Search results for "Bars"

showing 10 items of 106 documents

Iridoid extracts from Ajuga iva increase the antioxidant enzyme activities in red blood cells of rats fed a cholesterol-rich diet.

2010

The lyophilized aqueous extract of Ajuga iva (Ai) is able to reduce oxidative stress, which may prevent lipid peroxidation in hypercholesterolemic rats. Iridoids (I) were isolated from Ai. We hypothesized that the antioxidant defense status in red blood cells (RBC) and tissues in rats fed a cholesterol-rich diet and treated with Ai may be correlated to these compounds. Male Wistar rats (n = 32) weighing 120 +/- 5 g were fed a diet containing 1% cholesterol for 15 days. After this phase, hypercholesterolemic (HC) rats were divided into groups, fed the same diet, and received either the same or different doses (5, 10, or 15 mg/kg body weight by intraperitoneal injection) of I for 15 days. Com…

Malemedicine.medical_specialtyAntioxidantErythrocytesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGlutathione reductaseHypercholesterolemiaAjugamedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutaseCholesterol Dietarychemistry.chemical_compoundEndocrinologyInternal medicinemedicineTBARSAnimalsIridoidsRats Wistarchemistry.chemical_classificationNutrition and DieteticsbiologyDose-Response Relationship DrugPlant ExtractsGlutathione peroxidaseAnticholesteremic AgentsMusclesMyocardiumGlutathioneRatsOxidative StressEndocrinologychemistryLiverbiology.proteinOxidative stressPhytotherapyNutrition research (New York, N.Y.)
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Changes in antioxidant defense status in hypercholesterolemic rats treated with Ajuga iva.

2007

Abstract The aim of the study was to investigate the effect of aqueous extract of Ajuga iva ( Ai ) on serum and tissues lipid peroxidation as well as antioxidant enzymes activities in red blood cells (RBC) and tissues, in high hypercholesterolemic rats (HC). Male Wistar rats ( n =12) were fed on 1% cholesterol-enriched diet for 15 d. After this adaptation phase, hypercholesterolemic rats (total cholesterol=6.5±0.6 mol/l) were divided into two groups fed the same diet and treated or not with Ai for 15 d. Thiobarbituric acid reactive substances (TBARS) concentrations in serum, LDL-HDL 1 , HDL 2 and HDL 3 were respectively, 5-, 7.8-, 2.3- and 5-fold lower in Ai treated than untreated hyperchol…

Malemedicine.medical_specialtyAntioxidantErythrocytesThiobarbituric acidmedicine.medical_treatmentGlutathione reductaseHypercholesterolemiaPharmaceutical ScienceAdipose tissueAjugaNitric OxideAjugaAntioxidantsLipid peroxidationCholesterol Dietarychemistry.chemical_compoundEatingInternal medicineDrug DiscoverymedicineTBARSAnimalsRats WistarPharmacologychemistry.chemical_classificationbiologyPlant ExtractsGlutathione peroxidaseBody WeightVitaminsbiology.organism_classificationGlutathioneRatsEndocrinologyCholesterolComplementary and alternative medicinechemistryBiochemistryMolecular MedicineLipid PeroxidationPhytotherapyPhytomedicine : international journal of phytotherapy and phytopharmacology
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Study of the Correlations among Some Parameters of the Oxidative Status, Gelatinases, and Their Inhibitors in a Group of Subjects with Metabolic Synd…

2014

Our aim was to examine some parameters of oxidative status, gelatinases, and their inhibitors and to evaluate their interrelationships in subjects with metabolic syndrome (MS). We enrolled 65 MS subjects, subdivided according to the presence or not of diabetes mellitus. We examined lipid peroxidation (expressed as thiobarbituric acid reacting substances, TBARS), protein oxidation (expressed as carbonyl groups), nitric oxide metabolites (NOx), total antioxidant status (TAS), MMP-2, MMP-9, TIMP-1, and TIMP-2. We found that MS subjects, diabetics and nondiabetics, showed an increase in TBARS, PC, and NOx. A significant decrease in TAS was observed only in nondiabetic MS subjects in comparison …

