconi anemia (FA) and ataxia-telangiectasia (AT) are rare inherited syndromes characterized by abnormal DNA damage response and caused by pathogenic variants in key DNA repair proteins that are also relevant in the pathogenesis of breast cancer and other cancer types. The risk of cancer in children with these diseases is poorly understood and has never been assessed in a population-based cohort before. METHODS We identified 421 patients with FA and 160 patients with AT diagnosed between 1973 and 2020 through German DNA repair disorder reference laboratories. We linked patients' laboratory data with childhood cancer data from the German Childhood Cancer Registry. RESULTS Among 421 …

MaleOncologyRegister basedCancer Researchmedicine.medical_specialtyTime FactorsAdolescentDNA damageAnemiaDNA repairRisk AssessmentAtaxia TelangiectasiaRisk FactorsFanconi anemiaGermanyNeoplasmsInternal medicinemedicineHumansRegistriesChildbusiness.industryIncidenceAge FactorsInfantCancerPrognosismedicine.diseaseFanconi AnemiaOncologyChild PreschoolAtaxia-telangiectasiaFemalebusinessCohort studyJournal of Clinical Oncology
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Imaging of Orthotopic Glioblastoma Xenografts in Mice Using a Clinical CT Scanner: Comparison with Micro-CT and Histology

2016

Purpose There is an increasing need for small animal in vivo imaging in murine orthotopic glioma models. Because dedicated small animal scanners are not available ubiquitously, the applicability of a clinical CT scanner for visualization and measurement of intracerebrally growing glioma xenografts in living mice was validated. Materials and Methods 2.5x106 U87MG cells were orthotopically implanted in NOD/SCID/ᵞc-/- mice (n = 9). Mice underwent contrast-enhanced (300 μl Iomeprol i.v.) imaging using a micro-CT (80 kV, 75 μAs, 360° rotation, 1,000 projections, scan time 33 s, resolution 40 x 40 x 53 μm) and a clinical CT scanner (4-row multislice detector; 120 kV, 150 mAs, slice thickness 0.5 …

MalePathologyCancer Treatmentlcsh:MedicineContrast MediaMice SCIDSignal-To-Noise RatioDiagnostic Radiology030218 nuclear medicine & medical imagingchemistry.chemical_compound0302 clinical medicineMice Inbred NODMedicine and Health Scienceslcsh:ScienceSmall AnimalsTomographyNeurological TumorsMice KnockoutMultidisciplinarymedicine.diagnostic_testBrain NeoplasmsRadiology and ImagingBrainGliomaMagnetic Resonance ImagingIn Vivo ImagingOncologyNeurology030220 oncology & carcinogenesisFemaleAnatomyPreclinical imagingResearch ArticleInterleukin Receptor Common gamma Subunitmedicine.medical_specialtyHistologyImaging TechniquesAnimal TypesTransplantation HeterologousIomeprolBrain tumorNeuroimagingResearch and Analysis Methods03 medical and health sciencesDiagnostic MedicineCell Line TumorGliomamedicineAnimalsHumansMultislicebusiness.industrylcsh:ROrganismsBiology and Life SciencesCancers and NeoplasmsReproducibility of ResultsMagnetic resonance imagingX-Ray Microtomographymedicine.diseaseComputed Axial TomographyIopamidolTransplantationSignal-to-noise ratio (imaging)chemistrylcsh:QGlioblastomabusinessNuclear medicineZoologyNeurosciencePLOS ONE
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A European family with histidine 58 transthyretin mutation in familial amyloid polyneuropathy

1997

1. IntroductionMore than 50 mutations of the transthyretin (TTR) [1]molecule resulting in different clinical forms of amyloidosisincluding familial amyloid polyneuropathy (FAP) havebeen reported to date. Within this FAP spectrum severaltransthyretin mutations are more frequent, others are rare.One mutation, the codon 58 histidine for leucine has pre-viously been recorded only in American subjects (Mary-land/German type), originally reported in a large kinship[2,3] and in another family from Ohio [4]. In the originaldescription of the Maryland/German type of amyloidosis[2], it was stated that the early immigrants in this pedigreewere from the Rhine river area, "nearly all of them from thelef…

MalePathologymedicine.medical_specialtyAtaxiaAmyloid Neuropathiesmedicine.disease_causeAtrophyLeucineGermanymedicineHumansPoint MutationPrealbuminHistidineCodonGenetics (clinical)Genes DominantMutationDysesthesiabiologybusiness.industryPoint mutationAmyloidosisMiddle Agedmedicine.diseaseUnited StatesTransthyretinNeurologyPediatrics Perinatology and Child Healthbiology.proteinNeurology (clinical)medicine.symptomRestriction fragment length polymorphismbusinessNeuromuscular Disorders
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SIL1 mutations and clinical spectrum in patients with Marinesco-Sjogren syndrome.

