Search results for "Biochemistry"

showing 10 items of 20172 documents

Overexpression of CYB5R3 and NQO1, two NAD+-producing enzymes, mimics aspects of caloric restriction

2018

© 2018 The Authors.

0301 basic medicineAgingCalorie restrictionInflammationmedicine.disease_cause03 medical and health sciencesDownregulation and upregulationmedicineCytochrome b5 reductaseCalorie restrictionchemistry.chemical_classification030102 biochemistry & molecular biologybiologyCYB5R3Cell BiologyCell biology030104 developmental biologyEnzymechemistrySirtuinbiology.proteinNQO1NAD+ kinasemedicine.symptomCarcinogenesisMetabolic homeostasis
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Role of miRNA in the Regulatory Mechanisms of Estrogens in Cardiovascular Ageing

2018

Cardiovascular diseases are a worldwide health problem and are the leading cause of mortality in developed countries. Together with experimental data, the lower incidence of cardiovascular diseases in women than in men of reproductive age points to the influence of sex hormones at the cardiovascular level and suggests that estrogens play a protective role against cardiovascular disease and that this role is also modified by ageing. Estrogens affect cardiovascular function via their specific estrogen receptors to trigger gene expression changes at the transcriptional level. In addition, emerging studies have proposed a role for microRNAs in the vascular effects mediated by estrogens. miRNAs …

0301 basic medicineAgingEstrogen receptorFisiologiaDiseaseReview Article030204 cardiovascular system & hematologyBioinformaticsBiochemistry03 medical and health sciences0302 clinical medicineGene expressionmicroRNAMedicineAnimalsHumanslcsh:QH573-671GeneSistema cardiovascularRegulation of gene expressionlcsh:Cytologybusiness.industryEstrogensCell BiologyGeneral MedicineMicroRNAs030104 developmental biologyGene Expression RegulationAgeingCardiovascular DiseasesbusinessHormone
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Drosophila Full-Length Amyloid Precursor Protein is Required for Visual Working Memory and Prevents Age-Related Memory Impairment

2018

Summary The β-amyloid precursor protein (APP) plays a central role in the etiology of Alzheimer's disease (AD). However, its normal physiological functions are still unclear. APP is cleaved by various secretases whereby sequential processing by the β- and γ-secretases produces the β-amyloid peptide that is accumulating in plaques that typify AD. In addition, this produces secreted N-terminal sAPPβ fragments and the APP intracellular domain (AICD). Alternative cleavage by α-secretase results in slightly longer secreted sAPPα fragments and the identical AICD. Whereas the AICD has been connected with transcriptional regulation, sAPPα fragments have been suggested to have a neurotrophic and neu…

0301 basic medicineAgingFasciclin 2Nerve Tissue ProteinsGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciences0302 clinical medicineAmyloid precursor proteinMemory impairmentAnimalsDrosophila ProteinsOlfactory memorybiologyWorking memoryfungiMembrane ProteinsLong-term potentiationCell biology030104 developmental biologyDrosophila melanogasterMemory Short-Termbiology.proteinVisual PerceptionAmyloid Precursor Protein SecretasesGeneral Agricultural and Biological SciencesAmyloid precursor protein secretase030217 neurology & neurosurgeryNeurotrophin
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Cardiac Nonmyocyte Cell Functions and Crosstalks in Response to Cardiotoxic Drugs

2017

The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on cardiotoxicity, but recently the contribution of the other nonmyocyte cardiac cells is becoming of growing interest. This review deals with the role of oxidative stress, induced by anticancer drugs, in cardiac nonmyocyte cells (fibroblasts, vascular cells, and immune cells). The alterations of functional interplays among t…

0301 basic medicineAgingHeart DiseasesAntineoplastic AgentsReview Article030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeBiochemistryMuscle Smooth Vascular03 medical and health sciences0302 clinical medicineImmune systemHumansMedicineMyocytes CardiacLymphocyteslcsh:QH573-671Cardiotoxicitybusiness.industrylcsh:CytologyCell BiologyGeneral MedicineFibroblastsCell functionOxidative Stress030104 developmental biologyResearch studiesMolecular targetscardiovascular systemReactive Oxygen SpeciesbusinessOxidative stress
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From lymphopoiesis to plasma cells differentiation, the age-related modifications of B cell compartment are influenced by “inflamm-ageing”

