Search results for "BLAST"

showing 10 items of 2136 documents

Connecting temporal identity to mitosis: the regulation of Hunchback in Drosophila neuroblast lineages.

2006

Both in vertebrates and invertebrates, neural stem cells generate different cell types at different times during development. It has been suggested that this process depends on temporal identity transitions of neural progenitors, but the underlying mechanism has not been resolved, yet. Recently, Drosophila neuroblasts (NBs) have been shown to be an excellent model system to investigate this subject. Here, changes in temporal identity are regulated by sequential and transient expression of transcription factors in the NB, such as Hunchback (Hb) and Kruppel (Kr). The temporal expression profile is maintained in the progeny. Hb is expressed first and thus defines the earliest identity in a giv…

Retinal Ganglion CellsCell typeReceptors SteroidKruppel-Like Transcription FactorsDown-RegulationMitosisNerve Tissue ProteinsBiologyCell fate determinationKrüppelNeuroblastAnimalsDrosophila ProteinsNuclear export signalMolecular BiologyMitosisTranscription factorGeneticsNeuronsModels GeneticNuclear ProteinsCell DifferentiationCell BiologyNeural stem cellDNA-Binding ProteinsProtein BiosynthesisDrosophilaDevelopmental BiologyTranscription FactorsCell cycle (Georgetown, Tex.)
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p27Kip1participates in the regulation of endoreplication in differentiating chick retinal ganglion cells

2015

Nuclear DNA duplication in the absence of cell division (i.e. endoreplication) leads to somatic polyploidy in eukaryotic cells. In contrast to some invertebrate neurons, whose nuclei may contain up to 200,000-fold the normal haploid DNA amount (C), polyploid neurons in higher vertebrates show only 4C DNA content. To explore the mechanism that prevents extra rounds of DNA synthesis in these latter cells we focused on the chick retina, where a population of tetraploid retinal ganglion cells (RGCs) has been described. We show that differentiating chick RGCs that express the neurotrophic receptors p75 and TrkB while lacking retinoblastoma protein, a feature of tetraploid RGCs, also express p27K…

Retinal Ganglion CellsretinaEndocycleCell divisionCellular differentiationChick EmbryoRetinoblastoma ProteinendoreduplicationMicevertebrateRNA Small InterferingpolyploidyMice KnockoutRGCeducation.field_of_studyCell DifferentiationEndoreduplicationCell cycleImmunohistochemistryNuclear DNAendocycleneurogenesiscell cycleRNA InterferenceCyclin-Dependent Kinase Inhibitor p27NeurogenesisPopulationDown-RegulationCell cycleBiologyRetinal ganglionRetinaPolyploidyReportAnimalsReceptor trkBEndoreduplicationeducationMolecular BiologyPloidiesDNA synthesisVertebrateCyclin-Dependent Kinase 4Cyclin-Dependent Kinase 6Cell BiologyMinichromosome Maintenance Complex Component 7Molecular biologyeye diseasessense organsChickensDevelopmental BiologyCell Cycle
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SANS (USH1G) expression in developing and mature mammalian retina

2008

AbstractThe human Usher syndrome (USH) is the most common form of combined deaf-blindness. Usher type I (USH1), the most severe form, is characterized by profound congenital deafness, constant vestibular dysfunction and prepubertal-onset of retinitis pigmentosa. Five corresponding genes of the six USH1 genes have been cloned so far. The USH1G gene encodes the SANS (scaffold protein containing ankyrin repeats and SAM domain) protein which consists of protein motifs known to mediate protein–protein interactions. Recent studies indicated SANS function as a scaffold protein in the protein interactome related to USH.Here, we generated specific antibodies for SANS protein expression analyses. Our…

Retinal degenerationScaffold proteinBlotting WesternNerve Tissue ProteinsBiologyRibbon synapseRats Inbred WKYPhotoreceptor cellRetinaMiceXenopus laevisAntibody SpecificityCiliogenesisConnecting ciliumRetinitis pigmentosamedicineAnimalsCiliaEye ProteinsCentrosomeRetinaCiliogenesisPhotoreceptor cellsCiliumImmune SeraCiliary BodyFibroblastsmedicine.diseaseSynapseSensory SystemsCell biologyRatsMice Inbred C57BLOphthalmologymedicine.anatomical_structureSynapsesRetinal developmentsense organsUsher SyndromesUsher syndromePhotoreceptor Cells VertebrateSynaptosomesVision Research
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Missing Evidences in Cancer Genetics: The Retinoblastoma Paradigm