Malemedicine.medical_specialtyGelatinasesSettore MED/09 - Medicina InternaArticle SubjectThiobarbituric acidImmunologymedicine.disease_causeProtein oxidationNitric OxideThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationchemistry.chemical_compoundOxidative Status Gelatinases Metabolic SyndromeInternal medicineDiabetes mellitusmedicineTBARSlcsh:PathologyHumansMetabolic SyndromeTissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1business.industryCell BiologyMiddle Agedmedicine.diseaseEndocrinologychemistryBiochemistryMatrix Metalloproteinase 9GelatinasesMatrix Metalloproteinase 2FemaleLipid PeroxidationMetabolic syndromebusinessOxidative stresslcsh:RB1-214Research ArticleMediators of Inflammation
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Protection effect of endurance training against reoxygenation-induced injuries in rat heart

1990

Endurance training by swimming (219-229 h) resulted in a significant protection against hypoxia/reoxygenation-induced injuries in Langendorff-perfused rat hearts. The protection was manifested as improved flow characteristics and a smaller release of creatine kinase into the perfusate. The concentration of thiobarbituric acid reactive substances (TBARS) was lower in the trained than in the respective control hearts. The trained hearts also showed a lower reoxygenation-induced increase in TBARS. The myocardium of the right ventricle and that of the left subepimyocardium were the most affected by reoxygenation. The swimming program induced a decrease in the activities of catalase and glutath…

Malemedicine.medical_specialtyHeart DiseasesPhysiologyThiobarbituric acidGlutathione reductaseGlucosephosphate DehydrogenaseSuperoxide dismutasechemistry.chemical_compoundEndurance trainingPhysical Conditioning AnimalPhysiology (medical)Internal medicineTBARSAnimalsMedicineHypoxiaSwimmingbiologySuperoxide Dismutasebusiness.industryMyocardiumRats Inbred StrainsGlutathioneThiobarbituratesGlutathioneRatsOxygenEndocrinologychemistryPhysical Endurancebiology.proteinGlutathione disulfideCreatine kinaseLipid PeroxidationbusinessJournal of Applied Physiology
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Silibinin modulates lipid homeostasis and inhibits nuclear factor kappa B activation in experimental nonalcoholic steatohepatitis.

2012

Nonalcoholic steatohepatitis (NASH) is associated with increased liver-related mortality. Disturbances in hepatic lipid homeostasis trigger oxidative stress and inflammation (ie, lipotoxicity), leading to the progression of NASH. This study aimed at identifying whether silibinin may influence the molecular events of lipotoxicity in a mouse model of NASH. Eight-week-old db/db mice were fed a methionine-choline deficient (MCD) diet for 4 weeks and treated daily with silibinin (20 mg/kg intraperitoneally) or vehicle. Liver expression and enzyme activity of stearoyl-CoA desaturase-1 and acyl-CoA oxidase, and expression of liver fatty acid-binding protein were assessed. Hepatic levels of reactiv…

Malemedicine.medical_specialtyMice ObeseSilibininmedicine.disease_causeAntioxidantsTranslational Research BiomedicalMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineNonalcoholic fatty liver diseasemedicineTBARSAnimalsHomeostasisNASH MCD Silibinin lipotoxicity.Reactive nitrogen speciesLiver injurychemistry.chemical_classificationReactive oxygen speciesAnti-Inflammatory Agents Non-SteroidalBiochemistry (medical)NF-kappa BPublic Health Environmental and Occupational HealthGeneral MedicineLipid Metabolismmedicine.diseaseCholine DeficiencyFatty LiverDisease Models AnimalOxidative StressEndocrinologyLiverchemistryLipotoxicitySilybinOxidative stressSilymarin
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Pregnenolone sulfate, a naturally occurring excitotoxin involved in delayed retinal cell death.