2013

Marinesco-Sjogren syndrome is a rare autosomal recessive multisystem disorder featuring cerebellar ataxia, early-onset cataracts, chronic myopathy, variable intellectual disability and delayed motor development. More recently, mutations in the SIL1 gene, which encodes an endoplasmic reticulum resident co-chaperone, were identified as the main cause of Marinesco-Sjogren syndrome. Here we describe the results of SIL1 mutation analysis in 62 patients presenting with early-onset ataxia, cataracts and myopathy or combinations of at least two of these. We obtained a mutation detection rate of 60% (15/25) among patients with the characteristic Marinesco-Sjogren syndrome triad (ataxia, cataracts, m…

MalePathologymedicine.medical_specialtyAtaxiaultrastructure [Muscle Skeletal]SIL1 protein humanAdolescentMarinesco–Sjögren syndromeDNA Mutational Analysisgenetics [Mutation]Bioinformaticsmedicine.disease_causepathology [Muscle Skeletal]physiopathology [Spinocerebellar Degenerations]Cataractspathology [Brain]Intellectual disabilitymedicineGuanine Nucleotide Exchange FactorsHumansddc:610MyopathyMuscle SkeletalCells CulturedRetrospective StudiesSpinocerebellar DegenerationsFamily HealthMutationB-LymphocytesCerebellar ataxiabusiness.industryBrainmedicine.diseasegenetics [Guanine Nucleotide Exchange Factors]Magnetic Resonance Imaging10124 Institute of Molecular Life Sciencesgenetics [Spinocerebellar Degenerations]2728 Neurology (clinical)pathology [Spinocerebellar Degenerations]Mutationultrastructure [Brain]570 Life sciences; biologyAllelic heterogeneityFemaleNeurology (clinical)Neurosciences & Neurologymedicine.symptombusinessBrain : a journal of neurology
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Malignant teratoma in Klippel-Feil syndrome: a case report and review of the literature

2015

Introduction: Klippel-Feil syndrome is characterized by a congenital fusion of cervical vertebrae. Intracranial teratomas are nongerminomatous germ cell tumors and they account for 0.3 to 0.9% of all intracranial tumors. Teratomas with malignant transformation refer to lesions which give rise to malignant cancer of somatic type. The association between tumors of dermoid origin and Klippel-Feil malformation is extremely rare. Only 23 other cases have so far been reported, and only one case of dermoid tumor with areas of dedifferentiation on squamous cell carcinoma has been described. Case presentation: We report the case of a 72-year-old white man with a 2-year history of gait and balance di…

MalePathologymedicine.medical_specialtyVertebral arteryKlippel–Feil syndromeCase ReportKlippel-Feil syndromeFourth ventricleKlippel–Feil syndromeMalignant transformationCervical spine Dermoid tumor Klippel–Feil syndrome Pathology Posterior fossa TeratomaPosterior fossamedicine.arteryCervical spinemedicinePathologyHumansAgedDermoid CystMedicine(all)medicine.diagnostic_testbusiness.industryCervical spine; Dermoid tumor; Klippel-Feil syndrome; Pathology; Posterior fossa; Teratoma; Aged; Cervical Vertebrae; Cranial Fossa Posterior; Dermoid Cyst; Gait Apraxia; Humans; Klippel-Feil Syndrome; Magnetic Resonance Imaging; Male; Teratoma; Tomography X-Ray Computed; Treatment Outcome; Medicine (all)Medicine (all)TeratomaMagnetic resonance imagingGeneral Medicinemedicine.diseaseMagnetic Resonance ImagingGait ApraxiaTreatment OutcomeDermoid cystCranial Fossa PosteriorCervical VertebraeGerm cell tumorsTeratomabusinessTomography X-Ray ComputedDermoid tumorHuman
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