2008

BACKGROUND: Retinoblastoma (Rb) is the most common primary malignant intraocular tumour in childhood. The "two hit" theory, formulated by Knudson in 1971 to explain the variegated clinical expression of the disease, led to the discovery of the so called tumour suppressor genes and the identification of the Rb1 as the prototype of such genes. Mutations of the Rb1 gene are now commonly believed to be the "cause" retinoblastoma, although epidemiological, clinical, and biological evidences argue against it. MATERIAL/METHODS: The Authors have performed a systematic review of available data concerning clinical and diagnostic aspects of retinoblastoma, including molecular genetics. Meta analysis o…

Retinoblastoma pRBCancer Researchlcsh:CytologyRetinoblastomaCell BiologyGeneral Medicinelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282Retinoblastoma ProteinPathology and Forensic MedicineMutationHumansMolecular MedicineGenetic Predisposition to Diseaselcsh:QH573-671Letter to the EditorCellular Oncology : the Official Journal of the International Society for Cellular Oncology
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Retinoblastoma: History of His Identification, Characterization and Treatment

2015

The first description of a tumor resembling retinoblastoma (RB) was provided on 1597 by Pieter Pauw, who described a malignancy invading the orbit, the temporal region, and the cranium, filled with a "substance similar to brain tissue mixed with thick blood and like crushed stone". Since then, a number of retinal tumors were described and named until the 1922 when Verhoeff called these tumors RB, a term that the American Ophthalmological Society adopted in 1926. In 1971 Knudson focused on RB, and proposed his ‘two-hit’ theory of the molecular etiology of RB. In 1986, the RB1 gene was identified and the ‘two-hit’ theory of Knudson was validated. Successively, new studies in developing retina…

Retinoblastomabusiness.industrymedicineCancer researchRetinoblastoma pediatric cancer RB1 gene retinoblastoma inheritance retinoblastoma therapyRb1 geneIdentification (biology)medicine.diseasebusinessPediatric cancer
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The existence of FGFR1-5-HT1A receptor heterocomplexes in midbrain 5-HT neurons of the rat: relevance for neuroplasticity.

2012

The ascending midbrain 5-HT neurons to the forebrain may be dysregulated in depression and have a reduced trophic support. Within situproximity ligation assay (PLA) and supported by coimmunoprecipitation and colocation of the FGFR1 and 5-HT1A immunoreactivities in the midbrain raphe cells, evidence for the existence of FGFR1–5-HT1A receptor heterocomplexes in the dorsal and median raphe nuclei of the Sprague Dawley rat as well as in the rat medullary raphe RN33B cells has been obtained. Especially after combined FGF-2 and 8-OH-DPAT treatment, a marked and significant increase in PLA clusters was found in the RN33B cells. Similar results were reached with the FRET technique in HEK293T cells,…

Retractedmedicine.medical_specialtySerotonin receptorsEncèfalSettore BIO/11 - Biologia MolecolareBiologySettore BIO/09 - FisiologiaReceptors de serotoninaMidbrainInternal medicineRatesmedicineReceptor5-HT receptorNeuronal Plasticity Receptor Fibroblast Growth Factor Receptor Serotonin 5-HT1A Serotonergic Neurons SerotoninRapheGeneral NeuroscienceEncephalonFibroblastsRatsEndocrinologymedicine.anatomical_structurenervous systemForebrainAutoreceptor5-HT1A receptorNeuron
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Gingival hyperplasia as an early manifestation of acute myeloid leukemia. A retrospective review

2019

Background We study the prevalence of acute myeloid leukemia (AML) among patients with severe gingival enlargement. Material and Methods We retrospective reviewed the clinical records of patients with severe gingival enlargement, between 2011 and 2018. The Saxer and Mühlemann index were used to measure inflammation and gingival bleeding. The degree of dental mobility was measured by the Nyman and Lindhe technique. Results A correlation analysis was carried out to test whether there were any associations among the different variables. In the sample of 117 patients the mean gingival bleeding index was ≥3 and the degree of dental mobility ≥2.3. 1.7% of patients, with severe gingival hyperplasi…