2017

Ageing is a complex process characterized by a general decline in physiological functions with increasing morbidity and mortality. The most important aspect of ageing is the chronic inflammatory status, named “inflamm-ageing”, strictly associated with the deterioration of the immune function, termed “immunosenescence”. Both are causes of increased susceptibility of elderly to infectious diseases, cancer, dementia, cardiovascular diseases and autoimmunity, and of a decreased response to vaccination. It has been widely demonstrated that ageing has a strong impact on the remodelling of the B cell branch of immune system. The first evident effect is the significant decrease in circulati…

0301 basic medicineAgingImmunosenescenceHealth StatusPlasma CellsNaive B cellAutoimmunityInflammationBiologyLymphocyte ActivationBiochemistry03 medical and health sciences0302 clinical medicineImmune systemAntigenAge-related diseasemedicineAnimalsHumansLymphopoiesisProgenitor cellMolecular BiologyCellular SenescenceB cellInflammationB cellB-LymphocytesLymphopoiesisCell DifferentiationImmunosenescenceInflamm-ageing030104 developmental biologymedicine.anatomical_structureNeurologyImmune SystemImmunologyInflammation Mediatorsmedicine.symptomExhausted/Senescent cell030215 immunologyBiotechnologyAgeing Research Reviews
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Protective Effects of Polyphenols Present in Mediterranean Diet on Endothelial Dysfunction

2020

Endothelial dysfunction tends to be the initial indicator in proinflammatory state and macro- and microvascular complications, such as atherosclerosis and cardiovascular diseases. It has been shown that certain compounds in diet can generate beneficial effects on cardiovascular disease due to its interactions with endothelial cells. Thus, this review is aimed at investigating whether certain polyphenols present in the Mediterranean diet, specifically catechin, quercetin, resveratrol, and urolithin, could exert positive effects on endothelial dysfunction. After analysis of numerous papers, we found that polyphenols aiding endothelial function is beneficial not only for patients with cardiova…

0301 basic medicineAgingMediterranean dietFisiologiaReview ArticleDisease030204 cardiovascular system & hematologyResveratrolPharmacologyDiet MediterraneanBiochemistryProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound0302 clinical medicineWeight lossDiabetes mellitusmedicineHumansEndothelial dysfunctionQH573-671business.industryEndothelial CellsPolyphenolsfood and beveragesCell BiologyGeneral Medicinemedicine.diseaseUrolithin030104 developmental biologychemistryDietamedicine.symptomCytologybusinessOxidative Medicine and Cellular Longevity
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Electrical activity controls area-specific expression of neuronal apoptosis in the mouse developing cerebral cortex

2017

Programmed cell death widely but heterogeneously affects the developing brain, causing the loss of up to 50% of neurons in rodents. However, whether this heterogeneity originates from neuronal identity and/or network-dependent processes is unknown. Here, we report that the primary motor cortex (M1) and primary somatosensory cortex (S1), two adjacent but functionally distinct areas, display striking differences in density of apoptotic neurons during the early postnatal period. These differences in rate of apoptosis negatively correlate with region-dependent levels of activity. Disrupting this activity either pharmacologically or by electrical stimulation alters the spatial pattern of apoptos…

0301 basic medicineAgingMouseStimulationCell CountSomatosensory systemMice0302 clinical medicineAnesthesiaBiology (General)whisker deafferentationCerebral CortexNeuronsNeocortexCaspase 3General NeuroscienceQRapoptosisMotor CortexGeneral MedicineAnatomyactivity patternsmedicine.anatomical_structurecell deathCerebral cortexMedicinePrimary motor cortexMotor cortexResearch ArticleProgrammed cell deathQH301-705.5ScienceBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesmedicineAnimalsSensory deprivationdevelopmentGeneral Immunology and MicrobiologySomatosensory CortexElectrophysiological Phenomena030104 developmental biologyDevelopmental Biology and Stem Cellsnervous systemAnimals NewbornNeuroscience030217 neurology & neurosurgeryNeuroscienceeLife
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The emerging role of Notch pathway in ageing: Focus on the related mechanisms in age-related diseases

