Search results for "BLOOD-BRAIN BARRIER"

showing 10 items of 141 documents

Involvement of Microglia in Neurodegenerative Diseases: Beneficial Effects of Docosahexahenoic Acid (DHA) Supplied by Food or Combined with Nanoparti…

2021

Neurodegenerative diseases represent a major public health issue and require better therapeutic management. The treatments developed mainly target neuronal activity. However, an inflammatory component must be considered, and microglia may constitute an important therapeutic target. Given the difficulty in developing molecules that can cross the blood–brain barrier, the use of food-derived molecules may be an interesting therapeutic avenue. Docosahexaenoic acid (DHA), an omega-3 polyunsaturated fatty acid (22:6 omega-3), has an inhibitory action on cell death and oxidative stress induced in the microglia. It also acts on the inflammatory activity of microglia. These data obtained in vitro or…

Programmed cell deathDocosahexaenoic AcidsQH301-705.5microgliaApoptosisInflammationReviewPharmacologyProtective AgentsInhibitory postsynaptic potentialmedicine.disease_causeCatalysisInorganic ChemistryDrug Delivery Systemsneurodegenerative diseasemedicineAnimalsHumansBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopychemistry.chemical_classificationMicrogliabusiness.industryOrganic ChemistryNeurodegenerative DiseasesGeneral Medicinedocosahexaenoic acidnanomedicineIn vitroComputer Science ApplicationsDisease Models AnimalOxidative StressChemistryTreatment Outcomemedicine.anatomical_structurechemistryBlood-Brain BarrierinflammationDocosahexaenoic acidDietary SupplementsNanoparticlesmedicine.symptombusinessOxidative stressPolyunsaturated fatty acidInternational Journal of Molecular Sciences
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Role of oxoproline in the regulation of neutral amino acid transport across the blood-brain barrier.

1996

Regulation of neutral amino acid transport was studied using isolated plasma membrane vesicles derived from the bovine blood-brain barrier. Neutral amino acids cross the blood-brain barrier by facilitative transport system L1, which may allow both desirable and undesirable amino acids to enter the brain. The sodium-dependent amino acid systems A and Bo,+ are located exclusively on abluminal membranes, in a position to pump unwanted amino acids out. gamma-Glutamyl transpeptidase, the first enzyme of the gamma-glutamyl cycle, is an integral protein of the luminal membrane of the blood-brain barrier. We demonstrate that oxoproline, an intracellular product of the gamma-glutamyl cycle, stimulat…

ProlineBiologyBlood–brain barrierBiochemistrySystem aNeutral amino acid transportmedicineAnimalsAmino AcidsMolecular BiologyIntegral membrane proteinchemistry.chemical_classificationCell MembraneBiological TransportCell BiologyAmino acidCapillariesKineticsMembraneEnzymemedicine.anatomical_structureBiochemistrychemistryBlood-Brain BarrierCattleEndothelium VascularIntracellularThe Journal of biological chemistry
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The ins and outs of cholesterol in the vertebrate retina

2010

Thematic Review Series: Lipids and Lipid Metabolism in the Eye; International audience; The vertebrate retina has multiple demands for utilization of cholesterol and must meet those demands either by synthesizing its own supply of cholesterol or by importing cholesterol from extraretinal sources, or both. Unlike the blood-brain barrier, the blood-retina barrier allows uptake of cholesterol from the circulation via a lipoprotein-based/receptor-mediated mechanism. Under normal conditions, cholesterol homeostasis is tightly regulated; also, cholesterol exists in the neural retina overwhelmingly in unesterified form, and sterol intermediates are present in minimal to negligible quantities. Howe…

QD415-436Degeneration (medical)BiologyBlood–brain barrierBiochemistryRetina03 medical and health scienceschemistry.chemical_compoundSmith-Lemli-Opitz syndrome0302 clinical medicineEndocrinology[SDV.IDA]Life Sciences [q-bio]/Food engineeringmedicineAnimalsHumans[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process Engineeringcholesterol/biosynthesis;eye/retina;Smith-Lemli-Opitz syndrome030304 developmental biologychemistry.chemical_classification0303 health sciencesRetinaCholesterolThematic ReviewBiological TransportCell Biologycholesterol/biosynthesismedicine.diseaseSterolDe novo synthesisCholesterolmedicine.anatomical_structureEnzymechemistryBiochemistryBlood-Brain BarrierSmith–Lemli–Opitz syndromeeye/retinaVertebrates030221 ophthalmology & optometrylipids (amino acids peptides and proteins)sense organs
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Permeability properties of a three-cell type in vitro model of blood-brain barrier.

