Search results for "Azoles"

showing 10 items of 899 documents

Isomer-nonspecific action of dichlorodiphenyltrichloroethane on aryl hydrocarbon receptor and G-protein-coupled receptor 30 intracellular signaling i…

2014

Abstract Extended residual persistence of the pesticide dichlorodiphenyltrichloroethane (DDT) raises concerns about its long-term neurotoxic effects. Little is known, however, about DDT toxicity during the early stages of neural development. This study demonstrated that DDT-induced apoptosis of mouse embryonic neuronal cells is a caspase-9-, caspase-3-, and GSK-3β-dependent process, which involves p,p’ -DDT-specific impairment of classical ERs. It also provided evidence for DDT-isomer-nonspecific alterations of AhR- and GPR30-mediated intracellular signaling, including changes in the levels of the receptor and receptor-regulated mRNAs, and also changes in the protein levels of the receptors…

GPR30Time FactorsGSK-3 betaEstrogen receptorApoptosisStimulationBiochemistryReceptors G-Protein-CoupledGlycogen Synthase Kinase 3MiceEndocrinologyneurotoxicityestrogenReceptorCells CulturedNeuronsbiologyCaspase 3estrogen receptorsCaspase InhibitorsCell biologycaspasesReceptors EstrogenQuinolinesGPERNeural developmentSignal Transductionmedicine.medical_specialtyAryl hydrocarbon receptor nuclear translocatorneuronal cell culturesDDT17-beta-estradiolIsomerismbeta-NaphthoflavoneInternal medicineparasitic diseasesCytochrome P-450 CYP1A1medicineAnimalsBcl-2BenzodioxolesRNA MessengerMolecular BiologyG protein-coupled receptorBenzoflavonesGlycogen Synthase Kinase 3 betaL-Lactate DehydrogenaseAryl hydrocarbon receptorPyrimidinesEndocrinologyReceptors Aryl Hydrocarbonbiology.proteinPyrazolesMolecular and Cellular Endocrinology
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Galectin-3 Impairment of MYCN-Dependent Apoptosis-Sensitive Phenotype Is Antagonized by Nutlin-3 in Neuroblastoma Cells

2012

MYCN amplification occurs in about 20-25% of human neuroblastomas and characterizes the majority of the high-risk cases, which display less than 50% prolonged survival rate despite intense multimodal treatment. Somehow paradoxically, MYCN also sensitizes neuroblastoma cells to apoptosis, understanding the molecular mechanisms of which might be relevant for the therapy of MYCN amplified neuroblastoma. We recently reported that the apoptosis-sensitive phenotype induced by MYCN is linked to stabilization of p53 and its proapoptotic kinase HIPK2. In MYCN primed neuroblastoma cells, further activation of both HIPK2 and p53 by Nutlin-3 leads to massive apoptosis in vitro and to tumor shrinkage an…

Galectin 3Cancer TreatmentGene Dosagelcsh:MedicineApoptosisProtein-Serine-Threonine KinaseBiochemistryPiperazineschemistry.chemical_compoundNeuroblastoma0302 clinical medicineMolecular Cell BiologyBasic Cancer ResearchSignaling in Cellular Processeslcsh:ScienceEnergy-Producing OrganellesApoptotic SignalingNuclear ProteinOncogene Proteins0303 health sciencesN-Myc Proto-Oncogene ProteinMultidisciplinaryCell DeathImidazolesOncogene ProteinNuclear ProteinsTransfectionNutlin3. Good healthGene Expression Regulation NeoplasticProtein TransportCell killingPhenotypeOncologyGalectin-3030220 oncology & carcinogenesisGene Knockdown TechniquesMedicineResearch ArticleSignal TransductionHumanBiologyBioenergeticsProtein Serine-Threonine KinasesN-Myc Proto-Oncogene ProteinModels Biological03 medical and health sciencesNeuroblastomaCell Line TumormedicineHumansBiologyImidazolePiperazineneoplasms030304 developmental biologylcsh:RGene AmplificationChemotherapy and Drug Treatmentmedicine.diseasechemistryCell cultureApoptosisPediatric OncologyCytoprotectionGene Knockdown TechniqueCancer researchlcsh:QTumor Suppressor Protein p53Carrier ProteinsCarrier ProteinDNA Damage
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The loss of muscle mass and sarcopenia: non hormonal intervention.