2016

Notch signaling is an evolutionarily conserved pathway, which is fundamental for the development of all tissues, organs and systems of human body. Recently, a considerable and still growing number of studies have highlighted the contribution of Notch signaling in various pathological processes of the adult life, such as age-related diseases. In particular, the Notch pathway has emerged as major player in the maintenance of tissue specific homeostasis, through the control of proliferation, migration, phenotypes and functions of tissue cells, as well as in the cross-talk between inflammatory cells and the innate immune system, and in onset of inflammatory age-related diseases. However, until …

0301 basic medicineAgingNotchNotch pathwayNotch signaling pathwayInflammationa signaling complex networkBiologyBiochemistryBiomarkers and targets for personalized treatmentBiomarkers and targets for personalized treatments03 medical and health sciencesAge relatedAge-related diseaseReceptorsmedicineA signaling complex network; Age-related diseases; Ageing; Biomarkers and targets for personalized treatments; Involved mechanisms; Notch pathway; Aging; Animals; Homeostasis; Humans; Inflammation; Inflammation Mediators; Receptors Notch; Signal TransductionAnimalsHomeostasisHumansMolecular BiologyInflammationInnate immune systemReceptors NotchSettore BIO/11Involved mechanismsAge-related diseases; Ageing; Biomarkers and targets for personalized treatments; Involved mechanisms; Notch pathway; a signaling complex networkPhenotypeInvolved mechanismAgeing030104 developmental biologyNeurologyAgeingImmunologymedicine.symptomSignal transductionInflammation MediatorsNeuroscienceHomeostasisAge-related diseasesBiotechnologySignal Transduction
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Frailty as a predictor of adverse outcomes in hospitalized older adults: A systematic review and meta-analysis.

2019

Frailty syndrome is prevalent among hospitalized older adults as are the occurrence of adverse outcomes. This systematic review and meta-analysis investigated whether frailty in older adults at hospital admission predicts adverse outcomes. Manual (ProQuest, conferences annals and references) and electronic searches (PUBMED, EMBASE, Web of Science, Lilacs, CINAHL, PsycINFO and Google Scholar) were performed. We included prospective studies of hospitalized older adults. Primary outcomes were functional decline at hospital discharge and mortality after discharge. Other data were considered secondary outcomes. Methodological quality was evaluated by the Quality Assessment Tool for Observational…

0301 basic medicineAgingPediatricsmedicine.medical_specialtyFrail ElderlyFrailty syndromeAdverse effectBiochemistry03 medical and health sciences0302 clinical medicineRisk FactorsPrevalenceMedicineHumansRisk factorOlder adultMortalityAdverse effectProspective cohort studyMolecular BiologyGeriatric AssessmentAgedAged 80 and overFrailtybusiness.industryLength of Staymedicine.disease030104 developmental biologyNeurologyMeta-analysisRelative riskCohortObservational studyFemalebusiness030217 neurology & neurosurgeryBiotechnologyAgeing research reviews
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Targeting Nitric Oxide with Natural Derived Compounds as a Therapeutic Strategy in Vascular Diseases

2016

Within the family of endogenous gasotransmitters, nitric oxide (NO) is the smallest gaseous intercellular messenger involved in the modulation of several processes, such as blood flow and platelet aggregation control, essential to maintain vascular homeostasis. NO is produced by nitric oxide synthases (NOS) and its effects are mediated by cGMP-dependent or cGMP-independent mechanisms. Growing evidence suggests a crosstalk between the NO signaling and the occurrence of oxidative stress in the onset and progression of vascular diseases, such as hypertension, heart failure, ischemia, and stroke. For these reasons, NO is considered as an emerging molecular target for developing therapeutic stra…

0301 basic medicineAgingPhytochemicalsIschemiaEndogenyReview Article030204 cardiovascular system & hematologyPharmacologyBiologyNitric Oxidemedicine.disease_causeCardiovascular SystemBiochemistryNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineAnimalsHumansVascular Diseasescell biology; aging; biochemistrylcsh:QH573-671GasotransmittersPlants Medicinallcsh:CytologyPolyphenolsCardiovascular AgentsCell BiologyGeneral Medicinemedicine.diseaseDietOxidative StressCrosstalk (biology)030104 developmental biologychemistryHeart failurePlant PreparationsOxidative stressIntracellularPhytotherapySignal TransductionOxidative Medicine and Cellular Longevity
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