2005

We previously found that RBE4.B brain capillary endothelial cells (BCECs) form a layer with blood‐brain barrier (BBB) properties if co‐cultured with neurons for at least one week. As astrocytes are known to modulate BBB functions, we further set a culture system that included RBE4.B BCECs, neurons and astrocytes. In order to test formation of BBB, we measured the amount of (3)H‐sucrose able to cross the BCEC layer in this three‐cell type model of BBB. Herein we report that both neurons and astrocytes induce a decrease in the permeability of the BCEC layer to sucrose. These effects are synergic as if BCECs are cultured with both neurons and astrocytes for 5 days, permeability to sucrose decr…

SucroseCell typeTime FactorsBlotting WesternVascular permeabilityBiologyBlood–brain barrierOccludinArticleCapillary PermeabilityOccludinmedicineAnimalsRats WistarCell Line TransformedNeuronsBrainEndothelial CellsMembrane ProteinsCell BiologyPermeationblood-brain barrier cortical neurons astrocytes brain capillary endothelial cells RBE4.B occludin.Coculture TechniquesRatsmedicine.anatomical_structurenervous systemMembrane proteinBiochemistryBlood-Brain BarrierPermeability (electromagnetism)Astrocytescardiovascular systembiology.proteinBiophysicsMolecular MedicineAntibody
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Gold nanoparticle interactions with endothelial cells cultured under physiological conditions

2017

PEGylated gold nanoparticles (AuNPs) have an extended circulation time after intravenous injection in vivo and exhibit favorable properties for biosensing, diagnostic imaging, and cancer treatment. No impact of PEGylated AuNPs on the barrier forming properties of endothelial cells (ECs) has been reported, but recent studies demonstrated that unexpected effects on erythrocytes are observed. Almost all studies to date have been with static-cultured ECs. Herein, ECs maintained under physiological cyclic stretch and flow conditions and used to generate a blood-brain barrier model were exposed to 20 nm PEGylated AuNPs. An evaluation of toxic effects, cell stress, the release profile of pro-infla…

SwineBiomedical EngineeringNanoparticleNanotechnology02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesPolyethylene GlycolsIn vivoHuman Umbilical Vein Endothelial CellsMedicineAnimalsHumansGeneral Materials ScienceParticle SizeCells Culturedbusiness.industryEndothelial Cells021001 nanoscience & nanotechnologyQPR10104 chemical sciencesCancer treatmentCell stressmedicine.anatomical_structureColloidal goldBlood-Brain BarrierBiophysicsNanoparticlesCirculation timeGold0210 nano-technologybusinessBlood stream
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Cytotoxicity and inhibition of P-glycoprotein by selected medicinal plants from Thailand.

2014

Abstract Ethnopharmacological relevance Thai medicine has a long tradition of tonifying medicinal plants. In the present investigation, we studied the flower extracts of Jasminum sambac, Mammea siamensis, Mesua ferrea, Michelia alba, Mimusops elengi, and Nelumbo nucifera and speculated that these plants might influence metabolism and substance flow in the body. Materials and methods Isolation of porcine brain capillary endothelial cells (PBCECs) as well as multidrug-resistance CEM/ADR5000 leukemia cells, MDA-M;B-231 breast cancer, U-251 brain tumor, and HCT-116 colon cancer cells were used. The calcein-acetoxymethylester (AM) assay was used to measure inhibition of P-glycoprotein transport.…

SwineMesua ferreaMimusops elengiFlowersPharmacologyBlood–brain barrierchemistry.chemical_compoundCell Line TumorNeoplasmsDrug DiscoverymedicineAnimalsHumansATP Binding Cassette Transporter Subfamily B Member 1CytotoxicityP-glycoproteinPharmacologyMedicine East Asian TraditionalPlants MedicinalbiologyTraditional medicinePlant ExtractsMammeaBrainEndothelial Cellsmedicine.diseasebiology.organism_classificationThailandAntineoplastic Agents PhytogenicDrug Resistance MultipleLeukemiamedicine.anatomical_structurechemistryBlood-Brain BarrierDrug Resistance Neoplasmbiology.proteinEndothelium VascularGrowth inhibitionJournal of ethnopharmacology
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THERAPEUTIC-LOADED LIPID NANOSTRUCTURES AND BRAIN DISEASES.

2012

Central Nervous System (CNS) diseases represent the largest and fastest growing area of unmet medical need since an alarming increase in brain disease incidence is going on. Despite major advances in neuroscience, many potential therapeutic agents are denied access to the CNS because of the existence of a physiological low permeable barrier, the Blood-Brain Barrier (BBB). To obtain an improvement of drug CNS performance, sophisticated approaches such as nanoparticulate systems are rapidly developing. In particular, in this chapter, the most recent data demonstrating the potential of lipid nanostructures, such as Solid Lipid Nanoparticles (SLN) and Nanostructured Lipid Carriers (NLC), to tra…

Targeted drug delivery systemsSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoSolid lipid nanoparticleNanostructured lipid carrierBlood-brain barrier
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Caspase-3 contributes to ZO-1 and Cl-5 tight-junction disruption in rapid anoxic neurovascular unit damage.