2011

Muscle aging is a key component of the increase in frailty in human populations. The generation of critical levels of power is a prerequisite to perform simple tasks of daily living, such as rising from a chair or climbing stairs. There is great scientific and social interest to determine which behaviors can lead to the maintenance of the muscle mass in young immobilized subjects and in the elderly. Several hormonal treatments have been proposed for the treatment of sarcopenia. However, the side effects associated to these treatments emphasize the need of finding non-toxic and non-hormonal treatments that help increase muscle strength, improve muscle function, and decrease the degree of dep…

Gerontologymedicine.medical_specialtyAgingSarcopeniaNon hormonalFrail ElderlyPopulationLongevityMuscle massBiochemistryBenzoatesLosartanAngiotensin Receptor AntagonistsEndocrinologyPhysical medicine and rehabilitationIntervention (counseling)GeneticsmedicineAnimalsHumansMuscle StrengthPPAR deltaTelmisartaneducationMolecular BiologyExerciseHeat-Shock ProteinsAgedAged 80 and overeducation.field_of_studybusiness.industryPublic healthTOR Serine-Threonine KinasesCell Biologymedicine.diseasePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaMuscle atrophyMuscular AtrophySarcopeniaMuscle strengthBenzimidazolesmedicine.symptombusinesshuman activitiesTranscription FactorsExperimental gerontology
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Extraction of β-blockers from urine with a polymeric monolith modified with 1-allyl-3-methylimidazolium chloride in spin column format

2020

Abstract A glycidyl methacrylate-based monolith was modified with imidazolium-based ionic liquid (IL) to be used as stationary phase for solid-phase extraction (SPE). The host monolithic support was prepared by in-situ UV polymerization in spin column format. Two approaches were developed to incorporate the IL into the polymeric monolithic matrix: generation of IL onto the surface monolith, and copolymerization by addition of the IL to the polymerization mixture, which gave the best results. The resulting sorbent materials were morphologically characterized and used for the isolation of five β-blockers from human urine samples. All SPE steps were accomplished by centrifugation, which reduce…

Glycidyl methacrylatePolymersSurface PropertiesAdrenergic beta-Antagonists02 engineering and technology01 natural sciencesHigh-performance liquid chromatographyAnalytical ChemistryMatrix (chemical analysis)chemistry.chemical_compoundSpin column-based nucleic acid purificationHumansSolid phase extractionParticle SizeMonolithDetection limitgeographygeography.geographical_feature_categoryChromatographyChemistrySolid Phase Extraction010401 analytical chemistryExtraction (chemistry)Imidazoles021001 nanoscience & nanotechnology0104 chemical sciencesAllyl CompoundsEpoxy CompoundsMethacrylates0210 nano-technologyTalanta
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Leflunomide (HWA 486), a novel immunomodulating compound for the treatment of autoimmune disorders and reactions leading to transplantation rejection.

1991

Leflunomide has been shown to be very effective in preventing and curing several autoimmune animal diseases. Further, this agent is as effective as cyclosporin A in preventing the rejection of skin and kidney transplants in rats. Preliminary results from patients suffering from severe cases of rheumatoid arthritis demonstrated that clinical and immunological parameters could be improved with leflunomide therapy. Mode of action studies revealed that this substance antagonizes the proliferation inducing activity of several cytokines and is cytostatic for certain cell types. In this light, we could show that tyrosine phosphorylation of the RR-SRC peptide substrate and the autophosphorylation o…

Graft RejectionImmunologyMolecular Sequence DataGraft vs Host DiseasePharmacologyToxicologyAutoimmune Diseaseschemistry.chemical_compoundEpidermal growth factorCyclosporin amedicineAnimalsHumansPharmacology (medical)Amino Acid SequenceMode of actionLeflunomidePharmacologybusiness.industryAnti-Inflammatory Agents Non-SteroidalTyrosine phosphorylationIsoxazolesmedicine.diseaseTransplantationDisease Models AnimalchemistryRheumatoid arthritisImmunologybusinessTyrosine kinaseImmunosuppressive AgentsLeflunomidemedicine.drugAgents and actions
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Effects of leflunomide on immune responses and models of inflammation.

1993

Leflunomide is an antiphlogistic and immunomodulating agent that has been shown to be effective in preventing and healing autoimmune disorders and reactions leading to organ graft rejection. From our preliminary clinical data [4], we now have hopes that these effects, observed in experimental animals, can truly be transferred to humans. Although we are far from understanding the mode of action of leflunomide, we are slowly gathering some insight. A good many of the immunosuppressive effects of leflunomide can be attributed to the antagonistic effects it has on responses to many cytokines, most likely through receptor expression and signal transduction (tyrosine kinase inhibition). The inhib…

Graft RejectionReceptor expressionImmunologyDrug Evaluation PreclinicalAutoimmune Diseaseschemistry.chemical_compoundMiceImmune systemMedicineAnimalsHumansLeflunomideInflammationImmunity Cellularbusiness.industryAnti-Inflammatory Agents Non-SteroidalAutoantibodyGeneral MedicineIsoxazolesProtein-Tyrosine KinasesRatsDisease Models AnimalchemistryImmunologyAntibody FormationCytokinesSignal transductionbusinessTyrosine kinaseImmunomodulating AgentHistamineImmunosuppressive AgentsLeflunomidemedicine.drugSpringer seminars in immunopathology
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Electrochemical synthesis of carbazoles by dehydrogenative coupling reaction

2020

Abstract A constant current protocol, employing undivided cells, a remarkably low supporting electrolyte concentration, inexpensive electrode materials, and a straightforward precursor synthesis enabling a novel access to N‐protected carbazoles by anodic N,C bond formation using directly generated amidyl radicals is reported. Scalability of the reaction is demonstrated and an easy deblocking of the benzoyl protecting group is presented.