2002

The present study was designed to investigate the neurosteroid pregnenolone sulfate (PS), known for its ability to modulate NMDA receptors and interfere with acute excitotoxicity, in delayed retinal cell death. Three hours after exposure of the isolated and intact retina to a 30-min PS pulse, DNA fragmentation as assessed by genomic DNA gel electrophoresis and a modified in situ terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end-labeling (TUNEL) method appeared concurrently with an increase in superoxide dismutase (SOD) activity and thiobarbituric acid-reactive substances (TBARS) levels. At 7 h, the increased amount of DNA laddering was accompanied by a higher number of TUN…

Malemedicine.medical_specialtyNeurotoxinsExcitotoxicityApoptosisDNA FragmentationDNA ladderingBiologymedicine.disease_causeBiochemistryReceptors N-Methyl-D-AspartateThiobarbituric Acid Reactive SubstancesRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundAdjuvants ImmunologicSuperoxidesInternal medicinemedicineTBARSIn Situ Nick-End LabelingAnimalsCycloheximideRats WistarProgesteroneProtein Synthesis InhibitorsTUNEL assayEstradiolL-Lactate DehydrogenaseDehydroepiandrosterone SulfateSuperoxide DismutaseRatsEndocrinologychemistryApoptosisPregnenolonePregnenoloneDNA fragmentationLipid PeroxidationPregnenolone sulfateReactive Oxygen Speciesmedicine.drugJournal of neurochemistry
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Erythrocyte deformability, plasma lipid peroxidation and plasma protein oxidation in a group of OSAS subjects

2016

Considering that obstructive sleep apnea syndrome (OSAS) is usually associated with endothelial dysfunction, atherosclerosis and cardiovascular disorders, our aim was to examine the erythrocyte deformability and the oxidative status in a group of OSAS subjects. We consecutively enrolled 48 subjects with OSAS defined after a 1-night cardiorespiratory sleep study, subsequently subdivided according to the apnea/hypopnea index (AHI) value in two subgroups: Low (L = 21 subjects with AHI30) and High (H = 27 subjects with AHI30). We evaluated the erythrocyte deformability, expressed as elongation index (EI) and the parameters of the oxidative status, such as lipid peroxidation (expressed as thioba…

Malemedicine.medical_specialtyPhysiologyLipid peroxidation030204 cardiovascular system & hematologymedicine.disease_causeProtein oxidationBioinformaticsLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBlood Proteinstomatognathic systemErythrocyte DeformabilityPhysiology (medical)Internal medicineErythrocyte deformability; Lipid peroxidation; OSAS; Protein oxidation; Blood Proteins; Erythrocyte Deformability; Female; Humans; Lipid Peroxidation; Male; Middle Aged; Oxidation-Reduction; Oxidative Stress; Sleep Apnea Obstructive; Physiology; Hematology; Cardiology and Cardiovascular Medicine; Physiology (medical)medicineTBARSHumansErythrocyte deformabilitySleep Apnea Obstructivebusiness.industryErythrocyte fragilityOSASOxidative StreBlood ProteinsHematologyMiddle Agedmedicine.diseaseBlood proteinsnervous system diseasesrespiratory tract diseasesOxidative StressEndocrinology030228 respiratory systemchemistryFemaleCardiology and Cardiovascular MedicinebusinessProtein oxidationOxidation-ReductionHypopneaOxidative stressHumanClinical Hemorheology and Microcirculation
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Elastase, myeloperoxidase, nitric oxide metabolites and oxidative status in subjects with clinical stable chronic renal failure on conservative treat…

2009

We evaluated, in a group of 41 CRF undialyzed subjects (29 men and 12 women, mean age 64.1 +/- 11.3 years), some parameters that reflect leukocyte activation (elastase, myeloperoxidase - MPO), plasma NO metabolites (NO(x)) and the oxidative status (lipid peroxidation expressed as thiobarbituric acid-reactive substances (TBARS) and total antioxidant status (TAS). Elastase was determined, on plasma separated from fasting venous blood, as elastase/alpha1-proteinase inhibitor complex. MPO was evaluated employing the Myeloperoxidase ELISA kit. The NO production was evaluated by a micromethod. The oxidation of polyunsaturated fatty acids was evaluated in plasma by detection of the thiobarbituric …