2011

BACKGROUND: Tight-junction (TJ) protein degradation is a decisive step in hypoxic blood-brain barrier (BBB) breakdown in stroke. In this study we elucidated the impact of acute cerebral ischemia on TJ protein arrangement and the role of the apoptotic effector protease caspase-3 in this context. METHODOLOGY/PRINCIPAL FINDINGS: We used an in vitro model of the neurovascular unit and the guinea pig whole brain preparation to analyze with immunohistochemical methods the BBB properties and neurovascular integrity. In both methodological approaches we observed rapid TJ protein disruptions after 30 min of oxygen and glucose deprivation or middle cerebral artery occlusion, which were accompanied by…

Time FactorsAnatomy and Physiologylcsh:MedicineMiceMolecular Cell BiologyPathologySignaling in Cellular ProcessesHypoxia Brainlcsh:ScienceCells CulturedNeuropathologyApoptotic SignalingMultidisciplinaryTight junctionCaspase 3ChemistryAnimal ModelsCell biologyTransport proteinProtein Transportmedicine.anatomical_structureNeurologyBlood-Brain BarrierMedicineResearch ArticleSignal TransductionClinical Research DesignCerebrovascular DiseasesGuinea PigsIschemiaContext (language use)Caspase 3Protein degradationBlood–brain barrierNeurological SystemTight JunctionsCapillary PermeabilityModel OrganismsDiagnostic MedicinemedicineAnimalsTransient Ischemic AttacksAnimal Models of DiseaseClaudinBiologyIschemic Strokelcsh:REndothelial CellsMembrane ProteinsPhosphoproteinsmedicine.diseaseAnatomical PathologyClaudinsImmunologyZonula Occludens-1 ProteinNervous System Componentslcsh:QPLoS ONE
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Neurons and ECM regulate occludin localization in brain endothelial cells

2000

We report that extracellular matrix and neurons modulate the expression of occludin, one of the main components of tight junctions, by rat brain endothelial cells (RBE4.B). Of the three extracellular matrix proteins which we tested (collagen I, collagen IV, and laminin), collagen IV stimulated at the best the expression of occludin mRNA. The corresponding protein, however, was not synthesized. Significant amounts of occludin accumulated only when RBE4.B cells were cultured on collagen IV-coated inserts, in the presence of cortical neurons, plated on laminin-coated companion wells. Finally, occludin segregated at the cell periphery, only when endothelial cells were co- cultured with neurons …

Time FactorsEndothelial cellsCellOccludinTight JunctionsExtracellular matrixRats Sprague-DawleyFetusLamininNeurofilament ProteinsOccludinSettore BIO/10 - BiochimicaGlial Fibrillary Acidic ProteinmedicineAnimalsRNA MessengerCells CulturedBlood-brain barrierNeuronsbiologyTight junctionGeneral NeuroscienceBrainMembrane ProteinsCortical NeuronsExtracellular matrixImmunohistochemistryCell biologyRatsEndothelial stem cellmedicine.anatomical_structureMembrane proteinCell cultureSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoCerebrovascular Circulationbiology.proteinSettore MED/26 - NeurologiaCollagenEndothelium VascularLamininNeuroscience
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Isolation and molecular characterization of brain microvascular endothelial cells from human brain tumors.

2002

Brain tumor formation and growth is accompanied by the proliferation and infiltration of blood capillaries. The phenotypes of endothelial cells that make up capillaries are known to differ not only in the tissues in which endothelial cells are located but also as a result of the microenvironment to which they are exposed. For this reason, primary cultures of brain endothelial cells were isolated from human brain tumors removed by surgery and compared with cells from normal tissue. The primary confluent monolayers that grew out of isolated capillary fragments consisted of closely associated, elongated, fusiform-shaped cells. But brain tumor-derived endothelial cells in culture exhibited sign…

Vascular Cell Adhesion Molecule-1Cell SeparationBiologyBlood–brain barrierAntigenvon Willebrand FactormedicineTumor Cells CulturedHumansCell adhesionCell SizeFluorescent DyesTight junctionBrain NeoplasmsBrainMembrane ProteinsCell BiologyGeneral MedicineHuman brainCarbocyaninesmedicine.diseaseIntercellular Adhesion Molecule-1PhosphoproteinsCell biologyMicroscopy Electronmedicine.anatomical_structureCell cultureBlood-Brain BarrierZonula Occludens-1 ProteinEndothelium VascularStem cellPlant LectinsE-SelectinInfiltration (medical)Developmental BiologyIn vitro cellulardevelopmental biology. Animal
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