Green chemistry540 Chemistry and allied sciencesDeblocking filterSupporting electrolyteRadicalSustainable Chemistry010402 general chemistryElectrochemistry01 natural sciencesCatalysisCoupling reactionNC couplingProtecting groupgreen chemistry010405 organic chemistryChemistryCommunicationOrganic Chemistryheterocyclic chemistryGeneral ChemistryCombinatorial chemistryCommunications0104 chemical sciencesAnodecarbazoleselectrochemistry540 Chemie
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Structure and physical properties of [mu-tris(1,4-bis(tetrazol-1-yl)butane-N4,N4 ')iron(II)] bis(hexafluorophosphate), a new Fe(II) spin-crossover co…

2004

[mu-Tris(1,4-bis(tetrazol-1-yl)butane-N4,N4')iron(II)] bis(hexafluorophosphate), [Fe(btzb)(3)](PF6)(2), crystallizes in a three-dimensional 3-fold interlocked structure featuring a sharp two-step spin-crossover behavior. The spin conversion takes place between 164 and 182 K showing a discontinuity at about T-1/2 = 174 K and a hysteresis of about 4 K between T-1/2 and the low-spin state. The spin transition has been independently followed by magnetic susceptibility measurements, Fe-57-Mossbauer spectroscopy, and variable temperature far and midrange FIR spectroscopy. The title compound crystallizes in the trigonal space group P (3) over bar (No. 147) with a unit cell content of one formula u…

HALOGENATED ETHYLTETRAZOLESIRON(II)StereochemistrySpin transitionCrystal structureMagnetic susceptibilityTHERMAL HYSTERESISInorganic ChemistryBond lengthchemistry.chemical_compoundCrystallographyLattice constantchemistryMOLECULAR-INTERACTIONSSpin crossoverMAGNETIC-PROPERTIESFormula unitHexafluorophosphateTHEORETICAL DESCRIPTIONMOSSBAUERPHASE-TRANSITIONCOMPLEXESPhysical and Theoretical ChemistryELASTIC INTERACTION
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The new era of 1,2,4-oxadiazoles

2009

The synthesis, the chemical and photochemical reactivity, and the use of 1,2,4-oxadiazoles in materials and as bioactive compounds have been reviewed. The material in this survey includes some historical background, general features, state-of-the-art applications together with a critical discussion about current limitations and suggestions for future developments.

HETEROCYCLES OXADIAZOLES BIOACTIVE COMPOUNDS PHOTOCHEMISTRY FIVE-MEMBERED HETEROAROMATICSChemistryOrganic ChemistryNanotechnologyPhotochemical reactivitySettore CHIM/06 - Chimica OrganicaPhysical and Theoretical ChemistryBiochemistryCritical discussion
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Synthesis and induction of G0–G1 phase arrest with apoptosis of 3,5-dimethyl-6-phenyl-8-(trifluoromethyl)-5,6-dihydropyrazolo[3,4-f][1,2,3,5]tetrazep…

2007

The multistep synthesis of 3,5-dimethyl-6-phenyl-8-(trifluoromethyl)-5,6-dihydropyrazolo[3,4-f][1,2,3,5]tetrazepin-4(3H)-one 15 has been carried out. The compound showed antiproliferative and apoptotic effects against K562, K562-R (imatinib mesilate resistant), HL60 and multidrug resistant (MDR) HL60 cell lines. Compound 15 showed a pro-apoptotic activity against HL60 and K562 resistant cell lines markedly higher than etoposide and busulfan, respectively. Flow cytometry studies carried out on K562 cells allowed to establish that 15 induces G0-G1 phase arrest followed by apoptosis.

HL60StereochemistryApoptosisHL-60 CellsAntiproliferative activityResting Phase Cell CycleChemical synthesisPyrazolo[34-f][1234]tetrazepinoneFlow cytometrychemistry.chemical_compoundhemic and lymphatic diseasesDrug DiscoverymedicineHumansCytotoxicityEtoposideG0-G1 arrestPharmacologyTrifluoromethylMolecular Structuremedicine.diagnostic_testOrganic ChemistryG1 PhaseApoptosiAzepinesGeneral MedicineSettore CHIM/08 - Chimica FarmaceuticaMolecular biologyMultiple drug resistancechemistryApoptosisDrug resistancePyrazoles1234-TetrazepinoneK562 Cellsmedicine.drugEuropean Journal of Medicinal Chemistry
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