Malemedicine.medical_specialtyPhysiologyRenal functionThiobarbituric Acid Reactive SubstancesLipid peroxidationchemistry.chemical_compoundchronic renal failurenitric oxidePhysiology (medical)Internal medicineTBARSLeukocytesMedicineHumanselastaseAgedPeroxidasechemistry.chemical_classificationCreatininebiologyPancreatic Elastasebusiness.industryElastaseHematologyMiddle Agedoxidative statuOxidative StressmyeloperoxidaseEndocrinologychemistrySpectrophotometryMyeloperoxidaseCase-Control StudiesImmunologybiology.proteinKidney Failure ChronicFemaleHemoglobinCardiology and Cardiovascular MedicinebusinessPolyunsaturated fatty acid
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Lipid peroxidation and nitric oxide metabolites in a group of subjects with obstructive sleep apnea syndrome.

2015

It is known that in OSAS the plasma lipid peroxidation has an opposite behavior in comparison with nitric oxide metabolites. In the re-examination of our survey of OSAS subjects we calculated the ratio between thiobarbituric acid reactive substances (TBARS) and nitric oxide metabolites (NOx) in relation to OSAS severity. The study has regarded 48 OSAS subjects subdivided in two subgroups according to the apnea/hypopnea index - AHI- (Low = 21 subjects with AHI30 and High = 27 subjects with AHI30). From the obtained data it is evident that the TBARS/NOx ratio is significantly higher in the H subgroup compared to L subgroup as well as this ratio is reduced in L subgroup in comparison with the …

Malemedicine.medical_specialtyPhysiologyThiobarbituric acidobstructive sleep apnea syndrome030204 cardiovascular system & hematologyNitric OxideNitric oxideLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicinestomatognathic systemnitric oxide metabolitePhysiology (medical)Internal medicineTBARSLipid peroxidation; nitric oxide metabolites; obstructive sleep apnea syndrome; Female; Humans; Lipid Peroxidation; Male; Middle Aged; Nitric Oxide; Sleep Apnea Obstructive; Physiology; Hematology; Cardiology and Cardiovascular Medicine; Physiology (medical)MedicineHumansNOxSleep Apnea Obstructivebusiness.industryApneaHematologyMiddle Agedmedicine.diseasenervous system diseasesrespiratory tract diseasesObstructive sleep apneaEndocrinology030228 respiratory systemchemistryFemaleLipid Peroxidationmedicine.symptomCardiology and Cardiovascular MedicinebusinessHypopneaHumanClinical hemorheology and microcirculation
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Effect of aqueous extract of Ajuga iva supplementation on plasma lipid profile and tissue antioxidant status in rats fed a high-cholesterol diet

2006

Abstract The present study was designed to explore the possible antioxidant and hypolipidemic effects of the aqueous extract of Ajuga iva (0.5% in the diet) in rats fed a high-cholesterol (1%) diet (HCD). The results indicated that the HCD-Ai versus HCD treatment led to many changes in biochemical parameters. They showed a decrease of plasma total cholesterol (TC) and VLDL-cholesterol but an increase of HDL2-cholesterol. The triacylglycerol contents were reduced in plasma and in VLDL. The lipid peroxidation determined by TBARS was decreased by 75% in plasma. TBARS in liver, heart and kidneys were highly reduced excepted in the adipose tissue. Ajuga iva treatment enhanced superoxide dismutas…

Malemedicine.medical_specialtyVery low-density lipoproteinAntioxidantLipoproteinsmedicine.medical_treatmentHypercholesterolemiaAdipose tissueAjugaThiobarbituric Acid Reactive SubstancesAntioxidantsAjugaCholesterol DietaryLipid peroxidationEatingchemistry.chemical_compoundInternal medicineDrug DiscoverymedicineTBARSAnimalsRats WistarPharmacologybiologyPlant ExtractsCholesterolBody WeightOrgan Sizebiology.organism_classificationLipidsDietEnzymesRatsEndocrinologychemistryDietary SupplementsIntestinal cholesterol absorptionlipids (amino acids peptides and proteins)Journal of Ethnopharmacology
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