Retrospective reviewmedicine.medical_specialtyAcute myeloblastic leukemiabusiness.industryResearchMyeloid leukemia030206 dentistry030204 cardiovascular system & hematologyOral healthHyperplasiamedicine.disease:CIENCIAS MÉDICAS [UNESCO]GastroenterologyCommunity and Preventive DentistryDyscrasiaGingival enlargement03 medical and health sciences0302 clinical medicineInternal medicineCorrelation analysisUNESCO::CIENCIAS MÉDICASmedicinebusinessGeneral Dentistry
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DICER and ZRF1 contribute to chromatin decondensation during nucleotide excision repair

2016

Abstract Repair of damaged DNA relies on the recruitment of DNA repair factors in a well orchestrated manner. As a prerequisite, the chromatin needs to be decondensed by chromatin remodelers to allow for binding of repair factors and for DNA repair to occur. Recent studies have implicated members of the SWI/SNF and INO80 families as well as PARP1 in nucleotide excision repair (NER). In this study, we report that the endonuclease DICER is implicated in chromatin decondensation during NER. In response to UV irradiation, DICER is recruited to chromatin in a ZRF1-mediated manner. The H2A–ubiquitin binding protein ZRF1 and DICER together impact on the chromatin conformation via PARP1. Moreover, …

Ribonuclease III0301 basic medicineDNA RepairUltraviolet RaysDNA damageDNA repairgenetic processesPoly (ADP-Ribose) Polymerase-1Genome Integrity Repair and ReplicationBiologyChromatin remodelingCell LineDEAD-box RNA HelicasesHistones03 medical and health scienceschemistry.chemical_compoundUbiquitinCell Line TumorGeneticsAnimalsHumansCaenorhabditis elegansOncogene ProteinsOsteoblastsUbiquitinfungiRNA-Binding ProteinsFibroblastsChromatin Assembly and DisassemblyMolecular biologyChromatinChromatinDNA-Binding Proteinsenzymes and coenzymes (carbohydrates)HEK293 Cells030104 developmental biologychemistrybiology.proteinDNADNA DamageMolecular ChaperonesNucleotide excision repairDicerNucleic Acids Research
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The topology of vitronectin: A complementary feature for neuroblastoma risk classification based on computer‐aided detection

2019

Tumors are complex networks of constantly interacting elements: tumor cells, stromal cells, immune and stem cells, blood/lympathic vessels, nerve fibers and extracellular matrix components. These elements can influence their microenvironment through mechanical and physical signals to promote tumor cell growth. To get a better understanding of tumor biology, cooperation between multidisciplinary fields is needed. Diverse mathematic computations and algorithms have been designed to find prognostic targets and enhance diagnostic assessment. In this work, we use computational digital tools to study the topology of vitronectin, a glycoprotein of the extracellular matrix. Vitronectin is linked to…

RiskCancer ResearchStromal celltopologyTumor Markers and SignaturesComputer scienceAngiogenesisTopologyTopologyvitronectinExtracellular matrixComputational biology03 medical and health sciencesNeuroblastoma0302 clinical medicinecomputational biologyNeuroblastNeuroblastomamedicineTumor MicroenvironmentHumansVitronectinCell ProliferationbiologyNeovascularization PathologicComplex networkmedicine.diseasePrognosisExtracellular MatrixOncology030220 oncology & carcinogenesisnetworksbiology.proteinVitronectinStem cellNetworksStromal CellsAlgorithmsInternational Journal of Cancer
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Non-response bias as a likely cause of the association between young maternal age at the time of delivery and the risk of cancer in the offspring.

2003

Some epidemiological studies have shown an association between young maternal age at the time of delivery and risk of cancer in the offspring. In a recent German case-control study, there was a twofold increase in the leukaemia risk for children whose mothers were aged < 20 years at the time of delivery. As the prevalence of younger mothers among control families was particularly low, data on maternal age distributions for the general population of Germany were obtained in order to examine the representativeness of the control sample. Despite the excellent sampling frame based on data from complete and up-to-date population registries and a satisfactory response rate among controls ( approx…

RiskPediatricsmedicine.medical_specialtyEpidemiologyOffspringPopulationCentral Nervous System NeoplasmsNeuroblastomaBiasEpidemiologyMedicineHumansNon-response biaseducationSampling frameResponse rate (survey)education.field_of_studybusiness.industryLymphoma Non-HodgkinCase-control studyInfant NewbornPrecursor Cell Lymphoblastic Leukemia-LymphomaSample size determinationCase-Control StudiesSample SizePediatrics Perinatology and Child HealthFemalebusinessDemographyMaternal AgePaediatric and perinatal epidemiology
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